ライフサイエンスコーパス: adrenal

1 ficant androsterone levels also in the fetal adrenal.

2 ogenesis enriched in beta-catenin-stabilized adrenals.

3 rom 100 pmol/g to 500 pmol/g) independent of adrenals.

4 unit of administered radioactivity were the adrenals (0.1835 mSv/MBq), the kidneys (0.1722 mSv/MBq),

5 The relationship between age and adrenal aldosterone synthase (CYP11B2) expression was ev

6 Adrenal and gonadal mitochondrial metabolic activity req

7 escribe AR expression in the human and mouse adrenal and highlight that the mouse is a viable model t

8 the activation of the hypothalamic-pituitary-adrenal and hypothalamic-pituitary-thyroid axes, as well

9 rine growth restriction (IUGR) with gonadal, adrenal, and bone marrow failure, predisposition to infe

10 m 21 tissue datasets (brain; blood; thyroid, adrenal, and pituitary glands).

11 althy by daily video health checks and body, adrenal, and spleen weights of 37d-flight (FLT) mice did

12 patients (91%) had familial, multiple, extra-adrenal, and/or malignant tumors and/or were younger tha

13 (DHEA) and its sulfate, DHEAS, are the major adrenal androgen precursors, but they are biologically i

14 Glucocorticoids inhibit adrenal androgen production.

15 The adrenals are a major endocrine site of production/secret

16 nstead of consecutive hypothalamus-pituitary-adrenal axis activation, we report that acute SCI in mic

17 ognition, anxiety and hypothalamic-pituitary-adrenal axis activation.

18 othesis that maternal hypothalamic-pituitary-adrenal axis activity, measured by hair cortisol concent

19 ation, or exacerbated hypothalamic-pituitary-adrenal axis activity.

20 Lower hypothalamic-pituitary-adrenal axis and autonomic reactivity to stress has also

21 ng alterations in the hypothalamic-pituitary-adrenal axis and inflammatory cytokines, which may contr

22 ted by inhibiting the hypothalamic-pituitary-adrenal axis and inflammatory responses to stress.

23 the microbiota on the hypothalamic-pituitary-adrenal axis and neuroimmune system.

24 the activation of the hypothalamus-pituitary-adrenal axis and the sympathetic nervous system followin

25 of opioid drugs on the hypothalamo-pituitary-adrenal axis and their negative effects on bone health a

26 we review the role of hypothalamic-pituitary-adrenal axis dysfunction in the neurobiology of ill-heal

27 Specifically, hypothalamic-pituitary-adrenal axis dysfunction, enhanced inflammation and oxid

28 ly, we found impaired hypothalamic-pituitary-adrenal axis feedback, blunted sympathetic responsivenes

29 The hypothalamic-pituitary-adrenal axis is a key regulatory pathway in the maintena

30 catecholaminergic and hypothalamic-pituitary-adrenal axis leads to splenic atrophy and contraction of

31 The hypothalamic-pituitary-adrenal axis modulates immunity in response to stress.

32 uch as by influencing hypothalamic-pituitary-adrenal axis regulation and cortisol dynamics, the auton

33 central component of hypothalamic-pituitary-adrenal axis regulation that prepares the organism for s

34 knockdown had reduced hypothalamic-pituitary-adrenal axis responses to both acute and chronic stress

35 gnificantly increased hypothalamic-pituitary-adrenal axis stress response and impaired sensorimotor g

36 ts in the adrenergic, hypothalamic-pituitary-adrenal axis, and neuropeptide Y systems.

37 sity ES at hindlimb regions drives the vagal-adrenal axis, producing anti-inflammatory effects that d

38 ogical changes in the hypothalamic-pituitary-adrenal axis, reward processing in the brain, and possib

39 the regulation of the hypothalamic-pituitary-adrenal axis, thereby affecting an individual's ability

40 nse by activating the hypothalamic-pituitary-adrenal axis.

41 ulatory effect in the hypothalamic-pituitary-adrenal axis.

