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1 tion and contribute to greater osteolysis in aggressive periodontitis.
2 aque of a patient diagnosed with generalized aggressive periodontitis.
3 ng toxin (Cdt) are associated with localized aggressive periodontitis.
4 periodontal pathogens from individuals with aggressive periodontitis.
5 f African American children with and without aggressive periodontitis.
6 gly associated with development of localized aggressive periodontitis.
7 rated implants in a patient with generalized aggressive periodontitis.
8 , especially those who exhibited generalized aggressive periodontitis.
9 dosis affecting the periodontium could mimic aggressive periodontitis.
10 ome may be at the basis of susceptibility to aggressive periodontitis.
11 ive causative agent of the disease localized aggressive periodontitis.
12 ditary gingival fibromatosis associated with aggressive periodontitis.
13 in patients (>50% smokers) with generalized aggressive periodontitis.
14 dition, the brother also exhibited localized aggressive periodontitis.
15 hway involving caspase-8 are significant for aggressive periodontitis.
16 ly nondiseased subjects and 51 subjects with aggressive periodontitis.
17 be expected to exhibit a more severe form of aggressive periodontitis.
18 in treatment of intrabony defects (IBDs) in aggressive periodontitis.
19 n the treatment of patients with generalized aggressive periodontitis.
20 th the current classification of chronic and aggressive periodontitis.
21 ssociated with susceptibility to chronic and aggressive periodontitis.
22 is the main periodontopathogen of localized aggressive periodontitis.
23 cillin and metronidazole in individuals with aggressive periodontitis.
24 nomycetemcomitans is implicated in localized aggressive periodontitis.
25 IL-17 may play a role in the pathogenesis of aggressive periodontitis.
26 might be detected in sera from patients with aggressive periodontitis.
27 strain, D7S-1, isolated from a patient with aggressive periodontitis.
28 comitans, an organism highly associated with aggressive periodontitis.
29 AgP) and 49 patients with generalized (GAgP) aggressive periodontitis.
31 itans is the causative organism of localized aggressive periodontitis, a rapidly progressing degenera
32 comitans is the etiologic agent of localized aggressive periodontitis, a rapidly progressing oral dis
33 ic periodontitis, plaque-induced gingivitis, aggressive periodontitis, acute gingival and periodontal
34 terleukin (IL)-1 influence the risk for both aggressive periodontitis (AgP) and chronic periodontitis
35 ingival plaque samples from individuals with aggressive periodontitis (AgP) and chronic periodontitis
36 dren, 7 to 19 years of age, with and without aggressive periodontitis (AgP) and compared to healthy u
37 patients with chronic periodontitis (CP) or aggressive periodontitis (AgP) and periodontally healthy
38 givitis (G), chronic periodontitis (CP), and aggressive periodontitis (AgP) based on clinical paramet
39 t planing for the treatment of patients with aggressive periodontitis (AgP) compared to placebo gel.
40 RT) enzyme in chronic periodontitis (CP) and aggressive periodontitis (AgP) compared with healthy ind
42 hile partially inhibiting IL-6 production in aggressive periodontitis (AgP) human gingival fibroblast
43 about the epidemiology and risk factors for aggressive periodontitis (AgP) in Latin American and Bra
47 nocytes, and this is striking in cultures of aggressive periodontitis (AgP) monocytes, where DC numbe
49 vative (EMD) therapy in intrabony defects in aggressive periodontitis (AgP) patients and compare them
52 l sample of the severe early-onset phenotype aggressive periodontitis (AgP) with the Illumina Immunoc
53 th generalized chronic periodontitis (CP) or aggressive periodontitis (AgP), and periodontally health
54 amples obtained from patients diagnosed with aggressive periodontitis (AgP), chronic periodontitis (C
65 ng a German and Dutch case-control sample of aggressive periodontitis (AgP, 896 cases, 7,104 controls
68 AS data from a German case-control sample of aggressive periodontitis (AgP; 651 cases, 4,001 controls
69 d 471 patients with periodontitis (including aggressive periodontitis [AgP], and chronic periodontiti
70 of patients with gingivitis, the group with aggressive periodontitis [AgP], and the group with chron
71 terium is strongly associated with localized aggressive periodontitis and adult periodontitis and is
73 llected 66 GCF samples from 15 patients with aggressive periodontitis and examined both the GCF sampl
74 comitans is a pathogen that causes localized aggressive periodontitis and extraoral infections includ
75 llus that has been associated with localized aggressive periodontitis and infections of the heart, br
76 thogens commonly associated with chronic and aggressive periodontitis and opportunistic pathogens may
78 rom 18 parents with a history of generalized aggressive periodontitis and their children (6-12 years
79 h naturally occurring chronic periodontitis, aggressive periodontitis, and feline chronic gingivostom
80 The study included 130 individuals: 40 with aggressive periodontitis (AP group), 40 with chronic per
81 e investigated whether leukocyte activity in aggressive periodontitis (AP) is increased compared with
82 s diagnosed with chronic periodontitis (CP), aggressive periodontitis (AP), and feline chronic gingiv
83 rs of vascular inflammation in patients with aggressive periodontitis are associated with elevated le
85 ar-old female was diagnosed with generalized aggressive periodontitis based on clinical and radiograp
86 was diagnosed with gingival fibromatosis and aggressive periodontitis based on the clinical and radio
89 isits with a primary diagnosis for acute and aggressive periodontitis, covered by Medicare insurance,
90 s (63 with chronic periodontitis and 27 with aggressive periodontitis) each contributed at least two
91 A total of 87 individuals, 20 generalized aggressive periodontitis (G-AgP), 20 chronic periodontit
94 edentulous patients treated for generalized aggressive periodontitis (GAgP) and in periodontally hea
