ライフサイエンスコーパス: affective

1 xternally focused, self-other, and cognitive-affective.

2 relationship between the 'Lovers of Modena' (affective?

3 e demonstrate that THC induces cognitive and affective abnormalities resembling neuropsychiatric symp

4 e studied functional interactions within the affective amygdala circuit.

5 One-month post-psilocybin, negative affective and amygdala response to facial affect stimuli

6 circuitry function and the course of future affective and anxiety symptoms in a naturalistic, transd

7 Affective and anxiety symptoms were reported at each vis

8 ted with a more severe, persistent course of affective and anxiety symptoms.

9 Affective and autism spectrum disorders exhibited a SNP

10 ation about its motivational states to guide affective and behavioral responses.

11 e powerfully blocked the development of both affective and cognitive abnormalities commonly associate

12 adolescent THC exposure induces long-lasting affective and cognitive abnormalities, mesocorticolimbic

13 loss of PFC GABAergic inhibitory control and affective and cognitive abnormalities.

14 motor function, it also plays wider roles in affective and cognitive behaviors.

15 Emotion perception is fundamental to affective and cognitive development and is thought to in

16 s an opportunity to investigate how sensory, affective and cognitive stimuli alter functional connect

17 ion-making as it integrates motor plans with affective and contextual limbic information.

18 C in motor evaluation and the ventral ACC in affective and contextual memory.

19 lamocortical "brain-state" coding to include affective and exploratory states and provide an entry po

20 )) is implicated in convergent sensorimotor, affective and interoceptive processing, we hypothesised

21 d great success in modeling some of the core affective and neurovegetative depressive symptoms, inclu

22 The presence of affective and psychotic illnesses was associated with gr

23 Brain-wide circuits that coordinate affective and social behaviours intersect in the amygdal

24 search provides robust evidence for positive affective and social consequences of psychedelic substan

25 el therapeutic approach for the treatment of affective and stress-related neuropsychiatric disorders.

26 Overall, these findings suggest that affective and value-related brain responses can predict

27 primary sensory cortices integrate sensory, affective, and cognitive signals.

28 r structure representing negative, positive, affective, and cognitive symptoms was identified as the

29 ientific questions regarding what cognitive, affective, and environment factors lead to successful be

30 s extensive disynaptic links with cognitive, affective, and motor forebrain circuits.

31 ic region that critically regulates negative affective- and stress-induced relapse.

32 Misdiagnosis or comorbidity with affective, anxiety and personality disorders are frequen

33 In three experiments, we operationalized the affective aspects of social interactions as ratings of i

34 subdivisions of the SPB tract that underlie affective aspects of touch and pain.

35 y spinal afferents and fear and anxiety (the affective aspects of visceral pain) are the domain of no

36 nse in structures implicating the overlap of affective (automatic) and cognitive (higher-order) empat

37 tral anterior cingulate cortex underlies the affective (aversive), but not the sensory-discriminative

38 social structures, have a propensity towards affective balance, but one unbalanced configuration resi

39 mited understanding of how lithium modulates affective behavior and lack of validated cellular and an

40 prolide on reproductive function, social and affective behavior, cognition, and brain activity in a r

41 n the effects of SSRIs on motor function and affective behavior, highlighting the potential benefits

42 t exerts sex-specific effects on social- and affective behavior, stress regulation, and neural activi

43 onship between newborn neuronal activity and affective behavior.

44 tribute to the CSD stress-induced changes in affective behavior.

45 ssity of IC projections to NAc during social affective behavior.

46 tion of BNST CRF neurons that drive negative affective behavior.

47 for the neural basis of persistent negative affective behavioral states.

48 mood disorder, characterized by distressing affective, behavioral, and somatic symptoms in the late

49 the impact of these interneuron subtypes in affective behaviors as well as in the effects of rapid-a

50 the importance of the cerebellum in driving affective behaviors that could contribute to neurologica

51 which modulate cognitive, motivational, and affective behaviors, are involved in TAND.

52 s of this network were relevant to sleep and affective behaviors, these findings implicate that dysre

53 f the stria terminalis (BNST) drive negative affective behaviors, thus agents that decrease activity

54 onal differentiation of AHNPCs that modulate affective behaviors.

55 ation and neurogenesis and the modulation of affective behaviors.SIGNIFICANCE STATEMENT Inheritance o

56 childhood maltreatment as well as cognitive-affective, behavioural and interpersonal factors.

