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1 ogen peroxide (H2O2) as toxic by-products of aerobic metabolism.
2 ic regulation of the H2O2 produced by normal aerobic metabolism.
3  from all other members by its facultatively aerobic metabolism.
4  in pH, indicated the presence of maintained aerobic metabolism.
5 tic loss of FIH promotes both glycolysis and aerobic metabolism.
6 le factor (FIH) enhances both glycolysis and aerobic metabolism.
7 ganism can use to fuel sustainable levels of aerobic metabolism.
8 pecies (ROS) are toxic by-products of normal aerobic metabolism.
9 egy for select cancers that are dependent on aerobic metabolism.
10 ormed as a natural byproduct of the cellular aerobic metabolism.
11 17,18), may be an inescapable consequence of aerobic metabolism.
12  specific inactivation of enzymes central to aerobic metabolism.
13 ar oxygen derivatives produced during normal aerobic metabolism.
14 cid (TCA) cycle is the epicenter of cellular aerobic metabolism.
15 en the presence of the microbial cluster and aerobic metabolism.
16  reactions in the cytosol to be oxidized via aerobic metabolism.
17 d in their expression of proteins supporting aerobic metabolism.
18 ipts encoding proteins uniquely required for aerobic metabolism.
19 exchange for utilization of oxygen-dependent aerobic metabolism.
20 ve oxygen species occurs as a consequence of aerobic metabolism.
21 y detoxifier of endogenous H2O2 generated by aerobic metabolism.
22 des also emerged before and with the rise of aerobic metabolism.
23 sion during the transition from anaerobic to aerobic metabolism.
24  subunits of the 2-oxoacid dehydrogenases of aerobic metabolism.
25  are generated as inadvertent by-products of aerobic metabolism.
26 timing of the evolutionary appearance of the aerobic metabolism.
27 H2O2), which is produced in all cells during aerobic metabolism.
28  the environment and from those generated by aerobic metabolism.
29 te than for the wild-type W3110 during fully aerobic metabolism.
30 ns such as MPP+ that target specific loss of aerobic metabolism.
31 and nutrition during preservation and allows aerobic metabolism.
32 reated RIF-1 tumors is due to an increase in aerobic metabolism.
33 yl CoA reservoirs in the mitochondria during aerobic metabolism.
34 esulted in suppression of both anaerobic and aerobic metabolism.
35 (ROS) and reactive nitrogen species (RNS) in aerobic metabolism.
36 l analysis of the gene products important in aerobic metabolism.
37 raditionally regarded as toxic byproducts of aerobic metabolism.
38  also encodes capacity for heterotrophic and aerobic metabolisms.
39 stemic perfusion, unrelated to elevations in aerobic metabolism, accounted only for approximately 5%
40                                              Aerobic metabolism also generates superoxide (O2()) and
41 enase enzymes involved in both anaerobic and aerobic metabolism and also regulates posttranslational
42 ell lines are highly glycolytic with minimal aerobic metabolism and altered mitochondrial physiology.
43 st damaging superoxide radicals generated by aerobic metabolism and as a consequence of inflammatory
44  intermediate provides a direct link between aerobic metabolism and bacterial stress responses, repre
45  oxygen species (ROS) produced during normal aerobic metabolism and by the innate immune systems.
46 species are produced as the direct result of aerobic metabolism and can cause damage to DNA, proteins
47 cytosol and regulate processes as diverse as aerobic metabolism and cell death by necrosis and apopto
48  and other chemicals and result in decreased aerobic metabolism and cell death.
49 osphere to one suitable for the evolution of aerobic metabolism and complex life.
50 ctive oxygen species (ROS) are byproducts of aerobic metabolism and contribute to both physiological
51 ithin the mitochondrial matrix that promotes aerobic metabolism and controls reactive oxygen species
52 yme Q6 biosynthesis, which in turn activated aerobic metabolism and enhanced oxidative stress resista
53 these data show that NOXase is important for aerobic metabolism and essential in environments high in
54  current findings indicate that p53 promotes aerobic metabolism and exercise capacity by using differ
55 nase are required for the use of glycerol in aerobic metabolism and gluconeogenesis.
