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1 was blocked by atenolol, a selective beta(1)-adrenergic antagonist.
2 tion of a beta(2) (but not beta(1) or alpha) adrenergic antagonist.
3 also delayed by treating mice with an alpha-adrenergic antagonist.
4 duced by therapy with phentolamine, an alpha-adrenergic antagonist.
5 Prazosin is a centrally active alpha(1) adrenergic antagonist.
6 fect was canceled by prior injection of beta-adrenergic antagonists.
7 otine was blocked by prior injection of beta-adrenergic antagonists.
8 tion was further indicated by the effects of adrenergic antagonists.
9 gical differences between these two alpha(2)-adrenergic antagonists.
10 adrenergic antagonist), or zinterol (a beta2-adrenergic antagonist) administered bilaterally into the
11 ntagonist, and yohimbine, a selective alpha2-adrenergic antagonist, also reduced L-NAME-induced const
12 trials in which the combination of an alpha1-adrenergic antagonist and ARI was used to treat symptoma
13 Freshly isolated strips were exposed to beta-adrenergic antagonists and agonists and assayed for cAMP
14 crements are reversed by propranolol, a beta-adrenergic antagonist, and abolished by selective bilate
15 combination of prazosin, a selective alpha1-adrenergic antagonist, and yohimbine, a selective alpha2
17 l) of either propranolol (a nonspecific beta-adrenergic antagonist), atenolol (a beta1-adrenergic ant
18 of a single injection of the specific alpha2-adrenergic antagonist atipamezole (1.5 mg/kg i.p.) after
20 ption partially blocked by intrathecal alpha-adrenergic antagonists, but the mechanism underlying thi
22 ne epinephrine and show that beta- and alpha-adrenergic antagonists can block the bacterial response
23 ilation of the cerebral vasculature with the adrenergic antagonist carvedilol in a randomized, cross-
28 was administered with and without the alpha-adrenergic antagonist dapiprazole, and its effects were
31 receptor antagonist) or propranolol (a beta-adrenergic antagonist) did not alter DA-stimulated clear
32 ed additional inhibitors, including alpha(1)-adrenergic antagonists doxazosin, terazosin, and alfuzos
33 atic hyperplasia can be treated with alpha 1-adrenergic-antagonist drugs that relax prostatic smooth
34 energic agonist, phenylephrine, and a beta-1 adrenergic antagonist, esmolol, in 14 patients with POTS
39 his effect was blocked by use of the beta(2)-adrenergic antagonist ICI 118,551 but not by the alpha-a
40 renol was inhibited completely by the beta 2-adrenergic antagonist ICI-118,551 at 100 nM, with a half
41 ate the physician regarding the use of alpha-adrenergic antagonists in the management of various pedi
43 ystemic co-administration of alpha- and beta-adrenergic antagonists in young (n = 9; 26 +/- 1 years o
44 g evidence suggests that beta-blockers (beta-adrenergic antagonists) increase brain clearance of thes
45 in-deficient and wild-type mice while a beta-adrenergic antagonist increases bone mass in wild-type a
46 ompletely blocked by prior injection of beta-adrenergic antagonists, indicating that beta-adrenergic
48 ions in vivo, we show that infusion of alpha-adrenergic antagonists into the NIf (nucleus interfacial
49 icial heart failure treatments, such as beta-adrenergic antagonists, involve improved contractility.
50 r its specific blockade using alpha1/alpha1B adrenergic antagonists led to selective sensitization of
52 mprovements in symptoms compared with alpha1-adrenergic antagonist monotherapy after as little as 9 m
53 e evident after 1 year when comparing alpha1-adrenergic antagonist monotherapy and combination medica
54 ean decreases between those receiving alpha1-adrenergic antagonist monotherapy and combination medica
56 ce was blocked by pretreatment with the beta-adrenergic antagonist, nadolol (0.5 mg/kg), but not by t
57 adrenergic antagonist, prazosin, an alpha 1-adrenergic antagonist, nor yohimbine, an alpha 2-specifi
58 tic benign prostatic hyperplasia over alpha1-adrenergic antagonist or ARI monotherapy, when prescribe
61 terial infusion of phentolamine (PHEN, alpha-adrenergic antagonist) or phenylephrine (PE, alpha(1)-ad
62 ta-adrenergic antagonist), atenolol (a beta1-adrenergic antagonist), or zinterol (a beta2-adrenergic
65 h the nonselective alpha-adrenergic and beta-adrenergic antagonists phentolamine and propranolol, or
68 timulation by norepinephrine and an alpha(1)-adrenergic antagonist prazosin is not restored by pertus
69 [Ca2+]i signaling was blocked by the alpha1-adrenergic antagonist prazosin, whereas signaling in the
73 9)-10(-6) m), in the presence of an alpha(1)-adrenergic antagonist, prazosin (10(-6) m), selectively
75 renaline release) and phentolamine (an alpha-adrenergic antagonist), profound vasodilatation was seen
78 is effect was completely blocked by the beta-adrenergic antagonist propranolol but was not affected b
80 (saline, 1, 3, 5 or 10 mg kg(-1) of the beta-adrenergic antagonist propranolol) were trained with a t
81 s (saline, 1, 3, 5 or 10 mg kg-1 of the beta-adrenergic antagonist propranolol) were trained with a t
82 n the NPE but not PE was blocked by the beta-adrenergic antagonist propranolol, the gap junction bloc
84 Moreover, a single injection of the beta-adrenergic antagonist, propranolol, prevented the noctur
85 er, incubation of RG20 cells with the alpha2-adrenergic antagonist rauwolscine or with pertussis toxi
86 Pretreatment with selective beta1- and beta2-adrenergic antagonists reduced the increase in phosphory
88 cycline chemotherapy with adjunctive beta(2)-adrenergic antagonists represents a potential therapeuti
90 of mice housed at 22 degrees C with a beta2-adrenergic antagonist reverses the norepinephrine-driven
94 ugh it is the most important target for beta-adrenergic antagonists, such as beta-blockers, relativel
95 usion of dobutamine or treatment with a beta-adrenergic antagonist, suggesting that RLC is constituti
97 tenuated by infusion of the selective alpha1 adrenergic antagonist terazosin into the basal forebrain
100 t to the microvessel preparation or an alpha-adrenergic antagonist to the myocytes and was augmented
102 ved vasoconstriction, phentolamine, an alpha-adrenergic antagonist was administered prior to MA treat
104 l melatonin formation and is induced by beta-adrenergic antagonists, which block melatonin production
105 in the heart and is the main target for beta-adrenergic antagonists, widely used in the treatment of
108 nd this suppression was reversed by alpha(2)-adrenergic antagonists with an order of inverse efficacy
109 gn, we orally administered either the alpha2-adrenergic antagonist yohimbine (increasing noradrenergi
110 ic, that of Dex being blocked by the alpha-2 adrenergic antagonist yohimbine, 5 mg/kg, i.p.; that of
112 of NE by approximately 30%, whereas alpha(2)-adrenergic antagonist, yohimbine, did not prevent the st