ライフサイエンスコーパス: adrenaline

1 amol, formoterol, fenoterol, clenbuterol, or adrenaline).

2 Upon arrival, 126 patients were treated with adrenaline.

3 27.6% of insect anaphylaxis received on-site adrenaline.

4 ulla (RVLM), which results in the release of adrenaline.

5 short-acting bronchodilators, and nebulized adrenaline.

6 e treatment for anaphylaxis is intramuscular adrenaline.

7 nol, and the low-affinity endogenous agonist adrenaline.

8 were corticoids and antihistamines, but not adrenaline.

9 ed their autoinjector needed another dose of adrenaline.

10 to survive polymyxin B following addition of adrenaline.

11 ng with the first purification of a hormone, adrenaline.

12 on and increases in plasma noradrenaline and adrenaline.

13 ted neurogenic contractions and responses to adrenaline.

14 duct always responded best to stimulation by adrenaline.

15 laxis requiring treatment with intramuscular adrenaline.

16 axis properly with the ability to administer adrenaline.

17 lar Ca(2+) pattern in response to glucose or adrenaline.

18 ds, 83% antihistamines, and 9% intramuscular adrenaline.

19 sponsible for conversion of noradrenaline to adrenaline.

20 ses modified the response to inhaled racemic adrenaline.

21 menoptera sting anaphylaxis is intramuscular adrenaline.

22 erenol, a beta-adrenergic agonist similar to adrenaline.

23 e or by a counter-regulatory hormone such as adrenaline.

24 s relieved by the intramuscular injection of adrenaline.

25 an explanatory meeting on auto-injection of adrenaline.

26 tered three cases of accidental injection of adrenaline.

27 Infusion of adrenaline (1 mug kg(-1) min(-1) ) increased minute vent

28 Hypoxaemia increased plasma adrenaline (26-fold) and noradrenaline (5-fold) in shams

29 d by hyperoxia (noradrenaline 50.7 +/- 5.2%, adrenaline 62.6 +/- 3.3%, cortisol 63.2 +/- 2.1%, growth

30 67% of patients received adrenaline, 85% oral antihistamines, and 89% received IV

31 ferential ratio of noradrenaline (NA) versus adrenaline (A) release secreted in response to various p

32 Anaphylaxis is a medical emergency with adrenaline acknowledged as the first-line therapy.

33 lyst for oxidation of ascorbic acid (AA) and adrenaline (AD).

34 vidence of a relationship between successful adrenaline administration and risk estimation.

35 Primary outcome was successful adrenaline administration at six weeks, assessed by an i

36 A survey with regard to the timing of adrenaline administration for anaphylaxis was conducted

37 drenaline self-injector (ASJ), and timing of adrenaline administration for anaphylaxis.

38 The rate of adrenaline administration in post-OIT-FDEIA group was si

39 Adrenaline administration is a top priority treatment fo

40 ine adherence ranged from 12.2% (n = 77) for adrenaline administration to 85.4% (540) for supplementa

41 design is a major determinant of successful adrenaline administration.

42 did not necessarily understand the timing of adrenaline administration.

43 a transient or stable BP increase induced by adrenaline administration.

44 There were no differences in adrenaline (ADR) at rest or with heavy exercise, but the

45 Noradrenaline (NA) and adrenaline (ADR) effluxes were monitored from ex vivo ad

46 by the catecholamines isoprenaline (Iso) and adrenaline (Adr) is regulated by V(M).

47 ial environment mediated by activation of an adrenaline/ADRB2/PKA/BAD antiapoptotic signaling pathway

48 iotensin-converting enzyme (ACE) inhibitors, adrenaline, allergic myocardial infarction, anaphylaxis,

49 tyltransferase, the synthesizing enzymes for adrenaline and acetylcholine, respectively.

50 on and elicit cardiorespiratory stimulation, adrenaline and adrenocorticotropic hormone (ACTH) releas

51 used extralobular duct was used to show that adrenaline and carbachol stimulated the duct through the

52 R showed reduced survival in the presence of adrenaline and complete restoration of growth upon addit

53 s were accompanied by increased fetal plasma adrenaline and cortisol, and reduced plasma insulin leve

54 Salmonella are able to sense adrenaline and downregulate the antimicrobial peptide re

55 l principal cell line (DC2) and increased by adrenaline and forskolin.

