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1 s benefit significantly reduces with greater age-related hearing loss.
2 Asymptomatic adults 50 years or older with age-related hearing loss.
3 South Carolina Longitudinal Cohort Study of Age-Related Hearing Loss.
4 ng to compromised temporal processing during age-related hearing loss.
5 se aspirin did not affect the progression of age-related hearing loss.
6 shed, as are polygenic risk contributions to age-related hearing loss.
7 ng effort is critical for early diagnosis of age-related hearing loss.
8 aring threshold shifts and the prevention of age-related hearing loss.
9 riving compromised temporal processing under age-related hearing loss.
10 glycoside antibiotic exposure, as well as in age-related hearing loss.
11 ility deteriorates, in a graded manner, with age-related hearing loss.
12 ing in social settings for older adults with age-related hearing loss.
13 ted at speech rates (4 Hz) in listeners with age-related hearing loss.
14 their hearing and usually the first sign of age-related hearing loss.
15 system could be a viable strategy to prevent age-related hearing loss.
16 r leads to neurodegeneration and exacerbates age-related hearing loss.
17 essing of the central auditory system during age-related hearing loss.
18 a well-known correlation between smoking and age-related hearing loss.
19 jury and disease, and a major determinant of age-related hearing loss.
20 psychiatric treatments for older adults with age-related hearing loss.
21 d characterization of a novel mouse model of age-related hearing loss.
22 lowers tHcy levels potentially ameliorating age-related hearing loss.
23 that they play a role in the progression of age-related hearing loss.
24 Eps8 and is a candidate gene for progressive age-related hearing loss.
25 nal changes in IHCs are a potential cause of age-related hearing loss.
26 association between 17 tagSNPs for MSRB3 and age-related hearing loss.
27 orms of deafness, as well as progressive and age-related hearing loss.
28 2 and Skp1 as potential genetic modifiers in age-related hearing loss.
29 as mutations in its gene cause deafness and age-related hearing loss.
30 ion/treatment of inner ear disorders such as age-related hearing loss.
31 s directly contributes to the progression of age-related hearing loss.
32 ure and in analyses excluding those with non-age-related hearing loss.
33 uity has been identified as one component of age-related hearing loss.
34 t environmental exposures may play a role in age-related hearing loss.
35 ying progressive sensorineural hearing loss (age-related hearing loss 5, ahl5) and audiogenic seizure
39 on to many inbred mouse strains, accelerates age-related hearing loss (AHL) and can worsen auditory p
44 iety of species and slows the progression of age-related hearing loss (AHL), a common age-related dis
45 of mice vary widely in onset and severity of age-related hearing loss (AHL), an important considerati
51 e whether deficiencies in Cu/Zn SOD increase age-related hearing loss and cochlear pathology, we coll
53 ing an opportunity to study both progressive age-related hearing loss and possible interventional the
55 nd noise-induced hearing loss because of its age-related hearing loss and susceptibility to acoustic
56 vironmental, and sex-related determinants of age-related hearing loss and to identify potential inter
58 ges and suffered a base to apex gradient and age-related hearing loss, and that mutations in cdh23 we
62 d outer hair cell loss characteristic of the age-related hearing loss (ARHL) in the background strain
73 ric hearing deficits are a common symptom of age-related hearing loss (ARHL), as are specific histopa
80 ty of sensorineural etiologies including (1) age-related hearing loss (ARHL); (2) neuropathy related
81 conceptual framework for understanding human age-related hearing loss (ARHL, or presbycusis) holds th
85 protective effects of caloric restriction on age-related hearing loss by promoting the mitochondrial
88 earing thresholds in these mice demonstrated age-related hearing loss in all homozygous-null, but not
91 se SIRT3 was essential for the prevention of age-related hearing loss in mice fed a calorically restr
92 ts an age-noise interaction that exacerbates age-related hearing loss in previously noise-damaged ear
94 thological evaluation.SIGNIFICANCE STATEMENT Age-related hearing loss is a common neurodegenerative d
98 del suggests that the primary factor in true age-related hearing loss is an energy-starved cochlear a
104 gets for intervention.SIGNIFICANCE STATEMENT Age-related hearing loss is one of the most common chron
106 erved in older adults.SIGNIFICANCE STATEMENT Age-related hearing loss is the most prevalent sensory i
110 ty risk factors and studied multiplex family age-related hearing loss (mARHL) and simplex/sporadic ag
111 he objective was to test the hypothesis that age-related hearing loss may be associated with poor vit
112 animal model by comparing noise-induced and age-related hearing loss (NIHL; AHL) in groups of CBA/Ca
113 m response (ABR) which confirmed that severe age-related hearing loss occurred in 8-month-old mice, w
115 ion cells, were consistent with the observed age-related hearing loss of these mice beginning with th
118 rol subjects and patients with noise- and/or age-related hearing loss (p < 0.05 at all dilutions test
119 ait in the pathophysiological progression of age-related hearing loss, possibly aimed at preserving f
120 ly subjects challenge the common belief that age-related hearing loss (presbyacusis) is based primari
126 in a mouse model (of both sexes) of "normal" age-related hearing loss revealed that early pathophysio
127 ed hearing loss (mARHL) and simplex/sporadic age-related hearing loss (sARHL) cases and controls with
129 ction, the role of these supporting cells in age-related hearing loss, the most common sensory defici
130 cell (OHC) degeneration is a common trait of age-related hearing loss, the objective of this study wa
135 er hair cells of the C57BL/6J mouse model of age-related hearing loss, which reveal that cholinergic