42 text of a hyperactive hypothalamic-pituitary-adrenal axis.

43 and controlled by the hypothalamic-pituitary-adrenal axis.

44 nabinoid systems; the hypothalamus-pituitary-adrenal axis; and adenosine and nitric oxide signaling.

45 Conclusion Adrenal calcifications identified on CT scans are common

46 The prevalence of adrenal calcifications in patients with fCCM was compare

47 Adrenal calcifications in patients with fCCM were more f

48 rred in 2.5% of patients, and none developed adrenal cancer.

49 We developed an in vitro human adrenal cell model derived from fetal adrenal specimens

50 We determine that, whereas adrenal cells can secrete SHH on lipoproteins, this form

51 Human fetal adrenal cells retain age-related stem- and endocrine-dif

52 citability and reduced secretory activity in adrenal chromaffin cells (CCs).

53 zing enzyme, phenyl-N-methyl transferase, by adrenal chromaffin cells and changes in cell cycle dynam

54 Adrenal chromaffin cells and sympathetic neurons synthes

55 identified genes differentially expressed by adrenal chromaffin cells and sympathetic neurons.

56 directly from neural crest precursors while adrenal chromaffin cells arise from neural crest-derived

57 gations of this phenomenon in neuroendocrine adrenal chromaffin cells show that a single 2-ns, 16 MV/

58 Exposing adrenal chromaffin cells to single 150 to 400 ns electri

59 sults demonstrate that electrostimulation of adrenal chromaffin cells with ultrashort pulses can be m

60 Deletion of a gene highly expressed by adrenal chromaffin cells, NIK-related kinase, a gene on

61 i-inflammatory effects that depend on NPY(+) adrenal chromaffin cells.

62 les in two sequential priming steps in mouse adrenal chromaffin cells.

63 o ablate NPY(+) noradrenergic neurons and/or adrenal chromaffin cells.

64 one tenth of the catecholamine compared with adrenal chromaffin vesicles.

65 and subtype differentiation was performed by adrenal computed tomography scanning and adrenal vein sa

66 evated circulating corticosterone and severe adrenal cortex disruption.

67 noninvasive method for the evaluation of the adrenal cortex function in pediatric asthmatic patients.

68 atients with bronchial asthma, a decrease in adrenal cortex function occurs.

69 ignificant structural disorganization of the adrenal cortex in both sexes, with increased adrenal cor

70 Thus, homeostatic maintenance of the adrenal cortex is dependent on varying levels of Wnt/bet

71 ttes that provide a structural framework for adrenal cortex morphogenesis and plasticity.

72 we show zona glomerulosa cells in the adult adrenal cortex organize into rosettes through adherens j

73 adrenal cortex in both sexes, with increased adrenal cortex size in female mice and increased cell pr

74 pha contributes to the maintenance of normal adrenal cortex structure and cell proliferation, by modu

75 a Wnt/beta-catenin signaling gradient in the adrenal cortex that is disrupted upon loss of ZNRF3.

76 pecific ablation of androgen receptor in the adrenal cortex with or without reduction of circulating

77 arious oronasal regions), bone, spinal cord, adrenal cortex, and the uro-anogenital region in the neo

78 ptor (AR) is widely expressed throughout the adrenal cortex, yet the wider role for androgen signalli

79 el to investigate androgen signalling in the adrenal cortex.

80 designer drugs (DREADDs) specifically in the adrenal cortex.

81 zone-specific steroid production within the adrenal cortex.

82 ssion, cell clearance and to protect against adrenal degeneration during ageing.

83 , which catalyzes peripheral conversion from adrenal dehydroepiandrosterone (DHEA) to potent androgen

84 Sprague-Dawley rats to test the SNS with 1) adrenal demedullation and 2) chemical sympathectomy, and

85 Adrenal demedullation and chemical sympathectomy had no

86 ed derivatives, including posterior-specific adrenal derivatives, and display partial capacity to gen

87 These adrenal-derived androgens all share an oxygen atom on ca

88 55 +/- 14; F/M = 1.1) were operated upon for adrenal disease.

