ライフサイエンスコーパス: adventitial

1 reas of patchy or diffuse intimal, medial or adventitial abnormalities with thickening/accumulation o

2 r of diseases of the cardiopulmonary system, adventitial and interstitial fibroblasts are subjected t

3 numbers of inflammatory cells, and extensive adventitial and intimal neovascularity.

4 87b can contribute to cell death and loss of adventitial and medial integrity during hypertension-ind

5 al coronary arteries and in nonproliferating adventitial and neointimal cells 14 days after angioplas

6 tia of ApoE(-/-) mice produced donor-derived adventitial and peri-adventitial microvessels after athe

7 ings and localized inflammatory cells to the adventitial and periadventitial domains of injured vesse

8 nding into periaortic vascular channels, and adventitial and periaortic inflammatory infiltrates were

9 titia is a dynamic microenvironment in which adventitial and perivascular adipose tissue cells initia

10 a and media, less attention has been paid to adventitial and perivascular responses and their potenti

11 PDGFRalpha(+) cells present in adventitial and urothelial layers of murine renal pelvis

12 The active CMV infections involved adventitial and, less frequently, intimal cells.

13 Our findings indicate that adventitial angiogenesis stimulates intimal thickening b

14 subcutaneous injection of vitamin D(3) or by adventitial application of calcium chloride.

15 Adventitial application of the O2--generating system xan

16 s included the measurement of neointimal and adventitial area and thickness.

17 - 0.03 mm; p < 0.001) and a 23% reduction in adventitial area normalized to vessel size (0.43 +/- 0.0

18 0 Gy, 0.00+/-0.01 mm(2); P<0.05) and larger adventitial areas (20 Gy, 2.25+/-0.75 mm(2); 30 Gy, 2.38

19 Tracheal adventitial arterioles (13.0 to 41.0 microns initial dia

20 r space can enter the artery either from the adventitial aspect or from the lumen after absorption by

21 s of medial disruption, predominantly on the adventitial aspect.

22 e progenitor cells provides new insight into adventitial biology and its participation in atheroscler

23 taining showed that Thy-1 was upregulated in adventitial blood vessels after balloon injury to the ca

24 es were drawn at the lumen-intimal and media-adventitial borders.

25 whether antagonism of PDGF signaling alters adventitial cell migration after balloon injury in rat c

26 d in a selective expansion of the outermost, adventitial cell population in the great vessels.

27 crest results in expansion restricted to an adventitial cell population of the developing great vess

28 Opn was also detected in this adventitial cell population, but in addition was express

29 We have previously shown that adventitial cell proliferation increases significantly 4

30 in-Cre transgene targets perivascular cells (adventitial cells and pericyte-like cells) in WAT, and N

31 es, including both neuronal and non-neuronal adventitial cells and smooth muscle.

32 e the characteristics of coronary medial and adventitial cells and to compare the responses of corona

33 ponse to vascular stress or injury, resident adventitial cells are often the first to be activated an

34 hat reduced PD-L1 expression in VZV-infected adventitial cells contribute to persistent vascular infl

35 eported that only occasional fibroblasts and adventitial cells derived from c-kit positive progenitor

36 ce analyses to test whether VZV infection of adventitial cells downregulates PD-L1 showed decreased P

37 ecent findings suggesting the involvement of adventitial cells in coronary repair have raised questio

38 n situ hybridization showed that peri-aortic adventitial cells in high fat-fed mice express Msx2.

39 ents were designed to study the migration of adventitial cells in response to mechanical injury of th

40 evious studies suggest that the migration of adventitial cells into the neointima after balloon angio

41 n the lung, in myocytes in the heart, and in adventitial cells lining the arteries including the aort

42 kedly reduced in the stroma of heart valves, adventitial cells of the aortic root, perivascular and i

43 zation of the vascular wall by proliferative adventitial cells that contribute to the repair.

44 Adventitial cells were stained in situ with PKH26, a flu

45 l, localized to subpopulations of medial and adventitial cells, and the expression of leptin by arter

46 medial cells and growth factor production by adventitial cells, both of which resulted in maladaptive

47 served that large numbers of fibroblasts and adventitial cells, some smooth muscle and endothelial ce

48 and bromodeoxyuridine labeling to activated adventitial cells, which translocated to neointima.

49 restricted to subpopulations of intimal and adventitial cells.

50 n of smooth muscle alpha-actin expression in adventitial cells.