56 g is essential for bacterial survival during aerobic metabolism and host infection.
57        Hydrogen peroxide is generated during aerobic metabolism and is capable of damaging critical b
58 on in a canine model of DCD: (1) facilitates aerobic metabolism and resuscitates the DCD heart, (2) p
59 evels of H2O2 both as a consequence of their aerobic metabolism and through the respiratory burst of
60 olic rate of oxygen (CMRO(2); i.e., index of aerobic metabolism), and lactate production (J(Lac); i.e
61 ic processes, a particularly mobile fuel for aerobic metabolism, and perhaps a mediator of redox stat
62     Reactive oxygen species are generated by aerobic metabolism, and their deleterious effects are bu
63     During acute inflammation, as in sepsis, aerobic metabolism appears to malfunction and switches t
64 timulation frequencies and were generated by aerobic metabolism (approximately 98%), with %DeltaJ(ATP
65                Thus, CCL5 actions on glucose aerobic metabolism are critical for mitochondrial functi
66  Numerous genes involved in biosynthesis and aerobic metabolism are repressed, whereas a high proport
67           Although ROS, normal byproducts of aerobic metabolism, are essential for various defense me
68 cells (RPTCs) that exhibit in vivo levels of aerobic metabolism, are not glycolytic, and retain highe
69 e, are constantly generated as byproducts of aerobic metabolism, as well as in response to endogenous
70 cies (ROS), which are the products of normal aerobic metabolism, as well as naturally occurring free
71                  We conclude that restricted aerobic metabolism at the thermal limits contributes to
72                        Oxygen is critical to aerobic metabolism, but excessive oxygen (hyperoxia) cau
73           Copper plays a fundamental role in aerobic metabolism, but its role is double-edged, given
74 chondrial calcium uniporter, which regulates aerobic metabolism by catalyzing mitochondrial Ca(2+) in
75  is validated by detecting the impairment of aerobic metabolism caused by 1.5 mM amobarbital.
76                                    Likewise, aerobic metabolism downregulates during growth arrest, a
77 ed in the DIV-BBB demonstrated predominantly aerobic metabolism evidenced by a robust increase in glu
78                       The data indicate that aerobic metabolism fails and cellular damage with degrad
79 ior studies have indicated the importance of aerobic metabolism for L. monocytogenes infection, these
80 idence for the importance of menaquinone and aerobic metabolism for L. monocytogenes pathogenesis.
81 nced spoilage and loss of lactic acid due to aerobic metabolism for maize silage.
82  studies hypothesize a metazoan affinity and aerobic metabolism for these taxa, whereas others propos
83  with superoxide produced by bacteria during aerobic metabolism, forming peroxynitrite, a known oxida
84                                              Aerobic metabolism generally suppresses hypoxia-sensitiv
85                                              Aerobic metabolism generates biologically challenging re
86 ], exercise duration, 6-minute walk), muscle aerobic metabolism (half-time of VO2 and near infrared r
87 that the scant superoxide (O2-) generated by aerobic metabolism harms even cells that contain abundan
88                  As oxygen is central to all aerobic metabolism, hypoxia is now recognized to contrib
89                  The subsequent evolution of aerobic metabolism in prokaryotes to form aerobic bacter
90 e AMZ core, which could periodically support aerobic metabolisms in a typically anoxic environment.
91 ) in older groundwaters that seem to support aerobic metabolisms in subsurface ecosystems at an unpre
92 ults indicated the exogenous GAs lowered the aerobic metabolism including the oil metabolisms during
93            Silage deterioration, a result of aerobic metabolism (including utilization of lactic acid
94         Herein, we explore how mitochondrial aerobic metabolism influences different aspects of organ
95                                       Normal aerobic metabolism is associated with the production of
96 ysis during malignant progression, even when aerobic metabolism is available.
