ライフサイエンスコーパス: all-trans-retinoic acid

1 noid metabolism by HSCs and was dependent on all-trans retinoic acid.

2 ls incubated with rapamycin or rapamycin and all-trans retinoic acid.

3 and is rapidly down-regulated in response to all-trans retinoic acid.

4 eatment of acute promyelocytic leukemia with all-trans retinoic acid.

5 and was rescued by treatment with exogenous all-trans retinoic acid.

6 tly, this is counteracted in the presence of all-trans retinoic acid.

7 The top hit identified in the screen was all-trans-retinoic acid.

8 ehyde, the immediate precursor for bioactive all-trans-retinoic acid.

9 The addition of all-trans retinoic acid, a commonly used agent for the t

10 Accordingly, all-trans-retinoic acid, alone or together with gemfibro

11 Treatment of APL blasts with all-trans retinoic acid also did not result in immediate

12 s important therapeutic agents; for example, all-trans retinoic acid, an activating ligand for retino

13 Patients had significantly lower levels of all-trans retinoic acid and 13-cis retinoic acid than co

14 l-trans retinol), and its geometric isomers, all-trans retinoic acid and 9-cis retinoic acid, in a fo

15 Upon treating APL with all-trans retinoic acid and achieving complete remission

16 patients with t(15;17) treated with extended all-trans retinoic acid and anthracycline-based chemothe

17 patients with APL homogeneously treated with all-trans retinoic acid and anthracycline-based chemothe

18 emains high despite the wide availability of all-trans retinoic acid and appears significantly higher

19 Thanks to modern treatment with all-trans retinoic acid and chemotherapy, acute promyelo

20 APL cells from these mice were responsive to all-trans retinoic acid and had virtually no differences

21 All patients received All-trans retinoic acid and idarubicin according to the

22 We hypothesize that topical all-trans retinoic acid and interferon alfa-2b may act s

23 er rates of apoptosis in FBAE cells than did all-trans retinoic acid and the control (P = 0.004).

24 th acute promyelocytic leukemia treated with all-trans retinoic acid and/or arsenic trioxide.

25 myelocytic leukemia (APL) who were receiving all-trans-retinoic acid and anthracycline-based chemothe

26 pregulated during differentiation induced by all-trans-retinoic acid and brain-derived neurotrophic f

27 d also increased Bcl2a1 expression, although all-trans-retinoic acid and ligands for other RXR partne

28 for rapid stimulation of dendritic growth by all-trans-retinoic acid and reveal that the ligand-depen

29 with other targeted therapies such as ATRA (all trans retinoic acid) and arsenic trioxide.

30 Novel mutual prodrugs (MPs) of ATRA (all- trans-retinoic acid) and HDIs (histone deacetylase

31 Finally, vitamin A (all-trans retinoic acid) and vitamin D (1,25-dihydroxyvi

32 Retinoids (i.e., vitamin A, all-trans retinoic acid, and related signaling molecules

33 1) concentration-dependent, 2) selective for all-trans-retinoic acid, and 3) requires the presence of

34 Treatment with all-trans retinoic acid antagonizes stress-induced activ

35 rging data indicates that retinoids, such as all trans retinoic acid (ATRA) and its precursor all tra

36 Additionally, we reveal that all trans retinoic acid (ATRA) induces VCP expression, c

37 can be dissociated by pharmacologic doses of all trans retinoic acid (ATRA) inducing differentiation

38 All trans retinoic acid (atRA) is one of the most potent

39 c conditions, TNIP1 expression is induced by all trans retinoic acid (ATRA).

40 dies, a phenotype reversed by treatment with all trans retinoic acid (ATRA).

41 ion with fibrillar fibronectin and show that all trans-retinoic acid (ATRA), which induces PSC quiesc

42 APL0406 trial showed that the combination of all- trans-retinoic acid (ATRA) and arsenic trioxide (AT

43 In nude mice models, combination of all-trans retinoic acid (ATRA) and AEG-1 knockdown syner

44 is a model for oncogene-targeted therapies: all-trans retinoic acid (ATRA) and arsenic both target a

45 We examined whether combining all-trans retinoic acid (ATRA) and arsenic trioxide (ATO

46 nockdown of NPM1 also sensitized OCI-AML3 to all-trans retinoic acid (ATRA) and cytarabine.