51 oronary arteries, respectively), and various adventitial cells.

52 strated rFGF2-SAP binding to medial SMCs and adventitial cells.

53 and neointima, but not within endothelial or adventitial cells.

54 ut on extensive damage, they are replaced by adventitial cells.

55 These intimomedial interface and adventitial changes may play a role in the natural histo

56 elastic lamina (IEL) rupture, and medial and adventitial changes, including inflammation, fibrosis, a

57 Almost all adventitial ChAT-I bundles and thin fibers, and VIP-I me

58 response 3 days after injury over the entire adventitial circumference.

59 actor-beta1 (TGF-beta1)-related increases in adventitial collagen and reductions in medial elastin, w

60 distribution, however the fiber families of adventitial collagen are obscured by their waviness at n

61 Adventitial collagen content at the site of injury was i

62 normally but neointimal lesion formation and adventitial collagen deposition in response to carotid a

63 ributes to favorable arterial remodeling and adventitial collagen deposition via a mechanism that is

64 Adventitial collagen deposition was apparent after 28 da

65 angiotensin II infusion caused marked aortic adventitial collagen deposition, as quantified by Masson

66 The adventitial collagen exists as large wavy bundles of fib

67 This is accompanied by increased adventitial collagen, which may act as an external "scaf

68 s of interstitial (matrix) and perivascular (adventitial) collagen were analyzed with polarization mi

69 significantly greater when detected from the adventitial compared with the intimal aspect of the arte

70 The altered perivascular adventitial compartment and its associated reticular net

71 h muscle layers in these diseases, the outer adventitial compartment is poorly explored.

72 es only expressed LacZ positive cells in the adventitial compartment; however, after injury in LacZ a

73 t, only diffuse fibrin was identified in the adventitial compartments of arteries from PAI-1(-/-) mic

74 Expanded adventitial compartments with perivascular infiltrates s

75 al reference and stented segment luminal and adventitial contours were imported and reconstructed.

76 We propose that mammalian perivascular adventitial 'cuffs' are conserved sites in multiple orga

77 Adventitial DCN is reduced in abdominal aortic aneurysm

78 Activation of adventitial DCs initiates and maintains T cell responses

79 umatica (PMR), a subclinical variant of GCA, adventitial DCs were mature and produced the chemokines

80 Test the effects of adventitial delivery of Multistem in the peri-infarct pe

81 ne kinase-MB or troponin associated with the adventitial delivery of MultiStem.

82 livery of MultiStem with a first-in-coronary adventitial delivery system to determine the effects of

83 otid artery intima-media thickness and inter-adventitial diameter were measured by ultrasonography an

84 ventitia, along with macrophage recruitment, adventitial expansion, and development of thoracic and s

85 fic for eNOS documented both endothelial and adventitial expression in Ad.CMVeNOS arteries, whereas v

86 l consequences of activating this pathway on adventitial fibroblast (AF) migration in vitro.

87 The locations of MARs in aortic adventitial fibroblast (AoAF) cells were very stable (r

88 In contrast, perivascular adventitial fibroblast expression of VCAM1 is upregulate

89 r ATP would modulate/mediate hypoxia-induced adventitial fibroblast growth.

90 rectly attenuates integrin-beta(3)-dependent adventitial fibroblast migration after inhibition of OPN

91 tibody (F11) pretreatment markedly inhibited adventitial fibroblast migration directed by exogenous O

92 observations provide direct demonstration of adventitial fibroblast migration into neointima of arter

93 s modulation by estrogen, and its effects on adventitial fibroblast migration.

94 rthermore, immunodepletion of p67(phox) from adventitial fibroblast particulates resulted in the loss

95 The emergence of a distinct adventitial fibroblast population with an epigenetically

96 Losartan reduced Ang II-induced VSMC and adventitial fibroblast proliferation but had no effect o

97 ively, our data demonstrate that ATP-induced adventitial fibroblast proliferation requires activation

98 rrent study was to characterize perivascular adventitial fibroblast states in inflammatory human skin

99 cell types (ie, endothelial, smooth muscle, adventitial fibroblast) undergo site- and time-dependent

100 re, using a technique to isolate and culture adventitial fibroblasts (AdvFBs) and vasa vasorum endoth

101 wth of medial smooth muscle cells (SMCs) and adventitial fibroblasts (AFBs) that we have shown expres

102 (VZV)-infected primary human brain vascular adventitial fibroblasts (BRAFs), levels of beta interfer

103 mpared to mock-infected human brain vascular adventitial fibroblasts (HBVAFs), perineural cells (HPNC

104 1 induced upregulation of collagen in aortic adventitial fibroblasts and enhanced the expression of c

105 ncreased extracellular ATP concentrations in adventitial fibroblasts and in lung microvascular endoth

106 Thus NAD(P)H oxidases in adventitial fibroblasts and macrophages appear to modula

107 ling in PH, and uncover a cross-talk between adventitial fibroblasts and macrophages in which paracri

108 re the NADH/NAD(+) ratio in bovine and human adventitial fibroblasts and mouse lung tissues.