57 and decision-making were assessed using the affective bias test (ABT), or judgement bias task (JBT)

58 objective measure of affective valence, the affective bias test [8], to show that 50 kHz ultrasonic

59 and other RAADs mediate a specific effect on affective bias which involves the mPFC.

60 Clinical studies suggest that negative affective biases (the process, whereby cognitive process

61 gical effects mediated through modulation of affective biases (where cognitive processes such as lear

62 in rats increases vulnerability to negative affective biases and impairs animals' ability to appropr

63 In adulthood, affective biases associated with reward learning and dec

64 Previous work has shown that affective biases in a rodent decision-making task are di

65 others relies on a common network of social-affective brain regions, with the medial prefrontal cort

66 ualized attention for both the cognitive and affective challenges of decision making.

67 However, affective changes reflective of anxiety- and depressive-

68 on appears to be important for mediating the affective component of itch.

69 STATEMENT The sensation of itch includes an affective component that leads to stress and anxiety in

70 debilitating condition with both sensory and affective components, the underlying brain circuitry of

71 Most of the research in the field of affective computing has focused on detecting and classif

72 ity to pain when compared with cognitive and affective conditions from twelve additional studies (N =

73 ere is ongoing controversy about the role of affective congruency during multisensory integration, i.

74 nteroceptive processes may contribute to the affective consequences of minor hypo-hydration.

75 emotional system - our innately evaluative "affective" constraints - is missing from the model.

76 ), may contribute to the initiation of basic affective/defensive responses via hypothalamic and brain

77 rain changes may relate to the cognitive and affective deficits remains to be determined with a large

78 ological systems in somatic versus cognitive-affective depressive symptoms which remains largely unex

79 n perception is fundamental to cognitive and affective development.

80 age of the general population, with seasonal affective disorder (SAD) representing the most common pr

81 to 32 patients with schizophrenia or schizo-affective disorder and 39 matched controls.

82 ing had a first-time hospital contact for an affective disorder between 1996 and 2015.

83 Bipolar disorder (BD) is a chronic affective disorder with extreme mood swings that include

84 der and 49.7% (95% CI 48.1-51.3) for bipolar affective disorder.

85 cidence rate ratio 1.44 [1.27-1.62]) and non-affective disorders (1.60 [1.44-1.77]) than women, but n

86 (2.8% [95% CI, 2.2% to 3.4%] vs. 0.7%), mood affective disorders (19.5% [CI, 18.0% to 21.0%] vs. 8.1%

87 ation registers reported higher rates of non-affective disorders (9.64 [2.72-31.82]), schizophrenia (

88 to treatment of many patients, debilitating affective disorders (other disorders including anxiety a

89 iety is a risk factor for the development of affective disorders and has been associated with decreas

90 Affective disorders arise from abnormal responses of the

91 e impact of our study is broadly relevant to affective disorders associated with disruption of reward

92 thelium as a target to treat respiratory and affective disorders associated with vascular diseases.

93 a common antecedent of adolescent and adult affective disorders involving the reward circuitry.

94 Sub-group analysis of cases with affective disorders revealed SNPs associated with suicid

95 nform inquiries ranging from the etiology of affective disorders to the neurological basis of emotion

96 In studies of anxiety and other affective disorders, objectively measured physiological

97 people may be more vulnerable to developing affective disorders, we investigated whether serotonin-r

98 tics with incident dementia in patients with affective disorders.

99 operiod and are consistently associated with affective disorders.

100 tion may contribute to memory distortions in affective disorders.

101 avenues for pharmacological interventions in affective disorders.

102 ence significant and disabling cognitive and affective disorders.

103 ervention that could circumvent pain-induced affective disorders.

104 are often therapeutically targeted to treat affective disorders.

105 underlying mechanisms in the development of affective disorders.

106 e reward circuitry underlies many aspects of affective disorders.

107 a significant role in alcohol use and other affective disorders; however, the genetically-defined ne

108 with genetic predisposition to stress and/or affective disturbances.

109 By focusing on the affective domain, we systematically investigate the impa

110 Here, we argue that affective dynamics are essential to include when constru

111 escending pain modulatory information in the affective/emotional pain pathway.

112 egions associated with cognitive rather than affective empathy, and greater social adversity was asso

113 itous, the neural mechanisms underlying this affective experience remain unclear.

114 th been shown to be associated with negative affective experience.

115 Pain is an integrated sensory and affective experience.

116 ng evening resting wakefulness is related to affective experiences in REM sleep dreams.

