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1 designer drugs (DREADDs) specifically in the adrenal cortex.
2 of the bovine adrenodoxin (Adx) gene in the adrenal cortex.
3 system, which results in hyperplasia of the adrenal cortex.
4 immunohistochemistry to the liver and to the adrenal cortex.
5 pathway different from that existing in the adrenal cortex.
6 nce of optimal steroidogenic capacity in the adrenal cortex.
7 -induced release of corticosteroids from the adrenal cortex.
8 deglycosylation of DLIF isolated from bovine adrenal cortex.
9 5 may have tumor suppressor functions in the adrenal cortex.
10 zone-specific steroid production within the adrenal cortex.
11 n or by autonomous cortisol release from the adrenal cortex.
12 me is caused by tumors or hyperplasia of the adrenal cortex.
13 n that leads to release of cortisol from the adrenal cortex.
14 dogenic cells in the zona fasciculata of the adrenal cortex.
15 alamic control of the anterior pituitary and adrenal cortex.
16 a is an aggressive, lethal malignancy of the adrenal cortex.
17 ple organs, including the testis, ovary, and adrenal cortex.
18 chain fatty acids, particularly in brain and adrenal cortex.
19 GP prior to the separation between gonad and adrenal cortex.
20 g in the release of glucocorticoids from the adrenal cortex.
21 timulation of aldosterone secretion from the adrenal cortex.
22 roidogenic tissues, including the gonads and adrenal cortex.
23 of several steroidogenic genes in the human adrenal cortex.
24 ase that is upregulated during growth of the adrenal cortex.
25 el to investigate androgen signalling in the adrenal cortex.
26 marily expressed in the liver, intestine and adrenal cortex.
27 ly modified and was similar to normal bovine adrenal cortex.
29 elevated glucocorticoid concentrations after adrenal cortex activation; and e) release of bacterial p
34 that PKA 1) induces metabolic changes in the adrenal cortex and 2) phosphorylates CtBP proteins, part
35 g in increased steroidogenic activity in the adrenal cortex and an elevation of cortisol levels in th
36 udes the release of glucocorticoids from the adrenal cortex and catecholamines from the adrenal medul
38 uclear receptor selectively expressed in the adrenal cortex and gonads, where it mediates the hormona
40 athway--in both embryonic development of the adrenal cortex and in maintenance of the adult organ.
43 a dual-specificity PDE that is expressed in adrenal cortex and is partially inhibited by tadalafil a
44 the expression of CYP2D16 in the guinea pig adrenal cortex and its relationship to adrenal xenobioti
45 orticoid/catecholamine) interactions in both adrenal cortex and medulla is much greater in fetal shee
47 ter usage of IL-18 gene and suggest that the adrenal cortex and not immune cells may be the source of
49 ituitary, with consequent stimulation of the adrenal cortex and release of glucocorticoids critical f
50 t levels in the inner zone of the guinea pig adrenal cortex and suggest a major role for this isozyme
52 und, type II PtdIns 4-kinase activity of the adrenal cortex and the type III enzyme of bovine brain.
55 otein specific to the inner zones of the rat adrenal cortex, and called inner zone antigen (IZAg), wa
56 ency that impairs cortisol production by the adrenal cortex, and can lead to sexual ambiguity in both
59 67 that is produced in human islets, testis, adrenal cortex, and perhaps other endocrine tissues, but
60 ng pancreatic islets, parathyroids, thyroid, adrenal cortex, and pituitary are seen by 16 months.
62 ally drives glucocorticoid production in the adrenal cortex, and the sympathetic-adrenal-medullary ax
63 arious oronasal regions), bone, spinal cord, adrenal cortex, and the uro-anogenital region in the neo
64 in the locus coeruleus, inferior olive, and adrenal cortex; and decreased AT(2) receptor binding in
66 roxylase, an intracellular key enzyme of the adrenal cortex, are found in >90% of patients, but these
67 e III PI 4-kinases were isolated from bovine adrenal cortex as [3H]WT-labeled 110- and 210-kDa protei
68 nco-developmental genes present in the fetal adrenal cortex, as well as local adrenal paracrine and a
69 PHA revealed diffuse binding patterns in the adrenal cortex, avid binding in some adenomas, and "hot
70 al alterations in the hypothalamic-pituitary-adrenal cortex axis and the sympathetic nervous system,
72 y in the zona fasciculata/reticularis of the adrenal cortex, but also in the Leydig cell, kidney and
73 pha is involved in similar regulation in the adrenal cortex, but it also substantially modulates gluc
74 thus of angiotensin, and (3) stimulation of adrenal cortex by angiotensin is consistent with all the
75 ation of SF-1-dependent transcription in the adrenal cortex by signaling molecules such as protein ki
77 A), the predominant androgen secreted by the adrenal cortex, can be converted to both potent androgen
80 iency of 21-hydroxylase is a disorder of the adrenal cortex characterised by cortisol deficiency, wit
81 l development and homeostatic renewal of the adrenal cortex depend on capsular stem cells and cortica
86 high levels in peripheral tissues, including adrenal cortex (E16-E21) and cochlear ganglia (E19-P3),
87 Here we report that VEGF treatment of bovine adrenal cortex endothelial cells resulted in a 5-fold in
90 nchiolar epithelium of the lung, myocardium, adrenal cortex, epithelium of the seminal vesicles, prox
91 in, is synthesized in mammalian placenta and adrenal cortex from cholesterol through the novel acidic
92 hways involved in the differentiation of the adrenal cortex from the adrenogonadal primordium (AGP) h
95 noninvasive method for the evaluation of the adrenal cortex function in pediatric asthmatic patients.