109 histochemical staining showed marker eGFP in adventitial fibroblasts and some macrophages, indicating

110 -generating activity was localized to aortic adventitial fibroblasts and was enhanced by the potent v

111 In normal skin, adventitial fibroblasts are distinguished by CD90 expres

112 Activated adventitial fibroblasts are endowed with synthetic capab

113 ndothelial and smooth muscle cells, vascular adventitial fibroblasts contain a substantial NAD(P)H ox

114 Migration of adventitial fibroblasts contributes to vascular remodeli

115 in response to stimulation, whereas coronary adventitial fibroblasts demonstrated several characteris

116 ne fibroblasts) approaches, we observed that adventitial fibroblasts derived from hypertensive pulmon

117 increase in p21 expression that occurred in adventitial fibroblasts during the cell cycle.

118 We found that adventitial fibroblasts from calves with severe hypoxia-

119 Pulmonary adventitial fibroblasts from chronically hypoxic hyperte

120 rative, apoptosis-resistant, proinflammatory adventitial fibroblasts from human and bovine hypertensi

121 ion in injured arteries as well as that from adventitial fibroblasts in vitro.

122 was performed in cardiopulmonary tissues and adventitial fibroblasts isolated from pulmonary arteries

123 ting stimulus for subsets of bovine neonatal adventitial fibroblasts largely through Galpha(i/o)-medi

124 ation in both aortic smooth muscle cells and adventitial fibroblasts may contribute to development of

125 er balloon injury of the rat carotid artery, adventitial fibroblasts migrate in a luminal direction a

126 shown to act as a proliferative stimulus for adventitial fibroblasts of the pulmonary artery.

127 In vitro, coculture of monocytes and aortic adventitial fibroblasts produced MCP-1- and IL-6-enriche

128 injury triggers differentiation of activated adventitial fibroblasts to myofibroblasts, which may con

129 GF attenuates remodeling and contribution of adventitial fibroblasts to neointima formation after bal

130 We additionally used PA adventitial fibroblasts to test the hypothesis that TG2

131 ession of factor(s) controlling migration of adventitial fibroblasts via an ER-dependent mechanism.

132 IRS1 expression in adventitial fibroblasts was predominantly nuclear and nu

133 Primary cultures of VSMCs and adventitial fibroblasts were derived from female Sprague

134 of vascular smooth muscle cells (VSMCs) and adventitial fibroblasts were derived from female Sprague

135 Pulmonary adventitial fibroblasts were isolated from calves and hu

136 Primate aortic smooth muscle cells and adventitial fibroblasts were seeded into collagen I gels

137 Primary syngeneic adventitial fibroblasts were stably transduced with retr

138 Indeed, inhibition of miR-487b protected adventitial fibroblasts, and also medial smooth muscle c

139 rix deposition, an increase in the number of adventitial fibroblasts, and intimal thickening.

140 ng immunoglobulins, cultured human pulmonary adventitial fibroblasts, and network medicine analysis o

141 cells, pulmonary artery smooth muscle cell, adventitial fibroblasts, and pulmonary and systemic infl

142 lymphoid tissue and, when bound to pulmonary adventitial fibroblasts, change their phenotype to one t

143 In primary rat and human arterial adventitial fibroblasts, inhibition of miR-487b leads to

144 In cultured adventitial fibroblasts, TGF-beta1 induced increases in

145 lar inflammation is perpetuated by activated adventitial fibroblasts, which, through sustained produc

146 re required for ATP-induced proliferation of adventitial fibroblasts.

147 of soluble factor(s) directing migration of adventitial fibroblasts.

148 ism whereby estrogen attenuates migration of adventitial fibroblasts.

149 ess includes the activation and migration of adventitial fibroblasts.