47 We used all-trans retinoic acid (ATRA) and histone deacetylase (

48 we have identified a novel pathway involving all-trans retinoic acid (ATRA) and its receptor (RARgamm

49 Here we show that all-trans retinoic acid (ATRA) and other agonists of the

50 treatment until the recent identification of all-trans retinoic acid (ATRA) as a Pin1 inhibitor.

51 The identification of all-trans retinoic acid (ATRA) as a potent Pin1 inhibito

52 g from 5 large clinical trials that included all-trans retinoic acid (ATRA) as part of induction, we

53 We show that treatment with all-trans retinoic acid (ATRA) at clinically achievable

54 ng hematologist because early institution of all-trans retinoic acid (ATRA) at the first suspicion of

55 effect was reversed for K1 and K10 by adding all-trans retinoic acid (ATRA) but increased for K2 in t

56 reatment of PML-RARalpha leukemic cells with all-trans retinoic acid (ATRA) causes them to differenti

57 neuroblastoma cells following treatment with all-trans retinoic acid (ATRA) compared to controls.

58 All patients received all-trans retinoic acid (ATRA) during induction, each co

59 The treatment of patients with all-trans retinoic acid (ATRA) effectively ameliorates t

60 , we demonstrated that treatment of AML with all-trans retinoic acid (ATRA) enhanced FRbeta expressio

61 that differentiate along this lineage after all-trans retinoic acid (ATRA) exposure.

62 x 10(9)/L (or for a maximum of 28 days) and all-trans retinoic acid (ATRA) for 90 days.

63 Recent studies have shown that all-trans retinoic acid (ATRA) had a potent activity in

64 All-trans retinoic acid (ATRA) has been used in several

65 eatment of acute promyelocytic patients with all-trans retinoic acid (ATRA) has improved the survival

66 of cancer stem cells through treatment with all-trans retinoic acid (ATRA) have yielded limited succ

67 mpounds to enhance the biological effects of all-trans retinoic acid (ATRA) in a retinoid-responsive

68 h clinical outcomes of patients treated with all-trans retinoic acid (ATRA) in combination with anthr

69 ibition relieved by pharmacological doses of all-trans retinoic acid (atRA) in culture and in vivo.

70 histone deacetylase inhibitor valproate and all-trans retinoic acid (ATRA) in treatment-naive elderl

71 Treatment with all-trans retinoic acid (ATRA) increased both the expres

72 All-trans retinoic acid (ATRA) increased IRF4 expression

73 The vitamin A metabolite all-trans retinoic acid (ATRA) induces a gut-homing phen

74 All-trans retinoic acid (ATRA) induces clinical remissio

75 All-trans retinoic acid (ATRA) induces differentiation i

76 udy provides the first evidence showing that all-trans retinoic acid (ATRA) induces the interaction a

77 We determined the mechanism by which all-trans retinoic acid (ATRA) inhibits experimental aut

78 In mammals, all-trans retinoic acid (ATRA) is instrumental to sperma

79 All-trans retinoic acid (ATRA) neutralizes the different

80 -cis RA, 9-cis retinoic acid (9-cis RA), and all-trans retinoic acid (ATRA) on cell proliferation, ap

81 H/+; Pdx-1-Cre (KPC) mice and the effects of all-trans retinoic acid (ATRA) on these processes.

82 ally, we evaluated the impact of exposure to all-trans retinoic acid (ATRA) on wild-type NK and CD38K

83 Comparison of monocytes stimulated with all-trans retinoic acid (ATRA) or 1,25-dihydroxyvitamin

84 t PLZF/RAR alpha leukemia, was responsive to all-trans retinoic acid (ATRA) or As2O3 treatments.