89 nded treatment for many benign and malignant adrenal diseases.

90 rst year of life induces early, asymptomatic adrenal disruption compatible with the combined inhibiti

91 Lopinavir was associated with dose-dependent adrenal dysfunction in infants.

92 or frequently monitoring young children with adrenal dysfunction or severe asthma that are treated wi

93 regulation of the thyroid, somatotropic and adrenal endocrine axes, possibly influencing homeostatic

94 ervous system and the hypothalamus-pituitary-adrenal endocrine axis.

95 ted that both 5alpha-dihydrotestosterone and adrenal explant culture supernatant induce nuclear trans

96 and brain lesion count were associated with adrenal findings in patients with fCCM.

97 Assessment of gonadal and adrenal function (particularly if high index of clinical

98 Herein, we characterized the adrenal function and morphology in Kiss1-/- mice that do

99 ndings suggest that KISS1 may play a role in adrenal function in mice and possibly adrenocortical ste

100 ients had long-term recurrence, while normal adrenal function was obtained in 16 (62%) patients.

101 type 2B and affords a good chance for normal adrenal function.

102 READD receptors triggered disorganization of adrenal functional zonation, with induction of Cyp11b2 i

103 g significant mRNA silencing in liver (65%), adrenal gland (37%), ovary (35%), and kidney (78%).

104 FCs in the left adrenal gland than the right adrenal gland and 50 of the 61 observed SFCs (82%) found

105 nance imaging excluded common lesions of the adrenal gland and showed lymphadenopathy around the majo

106 producing zona glomerulosa (zG) cells of the adrenal gland arrange in distinct multi-cellular rosette

107 er featuring cardiac arrhythmias, autism and adrenal gland dysfunction that originates from a de novo

108 hythmias, autism spectrum disorder (ASD) and adrenal gland dysfunction.

109 g puberty, the synthesis of androgens by the adrenal gland has been considered of little physiologic

110 ed with controls, aged HCM females exhibited adrenal gland hypertrophy, reduced volume in mood-relate

111 of catecholamine-containing vesicles of the adrenal gland into a release-ready state.

112 The adrenal gland is a multiendocrine organ with a steroidog

113 The adrenal gland is a source of sex steroid precursors, and

114 The adrenal gland represents a useful model to address this

115 of 19 patients having more SFCs in the left adrenal gland than the right adrenal gland and 50 of the

116 s and the morphologic characteristics of the adrenal gland were recorded.

117 e role of the brain, neuroendocrine factors (adrenal gland) and gastrointestinal systems (colon) in p

118 l erythropoiesis (which notably included the adrenal gland), and integration with mouse developmental

119 Together, these data suggest that the liver, adrenal gland, and lymphatic organs are important sites

120 l motor nerve to the vicinity of the forming adrenal gland, where they detach from the nerve and form

121 ifically for HHV-6A), testis, esophagus, and adrenal gland.

122 ne silencing in lung, muscle, fat, heart and adrenal gland.

123 brane in the liver, blood-brain barrier, and adrenal gland.

124 n human serum and epidermis, and the porcine adrenal gland.

125 pinal mediastinum, retroperitoneum, neck and adrenal gland.

126 the 61 observed SFCs (82%) found in the left adrenal gland.

127 omocytoma and paraganglioma than for healthy adrenal glands (11.9 +/- 2.0 vs. 9.9 +/- 1.5 vs. 3.7 +/-

128 (Epstein-Barr) and the thymus, kidneys, and adrenal glands (human herpesvirus 6).

129 t siRNA accumulation in muscle, lung, heart, adrenal glands and fat.

