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1 y benign tumors of the ovary and uterus, and adrenal adenoma.
2 f hypertension, especially when caused by an adrenal adenoma.
3  or 2020 as well as 124 366 controls without adrenal adenoma.
4 teral renal-adrenal fusion with a concurrent adrenal adenoma.
5 suppression test) in 20-50% of patients with adrenal adenomas.
6 (53.1%) of whom had KCNJ5 mutations in their adrenal adenomas.
7 ns resulted in unilateral cortisol-producing adrenal adenomas.
8 sence and amount of histologic lipid in many adrenal adenomas accounts for their low attenuation on u
9 e adrenal gland that consisted of contiguous adrenal adenoma and metastasis, which represented a coll
10 ens from 10 patients with cortisol-producing adrenal adenomas and evaluated recurrent mutations in ca
11 ges show differences between the behavior of adrenal adenomas and metastases in oncologic patients st
12 r is a good tool for differentiating between adrenal adenomas and metastases, in both the arterial an
13 sity analysis allows differentiation between adrenal adenomas and nonadenomas, reflecting an improved
14 rcentage of lipid-rich cortical cells in the adrenal adenomas and the unenhanced CT attenuation numbe
15 caused by a unilateral aldosterone-producing adrenal adenoma (APA).
16 , KCNJ5, as a cause of aldosterone-producing adrenal adenomas (APAs) and one inherited KCNJ5 mutation
17             Unilateral aldosterone-producing adrenal adenomas (APAs) are the potentially curable caus
18 docrine tumors such as aldosterone-producing adrenal adenomas (APAs), a cause of severe hypertension,
19                                A subgroup of adrenal adenomas are larger, more heterogeneous, and mor
20  the 10-HU threshold method for diagnosis of adrenal adenomas at enhanced CT, with specificity mainta
21                                              Adrenal adenomas can be readily differentiated from nona
22 uced sensitivity for the characterization of adrenal adenomas compared with results from prior studie
23 included 17 726 patients with a diagnosis of adrenal adenoma in Sweden from 2005 to 2019 who were ide
24 eview of 2 years of clinical CT records, 223 adrenal adenomas in 193 patients (115 with contrast mate
25    The higher estimates encourage search for adrenal adenomas in patients with elevated ratios of pla
26 molecular pathogenesis of cortisol-producing adrenal adenomas is not well understood.
27               It is unclear if nonfunctional adrenal adenomas (NFAAs) are associated with increased m
28 ear-old woman had hyperaldosteronemia and an adrenal adenoma that showed no evidence of lipid on in-p
29  the mass was a pheochromocytoma, a cortical adrenal adenoma was histologically proven.
30 ells in histologic sections from 20 resected adrenal adenomas was assessed.
31 esent a case of a patient with a preexisting adrenal adenoma who only presented with clinical signs o
32 tudy, we selected a control (patient with an adrenal adenoma) with a nodule of similar size.