ライフサイエンスコーパス: aggression

1 pC1d neurons as core nodes regulating female aggression.

2 eral prefrontal cortex (DLPFC) in regulating aggression.

3 after attack and traumatic stress to enhance aggression.

4 e preferences, can shape biases in male-male aggression.

5 rons selectively modulates the escalation of aggression.

6 our understanding of how the LS may regulate aggression.

7 control sex recognition, mating, or maternal aggression.

8 ses blocks the effect of traumatic stress on aggression.

9 etween appetitive and consummatory phases of aggression.

10 ed, and genetic inactivation reduced, female aggression.

11 n the control of social behaviors, including aggression.

12 of these neurons blocked the enhancement of aggression.

13 rovide insights into the genetic etiology of aggression.

14 access certain areas with a reduced risk of aggression.

15 ess and the biological bases for interfemale aggression.

16 le mating behavior, general arousal, or male aggression.

17 ey elements in fine-tuning the regulation of aggression.

18 ve disorders in the female targets of female aggression.

19 m (LS) plays an important role in regulating aggression.

20 lencing reduces and its activation increases aggression.

21 imited to-peaceful resistance and collective aggression.

22 euron: its activation leads to a decrease in aggression.

23 rtant to assess LS involvement in regulating aggression.

24 re-escalation phase but not during escalated aggression.

25 at tempers rather than fuels poverty-induced aggression.

26 and Self-control in Humans (CLASH) model of aggression.

27 environments to rigid routine behaviors and aggression.

28 tions from the range cause impulsiveness and aggression.

29 male prairie voles displaying affiliation or aggression.

30 any tumors and their correlation with cancer aggression.

31 vl(Esr1+) cells are indispensable for female aggression.

32 were seen to initiate more and receive less aggression.

33 er warming would heighten average individual aggression.

34 uired for P1-evoked persistent courtship and aggression.

35 connotes social status and imminent physical aggression.

36 ative feedback was associated with decreased aggression.

37 erarchically organized social signals during aggression.

38 nsive range overlap, and limited inter-group aggression.

39 luding starvation stress resistance and male aggression.

40 locus for isolation-dependent enhancement of aggression.

41 ldhood trauma was associated with subsequent aggression.

42 o developmental challenges and environmental aggressions.

43 of the sexual coercion hypothesis [7]: male aggression (1) is greatest against cycling females, (2)

44 ividuals and in so doing avoid the threat of aggression [20-23].

45 staff intervention in response to escalated aggression, a continuum between support and control.

46 In explaining variation in violent aggression across populations, the age structures of tho

47 escribe the recently proposed phenomenon of "aggression addiction." Next, we discuss the pharmacologi

48 Behavioral aggression after social evaluation decreased over time,

49 Regulating aggression after social feedback is an important prerequ

50 le among the V group significantly predicted aggression against people.

51 Existing literature on mouse aggression almost exclusively examines territorial aggre

52 othesis that externalizing behaviors such as aggression, although in many respects detrimental, may b

53 t, as refuge-shortage increases, the rate of aggression among gobies increases and parasitism intensi

54 early age of allocation to a group decreases aggression and 3) manipulation increases aggression in m

55 Physical abuse predicted both aggression and affective lability as well as SI variabil

56 Analysis of interventions targeting aggression and agitation (148 studies [21 686 patients])

57 han pharmacologic interventions for reducing aggression and agitation in adults with dementia.

58 Social aggression and avoidance are defensive behaviors express

59 ntromedial hypothalamus controls both social aggression and avoidance, suggesting that it may encode

60 e explanations for the link between observed aggression and bystander behavior, including priming or

61 Aggression and callous, uncaring, and unemotional (CU) t

62 teractions, how the crosstalk impinges on OC aggression and chemoresistance are not well-characterize

63 an also promote negative outcomes, including aggression and coercion.

64 ion of these GAD2 neurons promoted male-male aggression and conditioned place preference for aggressi

65 t patterns characteristic of female and male aggression and confirmed that the effect was female-spec

66 Aggression and courtship behavior were examined of wild

67 gic, and neuroendocrine genetic variation in aggression and CU-traits.

68 hai2 ablation causes reduced infant-directed aggression and decreased activity in MeA neurons during

69 ednraa(-/-) mutants exhibit heightened aggression and decreased whole-body cortisol levels sugg

70 childhood intervention designed to decrease aggression and delinquency in at-risk kindergarteners.