130 lungs, hypoplastic/ectopic kidneys, aplastic adrenal glands and spleen, as well as atretic trachea an

131 Following infection, both liver and adrenal glands exhibited significant and early downregul

132 11B2) expression was evaluated in 127 normal adrenal glands from deceased kidney donors (age, 9 month

133 The impact of prolonged treatment on the adrenal glands may require further attention.

134 ed by removal of the catecholamine-producing adrenal glands prior to endotoxic shock.

135 X disease occurred almost exclusively in the adrenal glands with frequently bilateral tumors.

136 cidates how host responses within the liver, adrenal glands, and lymphoid tissues contribute to EBOV

137 understanding of tissue injury in the liver, adrenal glands, and lymphoid tissues remains limited.

138 e the transcriptomes of human embryos, fetal adrenal glands, and neuroblastoma at single-cell level a

139 s in significant damage to the liver and the adrenal glands, little is known about the molecular sign

140 KV RNA in the brain, thymus, lungs, kidneys, adrenal glands, spleen, liver, and small intestine.

141 oxoglutarate receptor 1 (OXGR1) expressed in adrenal glands.

142 multiple brain regions, skeletal muscle, and adrenal glands.

143 the HPA stress response in the pituitary or adrenal glands.

144 induced the production of dopamine from the adrenal glands.

145 art, lungs, spleen, bone marrow, thyroid and adrenal glands.

146 osette formation underlies the maturation of adrenal glomerular structure postnatally.

147 renin-angiotensin-aldosterone system and the adrenal glucocorticoid pathway, with a smaller fraction

148 totoxic effects in combination with elevated adrenal glucocorticoids and inflammatory cytokines as we

149 ploy explant cultures of human fetal organs (adrenals, gonads, genital skin) from the major period of

150 ular fracture, one sternal fracture, and one adrenal hematoma.

151 Adrenal-hormone profiles were compared at weeks 6 and 26

152 ition may involve the hypothalamic-pituitary-adrenal (HPA) and hypothalamic-pituitary-gonadal (HPG) a

153 t of their effects on hypothalamic pituitary-adrenal (HPA) axis activation, aversive conditioning, or

154 en functioning of the hypothalamic pituitary adrenal (HPA) axis and cognitive capability at older age

155 Hypothalamic-pituitary-adrenal (HPA) axis dysfunction contributes to numerous h

156 sturbance and promote hypothalamic-pituitary-adrenal (HPA) axis dysregulation, a key feature of affec

157 tional reactivity and hypothalamic-pituitary-adrenal (HPA) axis functioning.

158 variation within the hypothalamic-pituitary-adrenal (HPA) axis has been linked to risk for depressio

159 Dysregulation of the hypothalamic-pituitary-adrenal (HPA) axis occurs early in Alzheimer's disease (

160 The hypothalamic-pituitary-adrenal (HPA) axis orchestrates the physiological respon

161 The hypothalamic-pituitary-adrenal (HPA) axis regulates responses to internal and e

162 The hypothalamic-pituitary-adrenal (HPA) axis, a neuroendocrine network that contro

163 on risk activates the hypothalamic-pituitary-adrenal (HPA) axis, and there is growing evidence that a

164 primary output of the hypothalamus-pituitary-adrenal (HPA) axis, has been hypothesized to be a mechan

165 by senescence of the hypothalamic-pituitary-adrenal (HPA) axis, leading to progressive dysregulation

166 to activation of the hypothalamic-pituitary-adrenal (HPA) axis, the sympathetic nervous system throu

167 tokines stimulate the hypothalamic-pituitary-adrenal (HPA) axis, triggering endogenous glucocorticoid

168 ent alteration of the hypothalamic-pituitary-adrenal (HPA) axis.

169 energic input and the hypothalamus-pituitary-adrenal (HPA) axis.

170 ish equivalent of the hypothalamic-pituitary-adrenal (HPA) axis.

171 ress response via the hypothalamic-pituitary-adrenal (HPA) axis.

172 ases in immobility and hypothalamo-pituitary-adrenal (HPA) output in male rats during tail suspension

173 cardiometabolic, and hypothalamic-pituitary-adrenal (HPA) systems) in first-episode psychosis (FEP).

