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1 her commonly used molecular markers, such as allozymes.
2 polygyne (multiple queen) social forms using allozymes.
3  polymorphism with respect to the common A/B allozymes.
4 fragment length polymorphism (RFLP) data and allozymes.
5 sly generated based on mitochondrial DNA and allozymes.
6 atellite loci (0.016) was lower than that of allozymes (0.036).
7 rison with the wild-type SULT2B1a, of the 13 allozymes, 11 showed reduced activity toward pregnenolon
8 on the sulfation of pregnenolone by SULT2B1a allozymes, 13 recombinant SULT2B1a allozymes were genera
9 ampling sites in a manner similar to that of allozyme allele frequencies in Aequipecten opercularis,
10 in the absence of latitudinal clines for PGM allozyme alleles, the lack of association of PGM allozym
11 amino acid polymorphism segregating with the allozyme alleles.
12 lation showed significant differentiation in allozymes among neighbouring subpopulations.
13                                              Allozyme analysis revealed a high degree of fixed hetero
14 ing substantial gametic disequilibrium among allozyme and complementary DNA (cDNA) markers encompassi
15 and UV-irradiated experimental regimes using allozyme and DNA restriction fragment length polymorphis
16 ologically indistinguishable, and surveys of allozyme and DNA sequence variation have revealed signif
17                                     Overall, allozyme and ISSR techniques yielded similar results, in
18                      The conclusion from the allozyme and life history data is that the flowering dog
19  divergence is consistent with evidence from allozyme and microsatellite data and is interpreted in t
20                              Comparison with allozyme and mitochondrial DNA restriction fragment leng
21                           As expected, mesic allozyme and morphological markers are much more common
22                                              Allozyme and PCR-based molecular markers have been widel
23 nt distribution of Fst estimates across both allozyme and RAPD loci.
24                             We have now used allozyme and restriction fragment length polymorphism an
25  measure the extent of founder effects using allozymes and microsatellites, and ask whether this is c
26 nvicta using two classes of nuclear markers, allozymes and microsatellites, and markers of the mitoch
27 ilibrium that underlies associations between allozymes and susceptibility.
28 e relies on linkage between genetic markers (allozymes) and resistance loci.
29 tus) in 310 loci, including microsatellites, allozymes, and single nucleotide polymorphisms.
30 ed with constructs for wild type and variant allozymes; and recombinant proteins were measured by qua
31 dy of nucleotide variation observed that PGM allozymes are a heterogeneous mixture of amino acid poly
32 mon allozyme mobility alleles, we find these allozymes are the result of many different amino acid ch
33 he derived A allele, and differences between allozymes are twice as among B alleles.
34        Much of the scepticism that surrounds allozyme-associated heterosis comes from inconsistent an
35  allozyme-associated heterosis, and that the allozyme-associated heterosis detected in this study was
36                                              Allozyme-associated heterosis for growth rate was detect
37  in order to evaluate possible mechanisms of allozyme-associated heterosis for growth rate.
38 uating the genetic mechanisms that may cause allozyme-associated heterosis in natural populations has
39 ry for predictive power and repeatability of allozyme-associated heterosis, and that the allozyme-ass
40 e importance of heterosis, and in particular allozyme-associated heterosis, in natural populations re
41 to human genetic diseases.We looked for ACO2 allozymes by DNA sequencing and genotyping in a populati
42                   The novel recombinant NAT2 allozymes catalyzed both N- and O-acetyltransferase acti
43 ever, strongly disagreed with those based on allozymes, chloroplast DNA, and morphological traits.
44 e-altering amino acid polymorphism, and both allozyme classes correspond to two haplogroups with mult
45                        In addition, specific allozyme classes within species contain several amino ac
46 n Semibalanus cariosus We show that the slow allozyme contains a derived charge-altering amino acid p
47                  Alleles at all six of these allozymes correlate with the timing of adult eclosion, a
48                 We demonstrate the method on allozyme data from two species of hybridizing trout, as
49                                     Previous allozyme data have suggested that there is a small but s
50 ess SA is observed for S locus data than for allozyme data in accordance with the hypothesis that SSI
51 e Me-2 locus and by F-statistics for all the allozyme data, is evidence of some local relatedness due
52 e replacement mutation that accounts for the allozyme difference.
