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1 all mortality between enrollment and 1 month after birth).
2 he role of calcineurin in promyelinating SCs after birth.
3 re and after the risk interval until 5 years after birth.
4 n of which decreases in skeletal muscle soon after birth.
5 l development, whereas the trend is reversed after birth.
6 tem cell (SSC) 'niche' in its periphery soon after birth.
7 t toward the sudden microbial exposure early after birth.
8 is and signal to maintain tissue homeostasis after birth.
9 maturation from postnatal day 14 to month 2 after birth.
10 rearrangements did not significantly expand after birth.
11 was administered to HIV-exposed infants soon after birth.
12 up (n = 57), cord clamping was at 60 seconds after birth.
13 ethality between the second and fourth weeks after birth.
14 ntly consumed breast milk for the first year after birth.
15 protein profiles during the first two weeks after birth.
16 ism that controls their maintenance in bones after birth.
17 isit, which was scheduled for the first week after birth.
18 ong in the embryo and progressively declines after birth.
19 to meet the antigenic challenges encountered after birth.
20 49 newborn infants recruited within 24 hours after birth.
21 can readily regenerate within a short period after birth.
22 ended in infants with suspected CMTC shortly after birth.
23 ighted MRI and thalamic proton MRS 4-14 days after birth.
24 ver from positive to negative about 24 years after birth.
25 n = 56), cord clamping was within 15 seconds after birth.
26 g loss at neonatal hearing screening shortly after birth.
27 um from 30 days after conception to 9 months after birth.
28 term neonates for at least 30 to 60 seconds after birth.
29 cell view of the adaptation to air breathing after birth.
30 foration, and blindness as early as 24 hours after birth.
31 l energy reserves to sustain the first weeks after birth.
32 cal conjugate vaccine at 2, 4, and 11 months after birth.
33 y failure and pulmonary hypertension shortly after birth.
34 nd/or Ezh2, at postnatal day 14 and 2 months after birth.
35 re generated to a large degree independently after birth.
36 ortion of space with defined boundaries soon after birth.
37 as well as in infants against ZIKV infection after birth.
38 iple axons to single innervation a few weeks after birth.
39 als have muscles that rapidly disappear soon after birth.
40 ours (27.7%), and more than 48 hours (36.7%) after birth.
41 evelopment and cardiomyocyte maturation soon after birth.
42 e family is essential for normal bone growth after birth.
43 ates had a severe illness by the seventh day after birth.
44 ylcarnitines and serum lactate, and die soon after birth.
45 continued to mature until approximately 3 wk after birth.
46 cular cell progenitors, but is downregulated after birth.
47 ygous mice than in wild-type mice at 21 days after birth.
48 uilibrium following a dynamic priming period after birth.
49 y's birth, and at 3, 6, 9, 12, and 18 months after birth.
50 ctors (Pbx) led to lethal PH in mice shortly after birth.
51 leukocytes extracted from cord blood shortly after birth.
52 ain protein product, and its levels decrease after birth.
53 ects remain unrepaired for prolonged periods after birth.
54 d (b) temporally through tamoxifen treatment after birth.
55 NTBC) at a dose of 1 mg/kg per day initially after birth.
56 the abdomen and pelvis was performed 6 days after birth.
57 results into a solidified compact myocardium after birth.
58 ks and/or birth weight <1500 g were enrolled after birth.
59 oncentrations were measured for up to 7 days after birth.
60 city for regeneration, which is lost shortly after birth.
61 ne may be functionally required in the brain after birth.
62 nce between NIRS SctO2 and aEEG for 78 hours after birth.
63 evelopment and shape gut microbiota assembly after birth.
64 ging scans were acquired in neonates shortly after birth.
65 mite (HDM) allergens from days 3, 15, or 60 after birth.
66 mic encephalopathy enrolled at 6 to 24 hours after birth.
67 knocked out Celsr3 in the hippocampus 1 week after birth.
68 on parental disease activity both before and after birth.
69 ine receptor CXCR6 and seed meninges shortly after birth.
70 ly, expression of GAS6 initiated only 3-4 wk after birth.
71 idity and mortality from infectious diseases after birth.
72 ated rate, often within the first few months after birth.
73 lls were generated during embryogenesis, not after birth.
74 d in profound deficits in maternal care soon after birth.
75 KO) mice and found that most died within 3 d after birth.
76 in 99.5% of infants within the first 30 days after birth.
77 uces target RNA expression for up to 4 weeks after birth.
78 infant and maternal outcomes up to 24 months after birth.
79 mples from infants were collected 4 and 9 mo after birth.
