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1 ergic receptor stimulation inhibits the beta-adrenergically activated Cl- current, demonstrating a no
2 lin-dependent protein kinase II (CaMKII), an adrenergically activated kinase that contributes to arrh
5 d a significantly reduced ability to undergo adrenergically induced reactivation, i.e., viral sheddin
7 ial dynamics is a physiological regulator of adrenergically-induced changes in energy expenditure.
9 at least in part to a reduction of the beta-adrenergically mediated (PKA-dependent) phosphorylation
10 en CASQ2 mutations and the predisposition to adrenergically mediated arrhythmias observed in patients
11 1 integrin cytoplasmic domains inhibited the adrenergically mediated atrial natriuretic factor respon
13 ered activity in infected cells, (2) reduced adrenergically mediated ventricular tachycardia in treat
14 ortant role in muscarinic inhibition of beta-adrenergically regulated Cl- channels in these cells.
15 of alpha-adrenergic stimulation on the beta-adrenergically regulated Cl- current in guinea pig ventr
16 gatory for muscarinic inhibition of the beta-adrenergically regulated ICa in ventricular myocytes.
18 Incorporation of the resulting measured beta-adrenergically stimulated calcium transients into the mo
19 gatory role in muscarinic inhibition of beta-adrenergically stimulated ion channels in cardiac sinoat
22 nherited cardiac arrhythmia characterized by adrenergically triggered arrhythmias, is inadequately tr