71 ls for dissecting mechanisms mediating tumor aggression and demonstrating the value of humanized mode

72 of the neuroendocrine basis of female social aggression and dominance in laboratory mice.

73 ckdown of Chrna7 in the DG enhanced baseline aggression and eliminated the serenic effects of both ni

74 the nest, suggesting an association between aggression and frequent-close exposure to humans and der

75 t, when controlling for the effect of higher aggression and impulsivity.

76 or the evolutionary perspective on out-group aggression and in-group cooperation.

77 onal ablation of Galphai2 enhances male-male aggression and increases neural activity in the medial a

78 imity can include competition for resources, aggression and kleptoparasitism meaning that the decisio

79 m harassment and punishment, by showing that aggression and matings are temporally decoupled.

80 analysis of chronological sequences between aggression and matings ruled out other coercive mechanis

81 nd control innate social behaviors including aggression and mounting.

82 PA(Esr1+) cells are excited during intermale aggression and promote attacks.

83 Appetitive aggression and relapse to aggression seeking can be mode

84 eveloped a mouse model of appetitive operant aggression and reported that adult male outbred CD-1 mic

85 ings place the PA as a key node in both male aggression and reproduction circuits.

86 ehaviors across different species, including aggression and sexual behavior.

87 fish, Gymnotus omarorum, display territorial aggression and social dominance independent of reproduct

88 als have implicated brain reward circuits in aggression and suggest that, in subsets of aggressive in

89 We aim to provide an overview of features of aggression and the relevant neural substrates across spe

90 investigate shared genetic etiology between aggression and these three CU-traits.

91 e various exceptions, a general rule is that aggression and violence increase as one moves closer to

92 t association in healthy subjects with trait aggression and with in vivo prefrontal physiology relate

93 la including feeding initiation, locomotion, aggression, and courtship, among many others.

94 activity in the cerebellar vermis regulates aggression, and further support the importance of the ce

95 ay a crucial role in mediating reproduction, aggression, and parental care across taxa.

96 with a special focus on feeding, courtship, aggression, and postmating behaviors.

97 ry serves as the foundation for the CLimate, Aggression, and Self-control in Humans (CLASH) model of

98 1KT, reproductive output, altered courtship, aggression, and sperm morphology compared to the lineage

99 ocks aggression priming, naturally occurring aggression, and traumatic stress-induced aggression incr

100 motivation and various forms of altruism and aggression are discussed.

101 ncluded that instinctive behaviors including aggression are mediated by hardwired brain circuitries t

102 n male mice, territorial and infant-directed aggression are tightly regulated by the vomeronasal orga

103 the concept of de-escalation of violence and aggression as described within the healthcare literature

104 e communities - is likely to be key to their aggression as it encourages them to dig in and fight alo

105 exploratory activities to limit conspecific aggression as the river dries.

106 amus and then the lateral septum to modulate aggression associated with mate guarding.

107 are assumed to have an emotional basis, e.g. aggression based on frustration.

108 he neural mechanisms that support changes in aggression based on the animal's internal state.

109 that shrub landcover significantly moderates aggression between nests, suggesting nests are more rela

110 First, we measured and compared intraslope aggression between same sex fly pairings collected from

111 Territorial aggression between species, for instance, is widely assu

112 ics model to demonstrate that when both male aggression biases and female mate preferences are formed

113 f agonistic behaviour (display, non-damaging aggression, biting and mutual fighting) is influenced by

114 ychopathy) scores, and with higher levels of aggression (but not rule-breaking).

115 Syrian hamsters show reliable territorial aggression, but after social defeat they exhibit a condi

116 ulation can elicit complex behaviors such as aggression, but how discrete motor components of such be

117 of weaned pigs significantly reduced piglet aggression by 30% and tended to increase feeding behavio

118 Aggression by dominant individuals within groups was the

119 Next, we tested interslope aggression by pairing flies from opposite slopes.

120 ape and human factors that may promote human-aggression by raptorial Black kites Milvus migrans in th

121 Bullying data support the CLASH model of aggression by suggesting that climate may moderate the f

122 In some cases, such as addictive drugs, aggression can be highly rewarding (appetitive) and cont

123 Heightened aggression can be serious concerns for the individual an

124 erventions.SIGNIFICANCE STATEMENT Heightened aggression can have devastating social consequences and

125 s such as extraordinary acts of altruism and aggression can often be best understood as reflecting va

126 ient deprivation in vitro and promoted tumor aggression, chemotherapy resistance, and MEK inhibitor r

127 ghlighting an evolutionarily conserved 'core aggression circuit' composed of four subcortical regions

128 ng a key pathway linking visual input to the aggression circuitry.

