1 ed force loss in the forelimbs, and terminal akinesis.

2 between normal wall motion, hypokinesis, and akinesis.

3 rized by ventricular dilatation and regional akinesis.

4 t 3 to 5 days, 95% had severe hypokinesis to akinesis at 4 weeks.

5 z) and grouped by location: infarct (area of akinesis by ICE), border (0.5-cm perimeter of akinetic a

6 y demonstrated basal hyperkinesis and apical akinesis, characteristic of Takotsubo cardiomyopathy.

7 -point whereas in the CSD group, the area of akinesis decreased (P=0.001).

8 , particularly in regions exhibiting resting akinesis/dyskinesis.

9                         The relative area of akinesis followed a similar pattern.

10 onstrates a significantly diminished area of akinesis in the CSD group (P=0.004).

11 sms, regions of wall thinning with segmental akinesis, in addition to typical electric and histologic

12 aluation of Symptomatic Heart Failure Study [AKINESIS]; NCT01291836).

13 ending artery or second diagonal branch, and akinesis of the infarcted segment were identified in all

14 ure, with widening in the LVOT, thinning and akinesis of the septal base, decrease in the angle betwe

15 nt for ejection fraction on day 1, extent of akinesis or dyskinesis, treatment regimen, Killip class,

16 0001), infarction zone nonviability based on akinesis unresponsive to low-dose dobutamine (P < .0001)