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1 tions that indicate reduced attention to the affected side.
2 subjective effort, particularly for the more-affected side.
3 more-affected side and the anode on the less-affected side.
4 is) were measured every 3 months on the less affected side.
5 ected side, whereas it was normal on the non-affected side.
6 mine-derived radioactivity was higher on the affected side.
7 ments in plasma dihydroxyphenylglycol on the affected side.
8 nificantly reduced contralateral to the most affected side.
9 ooting was significantly smaller on the more affected side.
10 able increases in the upper limb in the less-affected side.
11 putamen mimicked that recognized on the most-affected side.
12 xposure to sensory information from the less-affected side.
13 panoplasty only or an additional ETBD of the affected side.
14 two measures in the clinically-defined least affected side.
15 pain syndrome exhibited a bias away from the affected side.
16 e individuals, but instead larger around the affected side.
18 uptake value ratio units higher on the more affected side (95% confidence interval, CI 0.11-0.24, P
19 mispheres, corresponding to affected and non-affected side and compared with 10 patients with primary
20 t worsening of parkinsonism in the primarily affected side and more noticeable increases in the upper
21 ticity protocol on both the affected and non-affected side and normal eye blink classical conditionin
22 tion was found between the FA values for the affected side and the ALS Functional Rating Scale Revise
23 otor cortex, placing the cathode on the more-affected side and the anode on the less-affected side.
26 indicates that signals from the healthy and affected side are still fused, but result in asymmetric
28 The mean MDS-UPDRS III score for the more affected side decreased from 19.9 at baseline to 9.9 at
31 compare between and within (affected vs. non-affected side) groups geometric temporal bone features (
32 wed evidence of facial nerve neuritis on the affected side (hypersignal on gadolinium-enhanced T1-wei
34 f a generalized attention bias away from the affected side of space in complex regional pain syndrome
35 emisphere ipsilateral to the clinically more affected side of the body, as measured by (123)I-ioflupa
36 n the latency of the transient VEPs from the affected side of the SC in late stages of the disease.
37 specifically on the clinically-defined most affected side (P < 0.05), and reduced locus coeruleus vo
40 were observed between TA-LG co-contraction (affected side, r = 0.557, p = 0.020; contralateral side,
41 two motor cortices with cathode on the more-affected side reduced noise and increased the willingnes
42 processed visual stimuli more slowly on the affected side (relative to the unaffected side) when the
43 However, for the clinically-defined most affected side, significant relationships were observed b
44 e affected hemisphere was larger and the non-affected side smaller in HME compared with non-HME child
45 mmonia-derived radioactivity was less on the affected side than on the unaffected side, whereas 6-[18
46 alf, and midupper arm of the clinically more affected side using high-resolution T2-weighted sequence
47 however, FA values and NAA/Cr ratios for the affected side were even stronger predictors of disease d
48 , in HME children NeuN cell densities on the affected side were increased in the molecular layer and
49 of origin (n = 11), cVEMP amplitudes of the affected side were significantly lower as compared to th
50 intracortical inhibition was reduced on the affected side, whereas it was normal on the non-affected
52 e significantly lower as compared to the non-affected side, while the force threshold level was incre
53 ith differences between the affected and non-affected sides within the MD-dg group were the sigmoid s