ライフサイエンスコーパス: albumin concentration

1 I, 0.42 to 0.74 per 1 g/dl increase in serum albumin concentration).

2 od urea nitrogen concentration and the serum albumin concentration.

3 ther factors that may affect muscle mass and albumin concentration.

4 greater prognostic accuracy than total serum albumin concentration.

5 d age-dependent PH associated with low serum-albumin concentration.

6 and manage conditions that reduce the serum albumin concentration.

7 ation than those with normal or higher serum albumin concentrations.

8 ischarge weight and had repleted their serum albumin concentrations.

9 ated with lower BMI but not with lower serum albumin concentrations.

10 which Hpn knockout mice manifest low plasma albumin concentrations.

11 nd baseline information about eGFR and urine albumin concentrations.

12 ere developed for measuring pH, glucose, and albumin concentrations.

13 erum total cholesterol, LDL cholesterol, and albumin concentrations.

14 mostly normal levels of serum bilirubin and albumin concentrations.

15 r 3 and blood urea nitrogen but higher serum albumin concentrations.

16 95% CI: 0.84, 0.99) for a 1-g/L higher serum albumin concentration].

17 (5.09 +/- 0.24) x 10(7) liters/mol at lower albumin concentrations (15 microm) to (0.54 +/- 0.05) x

18 8.1 yr; Child-Pugh score, 8.5 +/- 1.0; serum albumin concentration, 3.0 +/- 0.6 g/dl) were studied in

19 (0.54 +/- 0.05) x 10(7) liters/mol at higher albumin concentrations (300 microm).

20 Physiologic albumin concentrations activated the PI3K/mTORC2/PKB/mTO

21 ch as total protein concentration of saliva, albumin concentration, amylase level, pH, sodium, calciu

22 The key dependent variables were serum albumin concentration and BMI.

23 and then we used it to assess the effect of albumin concentration and buffer composition on binding.

24 irubin is not constant, but varies with both albumin concentration and buffer composition.

25 There was no association between serum albumin concentration and changes in CD4(+) T-cell count

26 Preoperative laboratory variables, including albumin concentration and donor-related information, wer

27 The inverse relationship between serum albumin concentration and its half-life suggested to ear

28 tacrolimus, including but not limited to low albumin concentration and low red blood cell count.

29 coefficients of variation (CVs) for urinary albumin concentration and other urinary variables.

30 The association between pretransplant albumin concentration and post-transplant 1-year surviva

31 ought to stem from the prevailing low plasma albumin concentration and the decreased transcapillary o

32 exhibited elevated PGE2, reduced circulating albumin concentrations and EP2-mediated immunosuppressio

33 ckout and control rats showed higher urinary albumin concentrations and reduced amounts of megalin an

34 clude altered fluid status, changes in serum albumin concentrations and renal and hepatic function, a

35 re noted for inflammatory (total protein and albumin concentrations) and hepatic (ALT, AST) parameter

36 debridement, inflammatory (total protein and albumin concentrations) and hepatic (AST) parameters ret

37 ng a 12 min walk, haemoglobin concentration, albumin concentration, and age-corrected resting heart r

38 osis on initial biopsy as well as age, serum albumin concentration, and CKD stage at onset affected E

39 ion with the IL28B CC genotype, had a higher albumin concentration, and had a lower HCV viral load at

40 eristics (age, sex, performance status, BMI, albumin concentration, and haemoglobin concentration), e

41 itrogen level, impaired sensorium, low serum albumin concentration, and partial thromboplastin time <

42 itrogen level, impaired sensorium, low serum albumin concentration, and partial thromboplastin time <

43 algorithm using platinum sensitivity, serum albumin concentration, and stage as stratification facto

44 te were associated with drug dose and plasma albumin concentration, and were lower among men and amon

45 Lower serum albumin concentrations appear to be associated with an i

46 ning test solutes and artificial plasma with albumin concentration approximately 4 g/dl was dialyzed

47 dy suggests that decreases with age in serum albumin concentrations are associated with muscle loss (

48 ([normal albumin] [observed albumin]), where albumin concentrations are in g/L; if given in g/dL, the

49 be adjusted for the effect of abnormal serum albumin concentrations as follows: adjusted anion gap =

50 normalization of ALT at 6 months, low serum albumin concentration at diagnosis, and age at presentat

51 The association between serum albumin concentration at the time of listing for lung tr

52 centrations, as well as increased mean serum albumin concentrations at 12 weeks, relative to placebo.