150 uscle cells (VSMCs), inflammatory cells, and adventitial fibroblasts.

151 at recombinant eNOS protein was expressed in adventitial fibroblasts.

152 ress markers of smooth muscle precursors and adventitial fibrocytes, respectively, by E13.5.

153 ive remodeling, which coincided with reduced adventitial fibrosis and collagen deposition.

154 TGF-beta isoforms as major factors mediating adventitial fibrosis and negative remodeling after vascu

155 able effect on late remodeling by preventing adventitial fibrosis at the injury site.

156 medial smooth muscle cell degeneration, and adventitial fibrosis.

157 intimal hyperplasia, medial hypertrophy, and adventitial fibrosis.

158 We describe a mechanistic model of adventitial function that brings together current knowle

159 Genetic fate tracing indicates that adventitial Gli1(+) MSC-like cells migrate into the medi

160 O2- derived from adventitial gp91(phox)-based NAD(P)H oxidase contributes

161 ocked by KMD3213 (alpha1A-AR antagonist) and adventitial growth by AH11110A (alpha1B-AR antagonist),

162 positive in several biopsy specimens within adventitial histiocytes-macrophages, but these results d

163 matic quantification of intimal, medial, and adventitial histopathological features in 598 human aort

164 duced proteoglycan deposition, and inhibited adventitial hypertrophy.

165 ch on the formation and structural nature of adventitial immune aggregates, potential mechanisms of c

166 three patterns: adventitial nodules, diffuse adventitial infiltrates, and neointimal infiltrates.

167 Medial and adventitial inflammation (P=0.01), medial fibrosis (P=0.

168 s characteristic of GCA, and 16 (36%) showed adventitial inflammation adjacent to viral antigen; no i

169 by increased oxidative stress, a promoter of adventitial inflammation and vasa vasorum neovasculariza

170 creased incidence of IEL rupture, medial and adventitial inflammation, medial fibrosis, and medial at

171 ntimal hemorrhage, and increased intimal and adventitial inflammation.

172 cterized by a strikingly increased number of adventitial inflammatory cells, highly proliferative, an

173 Histology demonstrated a marked adventitial inflammatory response in all allografts, wit

174 Our hypothesis was that adventitial injection of rapamycin nanoparticles would b

175 re randomized to the double-injury model and adventitial injection of saline (n=2) or 500 mug of nano

176 An intraluminal microinfusion catheter for adventitial injection represents an alternative to stent

177 Yorkshire pigs (20-25 kg; n=7) through intra-adventitial injections of collagenase (5 mL, 0.35 mg/mL)

178 mooth muscle cells (VSMCs) and in associated adventitial/interstitial fibroblasts of intramyocardial

179 centric layers of muscle cells and the outer adventitial layer are assembled and patterned around end

180 n demonstrated active invasion of the aortic adventitial layer by P. gingivalis.

181 with vessel walls stripped of the intimal or adventitial layer identified dendritic cells at the medi

182 hog (Shh) signaling domain restricted to the adventitial layer of artery wall that supports resident

183 ta and 37 in allograft) were analyzed in the adventitial layer with a total number of 8568 vectors pr

184 but also induces profound remodelling of the adventitial layer.

185 Macrophages were present primarily in the adventitial layer; B lymphocytes were notably absent.

186 mphocyte (P<0.0179) invasion into medial and adventitial layers and inhibited associated depletion of

187 its mesenchymal cells to form the medial and adventitial layers of arterioles and venules during the

188 We found PDGFRalpha(+) ICs in the adventitial layers of the pelvis, the muscle layer of th

189 Observed reductions in early adventitial leukocyte infiltration and late medial cell

190 There were significantly fewer adventitial leukocytes at 3 days, P<0.001, but no differ

191 Beneath these lesions, adventitial leukocytes organize in clusters that resembl

192 e dose heterogeneity at both the intimal and adventitial levels.

193 c and inflammation-related transcripts in an adventitial lymphatic EC (LEC) population in a ligand-de

194 Phenotypic transformation of intimal and adventitial lymphatics in atherosclerosis: a regulatory

195 Despite increased VEGF-C, we found that adventitial lymphatics regress during the course of form

196 ed VEGF-C in the atherosclerotic aortas, how adventitial lymphatics regress.

197 hts to previously unknown dynamic changes of adventitial lymphatics.