56 a large body of evidence indicates that (nor)adrenaline and glucocorticoid release induced by acute s

57 ted to govern activity of PP1 in response to adrenaline and insulin.

58 cluding training updates for self-injectable adrenaline and nasal spray use.

59 ne concentration was restored, whilst plasma adrenaline and neuropeptide Y (NPY) concentrations were

60 Adrenaline and noradrenaline concentrations were lower i

61 ressure (radial artery catheter), and plasma adrenaline and noradrenaline concentrations were measure

62 manipulated plasma catecholamines (combined adrenaline and noradrenaline concentrations) to three le

63 Adrenaline and noradrenaline correlated with syndecan-1

64 vous system and secreting the catecholamines adrenaline and noradrenaline in the 'fight-or-flight' re

65 S variants may allow modulation of endocrine adrenaline and noradrenaline release.

66 e and respond to the host NE stress hormones adrenaline and noradrenaline to modulate virulence.

67 -3 and the host neuroendocrine (NE) hormones adrenaline and noradrenaline were reported to display cr

68 Escherichia coli O157:H7, the catecholamines adrenaline and noradrenaline were shown to act synergist

69 t exercise in both groups, concentrations of adrenaline and noradrenaline were unchanged through low-

70 The second group comprised adrenaline and noradrenaline which displayed higher intr

71 sma concentrations of cortisol, vasopressin, adrenaline and noradrenaline, and falls in the fetal : m

72 fere with signaling from the stress hormones adrenaline and noradrenaline, have a lower incidence of

73 (beta2-AR), which binds the stress mediators adrenaline and noradrenaline, in modulating host respons

74 octopamine, the invertebrate counterpart of adrenaline and noradrenaline, in synaptic and behavioral

75 Adrenaline and noradrenaline, the main effectors of the

76 inding catecholamine agonist ligands such as adrenaline and noradrenaline.

77 elease tyramine, an invertebrate analogue of adrenaline and noradrenaline.

78 contractions produced by nerve stimulation, adrenaline and NPY, but not ATP.

79 .45 +/- 1.59 ml h-1; P < 0.01 relative to Jv adrenaline and P < 0.005 relative to Jv dichlorobenzamil

80 erns by modulating neurotransmitters such as adrenaline and serotonin.

81 in the infants treated with inhaled racemic adrenaline and those treated with inhaled saline (P>0.1

82 at lead to a lack of response to the initial adrenaline and thus RA are unclear.

83 rease in fetal plasma noradrenaline, but not adrenaline and vasopressin concentrations relative to sh

84 , in plasma concentrations of noradrenaline, adrenaline and vasopressin, and in the maternal-to-fetal

85 , L-NE is converted to L-epinephrine (L-Epi, adrenaline) and released as the primary neurotransmitter

86 Subjective fatigue, adrenaline, and body temperature variations during two 7

87 istration of anesthetics such as tetracaine, adrenaline, and cocaine and lidocaine, epinephrine, and

88 3,4-hydroxyphenylalanine [l-dopa], dopamine, adrenaline, and noradrenaline) elevate FUS1 and RLM1 tra

89 stress related hormones including cortisol, adrenaline, and serotonin were abnormally observed in th

90 base excess, platelet count and hemoglobin, adrenaline, and syndecan-1 were the only independent pre

91 atients used oral antihistamine, six inhaled adrenaline, and ten took no treatment.

92 vere anaphylaxis refractory to intramuscular adrenaline, and to consider a framework for managing the

93 Hypoxia increased venous [adrenaline] and [noradrenaline] but not [dopamine] at a

94 Reports on accidental auto-injection of adrenaline are few.

95 ction (no grade 3 or above); 10 (41.7%) used adrenaline as a rescue medication.

96 assess the effectiveness of inhaled racemic adrenaline as compared with inhaled saline and the strat

97 ly be effectively managed with intramuscular adrenaline as first line treatment.

98 mmediately following its first use, cases of adrenaline-associated sepsis were reported.

99 nged a second cohort of lambs with exogenous adrenaline at 21 dA.

100 in humans, but it is unclear if circulating adrenaline attenuates peripheral vasoconstriction during

101 opose indications for the prescription of an adrenaline auto-injector (AAI), and to discuss other for

102 Essential training for emergency adrenaline auto-injector administration alone provides a

103 h a written emergency management plan and an adrenaline auto-injector and educated to its use.