174 the physiology of the hypothalamic-pituitary-adrenal (HPA), hypothalamic-pituitary thyroid (HPT), and

175 Hypothalamic-pituitary-adrenal (HPA)-axis hyperactivity and inflammation are th

176 rations (a measure of hypothalamic-pituitary-adrenal [HPA] axis activation and the stress response).

177 nocorticotropic hormone, indicating primary (adrenal) hypercortisolism.

178 Congenital adrenal hyperplasia (CAH), resulting from mutations in C

179 nadequately treated patients with congenital adrenal hyperplasia (CAH).

180 ntly been discovered in primary macronodular adrenal hyperplasia (PMAH), a cause of Cushing syndrome.

181 Congenital adrenal hyperplasia due to P450 oxidoreductase deficienc

182 virilization of girls affected by congenital adrenal hyperplasia due to P450 oxidoreductase deficienc

183 Congenital adrenal hyperplasia is a group of autosomal recessive di

184 fic loss of ZNRF3, but not RNF43, results in adrenal hyperplasia that depends on Porcupine-mediated W

185 erone, with increased activity in congenital adrenal hyperplasia variants associated with 17alpha-hyd

186 l lipodystrophy and non classical congenital adrenal hyperplasia, and an essential splice site mutati

187 rders of androgen excess, such as congenital adrenal hyperplasia, premature adrenarche and polycystic

188 21A2, the disease-causing gene in congenital adrenal hyperplasia, we now provide a full structural ex

189 nical trials for the treatment of congenital adrenal hyperplasia.

190 e-P450-17A1, dosage-sensitive, sex-reversal, adrenal hypoplasia-critical region on chromosome X prote

191 actors for adrenocortical carcinoma (ACC) in adrenal incidentaloma (AI).

192 Adrenal incidentalomas are mostly benign nonfunctioning

193 te kidney injury, colitis, hypokalaemia, and adrenal insufficiency (n=1 each).

194 ults (n=1), prolonged QT interval (n=2), and adrenal insufficiency (n=1).

195 neumonitis (two [5%] and one [3%] patients), adrenal insufficiency (one [3%] and two [5%] patients),

196 Primary adrenal insufficiency is life threatening and can presen

197 Here, we have described a primary adrenal insufficiency syndrome and steroid-resistant nep

198 g., skin atrophy, osteoporosis, Addison-like adrenal insufficiency, fatty liver, and type 2 diabetes

199 each with constitutional symptoms, colitis, adrenal insufficiency, hyperglycaemia, and hypokalaemia.

200 ion, atrial fibrillation, hypophosphataemia, adrenal insufficiency, transaminitis, and infections.

201 had immune-related SAEs, including two with adrenal insufficiency, two with pneumonitis, and one wit

202 han 500 ms, and three patients had suspected adrenal insufficiency.

203 ver toxicity, corrected QT prolongation, and adrenal insufficiency.

204 lue, and 3mug/dL or less is considered to be adrenal insufficiency.

205 on the type of lymphoma and/ or presence of adrenal insufficiency.

206 unconjugated androgen produced by the human adrenals is 11beta-hydroxyandrostenedione (11OHA4).

207 decreases the limbic-hypothalamic-pituitary-adrenal (LHPA) axis activity in the offspring.

208 SCs into either human Leydig-like (hLLCs) or adrenal-like cells (hALCs) using chemically defined cult

209 addition to functioning at a distance, extra-adrenal (local) production allows GCs to act as paracrin

210 Primary adrenal lymphomas are a very rare type of extranodal lym

211 , renal primary versus other (P < 0.01), and adrenal margin status (P < 0.01).