85 ute promyelocytic leukemia by treatment with all-trans retinoic acid (ATRA) plus arsenic trioxide (AT

86 We recently reported that all-trans retinoic acid (atRA) rapidly (within minutes)

87 This study found that all-trans retinoic acid (atRA) reverses the effects of H

88 All-trans retinoic acid (atRA) reverses the HIV-induced

89 Treatment of monocytes with all-trans retinoic acid (ATRA) significantly decreased b

90 that the eyeballs are a source of diffusible all-trans retinoic acid (ATRA) that allow their targetin

91 We investigated the benefit of adding all-trans retinoic acid (ATRA) to chemotherapy for young

92 We used all-trans retinoic acid (ATRA) to differentiate MDSCs in

93 Dendritic cells (DCs) use all-trans retinoic acid (ATRA) to promote characteristic

94 ay initiated from the nongenomic activity of all-trans retinoic acid (atRA) to stimulate complex form

95 While all-trans retinoic acid (ATRA) treatment in acute promye

96 We have also found that all-trans retinoic acid (ATRA) treatment increased AGAP2

97 Interestingly, all-trans retinoic acid (ATRA) treatment induced potent

98 In acute promyelocytic leukemia (APL), all-trans retinoic acid (ATRA) treatment induces granulo

99 All-trans retinoic acid (ATRA) upregulated TRAIL-R1 expr

100 All-trans retinoic acid (ATRA) was negatively connected

101 The effects of all-trans retinoic acid (ATRA) were studied in LSL-KrasG

102 rly vulnerable to a novel strategy combining all-trans retinoic acid (ATRA) with arsenic trioxide (AT

103 All-trans retinoic acid (ATRA) with chemotherapy is the

104 All-trans retinoic acid (ATRA), a derivative of vitamin

105 All-trans retinoic acid (ATRA), a natural retinoid, arre

106 All-trans retinoic acid (ATRA), a potent derivative of v

107 dy was to evaluate the therapeutic effect of all-trans retinoic acid (ATRA), an active metabolite of

108 d with daunorubicin, cytarabine (Ara-C), and all-trans retinoic acid (ATRA), and complete remission w

109 ) in response to its natural agonist ligand, all-trans retinoic acid (atRA), and is repressed by SHP.

110 onducted to examine the interactive roles of all-trans retinoic acid (ATRA), Ets-1, Sp1, and histone

111 Its biologically active metabolite, all-trans retinoic acid (ATRA), exhibits a potent antivi

112 on the short half-life vitamin A metabolite, all-trans retinoic acid (atRA), in an amount sufficient

113 with phosphate-buffered saline (PBS), UDCA, all-trans retinoic acid (atRA), or UDCA and atRA by gava

114 e present standard of care, chemotherapy and all-trans retinoic acid (ATRA), results in a high propor

115 All-trans retinoic acid (atRA), the active derivative of

116 In response to challenge with all-trans retinoic acid (atRA), the balance of daughter

117 promoter and is stimulated by treatment with all-trans retinoic acid (ATRA), the biologically active

118 es for metabolism into an active metabolite, all-trans retinoic acid (atRA), where atRA is essential

119 or source of the immunoregulatory metabolite all-trans retinoic acid (ATRA), which may contribute to

120 mechanism-based screening, here we find that all-trans retinoic acid (ATRA)--a therapy for acute prom

121 Despite the great success of all-trans retinoic acid (ATRA)-based therapy, which resu

122 In this study, we present an effective model All-Trans Retinoic Acid (ATRA)-induced differentiation o

123 evels of NTAL were associated with increased all-trans retinoic acid (ATRA)-induced differentiation,

124 s depleted of Ajuba are highly sensitized to all-trans retinoic acid (atRA)-induced transcription and

125 Mutations in the all-trans retinoic acid (ATRA)-targeted ligand binding d

126 d substantially in the marrow and spleens of all-trans retinoic acid (ATRA)-treated C57BL6 mice, whil

127 investigated the gene expression profile of all-trans retinoic acid (ATRA)-treated human CD4(+) T ce

128 sion is directly and potently upregulated by all-trans retinoic acid (atRA).

129 stoma (SK-N-SH) cells to neuronal cells with all-trans retinoic acid (ATRA).

