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1 ted predominately by activation of group III afferent fibers).
2 chanism (e.g., inhibition of exocytosis from afferent fibers).
3 tonin gene-related peptide (CGRP) (marker of afferent fibers).
4 s provides the entire input to each cochlear afferent fiber.
5 from their ribbon synapses onto the auditory afferent fiber.
6 ibutes by observing the activity of a single afferent fiber.
7 formation arriving from Adelta and C primary afferent fibers.
8  inhibits exocytosis from pulpal peptidergic afferent fibers.
9 nsed by vagal, spinal, and intrinsic primary afferent fibers.
10 rrents elicited by electrical stimulation of afferent fibers.
11 ly located near central terminals of primary afferent fibers.
12 rization-induced suppression of excitation") afferent fibers.
13 /7-immunopositive terminals are from primary afferent fibers.
14 90 min of the study to block parasympathetic afferent fibers.
15 iors, suggesting that ET-1 activates primary afferent fibers.
16 ity of the branches were produced by type II afferent fibers.
17 d after electrical stimulation of peripheral afferent fibers.
18 eatic secretion via intestinal vagal mucosal afferent fibers.
19 til about birth, a time after the arrival of afferent fibers.
20 for the effects of L-ARG on the RA of crista afferent fibers.
21 l fusiform cells, granule cells, and primary afferent fibers.
22 geneous population of small diameter primary afferent fibers.
23 single unit activity of 54% of hepatic vagal afferent fibers.
24 presentations of fat USs may depend on vagal afferent fibers.
25 nsitive, substance P (SP)-containing primary afferent fibers.
26 lves CCK-A receptors and capsaicin-sensitive afferent fibers.
27 g in the terminals of small-diameter primary afferent fibers.
28 ic changes within the cells of origin of the afferent fibers.
29  s) did not differ between the two groups of afferent fibers.
30 nent of the response to burst stimulation of afferent fibers.
31 nals are mainly endings of thinly myelinated afferent fibers.
32 ase staining, and the pattern of cholinergic afferent fibers.
33  round vesicles, formed synapses with radial afferent fibers.
34 ne of the three proteins were present in all afferent fibers.
35 ors associated with the membranes of primary afferent fibers.
36 tral terminal projections of abdominal vagal afferent fibers.
37  neurons, and glutamate release onto Type II afferent fibers.
38  of capsaicin-sensitive, nociceptive primary afferent fibers.
39 e)- and small (nociceptive)-diameter primary afferent fibers.
40 ts ability to trigger phase-locked spikes in afferent fibers.
41  CR immunoreactivity of cell populations and afferent fibers.
42 t pruritogens activate the same or different afferent fibers.
43 ically through the recruitment of myelinated afferent fibers.
44 luconeogenesis to the brain depends on vagal afferent fibers.
45 dial to the solitary tract contained labeled afferent fibers.
46 manipulations of type I and type II cochlear afferent fibers.
47 li is not a common property of gastric vagal afferent fibers.
48 tivates P2X2 and P2X3 receptors on gustatory afferent fibers.
49 r neurons, receiving direct input from vagal afferent fibers.
50 lectrical stimulation of ipsilateral primary afferent fibers.
51  channel expressed on a subset of peripheral afferent fibers.
52  the site of termination of the chemosensory afferent fibers.
53 yers of termination of the primary gustatory afferent fibers.
54 orsal horn-the target zone of many cutaneous afferent fibers.
55 al cord dorsal horn by A delta and C primary afferent fibers.
56 ts, and (4) excitation of intestinal sensory afferent fibers.
57 0-kD neurofilament-immunopositive myelinated afferent fibers, a subpopulation of which did not expres
58 ul sensory inputs mediated by large-diameter afferent fibers (AB-fibers) can attenuate noxious signal
59 l capsaicin-treated animals (NNCAP, group IV afferent fibers ablated).
60 y measure dendritic and somatic responses to afferent fiber activation in two different populations o
61                                     Auditory afferent fiber activity is driven by high-fidelity infor
62  with a piezoelectric device while recording afferent fiber activity.
63 ory cells, in addition to exciting gustatory afferent fibers, also activates glial-like Type I taste
64             This ATP excites primary sensory afferent fibers and also stimulates neighboring taste bu
65 dose inducing functional ablation of sensory afferent fibers and by devazepide, a CCK-A receptor anta
66  synapses, including those formed by sensory afferent fibers and by intrinsic interneurons.