212 uation of a patient with melanoma in whom an adrenal mass was detected on CT and MR during follow-up

213 describe a patient with melanoma in whom an adrenal mass was detected on CT and MRI during post-surg

214 Ultrasonography detected an adrenal mass, and magnetic resonance imaging excluded co

215 F-FDG PET/CT showed bilateral hypermetabolic adrenal masses, gastric ulcer, small hypermetabolic aden

216 ve oxygen species (ROS) levels in the CB and adrenal medulla (AM).

217 In rats, cortical influences over the adrenal medulla and the kidney originate mainly from 2 m

218 from adrenomedullary chromaffin cells in the adrenal medulla or in sympathetic, paravertebral ganglia

219 In monkeys, the cortical influence over the adrenal medulla originates from 3 distinct networks that

220 The number of chromaffin cells in the adrenal medulla was also decreased, indicating a broad d

221 strate that leptin stimulates a hypothalamus-adrenal medulla-BAT axis, which is necessary and suffici

222 Increased adrenal medulla-derived plasma catecholamines were neces

223 tions with a major sympathetic effector, the adrenal medulla.

224 ow we think and feel and the function of the adrenal medulla.

225 epinephrine and norepinephrine released from adrenal medullary chromaffin cells and norepinephrine re

226 y axis), heart rate variability (sympathetic adrenal medullary system), EEG event-related potentials

227 survival is observed in patients undergoing adrenal metastasectomy and should be considered for subj

228 Patients who underwent adrenal metastasectomy from 1994 to 2015 were identified

229 In selected patients undergoing adrenal metastasectomy, there were no significant differ

230 and identify subpopulations who benefit from adrenal metastasectomy.

231 .3 years for patients with concomitant extra-adrenal metastases and 3.0 years for patients with isola

232 nts with (n = 83) and without (n = 91) extra-adrenal metastases did not differ with respect to age, a

233 Adrenalectomy has been used to treat adrenal metastases in select patients.

234 xamine a multi-institutional experience with adrenal metastases to describe survival outcomes and ide

235 patients with and without concomitant extra-adrenal metastases.

236 uld be considered for subjects with isolated adrenal metastases.

237 ses and 3.0 years for patients with isolated adrenal metastases; P = 0.816) and EFS (9.39 vs 9.59 mon

238 .74, P = 0.031), and incomplete resection of adrenal metastasis (R1 margins; HR: 1.62, P = 0.034; R2

239 The median time to detection of adrenal metastasis after initial diagnosis of the primar

240 se preliminary results is required.Keywords: Adrenal, MR-Imaging, UrinarySupplemental material is ava

241 ssay showed that chromatin obtained from the adrenals of TG mice containing the intron conversion bin

242 On multivariable analysis, extra-adrenal oligometastatic disease at adrenalectomy (HR: 1.

243 Extra-adrenal oligometastatic disease at initial presentation

244 inhibition augmented posttraining pituitary-adrenal output and enhanced the memory for inhibitory av

245 of pACC and to monitor DHEAS levels in other adrenal pathologies.

246 association between adrenal restrictive and adrenal permissive alleles and FEV(1)PP in patients with

247 9.8 vs. 63.4 (P < 0.001), and for homozygous adrenal permissive genotype, it is 66.7 vs. 67.7 (P = 0.

248 In patients with the homozygous adrenal permissive genotype, there was no FEV(1)PP diffe

249 1245A) allele limits conversion, whereas the adrenal permissive HSD3B1(1245C) allele increases DHEA m

250 enal structure and function, we explored the adrenal phenotype of male and female Raralpha knockout m

251 invasive methods to measure the hypothalamic-adrenal-pituitary axis (N = 173), immune and inflammator

252 ent of rats recruited STX5 and alpha-SNAP to adrenal PMs and mitochondria.

253 In a mitochondrial reconstitution assay, adrenal PMs supported steroidogenesis in the absence of

254 achment protein (alpha-SNAP) are enriched in adrenal PMs, and adrenocorticotropic hormone treatment o

255 We further found that AKG stimulates the adrenal release of adrenaline through 2-oxoglutarate rec

256 he wider role for androgen signalling in the adrenal remains underexplored.

