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1  including psoriasis, atopic dermatitis, and allergic contact dermatitis.
2 f occupational skin disease are irritant and allergic contact dermatitis.
3 Pd(2+) ions commonly trigger T cell-mediated allergic contact dermatitis.
4 atory diseases such as atopic dermatitis and allergic contact dermatitis.
5 r the successful diagnosis and management of allergic contact dermatitis.
6 inones as preservatives is a global cause of allergic contact dermatitis.
7  a role in psoriasis, atopic dermatitis, and allergic contact dermatitis.
8 pment of new therapeutic approaches to treat allergic contact dermatitis.
9 ll activation through MRGPRB2 drives itch in allergic contact dermatitis.
10 modulatory effect of these NPs in a model of allergic contact dermatitis.
11 e a potential therapeutic avenue in treating allergic contact dermatitis.
12  NAAA inhibitor, ARN077, in a mouse model of allergic contact dermatitis.
13  and characterize the role of MCs in chronic allergic contact dermatitis.
14 ty (CHS) of murine skin serves as a model of allergic contact dermatitis.
15  may notably influence the course of chronic allergic contact dermatitis.
16 onfirmed the generation of skin TRM cells in allergic contact dermatitis.
17 ffects the risk of contact sensitization and allergic contact dermatitis.
18  immunological processes like irritative and allergic contact dermatitis.
19 t and cosmetics, and they are known to cause allergic contact dermatitis.
20  and in experimental animal studies to evoke allergic contact dermatitis.
21 ated with the development of T-cell-mediated allergic contact dermatitis.
22  lesional skin of patients with psoriasis or allergic contact dermatitis.
23  CD4+ and CD8+ T cells from individuals with allergic contact dermatitis.
24 contact dermatitis and oxazolone, a model of allergic contact dermatitis.
25 f psoriasis, fixed drug eruption, atopic and allergic contact dermatitis.
26 re examined utilizing models of irritant and allergic contact dermatitis.
27 contact hypersensitivity, an animal model of allergic contact dermatitis, a common pruritic disorder
28       We studied the role of cathelicidin in allergic contact dermatitis, a model requiring dendritic
29         However, not all individuals develop allergic contact dermatitis (ACD) although they are regu
30 contact allergens or irritants, resulting in allergic contact dermatitis (ACD) and/or irritant contac
31 ug, and we sometimes encounter patients with allergic contact dermatitis (ACD) caused by EFCZ solutio
32      The role of the innate immune system in allergic contact dermatitis (ACD) has traditionally been
33                   Atopic dermatitis (AD) and allergic contact dermatitis (ACD) have a dynamic relatio
34  Immune mechanisms underlying elicitation in allergic contact dermatitis (ACD) have yet to be fully e
35 ce to haptens is an efficient way to prevent allergic contact dermatitis (ACD) in mice.
36  reviews the recent literature pertaining to allergic contact dermatitis (ACD) in the pediatric popul
37                                              Allergic contact dermatitis (ACD) is a common T-cell med
38                                              Allergic contact dermatitis (ACD) is a prevalent and poo
39                                              Allergic contact dermatitis (ACD) is a pruritic skin dis
40                                              Allergic contact dermatitis (ACD) is classically describ
41                                              Allergic contact dermatitis (ACD) is the most common occ
42                                              Allergic contact dermatitis (ACD) is well recognized as
43                                     Further, allergic contact dermatitis (ACD) model was used to eval
44 o higher skin permeation was investigated in allergic contact dermatitis (ACD) mouse model.
45 umor-suppressive to chronic, tumor-promoting allergic contact dermatitis (ACD) revealed how tumor-pro
46  (NKT cells) in cellular infiltrate of human allergic contact dermatitis (ACD) skin challenge sites.
47 xisting irritant contact dermatitis (ICD) or allergic contact dermatitis (ACD) that is sometimes clin
48         We evaluated the effect of OA-NO2 on allergic contact dermatitis (ACD) using an established m
49 y, a crucial role of Th2 responses in nickel allergic contact dermatitis (ACD) was demonstrated.