67 f mGlu1alpha receptors on peripheral primary afferent fibers and determine the behavioral effects of
68 lized spine-like contacts between individual afferent fibers and hair cells that are surrounded by la
69 en functional properties of mechanoreceptive afferent fibers and intrinsic discharge properties of la
70 received monosynaptic innervation from vagal afferent fibers and LepR neurons exhibited large synapti
71 BS may activate 5-HT3 receptors on extrinsic afferent fibers and motor neurons, giving rise to viscer
72 DA receptors have been demonstrated on vagal afferent fibers and on second-order neurons in the media
73 eceptors have been found on both the sensory afferent fibers and on the GCs.
74 gthening of the polysynaptic pathway between afferent fibers and pattern-initiating neurons of the bu
75 e peripheral auditory pathway with misrouted afferent fibers and reduced synaptic contacts with hair
76 receive primary inputs from vibrissa sensory afferent fibers and send monosynaptic connections to fac
77 ological data revealed that numbers of vagal afferent fibers and synapses in the NTS were significant
78                     Plastic changes in vagal afferent fibers and synapses were investigated at the mo
79 The distribution of gravity-sensing, otolith afferent fibers and terminals was studied in the vestibu
80 tudy to identify the type(s) of VR1-positive afferent fibers and terminals.
81 ators of sensory information between primary afferent fibers and the spinal cord, activate Erk in dor
82  likely attributable to damage to myelinated afferent fibers and their abnormal sprouting in lamina I
83                                              Afferent fibers and their terminal distributions within
84 on at superficial lamina IA among horizontal afferent fibers and unique axonal targeting pattern sugg
85 support for the role of the thalamus and its afferent fibers and visual attentional processes in the
86 ons via OTR activation in CGRPergic (primary afferent fibers) and GABAergic cells.
87 ses suggested normal function of hair cells, afferent fibers, and efferent feedback.
88 cells, on peripheral nerves, on fine sensory afferent fibers, and on brain endothelial cells, respect
89     First, we briefly discuss the receptors, afferent fibers, and pathways involved in conveying tact
90 udy found that both VIP and PACAP containing afferent fibers are abundant in the BSTLd (dorsolateral
91                      Subpopulations of these afferent fibers are activated by capsaicin.
92               Our results suggest that large afferent fibers are critical for the timing of automatic
93  and glutamatergic responses in CVNs, C-type afferent fibers are critical to the afferent stimulation
94 pite the normal presence of Bdnf transcript, afferent fibers are disoriented near the organ of Corti.
95 ggesting that both hair cells and vestibular afferent fibers are normally recruited by GVS.
96 s, then the LOC terminals synapsing with IHC afferent fibers are not particularly rich in these pepti
97 isease, in which the cone-photoreceptors and afferent fibers are preserved but do not function, as a
98 tric fish Eigenmannia, P- and T-type primary afferent fibers are specialized for encoding the amplitu
99                Additionally, evoked EPSCs in afferent fibers are unaffected by glutamate transporter
100     In addition, efferent axons that use the afferent fibers as a scaffold during pathfinding also sh
101 ional terminals, presumed to be from primary afferent fibers, at the center of glomerular arrangement
102  of CCKA receptors in the terminals of vagal afferent fibers because these receptors have been implic
103                 In control mice, the labeled afferent fibers belong to fine, beaded axons diffusely d
104 a severe reduction in axonal arborization in afferent fibers, but no deficit in neurogenesis.
105 terminals are mainly endings of unmyelinated afferent fibers; C2 terminals are mainly endings of thin
106 rimary sensory circuits from locus coeruleus afferent fibers can produce a spectrum of modulatory act
107 eding, (2) capsaicin-sensitive hepatic vagal afferent fibers carry the signal that stimulates feeding
108 nnervated by neuropeptide-containing primary afferent fibers chiefly from the trigeminal nerve.
109 istent with the notion that group IV phrenic afferent fibers comprise the afferent arm of a fatigue-i
110 anistic insight into the subtypes of primary afferent fibers damaged by chemotherapy.
111 tment groups before histological analysis of afferent fiber density at the injection sites.
112                     The activity of auditory afferent fibers depends strongly on the frequency of sti
113  by application of capsaicin, which destroys afferent fibers, directly to the cervical vagi.
114 or prolonged stimuli, like tetanic bursts of afferent fiber discharge at high frequencies.
