ライフサイエンスコーパス: afebrile

1 The patient was afebrile.

2 e initially afebrile and 6 patients remained afebrile.

3 566 (34%) were febrile, and 1,125 (66%) were afebrile.

4 At admission, 25% (19/76) of children were afebrile.

5 nd 16 (17%) and six (12%), if the patient is afebrile.

6 Both patients were afebrile.

7 rate and respiratory rate, but she remained afebrile.

8 findings were unremarkable, the patient was afebrile (36.8 degrees C), and other vital observations

9 d brain (30/81; 37%); 29 (36%) patients were afebrile; 46/81 (57%) had disseminated infections.

10 arum and 36 had P. vivax infections) and 162 afebrile adults (of whom 20 had P. falciparum and 20 had

11 , we enrolled healthy, malaria-asymptomatic, afebrile, adults (age 18-59 years) and older children (a

12 Samples from 797 afebrile and 457 febrile outpatients from two provinces

13 hea, although 21% of patients were initially afebrile and 6 patients remained afebrile.

14 ymmetric, descending flaccid paralysis in an afebrile and alert patient without sensory findings.

15 He remained afebrile and asymptomatic.

16 Adults were often afebrile and complained of shortness of breath, chills,

17 Patients were grouped into afebrile and febrile groups, based on initial pulmonary

18 of these two antigens did not differ between afebrile and febrile patients.

19 Physical examination revealed he was afebrile and had normal pulse oxygen saturation.

20 he start of therapy, 220 (30%) patients were afebrile and had normal white blood cell counts.

21 ents with gram-negative bacteremia, who were afebrile and hemodynamically stable for at least 48 hour

22 her had neutropenic fever, and the third was afebrile and non-neutropenic at the time of presentation

23 On clinical examination, she was afebrile and tachypneic.

24 ve therapy (80% v 53%); and (3) treatment of afebrile and uncomplicated febrile neutropenia (30% v 60

25 st likely to reduce CSF use for treatment of afebrile and uncomplicated febrile neutropenia.

26 She was afebrile, and her physical examination and routine labor

27 On physical examination, he was afebrile, and the digital rectal examination was not pai

28 He was afebrile, and the physical examination was notable for m

29 The patient was afebrile at admission.

30 s and Treg cells using flow cytometry in 168 afebrile children (of whom 15 had P. falciparum and 36 h

31 e illnesses other than KD to those of normal afebrile children and to KD patients.

32 om both virus-negative afebrile controls and afebrile children with the same viruses present in the f

33 blood culture-negative febrile controls, and afebrile community controls for most antigens.

34 le patients over the course of 3 months, and afebrile community controls.

35 ruited children with suspected infection and afebrile control participants at hospitals in 10 Europea

36 ve febrile children from both virus-negative afebrile controls and afebrile children with the same vi

37 ion or with acute bacterial infection and 22 afebrile controls.

38 NP specimens of febrile patients compared to afebrile controls.

39 P) specimens of febrile patients compared to afebrile controls.

40 y considered only admissions for febrile and afebrile convulsions.

41 ts with PKC also have a history of infantile afebrile convulsions.

42 5 of 10 on a visual analog scale, and being afebrile during the entire stay.

43 for emergency treatment of acute febrile and afebrile (epileptic) seizures in children.

44 Almost half of afebrile episodes (47.6% [204/429]) were RDT positive.

45 Fifteen affected members presented with afebrile focal or generalized tonic-clonic seizures duri

46 e the age of 6 months, and presentation with afebrile focal seizures were significantly associated wi

47 Subjects were tested when afebrile for (i) psychophysical loudness adaptation to c

48 ve bacteria in blood culture(s) if they were afebrile for 24 hours without evidence for complicated i

49 100 mg per kilogram per day until they were afebrile for 48 hours and 3 to 5 mg per kilogram per day

50 However, post-infectious refractory afebrile form of seizures in previously healthy children

51 exclude febrile HCWs from working, but allow afebrile HCWs with respiratory symptoms to have contact

52 Afebrile healthy infants served as controls.

53 nd 190 without bacterial infections), and 19 afebrile healthy infants.

54 er in febrile HRP2-positive patients than in afebrile HRP2-positive patients.