130 rated insensitive response to this effect of all-trans retinoic acid (ATRA).

131 emia activity and may restore sensitivity to all-trans retinoic acid (ATRA).

132 ctive up-regulation in the leukemic cells by all-trans retinoic acid (ATRA).

133 sensitivity to another known limb teratogen, all-trans retinoic acid (atRA).

134 icrog (low-dose) or 1,000 microg (high-dose) all-trans retinoic acid (ATRA)/kg body weight in corn oi

135 PURPOSE Event-free survival following all-trans-retinoic acid (ATRA) -based therapy for acute

136 PML-RARa-associated APL is sensitive to both all-trans-retinoic acid (ATRA) and arsenic trioxide (ATO

137 te promyelocytic leukemia (APL) treated with all-trans-retinoic acid (ATRA) and arsenic trioxide (ATO

138 The use of all-trans-retinoic acid (ATRA) and arsenic trioxide, bot

139 Synergistic actions between all-trans-retinoic acid (atRA) and interferon gamma (IFN

140 hrome P450 CYP26 enzymes are responsible for all-trans-retinoic acid (atRA) clearance.

141 All-trans-retinoic acid (ATRA) combined with chemotherap

142 All-trans-retinoic acid (ATRA) did not stimulate osteocl

143 All-trans-retinoic acid (atRA) has been implicated in th

144 All-trans-retinoic acid (ATRA) has been shown to act phy

145 All-trans-retinoic acid (ATRA) has been shown to arrest

146 (AML), the use of the differentiation agent all-trans-retinoic acid (ATRA) has revolutionized the th

147 The all-trans-retinoic acid (atRA) hydroxylase Cyp26a1 is es

148 Treatment of APL with all-trans-retinoic acid (ATRA) induces disease remission

149 All-trans-retinoic acid (ATRA) induces growth arrest of

150 Although all-trans-retinoic acid (atRA) is a key regulator of int

151 All-trans-retinoic acid (ATRA) is a natural compound pro

152 All-trans-retinoic acid (ATRA) is an active vitamin A de

153 All-trans-retinoic acid (atRA) is an important morphogen

154 All-trans-retinoic acid (atRA) is the active metabolite

155 All-trans-retinoic acid (atRA) is the active metabolite

156 The all-trans-retinoic acid (atRA) isomer, 9-cis-retinoic ac

157 26 family is believed to be responsible for all-trans-retinoic acid (atRA) metabolism and eliminatio

158 Lcn2 KO mice exhibited a blunted effect of all-trans-retinoic acid (ATRA) on body weight and fat ma

159 The combination of all-trans-retinoic acid (ATRA) plus arsenic trioxide (AT

160 l compared efficacy and toxicity of standard all-trans-retinoic acid (ATRA) plus chemotherapy versus

161 We investigated the actions of all-trans-retinoic acid (atRA) signaling in pancreatic b

162 All-trans-retinoic acid (atRA) stimulates neurogenesis,

163 All-trans-retinoic acid (atRA) supports embryonic develo

164 ntained only about a 0.6 nm concentration of all-trans-retinoic acid (atRA) that is the most active n

165 successfully treated with therapy utilizing all-trans-retinoic acid (ATRA) to differentiate leukemic

166 ytic leukemia patients and cell lines during all-trans-retinoic acid (ATRA) treatment by using a miRN

167 All-trans-retinoic acid (atRA), an autacoid derived from

168 ly after recognition of the effectiveness of all-trans-retinoic acid (ATRA), anthracycline-based chem

169 Vitamin A derivatives, including all-trans-retinoic acid (ATRA), have a well-established

170 his defect can be overcome by treatment with all-trans-retinoic acid (ATRA), leading to complete clin

171 All-trans-retinoic acid (ATRA), retinol, retinalaldehyde

172 All-trans-retinoic acid (atRA), the active metabolite of

173 All-trans-retinoic acid (atRA), the active metabolite of

174 All-trans-retinoic acid (atRA), the active metabolite of

175 All-trans-retinoic acid (atRA), the major active metabol

176 Subsequent oxidation produces all-trans-retinoic acid (ATRA), which functions as a lig

177 tly, different statins were found to enhance all-trans-retinoic acid (ATRA)-dependent differentiation

