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1 ened beverage (SSB) consumption is linked to adverse health outcomes.
2 ium intake and high blood pressure and other adverse health outcomes.
3 CA) increases stress sensitivity and risk of adverse health outcomes.
4 affeine has been linked to both positive and adverse health outcomes.
5 ductive infections, preterm birth, and other adverse health outcomes.
6 ations was associated with a higher risk for adverse health outcomes.
7 ch use to patients may not outweigh costs or adverse health outcomes.
8 mmigration may be particularly vulnerable to adverse health outcomes.
9 hat show the association between frailty and adverse health outcomes.
10 re known to be at risk for treatment-related adverse health outcomes.
11 senic (iAs) causes cancer and possibly other adverse health outcomes.
12 will reduce PM emissions but not necessarily adverse health outcomes.
13 he developing fetus and child from potential adverse health outcomes.
14 decrease the risk of hypertension and other adverse health outcomes.
15 ssing may place a child at a bigger risk for adverse health outcomes.
16 ve of BV status and correlates strongly with adverse health outcomes.
17 re, and is associated with increased risk of adverse health outcomes.
18 at lower concentrations) and a wide range of adverse health outcomes.
19 m of the vitamin in blood is associated with adverse health outcomes.
20 upport for self-management may contribute to adverse health outcomes.
21 a high lifetime risk of a broad spectrum of adverse health outcomes.
22 gical studies on the association of BPA with adverse health outcomes.
23 nce problems, adverse drug events, and other adverse health outcomes.
24 Reduced fetal growth increases the risk for adverse health outcomes.
25 and identifies a state of vulnerability for adverse health outcomes.
26 s an early warning system of risk for future adverse health outcomes.
27 ism; impaired methylation is associated with adverse health outcomes.
28 e of a healthy vaginal ecosystem, can reduce adverse health outcomes.
29 t (DBP) exposure has been linked to multiple adverse health outcomes.
30 s a strong and independent predictor of such adverse health outcomes.
31 poverty), inability to obtain services, and adverse health outcomes.
32 nowledge of the links between overweight and adverse health outcomes.
33 n stress-related personality differences and adverse health outcomes.
34 reliable and unrealistic estimates of future adverse health outcomes.
35 sue homeostasis, increased vulnerability and adverse health outcomes.
36 eczema may be related to multiple subsequent adverse health outcomes.
37 prevalent condition that is associated with adverse health outcomes.
38 cient vaginal microbiota, is associated with adverse health outcomes.
39 ctuation is associated with greater risks of adverse health outcomes.
40 ke in Europe, concerning as UPF is linked to adverse health outcomes.
41 CKD is common, costly, and associated with adverse health outcomes.
42 transition are warranted, as well as defined adverse health outcomes.
43 us may play a role in their association with adverse health outcomes.
44 Overweight and obesity are associated with adverse health outcomes.
45 ensive circadian rhythm desynchrony promotes adverse health outcomes.
46 Early menarche is associated with adverse health outcomes.
47 (PFASs) have been associated with a range of adverse health outcomes.
48 sed disease risk is important for mitigating adverse health outcomes.
49 y recognized as significant risk factors for adverse health outcomes.
50 ns between experiences of discrimination and adverse health outcomes.
51 iated with increased likelihood of reporting adverse health outcomes.
52 s of urban air pollution, is associated with adverse health outcomes.
53 ty supervision-is associated with a range of adverse health outcomes.
54 inflammatory response, is a risk factor for adverse health outcomes.
55 Food insecurity is associated with numerous adverse health outcomes.
56 state associated with accelerated aging and adverse health outcomes.
57 iness have individually been associated with adverse health outcomes.
58 ns, with low health literacy associated with adverse health outcomes.
59 ther nonuse of such care was associated with adverse health outcomes.
60 xamine the association between drug dose and adverse health outcomes.
61 gle to afford prescription drugs, leading to adverse health outcomes.
62 opefully, reduce rates of and disparities in adverse health outcomes.
63 ing radon and its progeny is associated with adverse health outcomes.
64 rt of the prevention and management of these adverse health outcomes.
65 ronicity of loneliness affects its impact on adverse health outcomes.
66 ld improve the strength of associations with adverse health outcomes.
67 ds (UPFs), which have been linked to several adverse health outcomes.
68 ation, and associated with increased risk of adverse health outcomes.
69 gs, LMB survivors were at increased risk for adverse health outcomes.
70 ructive sleep apnea (OSA) is associated with adverse health outcomes.
71 ned beverage consumption and higher risks of adverse health outcomes.
