ライフサイエンスコーパス: allergy

1 "probable food allergy" or "probable no food allergy".

2 in PBMCs from individuals with milk and egg allergy.

3 s and lead to novel means of treating peanut allergy.

4 linicians improve prediction of grass pollen allergy.

5 xistent peanut, tree nut, and/or sesame seed allergy.

6 tween healthy volunteers and volunteers with allergy.

7 ide the greatest accuracy to diagnose peanut allergy.

8 ally studied, especially in subjects without allergy.

9 d in the effector phase of IgE-mediated food allergy.

10 rhinitis, asthma, and challenge-proved food allergy.

11 ne the relationship between HMO profiles and allergy.

12 ay be a relevant approach to treating cashew allergy.

13 nd immunologists who manage infants for food allergy.

14 he value of emerging therapies to treat food allergy.

15 n intact epithelial barrier in prevention of allergy.

16 tocytosis and experimental IgE-mediated food allergy.

17 lergies, anaphylaxis, insect venom, and drug allergy.

18 g sera from 18 subjects with a proven almond allergy.

19 a promising alternative for preventing milk allergy.

20 diagnostic marker allergen for poultry meat allergy.

21 understanding of reaction severity in peanut allergy.

22 n of allergy in a murine model of cow's milk allergy.

23 l population has a reported penicillin (PCN) allergy.

24 m 48 Ara h 2-sensitized patients with peanut allergy.

25 architecture of reaction severity in peanut allergy.

26 en and can affect prevalence of occupational allergy.

27 ulation in mast cells, implicating a role in allergy.

28 inate the principal responses which underlie allergy.

29 reflect the low frequency of true penicillin allergy.

30 inform strategies for the prevention of food allergy.

31 I gene HLA-B in the occurrence of penicillin allergy.

32 ic diseases and in 90% regarding respiratory allergies.

33 is a proven, safe treatment for respiratory allergies.

34 He did not have any allergies.

35 mber of people suffer from IgE-mediated food allergies.

36 loped to guide clinical diagnosis of contact allergies.

37 , frequently occurring with nasal polyps and allergies.

38 ng chronic inflammatory disorders, including allergies.

39 ave a high prevalence of reported antibiotic allergies.

40 ergy and from non-atopic individuals without allergies.

41 e limited applicability for users with metal allergies.

42 iated with an increased odds of multiple nut allergies.

43 iotics for patients with specific antibiotic allergies.

44 ially beneficial immunomodulatory effects in allergies.

45 nt resources to address inpatient penicillin allergies.

46 ancial gain and food-toxicities such as meat allergies.

47 third of the world's population suffers from allergies(1).

48 Stratification of patients with HDM allergy according to molecular sensitization profiles an

49 Egg allergy affects almost 1 in 10 Australian infants.

50 deficiency are often present, in some cases allergy alone is the presenting symptom, suggesting that

51 to PCNs alone, 5 (9.4%) had a cephalosporin allergy alone, and 2 (3.8%) had reported allergies to bo

52 erapy, asthma, dermatological diseases, food allergies, anaphylaxis, insect venom, and drug allergy.

53 ast cell (MC)-associated diseases, including allergy/anaphylaxis and neuroinflammatory pain disorders

54 ng AYA to let their friends know about their allergies and asthma.

55 ecipients, 634 (29%) reported any antibiotic allergy and 347 (16%) reported BLAs.

56 ast cells and for the manifestations of food allergy and anaphylaxis.

57 Implementation of a perioperative allergy and antibiotic assessment tool in patients with

58 Specific allergy and asthma transition recommendations include (a

59 hypersensitivity by the European Academy of Allergy and Clinical Immunology (EAACI) differentiates b

60 A recent survey of the European Academy of Allergy and Clinical Immunology (EAACI) Drug Allergy Int

61 A position paper by the European Academy of Allergy and Clinical Immunology (EAACI) proposed season

62 The European Academy of Allergy and Clinical Immunology created a task force to

63 e evidence to inform the European Academy of Allergy and Clinical Immunology's (EAACI) anaphylaxis gu

64 of total IgE from individuals with a peanut allergy and from non-atopic individuals without allergie

65 ther understanding of the mechanisms of food allergy and identification of biomarkers to distinguish

66 Clinician-educators in the field of allergy and immunology (A/I) in the United States teach

67 ma, Allergy & Immunology, board certified in allergy and immunology.