97 cation, the dehydrogenases are inactive, and aerobic metabolism is blocked.
98                              A key enzyme in aerobic metabolism is cytochrome c oxidase (CcO), which
99 ll exceeds the PHX enzymes of glycolysis, an aerobic metabolism is preferred.
100  chain, we demonstrated that the reliance on aerobic metabolism is reduced at thermal limits.
101 enes, but not those for ammonia oxidation or aerobic metabolism, is identified.
102 at CO(2) is far more than a waste product of aerobic metabolism leading to acidosis and that it elici
103     However, in the cyanobacterial ancestor, aerobic metabolism likely predated the GOE, which may ha
104 om the effects of oxidative stress caused by aerobic metabolism may contribute to the ability of Neis
105              Thus, genes/operons involved in aerobic metabolism, NO. detoxification, flagellar biosyn
106 elucidate a putative role for this enzyme in aerobic metabolism, NOXase-deficient mutants were constr
107 out the domains of life that is required for aerobic metabolism of 2-oxoacids and for C(1) metabolism
108 of low-flow ischemia and mixed anaerobic and aerobic metabolism of an acutely infarcting region; (2)
109 gical constraints on oxygen supply affecting aerobic metabolism of aquatic ectotherms, ecological the
110 theoretical possibility of a switch to fully aerobic metabolism of glucose in the chemostat under con
111 r neurotransmitters are synthesized from the aerobic metabolism of glucose; anoxia-induced impairment
112           Consequently, the toxic affects of aerobic metabolism on biogenesis and function of the rib
113 uspected capabilities, including evidence of aerobic metabolism, one-carbon and complex carbon metabo
114 to analyze and predict allometric scaling of aerobic metabolism over a remarkable 27 orders of magnit
115 ance is limited by their ability to maintain aerobic metabolism (oxygen- and capacity-limited toleran
116  introduces metabolic adaptations within the aerobic metabolism pathways, affecting tricarboxylic aci
117                                              Aerobic metabolism produces reactive oxygen species, inc
118                             We conclude that aerobic metabolism produces toxic levels of H(2)O(2) in
119  effective preservation strategy to maintain aerobic metabolism, protect mitochondria, and achieve an
120 the high energy requirements and reliance on aerobic metabolism render it particularly susceptible to
121 ected utilization of glucose in overflow and aerobic metabolism satisfies the energetic and redox dem
122 e), we found that superoxide, a byproduct of aerobic metabolism, significantly destabilized the [2Fe-
123 tained lower levels of CO and MAP maintained aerobic metabolism sufficient to support DO(2).
124                         During physiological aerobic metabolism, the epidermis undergoes significant
125                                              Aerobic metabolism therefore makes use of reactive molec
126 ular metabolite diffusion on skeletal muscle aerobic metabolism through the application of the effect
127  C provides O(2) delivery (DO(2)) to support aerobic metabolism throughout a 3-h period.
128 oli, which conferred resistance by using its aerobic metabolism to compete with Salmonella for resour
129 ammed cellular metabolism from mitochondrial aerobic metabolism to glycolysis, resulting in a remarka
130 isplay decreased proton leak and switch from aerobic metabolism to glycolysis.
131 roposed to depend on the ability to maintain aerobic metabolism, too.
132  Since eROS is an unavoidable consequence of aerobic metabolism, understanding how Mtb manages eROS l
133       A corollary of this hypothesis is that aerobic metabolism underwrites complex biological functi
134 re ancestrally aerobic and thus propose that aerobic metabolism using HPQs significantly predates Ear
135  oxygen (95%) results in a greater degree of aerobic metabolism versus aeration (21%) in the nonphysi
136 duction by spheroids upon suppression of the aerobic metabolism was observed.
137 ng rate), pulmonary ventilation and systemic aerobic metabolism were only altered in whole-body heati
138 trong contemporary functional association of aerobic metabolism with both physical capacity and healt
139  contact with CAFs, were reprogrammed toward aerobic metabolism, with a decrease in GLUT1 expression

 
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