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1 who exhibited airway wall thickening without airway narrowing.
2 erapeutic target in CF and other diseases of airway narrowing.
3 ; in allergic mice, gVPLA2 caused persistent airway narrowing.
4 nges in enhanced pause (Penh) as an index of airway narrowing.
5 MCh challenge are associated with conducting airway narrowing.
6 ilies also suggests a propensity for dynamic airway narrowing.
7 e hyperreactive to stimuli causing extensive airway narrowing.
8 nflammatory disease associated with episodic airway narrowing.
9 ic disease characterized by life-threatening airway narrowing.
10 n of integrin alpha9beta1 increased in vitro airway narrowing and airway smooth muscle contraction in
11                              They both cause airway narrowing and are increasing in incidence through
12 s been linked to complex behaviour in sudden airway narrowing and avalanche-like reopening.
13 ctively structural abnormalities involved in airway narrowing and bronchial reactivity, particularly
14 ubjects, asthmatics have (1) more collateral airway narrowing and closure and lower segmental complia
15 truct, to our knowledge, a new model wherein airway narrowing and closure dynamics are modulated by c
16 ides differential information on predominant airway narrowing and loss in COPD.
17 eveloped a method to simultaneously quantify airway narrowing and muscle shortening using 2-photon mi
18 s via the regulation of airway inflammation, airway narrowing and remodelling.
19 n obtaining objective evidence of reversible airways narrowing and inflammation.
20 cked fully gVPLA2-induced cell migration and airway narrowing as marked by reduction of migrating leu
21  simulate the impact of different degrees of airway narrowing at different levels of the airway tree
22 airway dilatation) at low airway volumes and airway narrowing at high airway volumes (P < 0.05).
23 , co-activation produced volume compression (airway narrowing) at large airway volumes (P < 0.05), bu
24                                   To confirm airway narrowing by MCh in this setting and to determine
25 of nonspecific agonists and that the induced airway narrowing can be reversed by the inhalation of a
26 duals develop this condition, the associated airway narrowing can materially interfere with performan
27 steinyl leukotrienes are potent mediators of airway narrowing derived from the lipoxygenation of arac
28                           The maintenance of airway narrowing despite MCh clearance in humans is attr
29                    With placebo, significant airway narrowing developed at all times (mean [+/-SE] de
30 nge during sleep, related to transient upper airway narrowing disrupting ventilation, and causing oxy
31 yngeal walls play an important role in upper airway narrowing during sleep in normal subjects.
32 arrowing in the small airways and that small airway narrowing has a marked impact on both asthma cont
33 eta6 integrin are protected from exaggerated airway narrowing in a model of allergic asthma.
34 f beta2AR-selective PAMs in the treatment of airway narrowing in asthma and other obstructive respira
35 s naturally to the suggestion that excessive airway narrowing in asthma may be associated with the de
36 d oscillation R5 - R20 as a measure of small airway narrowing in asthma, and to investigate the role
37 cluding cough, and are a potent stimulus for airway narrowing in asthmatic patients, but the mechanis
38  trapping, with a contribution from proximal airway narrowing in asthmatic patients.
39  gVPLA2 mutant with reduced activity, caused airway narrowing in immune-sensitized mice.
40 on of goblet cells may contribute to chronic airway narrowing in moderate asthma.
41                  Neuronal VAChT staining and airway narrowing in response to electrical field stimula
42         Asthma is characterized by increased airway narrowing in response to nonspecific stimuli.
43 id peroxidation is associated with pulmonary airway narrowing in the general population.
44                                              Airway narrowing in the RP region was associated with a
45 sthma, the mechanisms by which they regulate airway narrowing in vivo remain to be elucidated.
46                                              Airway narrowing is maintained for a prolonged period af
47      Compared with subjects with predominant airway narrowing (n = 2,914; 66.3%), those with predomin
48 cells may contribute at least in part to the airway narrowing observed in patients with asthma.
49 l area, particularly during inspiration; (2) airway narrowing occurred during inspiration without evi
50 els were used to predict the impact on small airway narrowing of type-2 targeting biologics using poo
51 rt (n = 177) to simulate the impact of small airway narrowing on asthma control and quality of life.
52 ape probability, but a significant effect of airway narrowing on regional deposition.
53 symptoms and the demonstration of reversible airways narrowing on lung function testing, which is dif
54 nd (2) after challenge, increased collateral airway narrowing or closure without a change in complian
55 ation is not correlated with the severity of airway narrowing or the severity of bronchial hyperrespo
56 thma, and to investigate the role that small airway narrowing plays in asthma.Methods: Patient-based
57 rs, we categorized subjects into predominant airway narrowing [positive (SA/V) more than 0] and predo
58 rrent knowledge about ASM and its effects on airway narrowing, remodeling, and inflammation in asthma
59 pt of airways hyperresponsiveness and distal airways narrowing, respectively.
60 e pooled trials equated to a predicted small airway narrowing reversal of approximately 40%.Conclusio
61                                              Airway narrowing showed no predilection for particular a
62 ed bronchoconstriction (EIB) describes acute airway narrowing that occurs as a result of exercise.
63 ion has been postulated to contribute to the airway narrowing that occurs following exercise or hyper
64                             With progressive airway narrowing, the ratio of the airway luminal surfac
65 ary disease and asthma by 1) contributing to airway narrowing through hyperplasia and hypertrophy and
66  hyperventilation (ISH) is believed to cause airway narrowing through noninflammatory mechanisms.
67 ogically and by differential cell count, and airway narrowing was measured by calibrated micrometry.
68  these changes include sputum production and airway narrowing, which lead to asthma exacerbations.
69  airways hyperresponsiveness; and (3) distal airway narrowing, which may be associated with histopath
70  phase reaction (EPR), that leads to maximal airway narrowing within 15-30 min, followed by a recover
71  inhibition of a2B1 mitigated IL-13-enhanced airway narrowing without altering muscle shortening by i
72       Because loss of T(H)17 cells inhibited airway narrowing without any obvious effects on airway i