53                                 The observed allozyme differences and surprisingly low estimates of a
54                                     No fixed allozyme differences distinguish the flowering dogwood f
55 on for transfection efficiency, the Trp(173) allozyme displayed 31%, Thr(287) 350%, Ile(306) 4.8%, an
56                                   The Arg146 allozyme displayed a significant decrease in protein qua
57 cells harboring these same cytosolic variant allozymes displayed significant increases in the EC(50)
58                  The effect of inbreeding on allozyme diversity is consistently larger than has been
59                                              Allozyme diversity was surveyed in 19 putative loci over
60                                    Levels of allozyme diversity were studied in five species of the a
61 n aggregation in vitro for the three variant allozymes encoded by alleles that include the two TPMT*3
62 ely congruent to those produced by mtDNA and allozymes, except that the satellite DNA yields much lon
63                   Patients with TPMT variant allozymes exhibit diminished levels of protein and/or en
64           The Ile(306) and Thr(287)/Ile(306) allozymes expressed too little activity for inclusion in
65 s virtually identical (0.036) to the average allozyme FST.
66 conitase, we wished to find common ACO2 gene allozymes, functional polymorphisms that might be associ
67                                          The allozyme genetic structure of five populations (from Jap
68             Previous work has shown that Mpi allozyme genotypes (fast and slow) have different freque
69  effect of ontogeny on relationships between allozyme genotypes and fresh weight was measured weekly
70                                 Four variant allozymes had K(m) values that differed significantly fr
71                     An early experiment with allozymes had shown linkage disequilibrium between these
72  showing low levels of allelic diversity (eg allozymes) has been well characterised and evaluated.
73                  Most work to date, based on allozymes, has involved pattern analysis for individual
74 umulative effect was an increase in apparent allozyme heterosis later in ontogeny coinciding with a s
75 he glaciated history of the region, very low allozyme heterozygosity was found in these taxa (Ho = 0.
76                           The rs5063 variant allozyme in transfected HEK293 cells was decreased to 55
77 racterize spatial genetic differentiation of allozymes in adult Delphinium nuttallianum plants within
78            Earlier studies of the common PGM allozymes in Drosophila melanogaster reported no in vitr
79 he failure to detect differences between PGM allozymes in functional studies.
80 bata) was widely studied in California using allozymes in the 1970s and interpreted as a case of ecot
81 lity of maternally (mtDNA) and biparentally (allozymes) inherited genes of 443 chamois (Rupicapra r.
82  Here, fixation, segregation, and linkage of allozymes is investigated in the progeny of self-fertili
83                The previously reported Est-6 allozyme latitudinal clines may be accounted for by the
84 ter, the investigators report the results of allozyme-level analyses of 2 lines derived from hybridiz
85 ulation that was heterozygous for two of the allozyme loci (Sharp Spring).
86 .e., all eight microsatellite loci, 12 of 16 allozyme loci and 44% of the 285 SNPs).
87 ytonuclear disequilibrium between 11 nuclear allozyme loci and both mitochondrial and chloroplast DNA
88 a population that was monomorphic for all 25 allozyme loci examined (Monkey Spring) than for fish fro
89                                      We used allozyme loci for a paternity analysis of 518 seeds prod
90 s k statistic to describe the variance among allozyme loci in 102 published data sets from fishes.
91               Electrophoretic analysis of 17 allozyme loci in 21 populations reveals significant freq
92  design was used to survey eight polymorphic allozyme loci in the parent and offspring generations of
93                   Allelic variation for five allozyme loci is also consistent with the Spain-rest of
94 ferences from the hawthorn host race for six allozyme loci mapping to three different chromosomes.
95 the genetic structure of the nuclear-encoded allozyme loci should reflect the movement of both seeds
96                                   Two of six allozyme loci showed a significant increase in apparent
97                                        Three allozyme loci showed significant associations with mtDNA
98                           Results from eight allozyme loci showed that there was considerable genetic
99 sure greater interpopulation divergence than allozyme loci suggested constraints on microsatellite ev
100   Allelic frequency differentiation at seven allozyme loci was relatively homogeneous across loci and
101  was evident in two of four populations, and allozyme loci were characterized by temporal variation i
102       Spatial autocorrelation analyses of 12 allozyme loci were used to compare genetic structure wit
103  same populations based on seven polymorphic allozyme loci yielded an estimate of Fst of 0.13.
104 well beyond the previously characterized six allozyme loci, covering substantial portions of chromoso
105 as not paralleled by allelic variation at 33 allozyme loci, which were used as nuclear DNA markers.
106 al Pennsylvania, based on variation at eight allozyme loci.
107 pulations at chloroplast markers but not for allozyme loci.
108 rkers and local heterozygote deficiencies at allozyme loci.
109 imates calculated from published data on six allozyme loci.
110 hort tandem repeat (microsatellite) loci and allozyme loci.
111 e loci directly or on loci tightly linked to allozyme loci.
