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1 spontaneous breathing or continuous positive airway pressure.
2 esity tolerate hemodynamically LRM with high airway pressure.
3 ay pressure, but can be controlled under low airway pressure.
4 less than 34 weeks under continuous positive airway pressure.
5 essure support on top of expiratory positive airway pressure.
6 ng CT scans were obtained at 5 and 45 cm H2O airway pressure.
7 irst hours of life under continuous positive airway pressure.
8 /- 7% (P < 0.0001) going from 5 to 45 cm H2O airway pressure.
9 ts who are intolerant to continuous positive airway pressure.
10 roduction of less invasive forms of positive airway pressure.
11 tomography measurements when increasing mean airway pressure.
12 reduced extravascular lung water and plateau airway pressure.
13 y small tidal volumes cycling at a high mean airway pressure.
14 -invasive ventilation or continuous positive airway pressure.
15 in the duration of nasal continuous positive airway pressure.
16 xia or respiratory acidosis and high plateau airway pressures.
17 ssessed at low (5 cmH2O) and high (45 cmH2O) airway pressures.
18 litude (50, 60, 70, 80, and 90 cm H2O), mean airway pressure (20, 30, and 40 cm H2O), test lung compl
19 kg predicted body weight, p < .001), in peak airway pressure (31-25 cm H2O, p < .001), and in the per
20 or the fraction of inspired oxygen 0.25/mean airway pressure 4 definition (i.e., increase in minimum
21 ng the fraction of inspired oxygen 0.25/mean airway pressure 4 thresholds to identify pediatric venti
22 derwent lung recruitment continuous positive airway pressure 40 cm H2O for 40 secs to normalize volum
23 e; the fraction of inspired oxygen 0.30/mean airway pressure 7 definition yielded ventilator-associat
24 ing the 75th to the 25th percentile for mean airway pressure; 95% CI, 1.10-1.74) after adjusting for
25 s, a group that has poor continuous positive airway pressure adherence and difficulty in achieving we
26 release ventilation of 0%, biphasic positive airway pressure/airway pressure release ventilation more
27                            Biphasic positive airway pressure/airway pressure release ventilation more
28 , and damage compared with biphasic positive airway pressure/airway pressure release ventilation more
29              Compared with biphasic positive airway pressure/airway pressure release ventilation of 0
30                            Biphasic positive airway pressure/airway pressure release ventilation was
31 distress syndrome in pigs, biphasic positive airway pressure/airway pressure release ventilation with
32 elease ventilation, 0%; 2) biphasic positive airway pressure/airway pressure release ventilation, > 0
33 e ventilation, > 0-30%; 3) biphasic positive airway pressure/airway pressure release ventilation, > 3
34 tilation, > 30-60%, and 4) biphasic positive airway pressure/airway pressure release ventilation, > 6
35  per group, 6 hr each): 1) biphasic positive airway pressure/airway pressure release ventilation, 0%;
36 monary outcomes than the continuous positive airway pressure alone group.
37 ethod (200 mg/kg), or 3) continuous positive airway pressure alone.
38            Driving pressures calculated from airway pressures alone (plateau airway pressure--positiv
39 ere allocated to receive continuous positive airway pressure and 118 to receive standard care.
40         The old modes of continuous positive airway pressure and bilevel positive airway pressure hav
41 itor them properly, especially understanding airway pressure and flow graphics.
42                                 ZVV segments airway pressure and flow recordings into individual brea
43 S including preinduction continuous positive airway pressure and postextubation NRS for high-risk ind
44 scle pressure can be estimated from the peak airway pressure and the percentage of assistance (gain).
45 ssure ventilation (i.e., continuous positive airway pressure and/or intubation).
46 ntilator volumes lower in patients with high airway pressures and poor compliance (8.4-10.6 mL/kg int
47 tects the cardiovascular system against high airway pressures and prevents lung overdistension.
48 f high frequency jet ventilation in reducing airway pressures and, perhaps, barotraumas are cited.