198 w of the contributions of the adventitia and adventitial lymphocytes to the development of atheroscle

199 d moderate to severe intimal, medial, and/or adventitial lymphocytic infiltration with intimal expans

200 d chemokine production, as well as transient adventitial macrophage accumulation and activation.

201 ry and characterization of resident vascular adventitial macrophage progenitor cells provides new ins

202 ansfer studies revealed that Sca-1(+)CD45(+) adventitial macrophage progenitor cells were not repleni

203 Rather adventitial macrophage progenitor cells were upregulated

204 rogenitors (EMP) contribute substantially to adventitial macrophages and give rise to a defined clust

205 Human adventitial macrophages displaying a CD14(+)/CD16(+) res

206 l hyperplasia with increased infiltration of adventitial macrophages expressing MCP-1.

207 response to AngII inflammation, increase in adventitial macrophages is driven by recruitment of BM m

208 In contrast to aortic adventitial macrophages, CD11c(+)MHC II(hi) DCs were poo

209 e CD45 isoform is more prevalent in resident adventitial macrophages.

210 migration and recruitment of mural cells and adventitial macrophages.

211 nd III collagen were largely found in either adventitial/medial or transmural locations.

212 Type VI collagen was adventitial or adventitial/medial.

213 Tofacitinib disrupted adventitial microvascular angiogenesis, reduced outgrowt

214 utral beta-galactosidase and, in contrast to adventitial microvessel endothelium, exhibited weak stai

215 endothelial cells) in large-vessel lumen and adventitial microvessel lumen of arteriopathic vessels.

216 produced donor-derived adventitial and peri-adventitial microvessels after atherogenic diet, suggest

217 Transduction of adventitial microvessels was enhanced by balloon injury

218 s Wnt2, Wnt5a, and Sca1 expression in aortic adventitial myofibroblast cultures.

219 metalloproteinase (MMP) activation in aortic adventitial myofibroblasts (AMFs) and A7r5 vascular smoo

220 We hypothesize that adventitial myofibroblasts are actively involved in the

221 Aortic adventitial myofibroblasts from SM22-Cre;Msx1(fl/fl);Msx

222 Despite the prominent role that adventitial myofibroblasts seem to have in the postangio

223 We have previously shown that adventitial myofibroblasts synthesize growth factors tha

224 ng demonstrated that TnC expression began in adventitial myofibroblasts three days after injury.

225 Other vascular cells, including adventitial myofibroblasts, calcifying vascular cells, s

226 e in the recruitment and/or proliferation of adventitial myofibroblasts, possibly through the release

227 regions of Msx2 immunoreactivity in adjacent adventitial myofibroblasts, suggesting a potential parac

228 pression early after vascular injury was the adventitial myofibroblasts.

229 Tenascin-C expression begins with adventitial myofibroblasts.

230 tion in alkaline phosphatase (TNAP)-positive adventitial myofibroblasts.

231 Adventitial nab-rapamycin injection was safe and signifi

232 tulated that the interaction between NO. and adventitial NAD(P)H oxidase-derived O2- contributes to i

233 Adventitial neovascularization occurs after balloon inju

234 This study suggests that adventitial neovascularization of vasa vasorum occurs in

235 ) is seen in perineurial cells that surround adventitial nerve bundles and form the peripheral nerve-

236 y histological and biochemical alteration in adventitial nerves and correlated with improved hemodyna

237 We identified a dominant adventitial niche around lung bronchi and larger vessels

238 These data indicate that adventitial niches are conserved sites where ASCs regula

239 16) and were distributed in three patterns: adventitial nodules, diffuse adventitial infiltrates, an

240 In middle cerebral arteries, all adventitial NOS-I bundles and fine fibers were coinciden

241 m gp91(phox-/-) mice, which lack significant adventitial O2-, exhibited greater EDR and were not affe

242 Adventitial oligoclonal resident B cells of atherosclero

243 Type VI collagen was adventitial or adventitial/medial.

244 racic aorta, which were oriented so that the adventitial or luminal surface could be preferentially e

245 nvestigations to test specific hypotheses on adventitial pathobiology.

246 The interaction between medial and adventitial pathology and the intimal atherosclerotic pr

247 er the dissection of aspects of perivascular adventitial pathology.

248 st in part, by limiting leukocyte entry from adventitial/periadventitial tissues into injured vessels

249 Transplantation of adventitial pericytes (APCs) improves recovery from tiss

250 onocytes/macrophages in the pulmonary artery adventitial/perivascular areas of animals and humans wit

251 smural panarteritis and TLR5 ligands promote adventitial perivasculitis.