72 ct on health information and are at risk for adverse health outcomes.
73 of high blood pressure and potentially other adverse health outcomes.
74 Poverty is strongly associated with numerous adverse health outcomes.
75 lution exposure has been linked with several adverse health outcomes.
76 ons in the mother, fetus, and placenta, plus adverse health outcomes.
77 sity students and is hypothesized to lead to adverse health outcomes.
78 interest because of their associations with adverse health outcomes.
79 laining reported associations between EA and adverse health outcomes.
80 ations between prenatal smoking exposure and adverse health outcomes.
81 ionship may confer the longest-term risk for adverse health outcomes.
82 that have predictive value for beneficial or adverse health outcomes.
83 eydig cell dysfunction (LCD), and associated adverse health outcomes.
84 imilar risk factors, was not associated with adverse health outcomes.
85 eather events that have been associated with adverse health outcomes.
86 Working long hours has been associated with adverse health outcomes.
87 ociated with cancer susceptibility and other adverse health outcomes.
88 ociated with functional measures or risk for adverse health outcomes.
89 -based multiorgan phenotypes associated with adverse health outcomes.
90 oxide (NO2) air pollution is associated with adverse health outcomes.
91 tencies; it is unclear how exposure leads to adverse health outcomes.
92 dwide but also are critical risk factors for adverse health outcomes.
93 ions that can lower SBPV and their impact on adverse health outcomes.
94 Insomnia is associated with several adverse health outcomes.
95 standards can potentially be used to predict adverse health outcomes.
96 socioeconomic deprivation is associated with adverse health outcomes.
97 irths are at substantially increased risk of adverse health outcomes.
98 itamin D deficiency has been associated with adverse health outcomes.
99 nnot be synthesized by humans, are linked to adverse health outcomes.
100 2.5 mum (PM2.5) in aerodynamic diameter and adverse health outcomes.
101 e action that may be relevant to a number of adverse health outcomes.
102 of biological systems and with it associated adverse health outcomes.
103 the aging population and are associated with adverse health outcomes.
104 s that climate change can be associated with adverse health outcomes.
105 aged social environments are associated with adverse health outcomes.
112 s associated with immunologic impairment and adverse health outcomes among adults infected with human
113 ion exposure has been linked to a variety of adverse health outcomes among minors; yet little is know
114 ined associations between PFOA exposures and adverse health outcomes among residents in highly expose
117 adian rhythms and is associated with several adverse health outcomes and chronic diseases such as can
118 phasizes the relationship between stigma and adverse health outcomes and health access measures.
119 pecially deep/organ space SSI, contribute to adverse health outcomes and health care costs across the
120 ement was associated with increased rates of adverse health outcomes and health care utilization.
121 from the vaginal microbiome associates with adverse health outcomes and is linked to increased risk
122 nded follow-up evaluation increases risk for adverse health outcomes and medical or legal issues.
123 iming is genetically correlated with several adverse health outcomes and Mendelian randomization anal
125 is a major public health problem leading to adverse health outcomes and neurodevelopmental abnormali
126 on the role of functional iron deficiency in adverse health outcomes and on iron deficiency screening
127 and initiation of antiviral therapy to avert adverse health outcomes and reduce onward transmission o
128 s to investigate the spatial distribution of adverse health outcomes and their relationship with soci
129 Childhood maltreatment is associated with adverse health outcomes and this risk can be transmitted
132 known endocrine disruptor linked to numerous adverse health outcomes and was, therefore, banned in fo
133 otential associations among TDCIPP exposure, adverse health outcomes, and DNA methylation status with
134 a higher incidence of activity limitations, adverse health outcomes, and limited access to health ca
135 sks including smoking, activity limitations, adverse health outcomes, and limited access to health ca
136 pollution has been implicated in a number of adverse health outcomes, and the effect of particulate m
138 High-risk medications that contribute to adverse health outcomes are frequently prescribed to old
141 Oz reactions leading to indoor exposures and adverse health outcomes are provided as well as an outlo
143 ween subclinical vitamin B-12 deficiency and adverse health outcomes as well as appropriate cutoffs f
144 h activity patterns are at a greater risk of adverse health outcomes associated with activity fragmen
145 Appreciating the quality and quantity of adverse health outcomes associated with CSA has helped c
147 xins in SA, relevant collection methods, and adverse health outcomes associated with cyanotoxin inhal
148 an 2.5 mum in diameter (PM(2.5)) to evaluate adverse health outcomes associated with particulate air
149 ng or highly suggestive level of evidence of adverse health outcomes associated with psychotropic med
150 enetic mechanisms in the pathogenesis of the adverse health outcomes associated with this important i
151 interesting mechanistic explanation of some adverse health outcomes associated with toxic cyanobacte
153 but recognized as a risk factor for several adverse health outcomes based on observational studies.
