1 idance was used for all masses surrounded by aerated lung.

2       The mean CT extent (+/-SD) of normally aerated lung, ground-glass opacification, and dense pare

3 ifferentially abundant in atelectasis versus aerated lung, mostly (n = 126) with less abundance toget

4                            Overdistension of aerated lung occurs during atelectasis is detectable usi

5 ients with radiographic evidence of residual aerated lung regions than in patients with diffuse bilat

6                          Perfusion to poorly aerated lung regions was unchanged or increased after in

7  in the upper lung regions and in the poorly aerated lung regions.

8  to greater distension-and thereby injury-of aerated lung regions; recruitment of atelectatic lung ma

9 ed with the corresponding fraction of poorly aerated lung tissue ( r = 0.62; p = 0.01) and of lung ti

10               Patients with greater normally aerated lung tissue in the ventral and medial-ventral re

11 The present findings suggest that the poorly aerated lung tissue is an important target of the perpet

12 al proteomics of atelectatic versus normally-aerated lung tissue to test the hypothesis that immune a

13  chest wall condition, at end-expiration non aerated lung tissue weight was increased by 116 +/- 68 %

14  in patients with ARDS because the amount of aerated lung varies considerably due to differences in i

15 s end-expiratory transpulmonary pressure and aerated lung volume.

16 mine the value of quantification of the well-aerated lung (WAL) obtained at admission chest CT to det

17  lung was traversed and approximately 50% if aerated lung was penetrated.

18  pneumothorax rate was 15% (16 of 105) if no aerated lung was traversed and approximately 50% if aera

19 as more than three times less frequent if no aerated lung was traversed.

20 may be attributed to the small volume of the aerated lung, which receives most of ventilation.