117 It is theorized to be linked to affective experiences, the creation of particularly resi

118 ard anticipation, behavioral inhibition, and affective faces, and their respective associations with

119 task, pupil dilation in response to viewing affective faces.

120 Symptoms occur in response to cognitive and affective factors that trigger fear of symptoms or lack

121 feelings (e.g., "angry," "dreamy") or broad affective features (e.g., valence, arousal) that they ma

122 ere asked to judge the emotion categories or affective features communicated by 2,519 speech samples

123 ive the recognition of emotions more so than affective features, including Valence.

124 opose an integrative microbiome neuro-immuno-affective framework of how emotional dysregulation and a

125 r social bonding by introducing temporal and affective frameworks, which facilitate movement synchron

126 This suggests that the affective function or valence mediated by a neural modul

127 analyses to investigate perturbed sleep and affective functions at the gene network level.

128 neuropsychiatric disorders and cognitive and affective functions.

129 bdivisions that serve distinct cognitive and affective functions.

130 estradiol as an example) might contribute to affective illness.

131 l regulation of affect and susceptibility to affective illness.

132 elp us better understand the neural basis of affective impairments in psychosis, informing prediction

133 s, in part, like a sensory structure for the affective import of stimuli and conveys this information

134 demographic, cognitive, motor, olfactory and affective information enabling the assessment of each co

135 executive function and memory impairments), affective information processing patterns (e.g., rigid,

136 lly regarded as a key site for cognitive and affective information processing, and the anteroventral

137 Cortical mechanisms of sensory and affective integration, however, remain poorly defined.

138 We investigated the neuronal basis of affective itch in mice, with a focus on the amygdala, th

139 eatures from these regions and user-reported affective labels.

140 Physical abuse predicted both aggression and affective lability as well as SI variability, but not im

141 Future affective lability factor scores were predicted in both

142 ons at baseline, were used to predict future affective lability factor scores, using regularized regr

143 cal and neural predictors of future-specific affective lability factors is a step toward identifying

144 nstability phenotype strongly contributes to affective lability in patients with BD.

145 We also assessed whether affective lability, aggressive or impulsive traits expla

146 This study tested whether childhood trauma, affective lability, and aggressive and impulsive traits

147 g of hierarchical information flow disrupted affective learning and conditioned responding.

148 The neurobiology underlying affective learning capitalizes on this protracted timeli

149 n AIE history demonstrated enhanced negative affective-like behavior in the novelty-induced hypophagi

150 ive in alleviating anxiogenic and depressive affective-like behaviors in both sexes.

151 rd center that are implicated in sensory and affective manifestations of chronic pain.

152 alleviate spontaneous pain, nociceptive and affective manifestations.

153 evidence for the 'affective modules' versus 'affective modes' hypotheses may be useful for advancing

154 t startle-evoked amygdala responding and its affective modulation may hold promise as an important no

155 An evaluation of evidence for the 'affective modules' versus 'affective modes' hypotheses m

156 rders, alcohol use disorders and bipolar and affective mood disorders.

157 tical role in cognitive processes, including affective motivational behaviors and hippocampus (HPC)-d

158 ng this nociceptive ensemble alleviated pain affective-motivational behaviors without altering the de

159 x (ACC) and striatum play important roles in affective-motivational pain processing and reward learni

160 ratings showed a clearer distinction between affective movements than the computed counterparts.

161 o anhedonia, and ketamine's modulation of an affective network to exert its action.

162 rders: the default mode network and negative affective network.

163 The cognitive and affective networks are located in regions of cingulate c

164 The cognitive and affective networks, present in monkeys, are largely abse

165 How the brain's affective neural circuits attribute this aversive qualit

166 ual system in the higher-order cognitive and affective neural systems.

167 ogy is one of the best understood systems in affective neuroscience and is an ideal target for addres

168 hold promise as an important novel tool for affective neuroscience and its clinical translation.

169 experts from the fields of human and animal affective neuroscience to discuss their viewpoints on ho

170 lational tool to study defensive behavior in affective neuroscience with relevance to a broad range o

171 eptive technologies offer a new paradigm for affective neuroscience, allowing controlled intervention

172 ence from well-being research, cognitive and affective neuroscience, and clinical psychology to highl

173 Internal states, including affective or homeostatic states, are important behaviora

174 uent alternative diagnoses were personality, affective or non-schizophrenia psychotic disorders.

175 be useful for advancing understanding of the affective organization of limbic circuitry.

176 ly important role in integrating sensory and affective pain signals.

177 roject rostrally to pathways associated with affective pain, such as parabrachial nucleus and medial

178 ve widespread inhibitory projections to many affective pain-processing centers.