53 ysis, the serum monoclonal protein and serum albumin concentrations at diagnosis were the only risk f

54 umber of people had non-detectable aflatoxin-albumin concentrations at harvest.

55 Recent studies have shown that not only albumin concentration but also albumin function is reduc

56 In both groups, albumin concentrations but not FSRs were lower in studie

57 er unit length, skinfold thickness and serum albumin concentration, but only in a sea lion colony exp

58 , C-reactive protein (CRP), haptoglobin, and albumin concentrations by radial immunodiffusion assays.

59 ntal studies have shown that a reduced serum albumin concentration can increase the volume of distrib

60 Serum albumin concentrations can identify adults initiating AR

61 with better sensitivity for samples with low albumin concentrations compared to routine biochemical a

62 Serum albumin concentrations decrease with age and values < 38

63 ence of human urine (HU), which contains low albumin concentrations, did not modify MIC values of two

64 ation, which represents the fact that plasma albumin concentration does not reflect its function.

65 amount of weight loss and no improvement in albumin concentrations during the first month after hosp

66 damages, leading to the concept of effective albumin concentration (eAlb), referring to the albumin p

67 For urinary albumin concentration, exclusion of one mislabeled pair

68 pHo changes, and the presence of physiologic albumin concentrations failed to attenuate the pHo-media

69 (10 %, 20 %, and 40 %) irrespective of total albumin concentration fluctuations within clinically rel

70 d)/days, where albumin 1 and 2 are the serum albumin concentrations (g/L) at the beginning and end of

71 Serum albumin concentration has been recognized as a marker of

72 ssessed whether individuals with a low serum albumin concentration have delayed progression to AIDS,

73 By contrast, mean aflatoxin-albumin concentration in intervention villages after 5 m

74 d plasmatic parameters except an increase in albumin concentration in septic rats compared with the b

75 impedance response is linearly dependent on albumin concentration in the clinically relevant range w

76 ors of poor outcome were adjusted for, serum albumin concentration in the hospital was a strong and i

77 re were no significant differences in plasma albumin concentrations in nonedematous and edematous chi

78 omplexes by physiologic and pathophysiologic albumin concentrations in proximal tubule cells.

79 High albumin concentrations in the proximal tubule of the kid

80 (as assessed by bronchoalveolar lavage fluid albumin concentration) in both NADPH oxidase-deficient m

81 The mean serum albumin concentration increased by 1.42 g/dL in the albu

82 In control villages mean aflatoxin-albumin concentration increased postharvest (from 5.5 pg

83 Urine albumin concentrations increased in all salsalate groups

84 l) porphine were determined as a function of albumin concentration, ionic strength in medium, pH, and

85 Pretransplant serum albumin concentration is a strong prognostic marker for

86 effects of the elevated circulating glycated albumin concentration is associated with reduction in pr

87 Low serum albumin concentration is associated with short-term surv

88 Serum albumin concentration is determined by its rate of synth

89 te phase protein (alpha2 macroglobulin), and albumin concentration is inversely proportional to that

90 the familiar scale of values that apply when albumin concentration is normal.

91 1.9 vs. 2.2 +/- 0.6; P =.03) and lower serum albumin concentrations (low: 2.8 +/- 0.1 vs. normal: 3.3

92 = 75 y, use of > or = 3 medications, and an albumin concentration < 35.0 g/L were significant predic

93 Ninety-six percent of the patients had serum albumin concentration < or = 3 SD below the mean of the

94 Nineteen percent had serum albumin concentrations < 35 g/L (3.5 g/dL).

95 nemia (< or = 38 g/L) or progressively lower albumin concentrations < 40 g/L.

96 f these compounds and/or inappropriately low albumin concentrations may blur the interpretation of th

97 As a fixed covariate, albumin concentrations measured shortly after HIV-1 sero

98 Prospective studies of serum albumin concentration measurement as a low-cost predicto

99 was considered the upper limit of normal for albumin concentration, Micral-Test, sulfosalicylic acid

100 increased risk of disability with low serum albumin concentrations observed in the elderly may actua

101 blood urea nitrogen of > or =24 mg/dL; serum albumin concentration of < or =4.0 g/dL (< or =40.0 g/ L

102 itically ill patients: 49% of them had serum albumin concentration of <20 g/L.