154 h a history of DMDD were more likely to have adverse health outcomes, be impoverished, have reported
157 ation (such as racism) have been linked with adverse health outcomes, but age discrimination has not
159 ommon in older adults and signifies multiple adverse health outcomes, but it often goes unrecognized.
161 c beverages is hypothesized to contribute to adverse health outcomes, but the beverages and populatio
164 leep problems have been linked to a range of adverse health outcomes, but there is limited knowledge
165 how associations between sleep duration with adverse health outcomes, but these have not systematical
166 pairs daily communication is associated with adverse health outcomes, but use of hearing aids by olde
167 ring studies are important for understanding adverse health outcomes caused by exposure to chemicals.
168 between HL or tinnitus and five prespecified adverse health outcomes (cognitive dysfunction, fatigue,
169 ndings of the reviewed studies, the risk for adverse health outcomes due to CEW exposure can be curre
170 millions of Americans suffer from a range of adverse health outcomes due to noise exposure, including
172 ed food was associated with a higher risk of adverse health outcomes, especially cardiometabolic, com
173 vorable multiorgan phenotype associated with adverse health outcomes, especially in elderly American
175 plain links between neighborhood factors and adverse health outcomes following pediatric liver transp
177 ated to significant short-term and long-term adverse health outcomes for both mothers and offspring.
178 ncy complication associated with significant adverse health outcomes for both women and infants.
179 r evidence that air pollution contributes to adverse health outcomes for children posttransplant and
181 ssion, malaria remains an important cause of adverse health outcomes for mothers and offspring, inclu
184 between particulate matter (PM) exposure and adverse health outcomes has been widely evaluated using
185 ds to determine a) whether the occurrence of adverse health outcomes has changed, and b) the extent t
186 tions between hormonal contraceptive use and adverse health outcomes has not been quantified in aggre
187 iations observed between passive smoking and adverse health outcomes have generated controversy.
188 nation are associated with increased risk of adverse health outcomes; however, it is unknown whether
189 ersistent chemicals associated with multiple adverse health outcomes; however, the biological pathway
193 ough body fat patterning has been related to adverse health outcomes in adults, its importance in chi
196 studies in Europe and California that found adverse health outcomes in children associated with mode
197 smoking is a major risk factor for multiple adverse health outcomes in children, but the underlying
200 ween major depression, early life stress and adverse health outcomes in diseases associated with infl
204 d with differences in the gut microbiota and adverse health outcomes in humans; however, little is kn
207 ure has been associated with a wide range of adverse health outcomes in limited epidemiologic studies
211 (SHS) has been associated with a variety of adverse health outcomes in nonsmokers, including emphyse
214 lear whether cannabis use is associated with adverse health outcomes in patients with COVID-19 when a
217 th system intervention to reduce the risk of adverse health outcomes in the newborn baby and, potenti
218 gestational weight gain (EGWG) is related to adverse health outcomes in the offspring; however, its e
219 s consistently document a high prevalence of adverse health outcomes in this population, including HI
220 etic and epigenetic bases of therapy-related adverse health outcomes in this vulnerable population.
221 bute to longstanding disparities and lead to adverse health outcomes in vulnerable populations, yet t
222 aginosis (BV) is a condition associated with adverse health outcomes in which bacteria reside in dire
224 aseline frailty was associated with multiple adverse health outcomes (incident CVD, DM, and bone dise
227 (ADHD) is associated with increased risks of adverse health outcomes including premature death, but i
228 rmone action, thereby increasing the risk of adverse health outcomes, including cancer, reproductive
232 air pollution and increased risk of various adverse health outcomes, including cardiopulmonary morta
233 and PM(10)), has been linked to a number of adverse health outcomes, including cardiovascular diseas
234 ociations between particle radioactivity and adverse health outcomes, including changes in blood pres
235 ions between alcohol intake and a variety of adverse health outcomes, including coronary heart diseas
236 rcadian disruption have been associated with adverse health outcomes, including diabetes and cardiova
237 ytes are associated with an elevated risk of adverse health outcomes, including hematologic malignanc
238 dently associated with an increased risk for adverse health outcomes, including impaired pulmonary fu
239 ing in many countries, is linked to multiple adverse health outcomes, including increased colorectal
241 ge (SGA) newborns have a higher frequency of adverse health outcomes, including linear and ponderal g
242 ing of solid biomass fuel is associated with adverse health outcomes, including low birth weight.