55 Clinical bias was greatest in febrile vs. afebrile intensive care unit patients.

56 Geometric mean iron absorptions in the afebrile malaria and hookworm groups were 12.9% and 32.2

57 zation ranged between 79.1% and 88.0% in the afebrile malaria and hookworm groups with no significant

58 In contrast, afebrile malaria causes inflammation, increases hepcidin

59 Interventions targeting afebrile malaria infections may be needed to address res

60 Treatment of afebrile malaria reduced inflammation (P < 0.001) and se

61 tion pre- and posttreatment in children with afebrile malaria, hookworm, and Schistosoma haematobium

62 , placebo-controlled noninferiority trial of afebrile men with presumed symptomatic UTI treated with

63 Among afebrile men with suspected UTI, treatment with ciproflo

64 ernative to a 14-day course for treatment of afebrile men with UTI.

65 es more likely to become injured compared to afebrile monkeys.

66 v 4.6%); and (3) secondary prophylaxis after afebrile neutropenia following chemotherapy for germ cel

67 Afebrile neutropenia is rarely followed by CSF initiatio

68 of either established febrile neutropenia or afebrile neutropenia remains uncertain.

69 d: development of fever and/or infections in afebrile neutropenic outpatients and recovery without co

70 Secondary outcomes included: in afebrile neutropenic outpatients, infection-related mort

71 ividuals presented with febrile and multiple afebrile, often focal seizure types, multifocal epilepti

72 especified criteria (hemodynamically stable, afebrile, oral intake tolerated, pain controlled, and fo

73 ulocyte colony-stimulating factor (G-CSF) in afebrile outpatients with severe chemotherapy-induced ne

74 TTMV was equally present in both febrile and afebrile patient specimens.

75 found in similar percentages of febrile and afebrile patient specimens.

76 Afebrile patients had lower rates of antibiotic administ

77 es F documented in the emergency department; afebrile patients lacked both.

78 gency department patients with septic shock, afebrile patients received lower rates of emergency depa

79 antibacterial therapy in clinically well and afebrile patients with low-risk FN if blood cultures rem

80 Routine therapeutic application of G-CSF in afebrile patients with severe neutropenia can reduce the

81 ge number of specimens from both febrile and afebrile patients, they were more prevalent in the plasm

82 time of high bacteremia that alternate with afebrile periods of relative well being during low bacte

83 ement in detecting low-density infections in afebrile pregnant women compared to cRDTs.

84 Nearly half of HCWs with influenza were afebrile prior to their diagnosis.

85 h or without any other clinical finding; and afebrile rash with or without any other clinical finding

86 y characterised by undifferentiated fever or afebrile rash, were missed.

87 ses presented with undifferentiated fever or afebrile rash.

88 In afebrile rats, alpha-MSH infusion caused a modest transi

89 In both febrile and afebrile rats, CCK-8 induced dose-dependent skin vasodil

90 n combination, affected body temperatures in afebrile rats.

91 emergency department IV fluids volume, being afebrile remained a significant predictor of in-hospital

92 Like the distribution of afebrile seizure duration in children, the distribution

93 followed by treatment-refractory febrile and afebrile seizures and psychomotor decline.

94 Eight individuals developed afebrile seizures between ages 5 and 13 years.

95 Afebrile seizures consisted of generalized tonic-clonic,

96 ; or with clusters of two or more febrile or afebrile seizures within a 24-h period were also eligibl

97 onged (>10 min) febrile seizures; febrile or afebrile status epilepticus (>30 min); or with clusters

98 Patients with afebrile status epilepticus had a variety of imaging abn

99 illnesses are larger than those in normative afebrile subjects but smaller than dimensions in patient

100 of intensification 2, the patient developed afebrile tachypnea, tachycardia, and an increasing oxyge

101 nces in clinical characteristics (history of afebrile v febrile neutropenia), drug characteristics (G

102 They are the principal etiologic agents of afebrile viral upper-respiratory-tract infections (the c

103 ation required the patient with asthma to be afebrile with normal chest x-ray and white blood cell co

104 The patient was afebrile with normal vital signs.

105 ive clusters, 59.7% were febrile, 20.2% were afebrile with other symptoms, and 20.2% were asymptomati

106 At the second ICU admission, he was afebrile, with a blood pressure of 160/72 mm Hg and puls

107 At the second ICU admission, the patient was afebrile, with a blood pressure of 160/72 mm Hg and puls

108 On physical examination, the patient was afebrile, with normal vital signs and normal Glasgow Com

109 clinical and radiologic improvement and were afebrile within 24 hrs after drainage.

110 Among afebrile women (n = 392), the geometric-mean parasite de