49 spiratory distress syndrome in whom airflow, airway pressure, and esophageal pressure were recorded d
50 ntilation treatment with continuous positive airway pressure, and other potential ocular and systemic
51 the managing clinician, the ventilator flow, airway pressure, and volume/time waveforms were continuo
52 pwise recruitment maneuver without sustained airway pressure appeared to associate with less biologic
53               Interventions such as positive airway pressure are recommended for those with excessive
54  ten pigs with acute lung injury at multiple airway pressures, as well as a theoretical model relatin
55      Non-invasive bubble continuous positive airway pressure (bCPAP) is considered a safe ventilation
56 ve ventilation delivered as bilevel positive airway pressure (BiPAP) is often used to avoid reintubat
57  complex and costly than continuous positive airway pressure but might be advantageous because it pro
58 hepatic vein cannot be controlled under high airway pressure, but can be controlled under low airway
59  fraction of inspired oxygen by 0.25 or mean airway pressure by 4), rates ranged from 2.9 to 3.2 per
60 0.20, 0.25, and 0.30) and daily minimum mean airway pressure (by 4, 5, 6, and 7 cm H2O).
61                        These include rate of airway pressure change (influenced by flow amplitude, in
62 cm H2O) resulted in an increase in change in airway pressure, change in pleural pressure, change in p
63 .0001) after 3 months of continuous positive airway pressure, compared with standard care alone (7.5
64 .0001) after 3 months of continuous positive airway pressure, compared with standard care alone (9.2
65                          Continuous positive airway pressure (CPAP) and mandibular advancement device
66  and the clinical use of continuous positive airway pressure (CPAP) and positive end-expiratory press
67  an alternative to nasal continuous positive airway pressure (CPAP) as a means of respiratory support
68                 Although continuous positive airway pressure (CPAP) can mitigate these risks, effecti
69 Meta-analysis found that continuous positive airway pressure (CPAP) compared with sham was significan
70 ms Questionnaire (SASQ), continuous positive airway pressure (CPAP) compliance, and physician decisio
71 and economic benefits of continuous positive airway pressure (CPAP) for moderate to severe obstructiv
72 lar alternative to nasal continuous positive airway pressure (CPAP) for noninvasive respiratory suppo
73 ether OSA treatment with continuous positive airway pressure (CPAP) has metabolic benefits.
74 rapy delivered by bubble continuous positive airway pressure (CPAP) improved outcomes compared with s
75                          Continuous positive airway pressure (CPAP) in asthma patients with concomita
76                          Continuous positive airway pressure (CPAP) is considered the treatment of ch
77                          Continuous positive airway pressure (CPAP) is the first-line treatment for p
78                          Continuous positive airway pressure (CPAP) is the treatment of choice in pat
79 'drops' from therapeutic continuous positive airway pressure (CPAP) levels.
80 ients who cannot tolerate continous positive airway pressure (CPAP) machines or intraoral devices.
81 gy study performed using continuous positive airway pressure (CPAP) manipulations indicated that the
82  Adherence to short-term continuous positive airway pressure (CPAP) may predict long-term use.
83  determine the effect of continuous positive airway pressure (CPAP) of patients with OSA on renal hem
84 ive ventilation (NIV) or continuous positive airway pressure (CPAP) on cardiac structure and function
85 ence about the effect of continuous positive airway pressure (CPAP) on glycemic control in patients w
86 n premature infants with continuous positive airway pressure (CPAP) preserves surfactant and keeps th
87 irway pressure (PAP), 3) continuous positive airway pressure (CPAP) rather than noninvasive ventilati
88                          Continuous positive airway pressure (CPAP) reduces blood pressure, but adher
89                          Continuous positive airway pressure (CPAP) therapy is the most common treatm
90 to examine the effect of continuous positive airway pressure (CPAP) therapy on atrial fibrillation (A
91           The effects of continuous positive airway pressure (CPAP) therapy on endothelial function a
92 rm studies indicate that continuous positive airway pressure (CPAP) therapy reduces blood pressure in
93 ents had been prescribed continuous positive airway pressure (CPAP) therapy to manage OSA and were id
94                Prolonged continuous positive airway pressure (CPAP) therapy with supplemental oxygen
95 ood pressure response to continuous positive airway pressure (CPAP) treatment is highly variable and
96 ence about the effect of continuous positive airway pressure (CPAP) treatment on blood pressure in pa
97 n women, and the role of continuous positive airway pressure (CPAP) treatment on this association.