243 al period has been associated with long-term adverse health outcomes, including mood disorders and ab
244 disorder (PTSD), and poses a major risk for adverse health outcomes, including neurodegeneration and
246 are associated with short-term and long-term adverse health outcomes, including physical, psychologic
247 Chronic exposure to wood smoke can lead to adverse health outcomes, including respiratory infection
248 n disruptions are implicated in a variety of adverse health outcomes, including substance use disorde
249 cal illness has been associated with several adverse health outcomes, including various aspects of me
250 t changes in AT density with ART may lead to adverse health outcomes independent of AT quantity.
251 However, the body of evidence on potential adverse health outcomes indicates the need for comprehen
253 and other contextual factors associated with adverse health outcomes is needed to improve prediction
254 advantage and psychosocial stressors lead to adverse health outcomes is unclear, particularly when ex
255 tal muscle deterioration, linked to numerous adverse health outcomes, is driven by impairments in mus
256 hether accelerated epigenetic aging leads to adverse health outcomes later in life, these data point
258 tter (PM(2.5)) increases the risk of several adverse health outcomes, less is known about its relatio
259 Main limitation was inability to include adverse health outcomes not involving hospital contact.
260 elated exposures might plausibly explain the adverse health outcomes observed in flight crew, but our
261 impede public health efforts to mitigate the adverse health outcomes of a sedentary lifestyle in an a
263 s a prevalent disorder associated with major adverse health outcomes: Patients with NAFLD may develop
264 been associated with increased risk of many adverse health outcomes, prompting federal drinking wate
265 ess than 2.5 microm; PM2.5) constituents and adverse health outcomes rather than focusing solely on t
267 pic fat, pathophysiology and contribution to adverse health outcomes, response to treatment, and less
268 nflammation, DSB, and emphysema and explains adverse health outcomes seen in smokers while implicatin
269 with complicated grief have greater risk of adverse health outcomes, should be diagnosed and assesse
270 nal association between monoHAA exposure and adverse health outcomes such as oxidative stress, inflam
272 iations between cigarette smoking in men and adverse health outcomes, such as CAD and CLD, with adjus
273 ism by which tobacco exposure predisposes to adverse health outcomes, such as cancers, osteoporosis,
276 -2 were at risk for both direct and indirect adverse health outcomes, supporting efforts of ongoing s
277 ith PM2.5 may appear more closely related to adverse health outcomes than other constituents even if
278 ate visits was more strongly associated with adverse health outcomes than those recorded at a single
279 seases often occur together, leading to more adverse health outcomes than when a disease of one of th
280 pite mounting evidence linking it to various adverse health outcomes that include heightened pain per
281 logy to estimate numbers of deaths and other adverse health outcomes that would have been avoided dur
282 e linked elevations in tropospheric ozone to adverse health outcomes, the effect of long-term exposur
283 bipolar disorder is associated with various adverse health outcomes, the relationship with violent c
284 ugh fast food consumption has been linked to adverse health outcomes, the relative contribution of fa
285 Despite widespread exposure and potential adverse health outcomes, they are poorly understood in t
286 red effects cannot be directly translated to adverse health outcomes, this study demonstrates the app
288 beverages (SSBs) has been linked to several adverse health outcomes, thus many countries introduced
289 em responses that increase the likelihood of adverse health outcomes, underscoring the importance of
290 posure to organophosphate (OP) pesticides to adverse health outcomes via molecular mechanisms, such a
291 ivors of childhood cancer, the prevalence of adverse health outcomes was high, and a systematic risk-
292 g clinical criteria, the crude prevalence of adverse health outcomes was highest for pulmonary (abnor
293 nges in rates and geographic distribution of adverse health outcomes were detected, and, in each inst
294 ovascular risk, cancer risk, and other major adverse health outcomes were not supported by high-quali
295 ted rates of urinary tract cancers and other adverse health outcomes, which may be attributable to en
296 scores for the adiposity factor predict many adverse health outcomes, while those for body size and c
297 hat of oxidative stress, for associations of adverse health outcomes with PM2.5 mass and supports con
298 norganic arsenic is associated with numerous adverse health outcomes, with susceptibility differing b
299 34 weeks' gestation) confers a high risk for adverse health outcomes, yet no universal screening stra
300 (TRAP) during pregnancy has been linked with adverse health outcomes, yet the biological mechanisms r