129 f sex, delayed sexual satiety, and increased aggression compared to wild-type controls, while TRPM8(-

130 bees from other colonies experienced reduced aggression compared with dsRNA-immunostimulated bees, fa

131 he pharmacological and circuit mechanisms of aggression conditioned place preference and aggression s

132 lopment of social evaluation sensitivity and aggression control in childhood.

133 -two types of neurons with opposing roles in aggression control.

134 Further understanding of the causes of mouse aggression could have a significant impact on a large nu

135 However, male-female aggression decreased in intrusion trials compared to oth

136 ape human disposition-cognitive flexibility, aggression, depression, and reward seeking-and manipulat

137 but a wide disparity exists in the levels of aggression displayed by the 2 sexes.

138 s pre-weaning has been suggested to decrease aggression during later life.

139 tic mechanisms underlying experience-induced aggression escalation, however, are poorly understood.

140 opamine signaling (increase of irritability, aggression, etc.), is the main cause of Captagon addicti

141 One form, appetitive aggression, exhibits symptomatology that mimics that of

142 e function in competition and thus survival, aggression exists widely across animal species, includin

143 dulate behaviors such as olfactory learning, aggression, feeding, and egg laying [4-14].

144 Injurious home-cage aggression (fighting) in mice affects both animal welfar

145 latitudinal gradients of heat, poverty, and aggression finds that heat-induced aggression is mediate

146 differentiate social cue-induced changes in aggression from long-term modulation of this phenotype.

147 g regarding the neural substrates underlying aggression generation, highlighting an evolutionarily co

148 groups exhibited markedly less within-group aggression, higher group cohesion and improved winter su

149 e the best for reducing even minor levels of aggression (i.e. wounding).

150 as long been implicated in the regulation of aggression in a wide variety of animal species.

151 mantine, or intra-PLmPFC memantine increased aggression in AHAs, but only in the absence of alcohol.

152 ohol produced a pathological-like pattern of aggression in AHAs; these mice shifted their bites to mo

153 , are used to treat psychosis, agitation and aggression in Alzheimer's disease.

154 racted with prior alcohol intake to escalate aggression in ANAs.

155 Aggression in borderline personality disorder (BPD) is t

156 uals were less likely to engage in escalated aggression in contest 2.

157 ve identified three cell types that regulate aggression in Drosophila: one type is sexually shared, a

158 ed by increased submission and an absence of aggression in future social interactions.

159 Aggression in group-housed laboratory mice is a serious

160 ChR signaling have also been shown to reduce aggression in human 15q13.3DS.

161 ng-standing debate about the significance of aggression in human nature is misconceived, because both

162 eful in determining the origins of out-group aggression in humans, they so far provide inconclusive e

163 on the prevalence and potential triggers of aggression in laboratory mice.

164 favored increased in-group prosociality over aggression in late human evolution.

165 alpha66 may be a histopathological marker of aggression in LSCC.

166 Aggression in male mice often leads to injury and death,

167 ses aggression and 3) manipulation increases aggression in male mice.

168 ulations of LS neurons and the regulation of aggression in male mice.SIGNIFICANCE STATEMENT Aggressio

169 Early play fighting mimics later aggression in many species, and may, therefore, be expec

170 lar Purkinje cells to evaluate the impact on aggression in mice.

171 ral procedures developed to probe appetitive aggression in mouse models, spanning from Pavlovian to o

172 at, cyp6g1/2, GB53860, inos) associated with aggression in previous studies.

173 Aggression in psychiatric wards is a continuing matter o

174 le outcomes matched levels of heterospecific aggression in the field, significantly exceeding chance

175 Key factors influencing the prevalence of aggression included strain; number of mice per cage; how

176 tic mechanisms underlying experience-induced aggression increase, however, are poorly understood.

177 ing aggression, and traumatic stress-induced aggression increase.

178 idence of territorial defence, with rates of aggression increasing towards the centre of home ranges.

179 sion almost exclusively examines territorial aggression induced by introducing a stimulus mouse into

180 MeApv-BNST pathways contributes to increased aggression induced by traumatic stress, and weakening sy

181 n, smoke exposure, to deprive individuals of aggression-inducing olfactory cues and evaluate whether

182 All grooming and aggression interactions between individuals were recorde

183 Yet, behavioral traits like aggression, intimidation, and coercion, which are associ

184 Aggression involves both sexually monomorphic and dimorp

185 ich comprises diverse symptoms like physical aggression, irritability, and delinquency.