104 tion is often judged unnecessary, as well as adrenaline auto-injector and venom immunotherapy prescri

105 Provision of adrenaline auto-injector devices and education on how an

106 but only a minority received the recommended adrenaline auto-injector for self-administration at disc

107 If an adrenaline auto-injector is prescribed, education on whe

108 help later than 30 min after symptom onset, adrenaline auto-injector prescription is a necessity.

109 rs (<16 years) with food allergy, trained in adrenaline auto-injector use, were recruited from a hosp

110 plan, and, where appropriate, prescribing an adrenaline auto-injector.

111 reaction, and 28% had not been prescribed an adrenaline auto-injector.

112 mend that at-risk patients are provided with adrenaline auto-injectors (AAIs).

113 The low rate of doctors prescribing adrenaline auto-injectors in the ED setting underlines t

114 Adrenaline auto-injectors were prescribed to 84 patients

115 Our findings suggest that while handling adrenaline auto-injectors, we should keep in mind the po

116 However, adrenaline autoinjector activation has remained fairly s

117 ood allergic children who were prescribed an adrenaline autoinjector and to assess whether it was use

118 d and sport (at least for 2 h), and carry an adrenaline autoinjector at all times.

119 Similarly, rates of adrenaline autoinjector usage in the school environment

120 rs require more effective guidance on proper adrenaline autoinjector use.

121 An adrenaline autoinjector was prescribed to 139 food aller

122 An adrenaline autoinjector was used by 41 (16.7%, 95% CI: 1

123 ood allergic children who were prescribed an adrenaline autoinjector were investigated.

124 should carry an emergency kit containing an adrenaline autoinjector, H1 -antihistamines, and cortico

125 alf felt confident in knowing when to use an adrenaline autoinjector.

126 s in quality of life compared to carrying an adrenaline autoinjector.

127 e of anaphylaxis treatment, and provision of adrenaline autoinjectors (AAI) has become a standard of

128 ious work has shown patients commonly misuse adrenaline autoinjectors (AAI).

129 Although paramedics can use adrenaline autoinjectors (AAIs) during their duties, the

130 Patients already prescribed adrenaline autoinjectors (AAIs) for anaphylaxis were exa

131 The number of adrenaline autoinjectors activated per 1000 students at

132 rgy centers (84% of patients were prescribed adrenaline autoinjectors following EAACI guidelines) and

133 all year levels and the annual usage rate of adrenaline autoinjectors in the school setting relative

134 Newer models of adrenaline autoinjectors may slightly increase the propo

135 Adrenaline autoinjectors prescription was less common in

136 yncratic nature of LTP allergy, the need for adrenaline autoinjectors should always be considered.

137 There has been a debate about when adrenaline autoinjectors should be prescribed and how ma

138 children/carers are unsure when to use their adrenaline autoinjectors, contributing to a low quality

139 food allergy, of whom 38 288 were prescribed adrenaline autoinjectors.

140 Overall, the adrenaline-bound receptor structure is similar to the ot

141 It unravels a strong underuse of adrenaline by emergency physicians, not reflecting treat

142 mice that cannot synthesize noradrenaline or adrenaline by inactivating the gene that encodes dopamin

143 This is the first study to demonstrate that adrenaline can indirectly activate the PDC in skeletal m

144 ansport by the submandibular salivary gland (adrenaline, carbachol, isoprenaline and forskolin) mobil

145 rupted time series and - only in relation to adrenaline - case series investigating the effectiveness

146 In seven experiments, 0.5 microg min-1 adrenaline caused a significant (P < 0.0005) reduction i

147 In eight experiments, 0.5 microg min-1 adrenaline caused a significant (P < 0.005) reduction in

148 Adrenaline caused a significant increase in heart rate i

149 imilar interactions are seen with the hPheOH.adrenaline complex and Ser23.

150 t also significantly reduced baseline plasma adrenaline concentration (403 +/- 69 compared with 73 +/

151 This is not reflected in plasma adrenaline concentrations because of reduced plasma clea

152 A fall in fetal plasma noradrenaline and adrenaline concentrations occurred during betamethasone

153 ffect on heart rate (HR), plasma lactate and adrenaline concentrations or oxygen uptake at rest and d

154 uroglycopenic symptoms, and higher levels of adrenaline, cortisol, and growth hormone.

155 omotes prostate carcinogenesis in mice in an adrenaline-dependent manner.