80 and bone area (BA) were measured within 2 wk after birth.
81 nocytes that colonize the tissue immediately after birth.
82 r5, but delays robust Foxl2 expression until after birth.
83 e detectable using molecular methods by 16 h after birth.
84 was administered to HIV-exposed infants soon after birth.
85 h weight, and 30.8% required hospitalization after birth.
86 replaced by adult hemoglobin S at about 1 y after birth.
87 ibling was preemptively transplanted shortly after birth.
88 oxygen consumption rate, increased primarily after birth.
89 stepwise fashion, reaching complete maturity after birth.
90 us, the LSt group is sexually dimorphic soon after birth.
91 als that reproduce months, years, or decades after birth.
92 nic and fetal tissues and markedly decreases after birth.
93 edispositions and oncogenic events occurring after birth.
94 e fetus to a more mature but quiescent state after birth.
95 he children at birth as well as at 1 and 2 y after birth.
96 formation of diversified NK cell repertoires after birth.
97 nd continuing with direct dosing of the pups after birth.
98 ve erythropoietin or placebo within 24 hours after birth.
99 cocorticoids in preparing the fetus for life after birth.
102 a functioning pulse oximeter by two minutes after birth, (2) Delayed intubation, (3) Normothermia on
103 (5/5), sensorineural deafness diagnosed soon after birth (5/5), congenital cataracts (4/5), and hypot
105 V virologic testing performed before 8 weeks after birth (adjusted relative risk [aRR] 1.03; 95% CI 0
107 postnatally acquired ZIKV for several months after birth, an encouraging result given the potentially
108 receptor, VPAC2R, reached detectable levels after birth and after the onset of circadian synchrony.
112 crobiome is established within a few minutes after birth and consists of stable multi-species communi
113 pha-cell proliferation rapidly declined soon after birth and continued to drop to very low levels in
114 nseling on IYCF during pregnancy until 12 mo after birth and daily use of SQ-LNS for infants 6-12 mo;
115 iffer among studies as the outcomes manifest after birth and depend on maturation processes that vary
117 allowed Spint2-deficient mice to gain weight after birth and dramatically increased their lifespan.
118 udinal study of 2,113 children enrolled soon after birth and followed up monthly for 18 months to ass
119 samples were collected beginning immediately after birth and for 1 year; the children were followed u
120 alveolar macrophage (AM) development shortly after birth and for maintenance of AM functions througho
121 d genetic background, Dnaaf2 homozygotes die after birth and have left/right defects among other phen
123 ith GACI are diagnosed early, generally soon after birth and in some cases before birth by fetal ultr
125 translocates to cardiomyocyte nuclei shortly after birth and mediates postnatal cell cycle arrest(3).
126 on resulting from p62 deficiency is manifest after birth and obesity subsequently develops despite no
127 the mechanisms of germ cell differentiation after birth and potentially explain the spatiotemporal R
128 gammadelta TCR repertoires develop directly after birth and present important differences among gamm
129 ility of a health-associated oral microbiome after birth and provides insights that may be important
131 supplementary respiratory drive immediately after birth and reveal key molecular components of a pep
132 ealed a degenerative myopathy that developed after birth and specifically affected smooth muscle in t
135 arrests cardiomyocyte proliferation shortly after birth and thereby eliminates regeneration after in
136 eries enable these arteries to close rapidly after birth and thus prevent blood loss in newborns.
137 orted questionnaires at 6 weeks and 6 months after birth and were included into the analyses of this
138 ntinence (SUI) and postnatal depression (PD) after birth, and (2) investigate both possible direction
139 and and mature in parallel with renal growth after birth, and are mainly derived from fetal liver mon
140 onstrated that the preterm infant liver soon after birth, and by extension the fetal liver, was metab
142 to interact with others measured within days after birth, and differences in infants' early social en
143 pression at different periods (before birth, after birth, and during child ages 1-5, 6-10, and 11-16
144 ytes lose the ability to proliferate shortly after birth, and further increase in cardiac mass is ach
146 unique (neomorphic) features already shortly after birth, and these features persist throughout postn
147 The maturation of the mammalian brain occurs after birth, and this stage of neuronal development is f
148 ferentiation of V-SVZ progenitors before and after birth, and we identified Sox6 as a potential downs
149 he postnatal mastery of suckling at 4 months after birth; and (2) thereafter, from preparation to the
157 specific IgE measured before conception than after birth [BHR: aRRR = 2.96 (95% CI: 1.92, 4.57) and 1
158 of peripheral T cells within the first 2 wk after birth but failed to populate the peripheral T cell
160 icularly for microcephaly infants, were poor after birth but showed improvement beyond 4 months of li
161 or microcephaly infants, were initially poor after birth but showed improvement beyond four months of
162 tted by the mother during a tight age window after birth but stable for life, resistant to many micro
163 xidative phosphorylation increased primarily after birth but was accompanied by prepartum increases i
164 ual cortex is adult-like within a few months after birth, but is subsequently refined through develop
165 sequencing has transformed genetic diagnosis after birth, but its usefulness for prenatal diagnosis i
166 antly among children from 5 through 13 weeks after birth, but thereafter gradually increased to 21 an
167 to the amniotic space at Embryonic Day 20 or after birth by intraperitoneal injection for 2 weeks.
