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1 d by trauma and predict the future course of affective symptoms.
2 phase of the menstrual cycle may precipitate affective symptoms.
3  mainly due to somatic rather than cognitive-affective symptoms.
4 us on drinking to self-medicate or cope with affective symptoms.
5  depressed mood was less frequent than other affective symptoms.
6 ompanied by additional somatic and cognitive/affective symptoms.
7 uggesting that they did not have significant affective symptoms.
8 der characterized by sensory, cognitive, and affective symptoms.
9 lationships with somatic than with cognitive-affective symptoms.
10 trate underlying cognitive and, potentially, affective symptoms.
11 because 1) most women with PP have prominent affective symptoms; 2) treatment response to lithium and
12                    The relationships between affective symptoms and blood immuno-metabolic biomarkers
13                                 Experiencing affective symptoms and conduct problems in childhood and
14 were associated with earlier age at onset of affective symptoms and syndromal bipolar disorder.
15                                          For affective symptoms (anxiety, sadness, irritability, mood
16                            Research suggests affective symptoms are associated with reduced habitual
17               This study focuses on negative affective symptoms associated with prolonged alcohol abs
18 th significant and sustained improvements in affective symptoms, BMI, and changes in neural circuitry
19 ming to identify neural predictors of future affective symptom course.
20 on, or anhedonia were used as the qualifying affective symptoms; depressive symptoms were eliminated
21                                              Affective symptoms differed significantly among groups;
22 s that can be targeted to alleviate negative affective symptoms during abstinence.
23        Participants recovering with residual affective symptoms experienced subsequent major affectiv
24 n when they were moderate to severe, whereas affective symptoms had the largest effect on interferenc
25 ough the etiologic relationship of panic and affective symptoms has not been determined.
26              Psychosis (hazard ratio=1.537), affective symptoms (hazard ratio=1.510), agitation/aggre
27          The primary long-term outcomes were affective symptoms (Hospital Anxiety and Depression Scal
28 nvariably associated with the development of affective symptoms in PD.
29  the PC firing and also motor, cognitive and affective symptoms in SCA2-58Q mice.
30 e conclude that the underlying physiology of affective symptoms in schizophrenia involves the hippoca
31  risk for specific psychotic experiences and affective symptoms in the general population.
32                                              Affective symptoms influence health status (health-relat
33 nitive disorders, which were associated with affective symptoms, negative self-evaluation, negative i
34 e systems and the amygdala play in mediating affective symptoms of acute withdrawal, but promising pr
35              PFC activation also reduces the affective symptoms of pain.
36 odulate some of the negative, cognitive, and affective symptoms of schizophrenia and is a potential t
37 (mGlu2/3) receptors ameliorate psychotic and affective symptoms of schizophrenia suggests that compou
38 the amygdala, a brain area implicated in the affective symptoms of stress-related psychiatric disorde
39 or neurovegetative, rather than cognitive or affective, symptoms of depression.
40 merge, whether they are causes or effects of affective symptoms, or whether specific cognitive system
41 lect neural mechanisms of risk for worsening affective symptoms, particularly depression, in youth ac
42 BT that would correlate with improvements in affective symptoms, postconcussive symptoms, and quality
43 ather than manic, and subsyndromal and minor affective symptoms predominate.
44                                 In contrast, affective symptoms reflecting areas of chronic dysphoria
45 stradiol and progesterone (E2 + P) can cause affective symptom reoccurrence in women with a history o
46 elationships between dimensional measures of affective symptom severity and fractional anisotropy in
47 der (BP-I) and does not relate disability to affective symptom severity and polarity or to bipolar II
48 lness featuring the full range (spectrum) of affective symptom severity and polarity.
49 ility fluctuates in parallel with changes in affective symptom severity in BP-I and BP-II.
50 ects of clozapine on positive, negative, and affective symptoms, social and occupational functioning,
51                                       Weekly affective symptom status ratings for 86 patients with BP
52                                       Weekly affective symptom status ratings were analyzed by polari
53 icidal intent was reduced by controlling for affective symptoms, suggesting that the effect of the fo
54 rceived hearing problems or for cognitive or affective symptoms that may be related to hearing loss.
55      Whether cannabis can cause psychotic or affective symptoms that persist beyond transient intoxic
56 controls (HC) and negatively associated with affective symptoms throughout the weight spectrum, indep
57 een markers of reward circuitry function and affective symptom trajectories.
58  typology of early-life conduct problems and affective symptoms was identified: (1) stable low, (2) t
59                                       Unlike affective symptoms, we know much less about the cellular
60 for the Assessment of Negative Symptoms, and affective symptoms were assessed with the Hamilton Depre
61 eater reductions in severity of positive and affective symptoms were seen with risperidone than with
62 mpulsive symptoms resolved the most quickly, affective symptoms were the most chronic, and cognitive
63  contrast, their associations with cognitive-affective symptoms were weak after adjustment for all co
64 2) = 0.027, P = .002), but not the cognitive-affective symptoms, were positively associated with inti
65 rs-negative symptoms, positive symptoms, and affective symptoms-were all significantly correlated in
66 ion), and specific psychotic experiences and affective symptoms, while controlling for early risk fac
67 s now known to be associated with a range of affective symptoms, with suicidality, and with worsening
68 ingle patient), mainly transient anxiety and affective symptoms worsening (20 SAEs).
69 hesised that self-reported health status and affective symptoms would map onto salience network regio