98 apeutic decision-making, continuous positive airway pressure (CPAP) treatment or a healthy habit asse
99 A who were intolerant to continuous positive airway pressure (CPAP) treatment, submitted to DISE betw
100 ve ventilation (NIV) and continuous positive airway pressure (CPAP) use in patients with OHS, informa
101 pressure associated with continuous positive airway pressure (CPAP) use, with smaller or uncontrolled
102  of early treatment with continuous positive airway pressure (CPAP) versus early surfactant treatment
103 similar to that of nasal continuous positive airway pressure (CPAP) when used as postextubation suppo
104 am) on morning BP, after continuous positive airway pressure (CPAP) withdrawal in patients with moder
105 We aimed to determine if continuous positive airway pressure (CPAP), a form of non-invasive ventilati
106 o receive treatment with continuous positive airway pressure (CPAP), a weight-loss intervention, or C
107 t for symptomatic OSA is continuous positive airway pressure (CPAP), but its value in patients withou
108 rough the application of continuous positive airway pressure (CPAP), which remains a primary therapeu
109 tilation (IPPV) or nasal continuous positive airway pressure (CPAP)--at the time of the first use of
110  the standard therapy of continuous positive airway pressure (CPAP).
111 reated and adherent with continuous positive airway pressure (CPAP).
112 Noninvasive ventilation (continuous positive airway pressure [CPAP] or noninvasive intermittent posit
113                                    Under low airway pressure, CVP did not increase or often decreased
114                                   Under high airway pressure, CVP was persistently higher than pneumo
115 and regression analyses were performed among airway pressure, CVP, and pneumoperitoneum pressure.
116                               Increasing the airway pressures decreased but did not abolish the exten
117 tment to total lung capacity, using stepwise airway pressure decrements.
118 n predicting 90-day mortality, baseline mean airway pressure demonstrated similar discriminative abil
119             We aimed to thoroughly determine airway pressure distribution, how this is influenced by
120 ume loop [in Joules]) and stress relaxation (airway pressure drop during an end-inspiratory pause [in
121    Pmusc/Eadi index was also calculated from airway pressure drop during end-expiratory occlusions.
122 ic simulations determined flow, velocity and airway pressure drops.
123 tubation risk factors included lower maximum airway pressure during airway occlusion (aPiMax) preextu
124 vation of the Pmusc/Eadi index from Eadi and airway pressure during an expiratory occlusion enables a
125 ntaneous breathing trials, and the change in airway pressure during an occlusion maneuver to measure
126                                        Flow, airway pressure, esophageal pressures, and peak electric
127 glets to investigate the continuous positive airway pressure failure rate with nebulized poractant al
128 for 2 hours (phase 2) or continuous positive airway pressure for 2 hours (phase 3), and then crossove
129 ve treatment of OSA with continuous positive airway pressure for 3 months significantly reduced sever
130 ressure of 30 cm H2O: 1) continuous positive airway pressure for 30 seconds (CPAP-30); 2) stepwise ai
131 alternative treatment to continuous positive airway pressure for patients with obstructive sleep apne
132 spiratory muscle strength (maximal change in airway pressure generated during airway occlusion [PiMax
133 t compensation for more negative inspiratory airway pressures generated during heavy exercise occurs
134 re 1.63 (SD 3.74) in the continuous positive airway pressure group and 1.44 (3.07) in the non-invasiv
135 olic blood pressure occurred in the positive airway pressure group than in the usual care group (-3.5
136 ion group and 107 in the continuous positive airway pressure group were included in the analysis.
137 ed (one allocated to the continuous positive airway pressure group) and all were unrelated to the int
138 years (4.36-6.32) in the continuous positive airway pressure group, and 5.55 years (4.53-6.50) in the
139 ion group and 115 to the continuous positive airway pressure group, of which 97 patients in the non-i
140 rthermore, unlike in the continuous positive airway pressure group, there were no cases of respirator
141  patients who adhered to continuous positive airway pressure &gt;/=4 hours daily.
142 , changes in compliance, and changes in mean airway pressure had little effect.
143 asal cannula therapy and continuous positive airway pressure had similar efficacy (RR, 1.11; 95% CI,
144               Given that continuous positive airway pressure has lower complexity and cost, continuou
145 ntilation treatment with continuous positive airway pressure have an increased risk of second eye inv
146 ositive airway pressure and bilevel positive airway pressure have been actively introduced in clinica
147 invasive ventilation and continuous positive airway pressure have similar long-term effectiveness.
148 s ventilatory consequences include increased airway pressures, hypercarbia, and decreased pulmonary c
149 months of treatment with continuous positive airway pressure improved the quality of life in patients
150 r whether treatment with continuous positive airway pressure improves daytime function in these patie
151 ructive sleep apnea with continuous positive airway pressure improves not only patient-reported outco
152  the main alternative to continuous positive airway pressure, improves endothelial function in patien
153 ume was decreased by lowering end-expiratory airway pressure in a stepwise manner.