68 conducted in patients reporting a penicillin allergy and in whom skin tests and/or specific IgE quant

69 n immunotherapy in birch pollen-related food allergy and indicate a dominant protective role of funct

70 ada (PDF-532926-2019), National Institute of Allergy and Infectious Disease (R21 AI145356, R21 AI1523

71 earch Foundation), the National Institute of Allergy and Infectious Diseases (NIAID), the European Un

72 US National Institute of Allergy and Infectious Diseases and National Cancer Inst

73 US National Institute of Allergy and Infectious Diseases and National Cancer Inst

74 arch that supports the National Institute of Allergy and Infectious Diseases research agenda.

75 amural Research at the National Institute of Allergy and Infectious Diseases, and National Institutes

76 In 2018, the National Institute of Allergy and Infectious Diseases, Division of Allergy, Im

77 National Institute of Allergy and Infectious Diseases, Johns Hopkins Health Sy

78 US National Institute of Allergy and Infectious Diseases.

79 Human Development and National Institute of Allergy and Infectious Diseases.

80 and Skin Diseases; the National Institute of Allergy and Infectious Diseases; Rheumatology Research F

81 Vespid venom allergy and male sex likewise augment the risk of severe

82 be a relevant and safe treatment for peanut allergy and may improve the quality of life for many pea

83 an unexpected helper role in promoting food allergy and may represent a target for drug development.

84 ical as described by Clinical & Experimental Allergy and other journals in 2019.

85 allergic rhinitis (SAR) and subjects without allergy and tested for cross-sectional and interseasonal

86 cells, determining immune responses such as allergy and tolerance.

87 re the clinical spectrum of non-IgE-mediated allergies, and part of its objectives was to establish d

88 n alter the course of childhood asthma, food allergy, and allergic rhinosinusitis.

89 rafficking in inflammatory immune responses, allergy, and cancer.

90 by cesarean section is associated with food allergy, and consumption of fermented milk products is a

91 isposition for intestinal inflammation, food allergy, and failure to thrive, often necessitating nutr

92 ma, allergic rhinitis, food allergy, contact allergy, and hand eczema.

93 ical processes, such as inflammation, fever, allergy, and pain, their roles in COVID-19 are poorly ch

94 ith progression from AD to IgE-mediated food allergy, and white race was associated with progression

95 In mice with allergy, anti-IL-4Ralpha provided long-lasting suppressi

96 Food allergies are a major clinical problem and are driven by

97 Allergies are highly prevalent, and allergic responses c

98 Peanut, tree nut, and sesame allergies are responsible for most life-threatening food

99 Allergies are typically endemic, complex and heterogeneo

100 e and safer immunotherapies to manage peanut allergy are in great demand despite extensive investigat

101 So far, the molecules causing poultry meat allergy are largely unknown.

102 It has been suggested that asthma and allergy are not highly represented as comorbid condition

103 ency department, a pharmacist-led penicillin allergy assessment via medical records review and patien

104 rding this practice, the American Academy of Allergy, Asthma & Immunology Primary Immunodeficiency Di

105 ears to be associated with a higher risk for allergy, asthma and wheezing.

106 sk of multiple comorbidities, including food allergy, asthma, allergic rhinitis, and mental health di

107 record data for clinical research concerning allergy, asthma, and immunology and highlights the poten

108 logical heterogeneity in research concerning allergy, asthma, and immunology.

109 ly egg introduction and (b) did not have egg allergy at 12 months.

110 e their associations with respiratory health/allergy at 8 years in the PARIS birth cohort.