50 eased itch in multiple preclinical models of allergic contact dermatitis (ACD), a pruritic inflammato
51                               Elicitation of allergic contact dermatitis (ACD), an inflammatory type
52 ell as irritant contact dermatitis (ICD) and allergic contact dermatitis (ACD), are common skin disea
53                             In many cases of allergic contact dermatitis (ACD), epidermal-resident me
54 t memory T (T(RM) ) cells are detrimental in allergic contact dermatitis (ACD), in which they contrib
55 nd inflammation are commonly associated with allergic contact dermatitis (ACD), it is not known if th
56                       Using a mouse model of allergic contact dermatitis (ACD), we tested the effects
57 ous chronic inflammatory diseases, including allergic contact dermatitis (ACD).
58 rgen used in hair dye that is known to cause allergic contact dermatitis (ACD).
59 levance of SUCNR1/GPR91 expression mediating allergic contact dermatitis (ACD).
60 sident memory T (T(RM)) cells play a role in allergic contact dermatitis (ACD).
61 pothesize that IL-9 also has a role in human allergic contact dermatitis (ACD).
62 cal, and immunologic similarities to chronic allergic contact dermatitis (ACD).
63 le Kv1.3 blocker (EC50=2 nM), could suppress allergic contact dermatitis (ACD).
64                                              Allergic contact dermatitis affects 20% of children at s
65 ergic reactions: food allergy, drug allergy, allergic contact dermatitis, allergic rhinitis and/or al
66                                              Allergic contact dermatitis and ICD were characterized b
67      A close association was also found with allergic contact dermatitis and increased specific IgE t
68  is an organic chemical hapten which induces allergic contact dermatitis and is used in the treatment
69                                              Allergic contact dermatitis and its animal model, contac
70 ases in which NKT cells play a role, such as allergic contact dermatitis and psoriasis.
71  patients (n = 703) presenting with possible allergic contact dermatitis and subsequently undergoing
72       Secondary outcomes included sources of allergic contact dermatitis and, for occupationally rela
73  transiently compromised, such as psoriasis, allergic contact dermatitis, and blistering disorders.
74 in diseases, including psoriasis, atopic and allergic contact dermatitis, and cutaneous T-cell lympho
75 y patient risk characteristics, incidence of allergic contact dermatitis, and incidence of wound comp
76  sensitizing chemicals in the development of allergic contact dermatitis, and suggest that the irrita
77 lergic background such as atopic dermatitis, allergic contact dermatitis, and urticaria are very comm
78 i-inflammatory activity in both irritant and allergic contact dermatitis animal models.
79                          Additional cases of allergic contact dermatitis are being reported with temp
80                        Atopic dermatitis and allergic contact dermatitis are both common skin disease
81 ich could be applicable for the treatment of allergic contact dermatitis, are still largely unknown.
82 s diagnosed with either atopic dermatitis or allergic contact dermatitis as well as in an inducible m
83 nd NPRA may provide effective treatments for allergic contact dermatitis-associated chronic pruritus.
84 by prominent barrier abnormalities (subacute allergic contact dermatitis, atopic dermatitis), topical
85  anaphylatoxin C5a is a critical mediator of allergic contact dermatitis, bridging essential aspects
86 to Ni(++) is one of the most common forms of allergic contact dermatitis, but how the T-cell receptor
87 rin barrier defects might also predispose to allergic contact dermatitis by allowing greater penetrat
88                  Herein, we report a case of allergic contact dermatitis caused by detergents contain
89                                              Allergic contact dermatitis (CD) is a chronic inflammato
90 eled MrgprA3(+) neurons under both naive and allergic contact dermatitis conditions.
91                We diagnosed the patient with allergic contact dermatitis due to the EITC and BITC pre
92 lgHrnr(-/-) mice are predisposed to mount an allergic contact dermatitis, especially at hapten thresh
93 patients suffering from psoriasis, atopic or allergic-contact dermatitis express CCR10.