99 placenta, brain, testis, ovary, spleen, and adrenal cortex have the highest levels of expression of
100 amplified from RNA of interrenal tissue (the adrenal cortex homolog) from the southern stingray (Dasy
101 y-adrenal (HPA) axis, resulting in increased adrenal cortex hyperplasia and corticosterone production
102 20alpha-DHOPA paralleled the development of adrenal cortex hyperplasia, hypercortisolism, and spleen
103 tary and counterregulative adaptation of the adrenal cortex in abused women without major depressive
104 ignificant structural disorganization of the adrenal cortex in both sexes, with increased adrenal cor
109 timulated SAPK activity 3-4-fold both in the adrenal cortex in vivo and in the Y1 adrenocortical cell
111 ybridization showed that, in contrast to the adrenal cortex, in spleen and duodenum IL-18 is primaril
112 secretion, adrenal hypertrophy, and a larger adrenal cortex indicating chronic activation of the hypo
113 egulation of these synthetic pathways in the adrenal cortex inner zone during acute inflammation.
114 logical control of cortisol synthesis in the adrenal cortex involves stimulation of adrenocorticotrop
116 goxin-like immunoreactive factor (DLIF) from adrenal cortex is an endogenous molecule with structural
121 ike states to ultimately generate fetal zone adrenal-cortex-like cells (FZLCs), as evidenced by histo
124 ontrary, FADS2 expression is elevated in the adrenal cortex of obese mice, and plasma corticosterone
126 Here we report that the patches in mosaic adrenal cortex of the mouse and rat are also fractal and
127 gh levels of glucocorticoids produced by the adrenal cortex of the stressed subject reduce neurogenes
128 we show zona glomerulosa cells in the adult adrenal cortex organize into rosettes through adherens j
132 effects of stress and with mediation by the adrenal cortex, restraint stress evoked repeated transie
133 adrenal cortex in both sexes, with increased adrenal cortex size in female mice and increased cell pr
134 impact of LGR4 inactivation was analyzed by adrenal cortex-specific ablation of Lgr4, using Lgr4fl/f
135 lysis showed that IGIF is synthesized in the adrenal cortex, specifically in the zona reticularis and
137 pha contributes to the maintenance of normal adrenal cortex structure and cell proliferation, by modu
138 rom the inner zone (zona reticularis) of the adrenal cortex than from the outer zones, coinciding wit
139 a Wnt/beta-catenin signaling gradient in the adrenal cortex that is disrupted upon loss of ZNRF3.
140 osolic PtdIns 4-kinase (cPI4K) in the bovine adrenal cortex that is distinguished from previously kno
141 n a sea of the other cell type, while in the adrenal cortex the pattern is one of alternating cords o
143 that during organogenesis in both liver and adrenal cortex, the same basic mechanism is directed to
144 It is expressed exclusively in the outer adrenal cortex, the site of cell proliferation, and in t
145 athology, and to disorders that affected the adrenal cortex, the spinal cord, the cerebellum and the
146 of anti-inflammatory glucocorticoids by the adrenal cortex, thereby representing an endogenous feedb
148 that PKA acts as a tumour suppressor in the adrenal cortex, through repression of WNT signalling.
150 apidly secrete catecholamines (CAs), and the adrenal cortex to release progesterone (PROG), which may
151 e from the pituitary, which then acts on the adrenal cortex to stimulate the secretion of corticoster
152 lls with nuclear p21 and p53 appeared in the adrenal cortex together with DNA damage detected by in s
153 investigate the role of beta-catenin in the adrenal cortex, we used two Sf1/Cre transgenes to inacti
154 reticularis and the zona fasciculata of the adrenal cortex, where its levels are elevated by acute s
155 Finally, mice deficient for SR-BI in the adrenal cortex, where the receptor provides lipoprotein-
158 from resistance to the action of ACTH on the adrenal cortex, which stimulates glucocorticoid producti
159 pecific ablation of androgen receptor in the adrenal cortex with or without reduction of circulating
160 , we compared the expression of IL-18 in the adrenal cortex with that of spleen and duodenum, two oth
161 Abnormal lipid storage is evident in the adrenal cortex, with characteristic lamellar-lipid inclu
162 ptor (AR) is widely expressed throughout the adrenal cortex, yet the wider role for androgen signalli