111 s have demonstrated that diagnosis of almond allergy based on extract-sIgE tests displays low specifi

112 With food allergy being a major health problem of increasing conce

113 nstrate that daily EPIT treatment for peanut allergy beyond 1 year leads to continued response from a

114 n concentrations as reported by the National Allergy Bureau across 25 U.S. regions from 2012-2017, in

115 llen AIT is not only related to birch pollen allergy but extends to pollen from other trees, especial

116 Clinicians should consider that this allergy can occur indoors, unlike allergies to other typ

117 n prevention measures offered in specialized allergy centers (84% of patients were prescribed adrenal

118 is study compares both clinical and in vitro allergy characteristics but also diagnostic performance

119 s (MeDALL [Mechanisms for the Development of Allergies] chip) and to timothy grass extract (ImmunoCAP

120 commended that patients visit our outpatient allergy clinic.

121 quently, 133 patients visited our outpatient allergy clinic.

122 formally trained practitioners in specialist allergy clinics.

123 munologically confirmed, were recruited from allergy clinics.

124 atitis compared to children from a high-risk allergy cohort with comparable rates of parental atopy.

125 nts or specialist nurses formally trained in allergy compared with consultants with no formal allergy

126 pecialist pound 150.9 (138.8-163.2), trained allergy consultants pound 218.7 (205.7-231.9), compared

127 dermatitis, asthma, allergic rhinitis, food allergy, contact allergy, and hand eczema.

128 Operationalizing penicillin allergy de-labeling as an aspect of ASP has become an in

129 Operationalizing penicillin allergy de-labeling into a new arm of antimicrobial stew

130 urrent approaches and barriers to penicillin allergy de-labeling.

131 Collaboration between emergency medicine and allergy departments may be helpful for improving the pat

132 PLUS Environmental and Genetic Influences on Allergy Development (GINIplus) (Germany), and Prevention

133 As infant feeding may influence allergy development, we aimed to identify groups of infa

134 M consensus guidance to develop a novel food allergy diagnosis algorithm using available information

135 Molecular allergy diagnosis is expected to improve accuracy, but i

136 rate for further development of an automated allergy diagnosis system.

137 I task force survey, the current practice of allergy diagnosis was shown to rely on skin tests as fir

138 elopments in allergen and epitope discovery, allergy diagnostics and immunotherapy.

139 tients with MDH, tested and confirmed at our allergy division, were retrospectively evaluated in term

140 the current recording system, and to a poor allergy education.

141 ing formal skin testing to tackle penicillin allergy efficiently within complex healthcare systems.

142 time, there is great promise for penicillin allergy evaluation and de-labeling as an individual and

143 The prevalence of food allergy (FA) among European school children is poorly de

144 rtance of patient history in diagnosing food allergy (FA) and the need for studies investigating its

145 We show that Treg cells in food allergy (FA) had decreased expression of transforming gr

146 The steep rise in food allergy (FA) has evoked environmental factors involved i

147 Food allergy (FA) is a significant public health issue, prope

148 s investigating its value using standardized allergy-focused questionnaires.

149 ormability of WPEDs enables their simple and allergy-free attachment to skin, where they can monitor

150 Patients with diagnosis of LTP allergy from the Allergy Unit of Hospital Regional Unive

151 e end of January 2020, no treatment for food allergies had been approved by the US Food and Drug Admi

152 Skin-based immunotherapy of type 1 allergies has recently been re-investigated as an altern

153 Food allergies have become a significant heath burden as prev

154 Occupational allergy health promotion strategies need to consider app

155 ical Association and the Joint Task Force on Allergy Immunology Practice Parameters.