186 using the Child Behavior Checklist: physical aggression, irritability, disobedient behavior, and deli

187 rostructure was not associated with physical aggression, irritability, or disobedient behavior.

188 gression in male mice.SIGNIFICANCE STATEMENT Aggression is a behavior that arose evolutionarily from

189 Aggression is a social behavior essential for securing r

190 Aggression is a universal social behavior important for

191 Heightened aggression is characteristic of multiple neuropsychiatri

192 Pathological aggression is commonly associated with psychiatric and n

193 Aggression is controlled by the olfactory system in many

194 the circuitry modulating appetitive operant aggression is limited.

195 erty, and aggression finds that heat-induced aggression is mediated by poverty and that heat tempers

196 on reward and relapse.SIGNIFICANCE STATEMENT Aggression is often comorbid with neuropsychiatric disea

197 ext showed that in a free-living population, aggression is predicted by allelic imbalance favoring th

198 The intensity of bacterial aggression is such that it can strongly affect communiti

199 One particular brain region involved in aggression is the lateral septum (LS).

200 While both sexes exhibit aggression, its neuronal underpinnings are understudied

201 ical locus for the generation of conspecific aggression, its role is complex, with neurons responding

202 and pelted to assess wounding with the pelt aggression lesion scale (PALS).

203 In the Drosophila model of aggression, males and females fight in same-sex pairings

204 whereas high-frequency stimulation enhances aggression, mimicking attack experience.

205 ults reveal a circuit and synaptic basis for aggression modulation by experience that can be potentia

206 s illustrate a circuit and synaptic basis of aggression modulation by experience, which can be potent

207 , robustness, and adaptation toward external aggressions, most critically to colonizing microorganism

208 nd balancing territorial and infant-directed aggression of male mice through bidirectional activation

209 the glycocalyx strongly correlates with the aggression of many cancer types.

210 late childhood showed stronger decreases in aggression over time.

211 ression and conditioned place preference for aggression-paired contexts.

212 Here we identify two aggression pathways between the posterior ventral segmen

213 In the aggression phase, men with BPD exhibited higher activity

214 advantage of adaptively divergent fruit fly aggression phenotypes from different environments.

215 Inappropriate and pathological aggression plays a leading role in the suffering and dea

216 stinence and subsequent tests for relapse to aggression preference and aggression seeking.

217 l circuitry revealed that the MeApv mediates aggression priming via synaptic connections with the ven

218 an increase in aggressive behavior, known as aggression priming, activates neurons within the posteri

219 naptic transmission in these circuits blocks aggression priming, naturally occurring aggression, and

220 ng a synaptic depression protocol suppresses aggression priming, whereas high-frequency stimulation e

221 Two major types of aggression, proactive and reactive, are associated with

222 cious but enhances and prolongs courtship or aggression promoted by female cues.

223 Shared common aggression-promoting (CAP) neurons mediate aggressive ap

224 homologous cell types, called male-specific aggression-promoting (MAP) neurons in males and fpC1 in

225 ctivity of sensory neurons in response to an aggression-promoting pheromone.

226 ression in brain scored higher on a lifetime aggression questionnaire and show diminished engagement

227 eported that they would respond with greater aggression (r = 0.59).

228 the home range consistently elicited higher aggression, regardless of the groups' history.

229 Little is known about the development of aggression regulation following social feedback during c

230 Aggression regulation was studied using the Social Netwo

231 oss of antidepressant efficacy and increased aggression-related behavior.

232 The mean facility-level prevalence of aggression-related incidents reported across facilities

233 A total of 788 incidents of aggression-related injuries were reported across a sampl

234 ne cage checks and recorded all incidents of aggression-related injuries.

235 osure show a strong, temporary inhibition of aggression relative to bees allowed to perceive normal s

236 ng sensory perception had minimal impacts on aggression-relevant gene expression, althought it did in

237 uits and patterns of activity that influence aggression remain unclear.

238 d that an ecological account of violence and aggression requires consideration of societal and cultur

239 rons, act as a critical modulator of operant aggression reward and aggression seeking.

240 to study cell-type and circuit mechanisms of aggression reward and relapse.SIGNIFICANCE STATEMENT Agg

241 We then tested them for relapse to aggression seeking after forced abstinence or punishment

242 Appetitive aggression and relapse to aggression seeking can be modeled in mice studies using

243 attack subordinate male mice and relapse to aggression seeking during abstinence.