154  improved after starting continuous positive airway pressure in asthmatics with moderate to severe ob
155 al recruitment, but decreased at the maximum airway pressure in nine patients, indicative of a reduct
156  analysis of the ISAACC (Continuous Positive Airway Pressure in Patients with ACS and OSA) study, inc
157 linical effectiveness of continuous positive airway pressure in patients with mild obstructive sleep
158 ntilation treatment with continuous positive airway pressure in patients with severe OSAS increased t
159 tilation was superior to continuous positive airway pressure in preventing extubation failure (RR, 0.
160 lation (Venturi mask and continuous positive airway pressure) in the majority of cases.
161  similar to that of CPAP-30; and 3) stepwise airway pressure increase (5 cm H2O/step, 5 s at each ste
162 essure for 30 seconds (CPAP-30); 2) stepwise airway pressure increase (5 cm H2O/step, 8.5 s at each s
163 ve intra-abdominal pressure 5 mm Hg, plateau airway pressure increased linearly by ~ 50% of the appli
164 ificantly improved at 24 hours, as were peak airway pressures, intrinsic positive end-expiratory pres
165                          Continuous positive airway pressure is generally first-line therapy despite
166                                 Because mean airway pressure is readily available on all mechanically
167                            Conclusions: High airway pressure is required to recruit lung atelectasis
168                          Continuous positive airway pressure is the treatment of choice, with adheren
169 proved allograft lung function based on peak airway pressure, less infiltrates/consolidation on micro
170 +10, +15) in a randomized order, with a mean airway pressure level determined by adding 5, 10, or 15
171 id not improve oxygenation whatever the mean airway pressure level.
172 ies that use lower end-inspiratory (plateau) airway pressures, lower tidal volumes (VT), and higher p
173                      Treatment with positive airway pressure lowers blood pressure, especially in pat
174 ter; both plans included continuous positive airway pressure, mandibular advancement splints, or cons
175 bjects were treated with continuous positive airway pressure (mean duration of 26 weeks), after which
176 wer complexity and cost, continuous positive airway pressure might be the preferred first-line positi
177 ients were switched to the bi-level positive airway pressure mode with 1 second of 24 cm H2O high pre
178 cycles with supporting a continuous positive airway pressure model.
179 auto-titrating CPAP (n = 122) or no positive airway pressure (n = 122).
180 sal cannula >2 L/min or noninvasive positive airway pressure (n = 617); and grade 3, invasive mechani
181 ort noninferior to nasal continuous positive airway pressure (nCPAP) or bilevel nCPAP (BiPAP) as a pr
182  is optimal: noninvasive continuous positive airway pressure (NCPAP) or intubate-surfactant-extubate
183                                          The airway pressure needed to achieve comparable dependent l
184 fidence interval, 0.96-0.99; p = .017), peak airway pressure (odds ratio per 5-cm H2O increase: 1.11;
185 a plateau pressure of >30 cm H2O, and a peak airway pressure of >35 cm H2O.
186 ticipants who died compared to a median mean airway pressure of 12 cm H2O (interquartile range, 10-14
187 ontrol) lung was kept on continuous positive airway pressure of 20 cm H2O, and CO2 was partially remo
188 ilator settings were an inspiratory positive airway pressure of 24 (IQR, 22-26) cm H2O, an expiratory
189 t recruitment maneuvers, targeted to maximal airway pressure of 30 cm H2O: 1) continuous positive air
190  (IQR, 22-26) cm H2O, an expiratory positive airway pressure of 4 (IQR, 4-5) cm H2O, and a backup rat
191  airway occlusion and on continuous positive airway pressure of 5 and pressure support of 10 above po
192 per kg of predicted bodyweight and a plateau airway pressure of less than 30 cm H2O.
193 cording to the protocol of Webb and Tierney (airway pressures of 14/0, 30/0, 45/10, 45/0 cm H2O).
194 raphy (CT) during breath-holding sessions at airway pressures of 5, 15, and 45 cm H2O and Cine-CTs on
195 the beneficial effect of continuous positive airway pressure on quality of life, mood, and work absen
196  extubation technique or continuous positive airway pressure only.