156 S members of the American Academy of Asthma, Allergy & Immunology, board certified in allergy and imm

157 Allergy and Infectious Diseases, Division of Allergy, Immunology and Transplantation, sponsored a wor

158 nal experts from the fields of tick biology, allergy, immunology, infectious disease, and dermatology

159 iation Institute and the Joint Task Force on Allergy-Immunology Practice Parameters to provide a tech

160 ent fever disorders clinic established in an Allergy/Immunology division at a tertiary pediatric care

161 There is evidence that allergy immunotherapy (AIT) prevents the progression of

162 pite involving the same causative allergens, allergy immunotherapy (AIT) treatment habits differ sign

163 ative species regarding diagnosis as well as allergy immunotherapy.

164 Peanut allergy imposes an adverse psychosocial impact on patien

165 Oral immunotherapy (OIT) for food allergy improves the quality of life (QOL) of children f

166 eoperative apical scarring and environmental allergies in a multivariable linear regression model to

167 diagnosis and management of non-IgE-mediated allergies in breastfed infants.

168 ing to the International Study of Asthma and Allergies in Childhood (ISAAC) questionnaire.

169 n the Human Immune System and Development of Allergies in Childhood (LISA)/German Infant Study on the

170 associations with subsequent development of allergies in infants and children.

171 Ragweed frequently causes seasonal allergies in North America and Europe.

172 of allergic rhinitis, asthma and other food allergies in PwPA were 50%, 42% and 79%, respectively.

173 echnology to provide long-term modulation of allergy in a murine model of cow's milk allergy.

174 with high sensitivity for IgE-mediated food allergy in clinical study participants who do not underg

175 This may be a useful tool for excluding food allergy in future clinical studies where OFC is not cond

176 fe for primary prevention of eczema and food allergy in high-risk infants (ISRCTN21528841).

177 otential confounders a family history of any allergy in parents, type of delivery, having siblings, k

178 ith allergic sensitization and clinical food allergy in the first year of life.

179 ove the predictive character of existing BLC-allergy in vitro tests.

180 unrecognized early indicator of grass pollen allergy, in addition to Phl p 1.

181 Allergy and Clinical Immunology (EAACI) Drug Allergy Interest Group (DAIG) on how European allergy sp

182 etween temporary and permanent resolution of allergies is needed before a cure, where reactivity to t

183 ingredient or technological adjuvant causing allergies is required by EU food legislation.

184 Peanut allergy is a growing public concern; however, little is

185 Food allergy is a major health problem affecting 5% to 10% of

186 Food allergy is associated with a high personal health and ec

187 cific immunotherapy for patients with peanut allergy is available.

188 Peanut allergy is characterized by the development of IgE again

189 e response and a modulatory impact on type I allergy is discussed.

190 More critically, cashew allergy is frequently associated with severe anaphylaxis

191 Food allergy is the most common cause of anaphylaxis.

192 The mammalian meat allergy known as the "alpha-Gal syndrome" relates to IgE

193 ative review of the literature of penicillin allergy label carriage, the adverse effects of penicilli

194 carriage, the adverse effects of penicillin allergy labels, and current approaches and barriers to p

195 For each food allergy, linear mixed-effects models were built to test

196 The potentially fatal consequence of food allergies makes managing them costly and anxiety-inducin

197 Due to over-reporting of BL allergies, many patients may not receive guideline-direc

198 ith suspected bacterial endophthalmitis, PCN allergy may not be an absolute contraindication to intra

199 ting in the Mechanisms of the Development of ALLergy [MeDALL] consortium were included in this meta-a

200 Humanized TCR/HLA-transgenic allergy mice were treated in vivo with recombinant IL-2

201 analysis of the following studies: Children, Allergy, Milieu, Stockholm, Epidemiology (BAMSE) (Sweden

202 es a tolerogenic immune response in the food allergy model and in human immune cells.

203 teins on the immune response in a mouse food allergy model and on human monocyte-derived dendritic ce

204 n reported as a causative agent of immediate allergy, most of them are adult women.

205 ion restrictions (n = 21 [36%]), followed by allergies (n = 13 [22%]).

206 atients presented with atopic symptoms (food allergy, n = 13; hay fever, n = 10; asthma, n = 7).