179 ot only policy discourse but also addressing affective partisan hostility.

180 d the translation of environmental stress to affective pathology.

181 Previous research has identified affective patterns using self-reports(3) and text analys

182 ice and associated the networks to sleep and affective phenotypes, providing a resource for integrate

183 for the expression of motivated behavior and affective phenotypes.

184 res for stimuli drawn from the International Affective Picture System (IAPS) in a population (n = 50)

185 Affective polarization has become a defining feature of

186 measures track the expression of affect, not affective preference for external stimuli such as music,

187 to measure diurnal and seasonal patterns of affective preference.

188 ncidence of cancer, metabolic disorders, and affective problems in humans.

189 dressing cognition as an embodied, enactive, affective process involving cultural affordances; (3) cl

190 on and the ability to regulate cognitive and affective processes based on context.

191 he neurobiology of future-oriented cognitive-affective processes critical to adaptive social function

192 from the gut induce maladaptive cognitive or affective processes that amplify symptom perception.

193 Sleep loss disrupts a broad spectrum of affective processes, from basic emotional operations (e.

194 for contextual memories (entorhinal cortex), affective processing (amygdala), and motor planning (dor

195 activation, differentiating cognitive versus affective processing and sensory versus higher-order cog

196 ffect and test their unique contributions to affective processing during anticipation of unpredictabl

197 nfralimbic cortex (IL) is a key structure in affective processing in rodents and activation of its hu

198 r additive roles than the ones postulated in affective processing so far, particularly in abstract co

199 lable options on each trial, we examined the affective properties of agreeing with a group majority b

200 ed by model-based (MB) operations, including affective prospection, replay, and planning.

201 yelination, and functional connectivity with affective psychopathology, cognition, and family environ

202 In the CHR group, they are associated with affective psychopathology, impairments in verbal memory,

203 son with the care of patients with other non-affective psychoses, despite evidence suggesting targete

204 he general population (the prevalence of non-affective psychosis was on average 16 times higher, majo

205 l mental illnesses: depression, anxiety, non-affective psychosis, affective psychosis, eating disorde

206 epression, anxiety, non-affective psychosis, affective psychosis, eating disorders, personality disor

207 rders (1.60 [1.44-1.77]) than women, but not affective psychotic disorders (0.87 [0.75-1.00]).

208 t are functionally required for the negative affective qualities of acute and chronic pain perception

209 e congruent or incongruent in terms of their affective quality.

210 Participants provided affective ratings for negative and neutral images while

211 n image recognition task where HRD, SCRs and affective ratings were recorded again.

212 tability at follow-up was assessed using the Affective Reactivity Index, and cortical thickness was q

213 havior may be motivated by purely hedonic or affective reasons, such as the willingness to maintain o

214 raders, focusing on links between changes in affective relations and trading performance.

215 A key issue focuses on which affective relationships are prone to change and how thei

216 n this protracted timeline to develop a rich affective repertoire in adulthood.

217 suomotor functions, bridging the gap between affective research on humans and non-human animals.

218 rather than witnesses, triggered a stronger affective response (striatal and prefrontal activation).

219 Affective responses depend on assigning value to environ

220 can significantly alter the meaning and the affective responses to events.

221 s suggests that asymmetry magnitude reflects affective responses to music, while asymmetry entropy re

222 of stimulus detection from stimulus-related affective responses.

223 rning, stress reactivity, and stress-induced affective responses.

224 Within affective science, the central line of inquiry, animated

225 ersonal closeness by increasing temporal and affective self-other overlaps.

226 ecific features (e.g. perceptive, sensitive, affective signals).

227 the mean amplitude of the LPP to assess the affective significance that participants attributed to t

228 prevents the expression of both somatic and affective signs of nicotine withdrawal.

229 al aims of the target article, we argue that Affective Social Learning completes TTOM by pointing out

230 Examining the neurobiology of the psychotic-affective spectrum may greatly advance biological determ

231 nd disrupts the positive association between affective state and singing behavior, as revealed using

232 For example, if I am experiencing an affective state and someone matches their physiological

233 direct effect on the person experiencing the affective state and that parental co-regulation may invo

234 is study reward and punisher manipulation of affective state appeared to alter decision-making by inf

235 e RMTg plays a distinct role in the negative affective state associated with acute withdrawal and may

236 to the rewards or punishers that induced the affective state in the first place.