170 rse environmental events in utero or shortly after birth can lead to abnormal brain development and i
171 Although the epicardium becomes dormant after birth, cardiac injury reactivates developmental ge
173 to growth by cardiomyocyte enlargement soon after birth, coinciding with a period in which most card
174 er noxious-evoked brain activity a few hours after birth compared to those born by elective cesarean
175 athy, hypothermia initiated at 6 to 24 hours after birth compared with noncooling resulted in a 76% p
176 tion of hydrocortisone between 7 and 14 days after birth, compared with placebo, did not improve the
178 t(+) mice engrafted with human tissues 1 day after birth (designated NSG-1d mice) were highly suscept
179 r, establishment of the commensal microbiome after birth dictates immune functionality and tissue hom
180 of onset is related to physiological changes after birth (e.g., hormonal, immunological, circadian),
181 ghts of 400 g to 999 g at less than 72 hours after birth; enrollment took place between July 14, 2011
182 t's clinical condition in the first 24 hours after birth (EOS calculator period [July 1, 2014, throug
183 ugh the peripartum period (the first 6 weeks after birth) (epoch 1) with the frequency during the pos
184 n throughout fetal development and disappear after birth, except in conditions of delayed maturation
185 at are briefly exposed to ascr#3 immediately after birth exhibit increased ascr#3-specific avoidance
187 ion vaccine were given at 2, 3, and 4 months after birth followed by a booster at 11 months and a 10-
188 fetal liver and become PPARalpha responsive after birth following an epigenetic switch triggered by
189 nockout mice (Podxl(-/-)), which die shortly after birth from anuria and hypertension, Podxl(DeltaPod
190 n fetal membranes (FM) collected immediately after birth from women delivering preterm, p-IRAK1 was s
193 that develops during pregnancy and resolves after birth has been recognized for over 50 years, but u
194 for breast cancer that peaked about 5 years after birth (HR, 1.80 [95% CI, 1.63 to 1.99]) before dec
195 response in relation to outcome in the 80 h after birth in a cohort of babies with NE undergoing 'fa
196 ribe the care received by neonates up to 2 h after birth in a sample of three countries in west Afric
204 Thalamic proton MRS measures acquired soon after birth in neonatal encephalopathy had the highest a
205 asing peptide (GRP) was elevated within days after birth in newborns exposed to hyperoxia who later d
207 microbial population of infants at 3-7 days after birth in the control group was transmitted from th
209 a higher prevalence of remission at 6 months after birth in the THPP and EUC group compared with the
211 new and increasingly sophisticated behaviors after birth is accompanied by dramatic increase of newly
212 32 degrees C in utero and in the first year after birth is associated with a 0.1% reduction in adult
213 in a single high dose during the first month after birth is readily acquired but not retained efficie
214 ely lower their body temperature in the days after birth, is an emerging practice in some West Africa
215 (Lmnb2), whose expression decreases in mice after birth, is essential for nuclear envelope breakdown
216 1a B-cell subset (present as early as 4 days after birth), late-onset lymphoid cancer development, an
217 f adipogenic and antimicrobial dFBs declined after birth, leading to an age-dependent loss of dermal
219 knockout of Vangl2 in the hippocampus 1 week after birth led to a large increase in synaptic density,
221 etal-derived Vgamma9Vdelta2 T cells promptly after birth, likely upon environmental phosphoantigen ex
225 de PIWI proteins to silence transposons but, after birth, most post-pubertal pachytene piRNAs map to
226 to 9.0; P = .01) as well as anxiety shortly after birth (mothers: 13% vs 48%; OR = 6.5; 95% CI, 3.3
227 fants had higher rates of depression shortly after birth (mothers: 6% vs 40%; odds ratio [OR] = 9.9;
229 f the peripheral tissue continues to develop after birth, neurons may modulate tissue inflammation in
232 ulative grass pollen exposure up to 3 months after birth, on FEV(1) , FVC, and FEV(1) /FVC ratio at 1
234 Altogether, our findings suggest that soon after birth peripersonal space may be already considered
236 he expression of ERRalpha and gamma in heart after birth (pn-csERRalpha/gamma [postnatal cardiac-spec
237 s (Miz1DeltaPOZ) causes a neuropathy at 90 d after birth [postnatal day (P) 90], with a subsequent sp
238 blood transcriptomic signature measured soon after birth, predicts adverse neurodevelopmental outcome