207 es in human diseases such as cancer, asthma, allergy, neurodegeneration, and autoimmune diseases they

208 tudy comprised all the patients attended the Allergy of the University Hospital of Montpellier (Franc

209 ract infection (47%), pharyngitis (18%), and allergy or asthma (11%).

210 line, search terms used included "penicillin allergy" or "penicillin hypersensitivity" alone or in co

211 ugh to categorize children as "probable food allergy" or "probable no food allergy".

212 and offspring's asthma with or without nasal allergies (outcomes) was analyzed by using 2-level logis

213 Non-IgE-mediated allergy, outside of food protein-induced allergic procto

214 nce of rhinitis (p < .001) and previous food allergy (p < .001), together with a higher proportion of

215 Peanut allergy (PA) is associated with marked quality-of-life (

216 d research has examined the impact of peanut allergy (PA) on children using validated instruments to

217 ) is the criterion standard to assess peanut allergy (PA), but it involves a risk of allergic reactio

218 Prevention and Incidence of Asthma and Mite Allergy (PIAMA) (The Netherlands).

219 als evaluating probiotic supplementation for allergy prevention in very preterm infants are needed.

220 rld and the application of this knowledge to allergy prevention strategies.

221 The allergy preventive effects of gut immune modulation by b

222 silonRIalpha; mice that additionally have an allergy-promoting IL-4Ralpha mutation; and hu cord blood

223 schistosome and invertebrates in inducing an allergy-protective effect, as proposed by the hygiene hy

224 The Food Allergy Quality of Life Questionnaire-Child Form (FAQLQ-

225 Hymenoptera venom allergy ranks among the top three causes of anaphylaxis

226 require interventions to prevent increasing allergy rates in countries undergoing rapid urbanization

227 racts is a risk factor for, and mediator of, allergy-related diseases (ARDs).

228 A syndrome of mammalian meat allergy relating to IgE specific for galactose-alpha-1,3

229 IgE-mediated food allergy remains a significant and growing problem across

230 accuracy, but its value in diagnosing almond allergy remains unknown.

231 Immunotherapy for food allergy requires prolonged treatment protocols and, in m

232 Successful prevention of food allergy requires the identification of the factors adver

233 Consortium for Food Allergy Research investigators previously reported 52-we

234 t tool in patients with reported beta-lactam allergy resulted in a pronounced and sustained increase

235 nstitution-based models of IgE-mediated food allergy revealed an IL-4 signaling-dependent cell-intrin

236 Here, we review the most important allergy risk factors associated with changes in our expo

237 in mice to be responsible for different egg allergy risk.

238 In this high-allergy-risk birth cohort, some profiles of HMOs were as

239 ollected from 285 mothers enrolled in a high-allergy-risk birth cohort, the Melbourne Atopy Cohort St

240 ith which could make it suitable for massive allergy screening campaigns to better define sensitizati

241 f Oral Tolerance to Peanut study, and Peanut Allergy Sensitization study participants by administerin

242 nsic effect on SI MMC9 accumulation and food allergy severity.

243 Hypersensitivity reaction rate after allergy skin testing (17%; 95% CI: 7%, 29%; zero of 21 s

244 e the effect of using an alternative GBCA or allergy skin testing on the risk of a breakthrough react

245 class, switching to an alternative GBCA, and allergy skin testing were assessed.

246 f many more mammals than have ever died from allergy, so justifying the positive role of IgE in our e

247 llergy Interest Group (DAIG) on how European allergy specialists deal with beta-lactam (BL) hypersens

248 ons were used to compare groups based on egg allergy status and comorbidities.

249 15-month or 2- to 3-year samples can predict allergy status at age 4(+) years.

250 clinically relevant IgE cutoffs, predicting allergy status on an "unseen" set of patients with area

251 We assessed Learning Early about Peanut Allergy study, Persistance of Oral Tolerance to Peanut s

252 prove the health and quality of life of food allergy sufferers.