156 Noradrenaline and adrenaline dose-dependently suppressed the release of IL

157 ngle wound infiltration with bupivacaine and adrenaline during cesarean delivery (intervention group)

158 Both adrenaline effects can be inhibited by the addition of t

159 recommend intramuscular injection of 500 mug adrenaline (epinephrine) for anaphylaxis in teenagers an

160 room air and 40% O2: (1) during intravenous adrenaline (epinephrine) infusion at 320 ng kg(-1) min(-

161 Although adrenaline (epinephrine) is a cornerstone of initial ana

162 Adrenaline (epinephrine) is the cornerstone of anaphylax

163 In contrast to SNS activity, tonic adrenaline (epinephrine) secretion from the adrenal medu

164 d show how the binding of an agonist ligand, adrenaline (epinephrine), causes conformational changes

165 t studies investigating the effectiveness of adrenaline (epinephrine), H1-antihistamines, systemic gl

166 ical conditions, willingness to always carry adrenaline, etc.), consideration may be given to allow t

167 ignaling induced by either thrombin, ADP, or adrenaline, examined by suppression of forskolin-stimula

168 ation with lipopolysaccharide on day 6, (nor)adrenaline-exposed cells showed increased TNF-alpha (tum

169 , we exposed human primary monocytes to (nor)adrenaline for 24 hours, after which cells were rested a

170 participants received more than one dose of adrenaline, for nine of these a health professional gave

171 um route, site and dose of administration of adrenaline from trials studying people with a history of

172 of a written action plan and self-injectable adrenaline if appropriate, and advice on avoidance.

173 The commonest reasons for not using adrenaline in anaphylaxis were 'thought adrenaline unnec

174 ntrinsic activity than the endogenous ligand adrenaline in cAMP accumulation, beta-arrestin-2 recruit

175 We sought to assess whether use of adrenaline in hemodynamically stable patients with anaph

176 64.4%), whereas when physicians administered adrenaline in patients, it resulted in circulatory (74.8

177 ns of vasopressin and noradrenaline, but not adrenaline in the fetus, and inversely related to the fe

178 ked by catecholaminergic challenge (caffeine/adrenaline) in S2814D(+/+) mice in vivo or programmed el

179 Plasma caffeine and adrenaline increased after caf, but not after decaf.

180 The results demonstrate that adrenaline increased glycogen phosphorylase activation a

181 Physiological concentrations of adrenaline increased the CO2 sensitivity of freshly diss

182 of epithelial sodium channels) abolished the adrenaline-induced absorption of lung liquid (mean Jv am

183 poral relationship, combined with a probable adrenaline-induced increase in metabolic rate (and there

184 5 x 10-5 M did not significantly inhibit the adrenaline-induced lung liquid absorption (Jv dichlorobe

185 ng times and are protected from collagen and adrenaline-induced thromboembolism.

186 = 10) before and during (1, 3, 7 and 15 min) adrenaline infusion (0.14 microg (kg body mass)(-1) min(

187 The PDC was activated following 7 min of adrenaline infusion (pre-infusion = 0.22 +/- 0.04 vs. 7

188 mg atropine), before and during intravenous adrenaline infusion at 80 ng kg(-1) min(-1) (ATR + 80 AD

189 Adrenaline infusion increased glycogen phosphorylase "a"

190 The present study examined the effect of adrenaline infusion on the activation status of glycogen

191 While peripheral intravenous adrenaline infusions should always be initiated only in

192 ve participants, 1 required an intramuscular adrenaline injection during step 3.

193 , only one patient required an intramuscular adrenaline injection, and 70% of OFC-positive patients h

194 reaction and treated him with intramuscular adrenaline injection, corticosteroid and antihistamine i

195 curred after exercise and did not respond to adrenaline injection.

196 Intramuscular adrenaline injections were administered to two patients

197 R versus 2.3% allergic participants reported adrenaline injector usage.

198 of the myofibrillar ATPase that occurs after adrenaline intervention.

199 ggested that administration of intramuscular adrenaline into the middle of vastus lateralis muscle is

200 Adrenaline is a fundamental circulating hormone for bodi

201 eement that rapid intramuscular injection of adrenaline is life-saving and constitutes the first-line

202 te bronchiolitis in infants, inhaled racemic adrenaline is not more effective than inhaled saline.