115 tivity, group IV, but not group III, phrenic afferent fibers discharged more (p<0.05) during rhythmic
116  We found that high-frequency stimulation of afferent fibers elicits synaptic currents mediated by al
117 cell, voltage-clamp recordings from IHCs and afferent fiber endings in excised postnatal rat cochleae
118 ents rather than in the terminals of primary afferent fibers, even though labeling overlapped with th
119                         Furthermore, LES rat afferent fiber-evoked APs showed a pronounced loss of te
120                                              Afferent fibers exhibited a wide heterogeneity in membra
121             The central terminals of primary afferent fibers experience depolarization upon activatio
122            To test whether primary gustatory afferent fibers express Ca(2+)-permeable AMPA/kainate re
123 th duodenal input transmitted by sympathetic afferent fibers expressing TRPV1; spinal neuronal respon
124 tion when activated by a small population of afferent fibers firing asynchronously at physiologically
125 sion electron microscopy showing that single afferent fibers follow a single dendrite mostly up to th
126 sion electron microscopy showing that single afferent fibers follow a single dendrite mostly up to th
127 of motoneurons, interneurons, and myelinated afferent fibers for antagonistic ankle muscles.
128  and internodal distance were quantified for afferent fibers from eight sites within the vestibular n
129 rainstem structure that receives the primary afferent fibers from electroreceptors in the skin.
130 ing the presynaptic terminals of cholinergic afferent fibers from projection neurons (Pn).
131 clei receive axosomatic endings from primary afferent fibers from the cochlea and have projections th
132                                  Sympathetic afferent fibers from the heart enter the upper thoracic
133 , layer Ia, which is the unique recipient of afferent fibers from the olfactory bulb.
134 s, which has resulted from the re-routing of afferent fibers from the third spinal nerve into the hyp
135                                          All afferent fibers had characteristic frequencies of 16-32
136                                Hepatic vagal afferent fibers have been implicated in the feeding resp
137 mination region for unmyelinated (C) primary afferent fibers; however, how the input it receives from
138 BA release evoked by electric stimulation of afferent fibers in acute brain slices and produces readi
139 tch-clamp recordings of hair cells and their afferent fibers in amphibian papillae of either male or
140 Electrophysiological recordings from primary afferent fibers in control and hyperalgesic mice with tu
141 s autoreceptors in terminals of Adelta and C afferent fibers in laminae I-III, presynaptic IGRs may p
142 , cytoarchitecture, and projection fields of afferent fibers in tandem revealed input target selectiv
143 rotid sinus and anodal block of baroreceptor afferent fibers in the carotid sinus nerve to examine th
144 ion of contraction-sensitive skeletal muscle afferent fibers in the cat; and (ii) synaptic input from
145 her naturalistic activation of barosensitive afferent fibers in the glossopharygneal and vagus nerves
146 itu hybridization to determine if neurons or afferent fibers in the hindbrain possess both TRH type 1
147 e provide evidence that Arc is translated in afferent fibers in the skin.
148 nsistent with our previous report, medullary afferent fibers in the solitary tract and spinal trigemi
149  are coexpressed on the terminals of primary afferent fibers in the spinal cord where they may mediat
150  low-frequency stimulation (5 Hz) of primary afferent fibers in the tractus solitarius resulted in a
151           The central projections of primary afferent fibers in the utricular nerve, which convey lin
152 egister with the major terminal fields of CT afferent fibers, in the central third of the rostral 1.0
153 c conditions, noxious stimulation of primary afferent fibers induces release of CatS from microglia,
154 e monosynaptic input from trigeminal primary afferent fibers innervating extracranial orofacial struc
155 cts of opioid receptor-selective agonists on afferent fibers innervating the colon.
156                          Discrete tracing of afferent fibers innervating the individual semicircular
157 extrinsic NOS-IR nerve fibers represented an afferent fiber input that was separate from the substanc
158 synaptic EPSP, and EPSP in cells receiving C-afferent fiber input.