103 als with hypoalbuminemia (defined as a serum albumin concentration of <35 g/L) at ART initiation had

104 in FcRn-deficient mice, and that the plasma albumin concentration of FcRn-deficient mice is less tha

105 07; P < .001) that of individuals with serum albumin concentrations of >/= 35 g/L, after multivariate

106 Secondary analyses suggested that serum albumin concentrations of <38 g/L were associated with i

107 nvestigate the impact of pretransplant serum albumin concentration on post-transplant outcome in hear

108 en at relatively low hydraulic pressures the albumin concentration on the tissue side of the glycocal

109 at is only minimally sensitive to glycation, albumin concentration, or redox potential, unlike other

110 (S6K) pathway, but pathophysiologically high albumin concentrations overactivated mTORC1 and inhibite

111 Variability was explained by the serum albumin concentration (P < .0001), concomitant use of fe

112 P < .001) and was related to a greater serum albumin concentration (P < .001) and to a lower exposure

113 lations between the hematocrit and the serum albumin concentration (P = 0.009) and between the hemato

114 Serum albumin concentration (p=0.43), thromboprophylaxis (p=0.

115 Albumin administration increased plasma albumin concentrations (p <.05 compared with placebo) an

116 After exclusion of urinary albumin concentration pairs with values below the detect

117 the nephrotic syndrome (as assessed by serum albumin concentration), preexisting thrombophilic states

118 tes, serum creatinine concentration, urinary albumin concentration, previous cardiovascular event, an

119 g affinity remained heavily dependent on the albumin concentration (range (5.37 +/- 0.26) x 10(7) lit

120 ased plasma, BAL albumin, and the BAL:plasma albumin concentration ratio.

121 y ratios (W/D) and tissue-to-plasma 125I-rat albumin concentration ratios (T/P) 8 h after the endotox

122 ting only studies in which, depending on the albumin concentration, real or extrapolated free concent

123 ntly during the study period, but only serum albumin concentrations showed a significant association

124 Additional adjustment for serum albumin concentration substantially attenuated the all-c

125 n and clinical outcomes as compared to total albumin concentration (tAlb).

126 y be considerably less avid at physiological albumin concentrations than previously believed.

127 For each 0.5 g/dL decrease in serum albumin concentration the 1-year and overall mortality r

128 s implied that for every 1 mg/dL increase in albumin concentration, the post-transplant 1-year mortal

129 herein that the time required for the tissue albumin concentration to increase to values for a new st

130 intervention significantly reduced the urine albumin concentration (UAC) at week 12 and 24 endpoints

131 e albumin:creatinine ratio (ACR) and urinary albumin concentration (UAC) obtained from a single sampl

132 The mean serum albumin concentration was 3.8 +/- 0.4 g per deciliter, t

133 In the albumin group, the target serum albumin concentration was 30 g per liter or more until d

134 Increasing albumin concentration was also found to reduce dissoluti

135 Serum albumin concentration was determined before and after tr

136 Serum albumin concentration was measured at ART initiation for

137 The median salivary albumin concentration was significantly lower in the tra

138 , lymph node metastasis) and decreased serum albumin concentration were unfavorable for long-term sur

139 tients with liver failure, the average serum albumin concentrations were 30.55 g/L.

140 Lower serum albumin concentrations were associated with advanced age

141 Serum albumin concentrations were good predictors of the degre

142 Plasma glycated albumin concentrations were lower 10 wk after fructose t

143 Height and weight and cholesterol and albumin concentrations were measured, and health care cl

144 Serum 25(OH)D, DBP, and albumin concentrations were measured.

145 Mean urine albumin concentrations were not significantly different

146 Albumin concentrations were predicted by injury severity

147 Variables in the model for lower serum albumin concentrations were sex, increased age, increase

148 s (NHB) and Mexican Americans, whereas urine albumin concentrations were significantly higher in NHB

149 oxygen partial pressures, and intra-alveolar albumin concentrations) were the same in knockout mice a

150 observation led to the concept of effective albumin concentration, which represents the fact that pl

151 wer free fetuin-A, plasma pyrophosphate, and albumin concentrations, which accounted for 49% of the v

152 mimicked all the effects of pathophysiologic albumin concentrations, which disrupt normal signal tran

153 ndings support the concept of the "effective albumin concentration," which implies that the global HS