203 If promptly administered, adrenaline is potentially life-saving.

204 Although adrenaline is recommended as first line treatment for an

205 Adrenaline is regularly used for first-line emergency ma

206 Intramuscular adrenaline is the gold standard treatment for anaphylaxi

207 Adrenaline is used by only a minority of patients experi

208 Epinephrine (adrenaline) is a medication widely used in the pediatric

209 time, when patients injected themselves with adrenaline, it resulted in laryngeal (78.4%) and circula

210 Addition of adrenaline led to an induction of key metal transport sy

211 Plasma noradrenaline and adrenaline levels rose rapidly with dramatic increases i

212 At a blood glucose of 3.8 mmol/L, plasma adrenaline levels were twice as high after caffeine than

213 Through iron transport, adrenaline may affect the oxidative stress balance of th

214 However, adrenaline may also serve in favour of the host defences

215 that a failure or delay in administration of adrenaline may increase the risk of death.

216 th 0.31 +/- 0.04 and 0.34 +/- 0.01 hours for adrenaline-mediated beta-arrestin-2 recruitment and GFP-

217 Physiological levels of adrenaline mimicked the effect of hypoglycaemia on venti

218 However, in granular ducts only adrenaline mobilized the entire IP3-sensitive pool where

219 adrenaline unnecessary' (54.4%) and 'unsure adrenaline necessary' (19.1%).

220 We conclude that, in fetal sheep, neither adrenaline nor cGMP stimulate lung liquid absorption by

221 blood gases, glucose and lactate and plasma adrenaline, noradrenaline and vasopressin concentration

222 Admission plasma levels of catecholamines (adrenaline, noradrenaline) and biomarkers reflecting end

223 rkers reflecting sympathoadrenal activation (adrenaline, noradrenaline), tissue/endothelial cell/glyc

224 study was to examine the in vivo effects of adrenaline, noradrenaline, and cortisol on number and fu

225 The addition of adrenaline, noradrenaline, hydrocortisone, or dexamethas

226 e treated with physiological doses of either adrenaline, noradrenaline, or cortisol via i.v. infusion

227 We wished to investigate the impact of adrenaline on the biology of Salmonella spp.

228 We have determined the effect of adrenaline on the transcriptome of the gut pathogen Salm

229 There were no differences in basal plasma adrenaline or cortisol concentrations between low and hi

230 -adrenergic receptors on the cell surface by adrenaline or noradrenaline leads to alterations in the

231 amine, the invertebrate homolog of mammalian adrenaline or noradrenaline, plays important roles in mo

232 g peripheral beta-adrenergic agonist akin to adrenaline, or saline.

233 quency (ln HF) power (P < 0.001) and reduced adrenaline (P < 0.001) and noradrenaline concentrations

234 Arterial adrenaline (P < 0.05) and venous noradrenaline (P < 0.05

235 .01) and greater Area-Under-Curve for plasma adrenaline (p < 0.05) compared to 300 mug, with no diffe

236 ma, and prehospital fluids (100 pg/mL higher adrenaline predicted 2.75 ng/mL higher syndecan-1, P < 0

237 in management include injecting epinephrine (adrenaline) promptly, providing high-flow supplemental o

238 These results suggest that adrenaline, rather than low glucose, is an adequate stim

239 These findings suggest that adrenaline release can account for the ventilatory hyper

240 Indeed, adrenaline release in response to acute stress is substa

241 proximately 40%) were identified to regulate adrenaline release in response to glucoprivation.

242 oprivation in the PeH or in the RVLM elicits adrenaline release in vivo and 2) whether direct activat

243 d CO2 sensitivity is abolished by preventing adrenaline release or blocking its receptors.

244 had high plasma noradrenaline but attenuated adrenaline release with higher Injury Severity Score, im

245 or orexin release in the RVLM modulates the adrenaline release.

246 t, autonomic pathways affecting glucagon and adrenaline release.

247 Amiloride (10-4 M) inhibited the adrenaline response (Jv amiloride, +5.46 +/- 1.09 ml h-1

248 Leukocytosis strongly correlated with the adrenaline response to hypoglycemia.

249 ats), but did not alter the magnitude of the adrenaline response to restraint.

250 was determined to be 124.3 +/- 0.77% of the adrenaline response.