237 KBFs thus lead to a negative affective state lasting at least 3-4 weeks, and manageme

238 Developing methods to test affective state without excluding cognitively impaired i

239 sed that IE would negatively impact putative affective state, with dogs with IE exhibiting a more pes

240 ng facial electromyography (EMG), a proxy of affective state.

241 Negative affective states affect quality of life for patients suf

242 ha asymmetry (FAA) is considered a marker of affective states and traits as well as affect regulation

243 Affective states are key determinants of animal welfare.

244 Stress-induced negative affective states are prevented by depotentiation of VH t

245 rats, one promising way of inducing positive affective states is by human-simulated rough and tumble

246 nimal affect; humans and rodents in negative affective states often show potentiated startle magnitud

247 The influence of affective states on decision-making is likely to be comp

248 e driven by excessive drug value in negative affective states, a habit driven by strong stimulus-resp

249 ced by immune modulators, neurotransmitters, affective states, and even the underlying disease proces

250 less able to regulate (i.e., inhibit) strong affective states, such as anger, in dreams.

251 Stress promotes negative affective states, which include anhedonia and passive co

252 ld 'seed' play in others, spreading positive affective states.

253 syndromes, such as delirious mania and mixed affective states.

254 ion features reflecting the agent's mood and affective states.

255 ipants also continuously reported their felt affective states.

256 to target the elicitation of a wide range of affective states.

257 ond interact to produce persistent shifts in affective states.

258 een implicated in the generation of negative affective states; however, the mechanisms by which stres

259 cardiovagal activity in response to negative affective stimuli was associated with greater activation

260 ifferentially activated during processing of affective stimuli, and resting-state functional MRI expe

261 effort required to process dynamic shifts in affective stimuli, and this relationship is exacerbated

262 c autonomic activity in response to negative affective stimuli.

263 been focused on neural responses to static, affective stimuli.

264 d, and reduced amygdala response to negative affective stimuli.

265 ming to identify neural predictors of future affective symptom course.

266 een markers of reward circuitry function and affective symptom trajectories.

267 This study focuses on negative affective symptoms associated with prolonged alcohol abs

268 e systems and the amygdala play in mediating affective symptoms of acute withdrawal, but promising pr

269 the amygdala, a brain area implicated in the affective symptoms of stress-related psychiatric disorde

270 contrast, their associations with cognitive-affective symptoms were weak after adjustment for all co

271 ingle patient), mainly transient anxiety and affective symptoms worsening (20 SAEs).

272 nitive disorders, which were associated with affective symptoms, negative self-evaluation, negative i

273 Unlike affective symptoms, we know much less about the cellular

274 der characterized by sensory, cognitive, and affective symptoms.

275 lationships with somatic than with cognitive-affective symptoms.

276 d by trauma and predict the future course of affective symptoms.

277 tudied with generic descriptive analysis and affective tests.

278 nitive ToM/cToM) and understanding emotions (affective ToM/aToM).

279 Responses to experienced and observed affective touch (i.e., being touched or watching others

280 role of the anterior insula for ipsilateral affective touch perception open new avenues of enquiry r

281 facial reactions to matched primary social (affective touch) and nonsocial (food) rewards were asses

282 of the posterior insula in the perception of affective touch.

283 adults: (i) responses to socially relevant, "affective" touch, and (ii) visual attention to emotional

284 We developed a method called affective tracking to reveal and quantify the enormous c

285 porting MAGL inhibition for the treatment of affective, trauma-related, and stress-related disorders;

286 ology (i.e., heart rate change) in detecting affective valence induction across a broad continuum of

287 solateral amygdala that encodes the negative affective valence of pain.

288 correspond to dynamic conditions of varying affective valence or intensities.

289 activation based multivariate predictions of affective valence with measures of heart rate (HR) decel

290 are exploring how to augment the theory with affective valence, take into account individual differen

291 we use a validated and objective measure of affective valence, the affective bias test [8], to show

292 duction across a broad continuum of conveyed affective valence.

293 nduced affect processing in the dimension of affective valence.

294 iked UCS are based on a transfer of positive affective value between the representations (i.e., from

295 nating between auditory signals of different affective value is critical for the survival and success

296 Neuroanatomically, this affective value is encoded at both cortical and subcorti

297 salis of Meynert (NBM), which decomposes the affective value of a conditioned stimulus (CS) into its

298 udinal persistence and dimensions (cognitive-affective versus somatic) of depressive symptoms over a

299 ure descriptors to investigate their role in affective whole-body movement perception.

300 ssation leads the occurrence of physical and affective withdrawal symptoms representing a major burde