178 nsformation and leukemogenesis, and inhibits all-trans-retinoic acid (ATRA)-induced AML cell differen

179 or (RAR) target gene expression and inhibits all-trans-retinoic acid (ATRA)-induced myeloid different

180 DR5) site in the OLFM4 promoter and mediates all-trans-retinoic acid (ATRA)-induced transactivation o

181 o several developmental genes, including the all-trans-retinoic acid (ATRA)-responsive ones, through

182 in the formation of the essential morphogen all-trans-retinoic acid (ATRA).

183 vous system require the vitamin A metabolite all-trans-retinoic acid (atRA).

184 lation of myeloid cell differentiation using all-trans-retinoic acid (ATRA).

185 hat targeting pancreatic stellate cells with all-trans-retinoic-acid (ATRA) reprograms pancreatic str

186 Vitamin A (all trans retinoic acid, ATRA) treated leukemic cells ha

187 re, the outcome of 155 patients treated with all-trans retinoic acid-based therapy on 3 clinical tria

188 beta-apo-8'-carotenal, 13-cis-retinoic acid, all-trans-retinoic acid, beta-carotene-5,6-epoxide, all-

189 Rdh10 catalyzes the first step of all-trans-retinoic acid biogenesis physiologically, conv

190 retinal dehydrogenases (critical enzymes for all-trans retinoic acid biosynthesis) and were significa

191 orms efficiently (V(m)/K(m)) in a pathway of all-trans-retinoic acid biosynthesis in cells and recogn

192 When differentiation was induced by all-trans retinoic acid, CEACAM6 expression strongly cor

193 te promyelocytic leukemia model treated with all-trans retinoic acid combined with the histone-deacet

194 ng to neutrophils and in vivo treatment with all-trans retinoic acid decreased plasminogen binding to

195 ibroblasts in response to 5 toxic compounds (all-trans retinoic acid, dexamethasone, doxorubicin, 5'-

196 sfully demonstrated using both ibuprofen and all-trans retinoic acid; drugs with anti-inflammatory an

197 ndrites, and knocking down RARalpha prevents all-trans-retinoic acid effects on dendritic growth.

198 tion cycles with idarubicin, cytarabine, and all-trans retinoic acid either with VPA or without (STAN

199 of acute promyelocytic leukemia (APL) in the all-trans retinoic acid era.

200 de for vision as well as the biosynthesis of all-trans-retinoic acid for differentiation and developm

201 The major active retinoid, all-trans retinoic acid, has long been recognized as cri

202 ith granulocyte colony-stimulating factor or all-trans-retinoic-acid have shown improvement in decrea

203 ion of all-trans-retinol for biosynthesis of all-trans-retinoic acid, however, initial assays suggest

204 Consistent with the role of all-trans retinoic acid in inducing gut-homing T cells,

205 found that response to targeted therapy with all-trans retinoic acid in vivo was dependent on NB inte

206 idence that vitamin A uptake is regulated by all-trans-retinoic acid in non-ocular tissues of mice.

207 o produce visual chromophore in the eyes and all-trans-retinoic acid in other tissues.

208 All-trans retinoic acid increased CD38 levels and decrea

209 uced macrophage differentiation, but blocked all-trans-retinoic acid induced granulocytic differentia

210 ls causes acute myeloid leukemia and impairs all-trans retinoic acid-induced granulocytic differentia

211 feron-induced mortality (GRIM)-19, as an IFN/all-trans retinoic acid-induced growth suppressor.

212 We found that the major vitamin A metabolite all-trans-retinoic acid induces histone acetylation at t

213 (PPAR) alpha, alone and in conjunction with all-trans-retinoic acid is capable of enhancing TFEB in

214 Administration of all-trans-retinoic acid led to a significant decrease in

215 dipose retinoid (retinol, retinyl ester, and all-trans-retinoic acid) levels were observed.