159 eurons, but also to regulate local growth of afferent fibers into target tissues and, in some cells,
160 nd behavior coincided with the projection of afferent fibers into the central vestibular nuclei, indi
161 on of NMDA responses to burst stimulation of afferent fibers is described, which required GABAA block
162 hat the modulatory function of noradrenergic afferent fibers is exerted through paracrine interaction
163 nerve growth factor (NGF)-responsive primary afferent fibers is thought to contribute to their hypera
164   During paired recordings of hair cells and afferent fibers, L-type voltage-gated Ca(2+) currents sh
165                         Shock stimulation of afferent fibers (lateral olfactory tract) and associatio
166 4.3 downregulation in nociceptive trigeminal afferent fibers may contribute to peripheral cold hypers
167  Based on their anatomic localization, these afferent fibers may function to transmit important senso
168 at receptor-Ca2+ channel coupling in primary afferent fibers may have little functional significance
169 response to colorectal distension, and colon afferent fiber mechanosensitivity were assessed in contr
170  induce long-lasting depression of A primary afferent fibers-mediated monosynaptic excitatory postsyn
171  preparation, cats were anesthetized and the afferent fibers mediating the pressor reflex entered the
172             Examination of over 400 isolated afferent fibers, more than 200 from each species, indica
173   Sublamination requires the activity of the afferent fibers, NMDA receptors, and nitric oxide syntha
174                         Synaptic currents in afferent fibers occurred with high failure rates of appr
175 erent fibers within the dorsal root ganglia, afferent fibers of the dorsal root, dorsal root entry zo
176      The distribution and synaptology of the afferent fibers of the glossopharyngeal nerve (IXN) in t
177  to identify putative synaptic contacts from afferent fibers of the two nerves onto individual projec
178 tivation in the brain, which is triggered by afferent fibers of the vagus nerve (VN).
179                                    Gustatory afferent fibers of the vagus nerve that innervate taste
180  to determine the effect of activating vagal afferent fibers on the discharge of median preoptic (MnP
181            We conclude that group III and IV afferent fibers originating from the skeletal muscle of
182 dpuppy, PACAP is found primarily in visceral afferent fibers, originating from cells in either the do
183 afferent detected at the terminal and in the afferent fiber outside of the corpuscle.
184                 Trigeminal, spinal and vagal afferent fibers overlap in C1-C2 segments.
185 ransmission from sympathetic and peptidergic afferent fibers participates in the regulation of pulpal
186 hat selective CST injury induces maladaptive afferent fiber plasticity remote from the lesion.
187 mply that, in conjunction with local primary afferent fiber plasticity, injury-induced sprouting of D
188 tio-temporal patterning of cutaneous sensory afferent fiber projections to the dorsal, but not the ve
189                                              Afferent fibers regrew through the transection site with
190 that activity-dependent production of Arc in afferent fibers regulates neurogenic inflammation potent
191  no effect; in that situation, the RA of the afferent fibers remained and the preparation still respo
192 r combined; in that situation, the RA of the afferent fibers remained.
193 ive colonic neurons, consistent with reduced afferent fiber responses to UTP and ADP in Na(v)1.9(-/-)
194          In addition, whereas stretch-evoked afferent fiber responses were enhanced by chemical infla
195 ith a significant and selective reduction in afferent fiber sensitivity to circumferential stretch of
196   Of interest, 10% of the trigeminal primary afferent fibers showed glycine localization.
197 e, paired recordings of hair cells and their afferent fibers showed that synaptic delays shortened an
198 ER) mice showed selective labeling of type I afferent fibers, Slc6a4-GFP mice labeled type II fibers
199                              Using single TS afferent fiber stimulation in slices from control and CI
200 synaptic current elicited in mPFC neurons by afferent fiber stimulation shows linear correlation with
201 secreting the enzyme, as a result of primary afferent fiber stimulation.
202 sed the synaptic transmission in response to afferent fibers stimulation.
203 activity generally associated with 8th nerve afferent fibers, strongly suggest that GABA serves an im
204 More specifically, how does the hair cell to afferent fiber synapse in bullfrog amphibian papilla adj
205 odel, suggest sustained release at hair cell afferent fiber synapse is dictated by Ca(2+)-dependent v
206 ling statistical properties of the hair cell-afferent fiber synapse suggested that the ability of one
207                                Cardiac vagal afferent fibers synapse in the nucleus tractus solitariu
208 ontaining glomus cells (GCs) and the sensory afferent fibers synapsing onto the GCs.
209 mpal GABA cells with extrinsic and intrinsic afferent fiber systems.
210 opy results, imply that unmyelinated primary afferent fibers terminating in the superficial dorsal ho
211                 Along this row the number of afferent fibers terminating on IHCs increases toward the
212 inea pig intrinsic cardiac ganglia are vagal afferent fibers that also contain NOS.