197                            Bi-level positive airway pressure or adaptive servo-ventilation can be use
198 rst 72 hours (the use of continuous positive airway pressure or high-flow nasal cannula for at least
199 is commonly treated with continuous positive airway pressure or non-invasive ventilation during sleep
200 high-flow nasal cannula, continuous positive airway pressure, or bilevel noninvasive ventilation in t
201 uired a mask, continuous or bilevel positive airway pressure, or mechanical ventilation were classifi
202 s include weight loss and exercise, positive airway pressure, oral appliances that hold the jaw forwa
203  respiratory system dynamic compliance, mean airway pressure, PaO2/FiO2 ratio, and oxygenation index
204 mbulatory patients with OHS receive positive airway pressure (PAP), 3) continuous positive airway pre
205 toms, adherence to using continuous positive airway pressure, patient satisfaction, and health care c
206                       The difference between airway pressure (Paw) and Pes is a valid estimate of tra
207 th at least 8 cm H2O positive end-expiratory airway pressure (PEEP), and bilateral infiltrates consis
208                     Central venous pressure, airway pressure, pericardial pressure, and pleural press
209 ntilation as a 35 cm H2O continuous positive airway pressure period lasting 3-4 seconds at different
210  ratio, tidal volume, respiratory rate, mean airway pressure, plateau pressure, and hemodynamic varia
211 0.3 +/- 0.1, and 0.3 +/- 0.1 mm Hg/mL/kg for airway pressure, pleural pressure, pericardial pressure,
212 1) to either 3 months of continuous positive airway pressure plus standard care (sleep counselling),
213 uscle pressure, estimated in cm H2O as (peak airway pressure-positive end-expiratory pressure)x[(100-
214 culated from airway pressures alone (plateau airway pressure--positive end-expiratory pressure) did n
215 icted body weight with corresponding plateau airway pressures (PPlat) less than or equal to 30 cm H2O
216 spontaneous breathing or continuous positive airway pressure; pressure support ventilation 5-12 cm H2
217  show that important qualitative features of airway pressure-radius hysteresis loops are highly depen
218 ed by adding 5, 10, or 15 cm H2O to the mean airway pressure recorded during conventional mechanical
219 o a "reference" P0.1 (P0.1ref) measured from airway pressure recording during an occlusion.Methods: A
220                          Continuous positive airway pressure reduced extubation failure in comparison
221 ventional mechanical ventilation (n = 15) or airway pressure release ventilation (n = 12) for 48 hrs
222                                              Airway pressure release ventilation also significantly r
223                   We compared the effects of airway pressure release ventilation and conventional mec
224         Mean airway pressures were higher in airway pressure release ventilation animals between 6 an
225              Respiratory rates were lower in airway pressure release ventilation at 24, 42, and 48 hr
226                                              Airway pressure release ventilation has been gaining a r
227                                  The role of airway pressure release ventilation in the management of
228 ation for acute hypoxic respiratory failure, airway pressure release ventilation is associated with a
229  release ventilation was conducted using the airway pressure release ventilation mode with an inspira
230 ween conventional mechanical ventilation and airway pressure release ventilation pigs.
231                 PaO2/Fio2 ratio was lower in airway pressure release ventilation vs. conventional mec
232                                              Airway pressure release ventilation was associated with
233                                              Airway pressure release ventilation was set with a P(Hig
234                                       During airway pressure release ventilation with low tidal volum
235 mode to prevent intubation and then go on to airway pressure release ventilation, high-frequency osci
236  caused by severe smoke inhalation in swine, airway pressure release ventilation-treated animals deve
237 ween conventional mechanical ventilation and airway pressure release ventilation.
238                          Intraoperative peak airway pressures remained within normal limits for all p
239 ation index (p < .05); and 1/PaO2/Fio2, mean airway pressure, serum pH, and Paco2 were associated wit
240  volume were simultaneously recorded at each airway pressure step.
241 eight, positive end-expiratory pressure, and airway pressure), subjects with an overweight fluid-bala
242 Eadi index obtained during an occlusion from airway pressure swing was tightly correlated with that d
243 ic bifurcations also exhibit higher proximal airway pressures than symmetric ones, but the improvemen
244 ife support, when combined with lower Vt and airway pressures than the current standard of care, may
245  the chest wall allows for calculation of an airway pressure that would place the lung at a desired v
246                           However, under low airway pressure, the risk of pulmonary gas embolism incr
247 icant in those who were adherent to positive airway pressure therapy (-4.4 mm Hg vs. -1.6 mm Hg; P =
248 re previously prescribed continuous positive airway pressure therapy (CPAP) but were dissatisfied wit
249 cant reduction in sleepiness in the positive airway pressure therapy group (P < 0.0001).