94                   The role of MCs in chronic allergic contact dermatitis has not been investigated, i
95    In the past decade, mechanisms underlying allergic contact dermatitis have been intensively invest
96 responsible for nitrile rubber glove-induced allergic contact dermatitis have not been fully elucidat
97            In addition, AFC inhibits in vivo allergic contact dermatitis in a mouse model utilizing s
98                      The documented rates of allergic contact dermatitis in children are on the rise.
99  epidemiology and clinical manifestations of allergic contact dermatitis in children.
100  reviews the recent literature pertaining to allergic contact dermatitis in the pediatric population.
101 ct of topically applied THC on DNFB-mediated allergic contact dermatitis in wild-type and CB1/2 recep
102 63(-/-) mice show a stronger inflammation in allergic contact dermatitis, indicating a regulatory rol
103 sible for prolonged itch in a mouse model of allergic contact dermatitis induced by squaric acid dibu
104 s included dyskeratosis follicularis Darier, allergic contact dermatitis, infectious folliculitis, va
105                                              Allergic contact dermatitis is a chronic T cell-driven i
106                                              Allergic contact dermatitis is a common disorder that ha
107                                              Allergic contact dermatitis is a common inflammatory ski
108                                              Allergic contact dermatitis is a common skin disease ass
109                                              Allergic contact dermatitis is commonly associated with
110 ults provide support for the hypothesis that allergic contact dermatitis is not a classic delayed typ
111                                              Allergic contact dermatitis is one of the most common oc
112                                              Allergic contact dermatitis is the most frequent occupat
113 ective mechanism in those who do not develop allergic contact dermatitis is tolerance induction by re
114    Chemically induced skin sensitization, or allergic contact dermatitis, is a common occupational an
115                                              Allergic contact dermatitis may affect as many as 20% of
116 reports have suggested that chemical-induced allergic contact dermatitis may not be a traditional typ
117 nding to EC on T-cell extravasation using an allergic contact dermatitis model in mice.
118                               Nickel-induced allergic contact dermatitis (nACD) remains a major occup
119                                              Allergic contact dermatitis occurs less frequently in th
120 , females have significantly higher rates of allergic contact dermatitis on the face.
121 ne (acute irritant contact dermatitis, acute allergic contact dermatitis) or by prominent barrier abn
122 ere observed in sensory neurons of mice with allergic contact dermatitis- or dry skin-elicited itch;
123 tor-alpha agonists in models of irritant and allergic contact dermatitis produced in mouse ears by to
124 h AD, despite a trend toward weaker clinical allergic contact dermatitis reactions.
125 tcomes of CD39 deficiency in irritant versus allergic contact dermatitis, reflecting its diverse role
126 shing the clinical phenotype of irritant and allergic contact dermatitis remains challenging.
127                                              Allergic contact dermatitis, such as in response to pois
128               In a well-established model of allergic contact dermatitis, the absence of Mincle leads
129  inflammatory responses in a mouse model for allergic contact dermatitis, the contact hypersensitivit
130                            The prevalence of allergic contact dermatitis to allergens (eg, fragrance)
131  this study we use an in vivo mouse model of allergic contact dermatitis to investigate how nanoparti
132                                Prevalence of allergic contact dermatitis to MCI/MI and MI.
133                             Individuals with allergic contact dermatitis to one topical corticosteroi
134 ral minoxidil for patients with a history of allergic contact dermatitis to topical minoxidil, the lo
135 ver, the frequency of DNCB sensitization and allergic contact dermatitis to topically applied mechlor
136  pathogenesis of rosacea, atopic dermatitis, allergic contact dermatitis, urticaria, and mastocytosis
137                                  Irritant or allergic contact dermatitis usually presents as an eczem
138 n the group using white petrolatum developed allergic contact dermatitis vs 4 patients (0.9%) in the
139 Additionally, chronic itch from DNFB-induced allergic contact dermatitis was decreased by Oprm1-Vgat
140  molecular and cellular mechanism underlying allergic contact dermatitis, we evaluated oxazolone-indu
141  IV hypersensitivity reactions, resulting in allergic contact dermatitis, which clinically resembles
142 s the most frequent cause of T cell-mediated allergic contact dermatitis worldwide.

 
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