252 Additionally, we identified a CD31-positive adventitial plexus after ligation of the carotid artery

253 In the rat carotid injury model, local adventitial polymer-based delivery of radiolabeled linea

254 We identified a vascular adventitial population containing macrophage progenitor

255 Adventitial progenitors express the stem cell markers, S

256 findings implicate Gli1(+) cells as critical adventitial progenitors in vascular remodeling after acu

257 with a reduction in neointima formation and adventitial reaction after balloon injury.

258 microscopic evidence of a pronounced chronic adventitial reaction, with perivascular infiltration pro

259 High rates of residual tumor in the adventitial region even inside the radiation fields will

260 or gadolinium chloride, prevented pulmonary adventitial remodeling (ie, production of collagen, fibr

261 facilitates migration of these cells during adventitial remodeling.

262 pand in number and are major contributors to adventitial remodeling.

263 ents, indicating an inhibitory effect on the adventitial response to injury.

264 eyond its content of macrophage progenitors, adventitial Sca-1(+)CD45(+) cells are also vasculogenic

265 xpress the stem cell markers, Sca1 and CD34 (adventitial sca1-positive progenitor cells [AdvSca1]), h

266 More recently, pars plana vitrectomy with adventitial sheathotomy has also been shown to be of ben

267 )) and patched-2 (Ptc2(lacZ)) reporter mice, adventitial Shh signaling activity was first detected at

268 Endogenous O2- was increased by treating the adventitial side of the aortas with Ang II (10 pmol/L),

269 In vivo, local application of VEGF to the adventitial side of the decellularized vessel increased

270 Arterial radiation resulted in a decrease in adventitial size, which was maximal for high-activity (3

271 s likely causes the transition of medial and adventitial smooth muscle cells (SMC) into classic myofi

272 Further study into the regulation of this adventitial source of O2- is important in elucidating th

273 , ILC2s were most intimately associated with adventitial stromal cells (ASCs), a mesenchymal fibrobla

274 EDR impairment was reversed by adventitial suffusion of superoxide dismutase (SOD) of a

275 This study suggests that adventitial superoxide anion can play a role in the path

276 adrenaline (NA) overflow at the blood vessel adventitial surface and vasoconstriction evoked by elect

277 ecifically, deformations are measured at the adventitial surface by tracking motions of a speckle pat

278 eparin uniformly and at a steady rate to the adventitial surface of balloon-injured rat carotid arter

279 Adventitial TGF-beta1-related oxidative stress may play

280 Irradiated vessels demonstrated more adventitial thickening and fibrosis.

281 [FGF-R1DN]), respectively, signaling reduced adventitial thickening induced by VEGF and PR39 to the l

282 t phase is defined by nonspecific medial and adventitial thickening of the pulmonary artery and is co

283 s initiated, although medial hypertrophy and adventitial thickening still developed.

284 ally by pulmonary artery medial hypertrophy, adventitial thickening, and neointimal proliferation.

285 ypes (intimal thickening, media hypertrophy, adventitial thickening, plexiform lesions, vascular prun

286 iance, decreased ankle-brachial indexes, and adventitial thickening.

287 h higher intimal thickness but not medial or adventitial thickness as measured by histology.

288 intimal area, maximal intimal thickness, and adventitial thickness were significantly reduced in both

289 in the number of macrophages present in the adventitial tissue underlying lesions.

290 nounced dilation and more modestly remodeled adventitial tissue.

291 ipidemic ApoE(-/-) and LDL-R(-/-) mice, with adventitial transfer experiments demonstrating their dur

292 Adventitial transplantation of MSCs decreases Mcp-1 gene

293 IP NCs arose directly from adventitial vasa vasorum and were anatomically and quant

294 In balloon-injured coronary arteries, adventitial vasa vasorum density was increased (3.16+/-0

295 rPAI-1(23) causes regression or collapse of adventitial vasa vasorum in hypercholesterolemic mice by

296 cellular and molecular mechanisms regulating adventitial vasa vasorum neovascularization, which occur

297 to examine the quantitative response of the adventitial vasa vasorum to balloon-induced coronary inj

298 of diffusion from the artery lumen and outer adventitial vasa vasorum, deposit proatherogenic plasma

299 of the atherosclerotic plaque and associated adventitial vasa vasorum.

300 Interestingly, adventitial vascularity significantly increased, suggest