257 ysiological processes underlying physiologic adrenal remodeling and pathologic alterations involved i

258 Totally eighteen adrenal rest tumors in 9 patients were diagnosed TART on

259 Testicular adrenal rest tumours (TARTs) are benign testicular masse

260 types were assessed, and association between adrenal restrictive and adrenal permissive alleles and F

261 GC patients homozygous for the adrenal restrictive genotype have lower FEV(1)PP compare

262 pendently confirmed: FEV(1)PP for homozygous adrenal restrictive genotype in GC vs. noGC is 49.8 vs.

263 The adrenal restrictive HSD3B1(1245) genotype is associated

264 The adrenal restrictive HSD3B1(1245A) allele limits conversi

265 g these genes, we show Fgfr2 is required for adrenal rosette formation by regulating adherens junctio

266 evels remain raised as a result of increased adrenal sensitivity.

267 esis in the human fetus proceeds through the adrenal sex steroid precursor dehydroepiandrosterone, wh

268 genes involved in the central nervous system-adrenal signaling axis (ZDHHC17, CADPS, PIK3C2G), vascul

269 Adrenal-sparing surgery is feasible in multiple endocrin

270 Adrenal-sparing surgery was done in 31 patients: three (

271 human adrenal cell model derived from fetal adrenal specimens at different gestational ages, consist

272 rol trafficking from PMs to mitochondria for adrenal steroid synthesis and underscore the importance

273 ders encompassing enzyme deficiencies in the adrenal steroidogenesis pathway that lead to impaired co

274 organoids show morphofunctional features and adrenal steroidogenic factor, steroid acute regulatory,

275 is, we developed a mathematical model of the adrenal steroidogenic regulatory network that accounts f

276 g AKG as a systemically derived molecule for adrenal stimulation of muscle hypertrophy and fat loss.

277 tem cells is independent of immune attack or adrenal stress hormones.

278 ssion of genes in the hypothalamic-pituitary-adrenal/stress system (e.g., Crhr1) is one of them.

279 To understand how RARalpha modulates adrenal structure and function, we explored the adrenal

280 s role in behaviour, HPA-axis regulation and adrenal structure.

281 other additional treatments when faced with adrenal suppression as a side effect of steroids or loss

282 pruritus, and they had striae and secondary adrenal suppression as side effects of steroids.

283 is devoid of the chemical moieties producing adrenal suppression.

284 uality Register for Thyroid, Parathyroid and Adrenal Surgery and the Swedish National Patient Registe

285 hould be referred to high-volume centers for adrenal surgery.

286 increases lumbar sympathetic nerve activity, adrenal sympathetic nerve activity and arterial blood pr

287 This in vitro human adrenal system represents a unique but preliminar model

288 Cs) are small lipid hormones produced by the adrenals that maintain organismal homeostasis.

289 ctors negatively associated with OS included adrenal tumor size (P < 0.01), renal primary versus othe

290 tastases did not differ with respect to age, adrenal tumor size, or margin status.

291 identalomas are mostly benign nonfunctioning adrenal tumors (NFATs) or adenomas causing mild autonomo

292 sures aimed at improving the clinical use of adrenal vein sampling and at developing alternative tech

293 by adrenal computed tomography scanning and adrenal vein sampling, using strict criteria to define s

294 nt of optimal results require subtyping with adrenal vein sampling, which, as it is technically chall

295 termined when the ratio of cortisol from the adrenal vein to the level of cortisol in the inferior ve

296 sed with unilateral primary aldosteronism by adrenal venous sampling who had undergone a total adrena

297 Analysis of freshly isolated bovine adrenal vesicles shows that the size and internal catech

298 nd AR-independent androgen signalling in the adrenal, we used a novel mouse model with a specific abl

299 We show that adrenal-wide ligand activation of Gq DREADD receptors tr

300 ralocorticoid aldosterone is produced in the adrenal zona glomerulosa (ZG) under the control of the r