253 ives a substantial reduction in grass pollen allergy symptoms and use of rescue medication, significa

254 origin and evolution of SLIT for respiratory allergy, taking into account the clinical context that s

255 ant for patients with anaphylaxis to undergo allergy testing after discharge from an emergency depart

256 Food allergy testing is not recommended in the routine evalua

257 Currently, diagnostic regimes rely on allergy testing, exposure specification, and follow-up v

258 stly by clinical history and peanut-specific allergy testing.

259 ed at baseline with detailed questionnaires, allergy tests, and lung function tests and were reassess

260 y suppress IgE-mediated anaphylaxis and food allergy than divalent variants of the same mAbs do.

261 care costs, and less likelihood of inducing allergy than gold.

262 tial risk factor for the development of food allergy, that is, D pteronyssinus allergens in breast mi

263 provoke immunity to infectious agents, as in allergy the patient is presensitized to the antigen.

264 imize diagnosis and treatment of tree pollen allergy, the experts recommend to focus diagnosis and re

265 However, the effect of anti-allergy therapy on MCNS remains unknown.

266 esented reaction only with peach and (b) LTP-Allergy: those that presented reaction with peach and at

267 B cells play an important role in allergies through secretion of IgE.

268 role in the development of IgE-mediated food allergies through the production of allergen-specific an

269 ed for the majority of individuals with food allergy through immunotherapy, continued ingestion of al

270 rin allergy alone, and 2 (3.8%) had reported allergies to both PCN and cephalosporin antibiotics.

271 that this allergy can occur indoors, unlike allergies to other types of venom.

272 ved intravitreal ceftazidime, 46 (86.8%) had allergies to PCNs alone, 5 (9.4%) had a cephalosporin al

273 Allergies to peanuts, tree nuts, and sesame seeds are am

274 Allergy to bites of blood-sucking insects, including bit

275 for IBH and could be useful for the study of allergy to biting midges in humans and other species.

276 Allergy to insects of the family Tabanidae (order Dipter

277 library and sera from patients with clinical allergy to pine nut in order to deepen into the allergen

278 Allergy to stainless steel was identified in 13.7%.

279 nd after sublingual immunotherapy (SLIT) for allergy to temperate grass pollen, predominantly to ryeg

280 rgy compared with consultants with no formal allergy training [Willingness to pay (WTP) estimates for

281 f hematopoietic stem cell transplantation on allergy transfer or cure of the disease.

282 Allergy transfer upon solid organ transplantation has be

283 ocking IgG1 antibodies in the early phase of allergy treatment.

284 with at least 1 confirmed nut or sesame seed allergy underwent sequential diagnostic food challenges

285 tric patients with clinically confirmed fish allergy underwent skin prick tests to salmon and catfish

286 ients with diagnosis of LTP allergy from the Allergy Unit of Hospital Regional Universitario de Malag

287 large number of patients suffering from food allergies, up until the end of January 2020, no treatmen

288 erpetiformis (DH), gluten ataxia (GA), wheat allergy (WA), and non-celiac gluten sensitivity (NCGS) a

289 No effect on offspring's asthma with nasal allergies was found.

290 preference value set for patients with food allergy was determined using the SF-6Dv2 generic questio

291 Food allergy was induced in young pigs using two protocols.

292 Peanut allergy was more common in London, cashew and pistachio

293 e common in London, cashew and pistachio nut allergies were more common in Geneva, and walnut and pec

294 more common in Geneva, and walnut and pecan allergies were more common in Valencia.

295 x; (b) Psychological factors-asthma and food allergy were associated with anxiety and depression, ato

296 Cumulative symptoms in those without allergy were lower than in those with SAR but followed t

297 Most patients with peanut allergy were sensitized to both Ara h 2 and Ara h 6.

298 llergic reactions in a mouse model of cashew allergy, which suggests that EPIT may be a relevant appr

299 n to Pru du 6 was highly specific for almond allergy, while frequencies of sensitization to legumins

300 80 adult patients with a reported penicillin allergy who received antibiotics at a community hospital