95 th chronic periodontitis (CP) or generalized aggressive periodontitis (GAgP) and their association wi
96 1 (TIMP-1) gene polymorphisms in generalized aggressive periodontitis (GAgP) and to assess the effect
97 lay a role in the development of generalized aggressive periodontitis (GAgP) are investigated using g
98 f this study is to study whether generalized aggressive periodontitis (GAgP) has similar survival rat
99 h chronic periodontitis (CP) and generalized aggressive periodontitis (GAgP) positive for anti-CL (16
100 h chronic periodontitis (CP) and generalized aggressive periodontitis (GAgP) to test the utility of G
102 ored the molecular signatures of generalized aggressive periodontitis (GAgP) using gingival tissues t
103 ntitis (CP), 14 individuals with generalized aggressive periodontitis (GAgP), 18 individuals with gin
104 onic periodontitis (CP), 20 with generalized aggressive periodontitis (GAgP), 20 with gingivitis, and
105 (GT) samples of 12 patients with generalized aggressive periodontitis (GAgP), 6 patients with localiz
106 odontitis (CP), 12 patients with generalized aggressive periodontitis (GAgP), 6 patients with localiz
107 is a primary etiologic agent of generalized aggressive periodontitis (GAgP), and gingipains, a group
108 es in patients with a history of generalized aggressive periodontitis (GAgP), chronic periodontitis (
109 grouped into control group (CG), generalized aggressive periodontitis (GAgP), or chronic periodontiti
114 ic periodontitis (GChP; n = 30), generalized aggressive periodontitis (GAgP; n = 30), and periodontal
115 Group Healthy), 20 patients with generalized aggressive periodontitis (Group AgP), and 20 patients wi
116 seased subjects and 63% of the subjects with aggressive periodontitis harbored multiple clones of E.
119 en proposed for the treatment of generalized aggressive periodontitis; however, its effectiveness and
120 ontrols (IL-6 -174 & VDR -1056); chronic and aggressive periodontitis (IL-1A -889); and periodontitis
123 tors may prevent or reduce the prevalence of aggressive periodontitis in this and similar populations
125 test, P < 0.05) in sites from patients with aggressive periodontitis, in sites with pocket depths of
126 c infections, and in patients with localized aggressive periodontitis, its presence is now observed a
127 y responses to infecting agents in localized aggressive periodontitis (LAgP) and weak responses in ge
128 earch indicated that patients with localized aggressive periodontitis (LAgP) had minimal proximal dec
129 in African-American children with localized aggressive periodontitis (LAgP) have been reported previ
130 used to predict susceptibility to localized aggressive periodontitis (LAgP) in an African-American p
135 that in healthy controls (NP) and localized aggressive periodontitis (LAgP) patients (6.8% and 3.2%)
136 globulin G2 (IgG2) are elevated in localized aggressive periodontitis (LAgP) patients, and secretory
137 onocytes in cultures prepared from localized aggressive periodontitis (LagP) patients, and these pati
138 ukocytes (PMNs) from subjects with localized aggressive periodontitis (LAgP) present multiple functio
140 l study of students susceptible to localized aggressive periodontitis (LAgP) was conducted to evaluat
141 iodontitis (GAgP), 6 patients with localized aggressive periodontitis (LAgP), 10 patients with chroni
142 iodontitis (GAgP), 6 patients with localized aggressive periodontitis (LAgP), 6 patients with gingivi
143 issues were probed with serum from localized aggressive periodontitis (LAgP), chronic periodontitis (
144 eutrophils (PMNs) of patients with localized aggressive periodontitis (LAgP), suggesting a genetic ba
147 cter actinomycetemcomitans-induced localized aggressive periodontitis (LAP) in African-American adole
149 ls from individuals diagnosed with localized aggressive periodontitis (LAP) present an in vivo phenot
150 in African American subjects with localized aggressive periodontitis (LAP) who had proximal bone los
151 ne severe form of periodontitis is localized aggressive periodontitis (LAP), a condition to which ind
152 ired PMN chemotaxis is observed in localized aggressive periodontitis (LAP), a familial disorder char
153 ity and is the causative agent for localized aggressive periodontitis (LAP), an aggressive form of pe
159 tans is frequently associated with localized aggressive periodontitis (LAP); however, longitudinal co
161 tin were significantly higher in generalized aggressive periodontitis patients who had elevated anti-
163 s that periodontal pathogens associated with aggressive periodontitis persist in extracrevicular loca
167 ter actinomycetemcomitans is associated with aggressive periodontitis resulting in premature tooth lo
169 GCF were higher in persons with chronic and aggressive periodontitis than in those with gingivitis o
170 GCF were higher in persons with chronic and aggressive periodontitis than in those with gingivitis,
171 al, biofilm-mediated disease are chronic and aggressive periodontitis, the latter being characterized
172 m 45 young adults diagnosed with generalized aggressive periodontitis to study Fc receptors, formyl p
173 of children and young adults with localized aggressive periodontitis treatment (LAgP) affecting prim
174 w of the inflammatory nature and severity of aggressive periodontitis, we hypothesized that IL-17 mig
175 ed by nondiseased subjects and subjects with aggressive periodontitis were 1.3 and 3.0, respectively.
176 ents who had a primary diagnosis of acute or aggressive periodontitis were associated with significan
177 Thirteen patients (36.9+/-7.4 years) with aggressive periodontitis were monitored before and up to
178 wo nondiseased subjects and 11 subjects with aggressive periodontitis were subsequently reexamined af
179 o present a 9-year-old female with localized aggressive periodontitis who had a history of type 1 DM
180 ents a patient with a history of generalized aggressive periodontitis who has been maintained with th
181 addresses antibiotic use during treatment of aggressive periodontitis with emphasis on juvenile disea