216 All-trans-retinoic acid may be an important molecular si

217 me myeloid leukemias respond dramatically to all-trans retinoic acid mediated differentiation therapy

218 Chiral pentamers of all-trans-retinoic acid molecules have been prepared on

219 Temporary discontinuation of all-trans retinoic acid or arsenic trioxide is indicated

220 at trigger PML-RARalpha degradation, such as all-trans retinoic acid or arsenic trioxide, restore nuc

221 All-trans retinoic acid or related compounds may also pl

222 this randomization were randomly assigned to all-trans-retinoic acid or not.

223 tion of SH-SY5Y human neuroblastoma cells by all-trans retinoic acid, or oxidative stress induced by

224 nal differentiation following treatment with all-trans retinoic acid (P = 3.1 x 10(-13) to 2.4 x 10(-

225 ll (Treg)-polarizing molecules TGF-beta1 and all-trans retinoic acid, particularly during states of i

226 vailable only in the context of conventional all-trans retinoic acid plus chemotherapy regimens.

227 sence or presence of the RAR-specific ligand all trans retinoic acid (RA).

228 s well as IL10 and the vitamin A metabolite; all-trans retinoic acid (RA [at-RA]) has been found to e

229 oth genes are transcriptionally activated by all-trans retinoic acid (RA) and display increased level

230 spinal cord phenotype using a combination of all-trans retinoic acid (RA) and epidermal growth factor

231 All-trans Retinoic acid (RA) and its derivatives are pot

232 r 1 (CYP26B1) regulates the concentration of all-trans retinoic acid (RA) and plays a key role in ger

233 Reduced generation of all-trans retinoic acid (RA) by CD103(+) intestinal dend

234 We demonstrate that all-trans retinoic acid (RA) induces FoxP3(+) adaptive T

235 60 model human myeloid leukemia cells, where all-trans retinoic acid (RA) induces granulocytic differ

236 We have previously shown that all-trans retinoic acid (RA) mediates activity blockade-

237 The major vitamin A metabolite all-trans retinoic acid (RA) not only enforces the gener

238 However, the effects of all-trans retinoic acid (RA) on cellular expression of V

239 pplication of active vitamin D3 (VD3) and/or all-trans retinoic acid (RA) on wild-type mouse skin ind

240 All-trans retinoic acid (RA) plays crucial roles in embr

241 mechanisms whereby the vitamin A metabolite all-trans retinoic acid (RA) promotes the formation of p

242 All-trans retinoic acid (RA) stimulates cellular prolife

243 ated the ability of the vitamin A metabolite all-trans retinoic acid (RA) to restore the defective im

244 Here, we report that all-trans retinoic acid (RA), a well-known developmental

245 Vitamin A and its active metabolite, all-trans retinoic acid (RA), regulate the antibody resp

246 rcinoma (EC) is relieved upon treatment with all-trans retinoic acid (RA), resulting in enhanced p53

247 Hox gene expression is activated by all-trans retinoic acid (RA), through binding to retinoi

248 sticity, in part by stimulating synthesis of all-trans retinoic acid (RA), which in turn increases AM

249 ndritic cells (DC) metabolize vitamin A into all-trans retinoic acid (RA), which is required to induc

250 with HtrA2 and augments cell death in an IFN/all-trans retinoic acid (RA)-dependent manner.

251 In all-trans retinoic acid (RA)-induced differentiation of

252 of glioblastoma stem-like cells (GBM-SCs) to all-trans retinoic acid (RA).

253 neuroblastoma cells following treatment with all-trans retinoic acid (RA).