213 r hair cells (IHCs) are mainly innervated by afferent fibers that convey sound information to the CNS
214 use sprouting of nociceptive pelvic visceral afferent fibers that correlates with increased hypertens
215 rried by action potentials of S-type primary afferent fibers that project to the inner cellular layer
216 selectively taken up by unmyelinated primary afferent fibers that terminate in the outer part of lami
217                     The vagus nerve contains afferent fibers that transmit sensory information from t
218 ion was afferent in origin, we traced apical afferent fibers that were retrogradely labeled by extrac
219                  The major class of cochlear afferent fibers, the type-I or radial-fiber (RF) populat
220 ve ivy cells and SM-expressing O-LM cells to afferent fiber theta burst stimulation.
221  142,801 decreased responses of pelvic nerve afferent fibers to colorectal distention.
222 that relays sensory information from primary afferent fibers to higher order cells of ELLS.
223 vated 5-HT(3) and 5-HT(2) receptors on vagal afferent fibers to mediate luminal factor-stimulated pan
224 ent duodenal factors stimulate vagal mucosal afferent fibers to mediate pancreatic enzyme secretion v
225 tion to the central nervous system via vagal afferent fibers to mediate pancreatic secretion.
226 endently attenuate responses of pelvic nerve afferent fibers to noxious colonic distension was unaffe
227 eculate that they may be branches of type II afferent fibers to outer hair cells and that a smaller p
228                                  Response in afferent fibers to peripheral stimulation is simulated b
229 tterns of peptidergic small-diameter primary afferent fibers to the cat sacrocaudal spinal cord, a re
230 e information is transmitted by baroreceptor afferent fibers to the central network by glutamate bind
231 tter from glomus cells activates the sensory afferent fibers to transmit information to the nucleus o
232 gustatory receptor cells and primary sensory afferent fibers transmit the output signal from taste bu
233                 These pulses stimulate vagal afferent fibers, transmitting signals to the brain and m
234 at climbing fibers and some other cerebellar afferent fibers transported the virus retrogradely from
235 not contacted by type I afferents; and last, afferent fiber type determines if and how efferents inne
236 tigate changes in the responses of meningeal afferent fibers using a preclinical model of migraine in
237 inal common pathway through which convergent afferent fibers using a variety of neurotransmitter agen
238 topic array of hair cells and transmitted to afferent fibers via fast, repetitive synaptic transmissi
239 t group III (n=7) and group IV (n=8) phrenic afferent fibers was recorded during rhythmic diaphragmat
240 elated peptide (CGRP)-immunoreactive primary afferent fibers was selectively altered at specific spin
241 from presynaptic hair cells and postsynaptic afferent fibers, we established that the rate of evoked
242 understand the effects of capsaicin on adult afferent fibers, we examined DRG neurons retrogradely la
243 ordings from adult frog hair cells and their afferent fibers, we show here that during sine-wave stim
244 ctrophysiological recordings of pelvic nerve afferent fibers, we studied drug effects on responses to
245                                     Group IV afferent fibers were ablated in neonatal Sprague-Dawley
246 ponses and neuronal activity in unmyelinated afferent fibers were assessed in monkey after topical ap
247                     Responses of rat jejunal afferent fibers were examined by electrophysiology.
248   CAPs of the large and the small myelinated afferent fibers were observed in response to SCS at all
249                      In contrast, myelinated afferent fibers were observed to be sprouting into lamin
250 tide (CGRP)-immunoreactive (CGRP-IR) primary afferent fibers were observed within the superficial lam
251                                      Labeled afferent fibers were of two types: type I fibers contact
252                Mechanosensitive pelvic nerve afferent fibers were recorded from the decentralized S1
253 nal evidence suggests that CCK acts on vagal afferent fibers, which may explain how CCK doses that pr
254 hanisms to maximize the dynamic range of its afferent fibers, which operate at the physiological limi
255 e activated by different distinct subsets of afferent fibers with minimal overlap.
256                   Rats denervated of primary afferent fibers with neonatal capsaicin injection and an
257  to correlate the influence of sensory vagal afferent fibers with the functional organization of mGlu
258 ceived direct ST inputs from only one or two afferent fibers, with one-half also receiving smaller am
259 r) was constitutively expressed on cells and afferent fibers within the dorsal root ganglia, afferent
260  Here, we describe that stimulation of GLP-1 afferent fibers within the PVN is sufficient to suppress

 
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