251 s whether they possessed registrations as an adrenaline self-injector (ASJ), and timing of adrenaline

252 Adrenaline sensing may provide an environmental cue for

253 ent signal transduction system is the likely adrenaline sensor mediating the antimicrobial peptide re

254 to local anaesthetic induced neurotoxicity: adrenaline significantly increases the neurotoxic effect

255 The stress hormone adrenaline stimulates beta(2)-adrenoreceptors that are e

256 chromosome, results in reduced expression of adrenaline-synthesizing enzyme, phenyl-N-methyl transfer

257 ld lower in the presence of isoprenaline and adrenaline than when salbutamol or terbutaline were pres

258 d that AKG stimulates the adrenal release of adrenaline through 2-oxoglutarate receptor 1 (OXGR1) exp

259 notable exception to this is the failure of adrenaline to have a direct effect on glycolysis.

260 ersely, it could be seen that the failure of adrenaline to maintain a constant glucose 6-phosphate co

261 The ability of adrenaline to mobilize NEFA was 55 +/- 15% lower (P < 0.

262 We also tested the ability of adrenaline to mobilize non-esterified fatty acids (NEFAs

263 years and increased adrenomedullary output (adrenaline) to stress at 2.5 years.

264 eripheral beta-adrenergic agonist similar to adrenaline, to induce sensations of palpitation and dysp

265 ing anaphylaxis, refractory to intramuscular adrenaline treatment, during supervised oral food challe

266 Epinephrine (adrenaline) treatment is underused in health care and co

267 that in human cardiac muscle unstimulated by adrenaline, troponin I is phosphorylated on Ser24.

268 sing adrenaline in anaphylaxis were 'thought adrenaline unnecessary' (54.4%) and 'unsure adrenaline n

269 Adrenaline use in hemodynamically stable anaphylaxis pat

270 Adrenaline use in hemodynamically stable anaphylaxis pat

271 lled trial to evaluate ability to administer adrenaline using different AAI devices.

272 We assessed ability to deliver adrenaline using their AAI in a simulated anaphylaxis sc

273 ntrolled trial that compared inhaled racemic adrenaline versus saline.

274 ercise to onset of severe symptoms requiring adrenaline was 32.5 min in the FDEIA group and 25 min in

275 Intramuscular adrenaline was administered in 17 SRs, but only 65% of t

276 examethasone fetuses, the increase in plasma adrenaline was attenuated during H1 and the increase in

277 In another trial (120 min duration), adrenaline was infused (AI) at 0.1 microgram kg-1 min-1

278 Treatment effect of inhaled adrenaline was not modified by virus type, load or coinf

279 thermore, no change in levels of circulating adrenaline was observed with L-NMMA.

280 the efficacy of the endogenous full agonist adrenaline was reduced by depolarization.

281 1 and r = 0.23, P < 0.001, respectively) but adrenaline was the only independent predictor of syndeca

282 Intramuscular adrenaline was used after 0.01% of doses (one participan

283 9-12 years, and >= 1 recent dispensation of adrenaline was used as a marker for current severe food

284 Adrenaline was used more often in patients with DIA than

285 onger hospital stay before death, and use of adrenaline were also significantly associated with poore

286 occurred upon addition of either glucagon or adrenaline were measured.

287 Isoprenaline and adrenaline were more efficacious in functional studies,

288 erse effects of accidental auto-injection of adrenaline were not observed in these three cases.

289 ncentrations of ACTH, AVP, noradrenaline and adrenaline were observed during hypoxaemia in both group

290 reas the inhibitory or excitatory actions of adrenaline were prevented by alpha1 or alpha2 antagonist

291 Adverse events induced by adrenaline were rare when the intramuscular route was us

292 ists such as adenosine diphosphate (ADP) and adrenaline were severely impaired.

293 consider how to best encourage the usage of adrenaline when clinically indicated in anaphylaxis.

294 reterm and term infants, use of epinephrine (adrenaline) when ventilation and compressions fail to st

295 in such cases using intravenous infusion of adrenaline which has been adopted for widespread use els

296 Many natural agonists such as adrenaline, which activates the beta2-adrenoceptor (beta

297 ine (p < .001) but an attenuated increase in adrenaline with increasing Injury Severity Score and low

298 actorial design, we compared inhaled racemic adrenaline with inhaled saline and on-demand inhalation

299 boration of neuroendocrine hormones, such as adrenaline, with infectious disease.

300 nable to treatment with low-dose intravenous adrenaline, with no reported adverse effects.