254 ivation of retinoic acid receptor (RAR) with all-trans-retinoic acid (RA) ameliorates glucose intoler

255 hrome P450 enzyme Cyp26a1, which metabolizes all-trans-retinoic acid (RA) and thereby reduces RA leve

256 Many biological activities of all-trans-retinoic acid (RA) are mediated by the ligand-

257 APL cell treatment with all-trans-retinoic acid (RA) degrades the chimeric, domi

258 is study was to examine the possibility that all-trans-retinoic acid (RA) in the eye is a signal rela

259 We have previously demonstrated that all-trans-retinoic acid (RA) induction of RIP140 constit

260 P26A1, a cytochrome P450 enzyme, metabolizes all-trans-retinoic acid (RA) into polar metabolites, e.g

261 All-trans-retinoic acid (RA) is a vitamin A metabolite t

262 al (HBE) cells to evaluate stabilities after all-trans-retinoic acid (RA) or cycloheximide treatments

263 The vitamin A metabolite all-trans-retinoic acid (RA) regulates multiple biologic

264 , but not Tbx18 or NFATC1, is activated with all-trans-retinoic acid (RA) treatment of isolated chick

265 We found that all-trans-retinoic acid (RA) treatment of mouse epiphyse

266 All-trans-retinoic acid (RA), a bioactive derivative of

267 We report herein that all-trans-retinoic acid (RA), an active metabolite of vi

268 Vitamin A metabolite, all-trans-retinoic acid (RA), induces cell growth, diffe

269 f vitamin A (retinol/ROL) can be oxidized to all-trans-retinoic acid (RA), which plays a critical rol

270 hat DGL-alpha and DGL-beta may contribute to all-trans-retinoic acid (RA)-induced neurite outgrowth i

271 Whereas both are all-trans-retinoic acid (RA)-responsive in ES cells, the

272 nhibitory and pro-differentiating effects of all-trans-retinoic acid (RA).

273 tival epithelial (HCjE) cell line grown with all-trans-retinoic acid (RA).

274 LDH1A3 complexed with NAD(+) and the product all-trans retinoic acid (REA).

275 In contrast, treatment of apc mutants with all-trans retinoic acid rescued retinal differentiation

276 drugs should be considered for treatment of all-trans retinoic acid-resistant APL patients as well a

277 induce cell death and induces expression of all-trans-retinoic acid-responsive genes that can be blo

278 Short-term treatment of mice with all-trans retinoic acid resulted in increased PreB lymph

279 All-trans-retinoic acid stimulates dendritic growth in h

280 ation of the retinoic acid receptor agonist, all-trans-retinoic acid, suggesting that the ability of

281 All -trans-Retinoic acid ( t-RA) regulates leukocyte dif

282 generate both all-trans-retinal (t-RAL) and all-trans-retinoic acid (t-RA) from the precursor all-tr

283 All-trans retinoic acid therapy of acute promyelocytic l

284 essed cell proliferation and synergized with all-trans retinoic acid to promote differentiation.

285 Topical treatment of human skin with all-trans retinoic acid (tRA) induces EGFR ligands hepar

286 All-trans retinoic acid (tRA) induces NIS gene expressio

287 All-trans-retinoic acid (tRA) markedly induces NIS activ

288 All trans-retinoic acid treatment of A404 cells induced

289 vation of c-SRC as early as 15 min following all-trans-retinoic acid treatment in LA-N-5 cells.

290 y, early forced maturation of MDSC by either all-trans-retinoic acid treatment or active immunorecept

291 Utilizing in-vitro experimental models of all-trans retinoic acid triggered myeloid leukemia diffe

292 Both promoters are partially regulated by all-trans retinoic acid via RARA and other RARs.

293 d for CCR9 by cell sorting or culturing with all-trans retinoic acid, we measured chemotaxis, intrace

294 Isotretinoin is a pro-drug for all-trans retinoic acid, which can induce long-term remi

295 t-associated dendritic cells (DC) synthesize all-trans retinoic acid, which is required for inducing

296 s-retinaldehyde, the visual chromophore, and all-trans-retinoic acid, which is involved in the regula

297 Furthermore, treatment of NB4 cells with all-trans-retinoic acid, which promotes PML-RARalpha deg

298 nergistic effects of valproic acid (VPA) and all-trans retinoic acid with chemotherapy.

299 with idarubicin, cytarabine, etoposide, and all-trans-retinoic acid with or without GO.

300 the retinoic acid receptor (RAR) compared to all-trans-retinoic acid, with select analogues also redu