240 isation or known neonatal death up to 7 days after birth), preterm delivery (<37 weeks' gestation), o
241 processes during endochondral bone formation after birth, recent studies have demonstrated the direct
242 Hypothermia initiated at less than 6 hours after birth reduces death or disability for infants with
243 that hypothermia initiated at 6 to 24 hours after birth reduces the risk of death or disability at 1
244 aneous head movements as early as 1-2 months after birth, relative to HR infants who showed more rapi
247 tion, vertical transmission of GBS during or after birth results in life-threatening neonatal infecti
249 spital discharge, infection more than 3 days after birth, severe retinopathy of prematurity, severe i
250 iorally determined contrast sensitivity soon after birth.SIGNIFICANCE STATEMENT Despite >50 years of
251 cient mice showed enlarged hearts before and after birth, similar to Nppa-Nppb compound knockout mice
252 hat although beta-catenin signaling declines after birth, Sox17 activation increases and remains high
253 arding HOXA5 functions in the nervous system after birth, such as a potential role in the establishme
254 lls complete their differentiation 2-3 weeks after birth, suggesting a link to postnatal maturation.
255 ough the kidney cannot generate new nephrons after birth, suggesting a low level of regenerative comp
256 ive knockout mice lacking Syt11 died shortly after birth, suggesting Syt11-mediated membrane transpor
257 tology and proteome over the first two weeks after birth support that the porcine vagina continues to
258 alcn in excitatory preBotC neurons died soon after birth; surviving mice developed apneas in adulthoo
259 between 10 (adolescent) and 15 (adult) weeks after birth, symptom initiation and progression upon rem
260 not disrupted in mutant embryos, but shortly after birth tenocytes lost differentiation markers and r
261 is a good understanding of the early events after birth that lead to the rapid increase in serum bil
264 strate that even though the gingiva develops after birth, the majority of gingival gammadeltaT cells
266 s demonstrate that except for a short period after birth, the TRN of the mouse lacks intrinsic GABAer
267 ases in the use of less invasive ventilation after birth, there was no significant decline in oxygen
268 unting usually begins in utero and continues after birth; therefore, its reduction must involve actio
270 d to extremely preterm infants from 24 hours after birth through 32 weeks of postmenstrual age did no
271 valuated oral microbiome communities shortly after birth, through adulthood, and up to 1 y of life in
274 g oral ADRbeta modifiers for the first month after birth to activate ADRbeta2 and antagonize ADRbeta1
275 s are programmed to develop in the intestine after birth to constitute a specialized microenvironment
276 hanges in RNA expression patterns before and after birth to identify signaling pathways selectively a
277 n of SMN has taken place either 1 or 24 days after birth to reflect early or later therapeutic interv
278 outcomes of individuals who spent from soon after birth to up to 43 months in severe deprivation in
279 e eyes, which naturally undergoes regression after birth, to gain mechanistic insights that could be
280 tly more likely to have skin-to-skin contact after birth, to have it for a longer time, and to breast
282 a probiotic combination or placebo from soon after birth until discharge from hospital or term correc
283 ibiotic use occurred between 24 and 72 hours after birth; use decreased from 0.5% to 0.4% (adjusted d
286 stop dividing during embryogenesis; and soon after birth, vestibular SCs in mammals transition to las
287 myocytes (CMs) become multinucleated shortly after birth via endoreplication and interrupted mitosis,
288 repertoires are shaped by microbial exposure after birth, we monitored the gamma-chain (TRG) and delt
289 solely responsible for pulmonary circulation after birth when the left ventricle (LV) becomes the sys
290 cteria phyla were already present at 5 weeks after birth, whereas many bacteria of the Firmicutes phy
291 the hematologic and neurologic complications after birth, whereas TCblR/CD320 gene defects appear to
292 ck-out mice gradually develop hypothyroidism after birth, which leads to reduced body weight and shor
293 F or saline into the BNST in the first weeks after birth while the LB dams were exposed to limited ne
297 out noticeable defects but die at about 3 wk after birth with signs of both general growth defects an
298 h a DGC-like morphology at approximately 7 d after birth, with a primary dendrite pointing to the mol