244 ound that aggression self-administration and aggression seeking induced higher Fos expression in NAc

245 them for aggression self-administration and aggression seeking on abstinence Day 1.

246 , operant aggression self-administration and aggression seeking on abstinence Day 1.

247 sts for relapse to aggression preference and aggression seeking.

248 for both aggression self-administration and aggression seeking.

249 decreased aggression self-administration and aggression seeking.

250 eurons in aggression self-administration and aggression seeking.

251 l modulator of operant aggression reward and aggression seeking.

252 R availability and from increased behavioral aggression seen in active smokers.

253 We found that aggression self-administration and aggression seeking in

254 uders (9 d, 12 trials/d) and tested them for aggression self-administration and aggression seeking on

255 bens are recruited for, and control, operant aggression self-administration and aggression seeking on

256 expressing neurons that is critical for both aggression self-administration and aggression seeking.

257 but not Drd2-, expressing neurons decreased aggression self-administration and aggression seeking.

258 eptor (Drd)1- and Drd2-expressing neurons in aggression self-administration and aggression seeking.

259 Here we used a novel mouse model of aggression self-administration and relapse, in combinati

260 aggression conditioned place preference and aggression self-administration, seeking, and relapse, hi

261 tinence or punishment-induced suppression of aggression self-administration.

262 raditional beekeeping approach that inhibits aggression, smoke exposure, to deprive individuals of ag

263 individuals were recorded, and grooming and aggression social networks were created for the 2 days p

264 wnstream lPAG(vGlut2) populations results in aggression-specific deficits.

265 The increased levels of aggression suggest that chiffchaffs, like humans, might

266 of RDL receptors in these neurons increases aggression, suggesting the possibility of a direct cross

267 ulation was studied using the Social Network Aggression Task.

268 ullying was defined as repeated, intentional aggression that is targeted at a person who cannot easil

269 SLOF Social acceptability, the BDHI Indirect Aggression, the BIS Motor Impulsiveness and the STAXI-2

270 asures of their exposure to various types of aggression, their attitudes towards seclusion and restra

271 phai2 regulate male-male and infant-directed aggression through distinct circuit mechanisms.

272 d of different ways, ranging from unprovoked aggression to actively feeding each other.

273 c hawk-dove model of the evolution of animal aggression to consider cases in which a subset of indivi

274 used with castrated male mice and tested for aggression toward female intruders.

275 omotion, demeaning comments toward patients, aggression toward health professionals, and HIPAA violat

276 reover, most males demonstrate avoidance and aggression toward infants, and have little to no involve

277 terpersonal rejection and directing physical aggression toward others.

278 igh-status individuals commit random acts of aggression toward subordinates [23] and even commit infa

279 ach other reproductively, and in some cases, aggression towards heterospecifics may be an adaptive re

280 n (LTD) diminishes, the behavioral effect of aggression training, implying a causal role for potentia

281 studies have established the phenomenon of "aggression training," in which the repeated experience o

282 5% of inbred C57BL/6 mice fail to respond to aggression training.

283 ing, and with a failure to exhibit LTP after aggression training.

284 ral judgements regarding the function of the aggression; trial-and-error; ingrained local custom (esp

285 ) Why is there so much variation in reactive aggression/violence between people living in the same en

286 network position on later skin lesions from aggression was assessed in domestic pigs.

287 Trait aggression was found to positively modulate connectivity

288 Reduced aggression was not a consequence of impaired motor funct

289 and female nervous systems, but the enhanced aggression was seen only in females.

290 Exposure to mild and severe physical aggression was unrelated to nurses' emotions.

291 sive view of the complex genetics underlying aggression, we undertook an integrated, cross-species ap

292 cial behaviors such as mating or territorial aggression; we further hypothesized social interactions,

293 Skin lesions from aggression were counted after a dyadic contest and at 24

294 s partner preference formation and selective aggression, were not disrupted by prenatal VPA exposure.

295 This suggests that reduced aggression when accessing areas within neighbours' home

296 (GMR26E01-Gal4) displaying greatly enhanced aggression when thermoactivated.

297 h each other and experienced lower levels of aggression when within the other's peripheral home range

298 Verbal aggression which appears targeted, demeaning or humiliat

299 n lead to the evolution of severe intergroup aggression, with negative consequences for population me

300 grooming) would increase after conflict, and aggression would decrease, to minimize conflict between