250 tal component summary scores in the positive airway pressure therapy group.
251 d at baseline and after 3 months of positive airway pressure therapy in a heterogeneous group of 52 c
252 tiation of autotitrating continuous positive airway pressure therapy in the home has greatly reduced
253                                     Positive airway pressure therapy is frequently used to treat obst
254      This trial showed no effect of positive airway pressure therapy on glycemic control in patients
255 ex of 15 or more events per hour to positive airway pressure therapy or to usual care.
256         In OSA patients, continuous positive airway pressure therapy resulted in reduction of the pos
257    However, it is not known whether positive airway pressure therapy results in improvements in the n
258                                     Positive airway pressure therapy was associated with significant
259                We hypothesized that positive airway pressure therapy would be associated with improve
260 ction in children after 3 months of positive airway pressure therapy, even in developmentally delayed
261         After 4 weeks of continuous positive airway pressure therapy, flow-mediated dilation and expr
262  expenditure (effort), arterial blood gases, airway pressure, tidal volume and its coefficient of var
263                              Analysis of the airway pressure-time curve (stress index) has been propo
264  and inflammation were assessed based on the airway pressure-time index, bronchoalveolar lavage (BAL)
265 nt of confirmed OSA with continuous positive airway pressure to reduce driving risk, rather than no t
266 t medications or empiric continuous positive airway pressure to reduce driving risk.
267 d by repeatedly lowering continuous positive airway pressure to subtherapeutic levels for 3 minutes d
268  and women (n = 82) with continuous positive airway pressure-treated obstructive sleep apnoea.
269 DATION 2: ACP recommends continuous positive airway pressure treatment as initial therapy for patient
270  should be combined with continuous positive airway pressure treatment during sleep.
271 n alternative therapy to continuous positive airway pressure treatment for patients diagnosed with OS
272                          Continuous positive airway pressure treatment improves the functional outcom
273 e might be the preferred first-line positive airway pressure treatment modality until more studies be
274                          Continuous positive airway pressure treatment was associated with reduction
275    Patients who received continuous positive airway pressure treatment were significantly less likely
276 idences of both OSA (AHI of >/=5 or positive airway pressure treatment) and OSA concomitant with habi
277  effects associated with continuous positive airway pressure treatment.
278  weeks of active or sham continuous positive airway pressure treatment.
279 ceived 8 weeks of active continuous positive airway pressure treatment.
280 ive sleep apnea refusing continuous positive airway pressure treatment.Methods: In an international,
281 sted of a 30-second breath hold at 40 cm H2O airway pressure under heavy sedation or paralysis.
282 spiratory distress syndrome, using high mean airway pressure under high-frequency oscillatory ventila
283 o, 0.98; 0.97-0.99, p = 0.018), initial mean airway pressure (unit odds ratio, 1.13; 1.02-1.28, p = 0
284 h-flow nasal cannula and continuous positive airway pressure use in a monitored setting to prevent re
285 were randomized to three continuous positive airway pressure-ventilated groups: 1) nebulized surfacta
286      We tested whether a continuous positive airway pressure ventilation strategy mitigates ventilato
287                         Median baseline mean airway pressure was 13 cm H2O (interquartile range, 10-1
288                                         Mean airway pressure was 18+/-3 cm H2O during conventional me
289 ulticenter prospective cohort, baseline mean airway pressure was independently associated with 90-day
290                                         Mean airway pressure was independently associated with 90-day
291                           The contour of the airway pressure waveform on a ventilator screen provides
292 of respiratory mechanics based on unmodified airway pressure were misleading regarding lung behavior
293                                         Mean airway pressures were higher in airway pressure release
294 ar mechanics (derived from alveolar size and airway pressure) were determined in noninjured (n = 9) a
295  was measured 3 times at each of 9 levels of airway pressure, which was increased in increments of 5
296  others were examined over a range of static airway pressures, which varied the extents of regional P
297 y, chronic alcohol abuse, shock, higher peak airway pressure while being mechanically ventilated, cur
298 = 1,500), 65 (4%) were missing baseline mean airway pressure, while 352 (23.5%) were missing baseline
299 infants were ventilated, continuous positive airway pressure without ventilation increased from 7% (1
300 onsiveness during transient manipulations of airway pressure, zolpidem did not alter pharyngeal muscl

 
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