112 ulation diversity is substantial for nuclear allozyme markers (mean P=0.610, A(e)=1.580, H(e)=0.277)
113 was derived from 251 microsatellite and four allozyme markers and covers 3350 cM (approximately 90% o
114 ts recombination fractions with ES1 and MDH2 allozyme markers consistent with previous localization o
115            Thus, fixation and segregation of allozyme markers in inbred oyster families deviates from
116 oral population structure of chloroplast and allozyme markers in the annual plant Phacelia dubia.
117                Spatial analysis of mtDNA and allozyme markers revealed strong geographical structure
118 ysis of 288 microsatellite polymorphisms, 13 allozyme markers, and phenotypic sex in four backcross f
119       The method is appropriate for use with allozymes, microsatellites, or restriction fragment leng
120 D. melanogaster and D. simulans share common allozyme mobility alleles, we find these allozymes are t
121 tected polymorphisms that would lead to ACO2 allozymes, nor have we observed differences in ACO2 isof
122 (m) values for sodium arsenite with the same allozymes of 11.8, 8.9, and 4.5mum.
123                           Of the recombinant allozymes, only the double mutant (Arg39Cys264) displaye
124  However, estimates of U based on changes in allozymes or DNA sequences and fitness traits are discor
125  collected from natural populations, and the allozyme phenotypes of infected hosts were compared to t
126  parallels the paradox described earlier for allozyme polymorphism and challenges simple equilibrium
127 an amino acid variant, leading to the common allozyme polymorphism at Gld, remains in low to intermed
128 utocorrelation (SA) treatment of S locus and allozyme polymorphism data for four loci indicates that
129 sential to understanding the significance of allozyme polymorphism in adaptation.
130 the Pro/Leu mutation responsible for the A/B allozyme polymorphism in each variant.
131 t polymorphism, which corresponds to the S-F allozyme polymorphism.
132 morphisms, one of which accounts for the S-F allozyme polymorphism.
133 lected site is responsible for the EST-6 F/S allozyme polymorphism; the same site is responsible for
134 s significant clinal differentiation for the allozyme polymorphisms as well as for many of the crypti
135 id mutations responsible for the common G6PD allozyme polymorphisms in Drosophila melanogaster have b
136 uency-dependent selection protecting natural allozyme polymorphisms through differential resource uti
137 e phosphoglucomutase (PGM) possesses several allozyme polymorphisms, it is unique in that it had been
138 ults are consistent with the hypothesis that allozyme population structure is primarily the result of
139 e loci was significantly higher than that of allozymes, probably because the former experience consid
140                Specifically, P134L and R259Q allozymes, reported to be involved in autosomal-recessiv
141                                          The allozyme results only partially supported the patterns r
142 s264, Thr364, and double variant Arg39Cys264 allozymes showed significant decreases in levels of acti
143 hila willistoni was the subject of intensive allozyme studies and the locus coding for alcohol dehydr
144 ogen metabolism and is highly polymorphic in allozyme studies of many species.
145                                              Allozyme studies revealed complete reproductive isolatio
146       A combination of field experiments and allozyme studies was used to test whether two spring-eph
147 ore than twice the level detected in earlier allozyme surveys.
148                                        Using allozyme techniques, we uncovered genetic differentiatio
149  on the sulfating activity of coded SULT2B1a allozymes toward Pregnenolone.
150                                          ADH allozyme type has no detectable effect on development.
151 s of QTL suggests that coding variants (e.g. allozymes) underlie a fraction of quantitative variation
152 ues, but there was nevertheless considerable allozyme variability in two of the four sets of inbred p
153  multiple continents for several phenotypes, allozyme variants, sequence variants, and chromosome inv
154  Canary Islands was investigated by studying allozyme variation at nine putative loci in five populat
155                                   We studied allozyme variation in metapopulations of two species of
156                                   We studied allozyme variation in rock pool metapopulations of two s
157 al outcrossing distances and the patterns of allozyme variation in T. erectum and T. grandiflorum pop
158                                        Here, allozyme variation is analysed in over 1100 individuals
159 bit a positive correlation between mtDNA and allozyme variation, suggesting that mtDNA diversity may
160 t contrasts sharply with previous studies of allozyme variation, which have showed significantly less
161 ll (South America) associated with the Est-6 allozyme variation.
162 hich are not perfectly associated with Est-6 allozyme variation.
163 s linked to Thr(287), so this double variant allozyme was also studied functionally.
164 The intracellular stability of the *1 and *2 allozymes was examined in insect as well as mammalian ce
165                       Using four polymorphic allozymes, we compared the outcrossing-rate estimates of
166                            Purified SULT2B1a allozymes were analyzed for sulfating activities towards
167  SULT2B1a allozymes, 13 recombinant SULT2B1a allozymes were generated, expressed, and purified using
168         The enzyme activities of the SULT1A1 allozymes were studied with a variety of substrates, inc
169                                GSTP1 variant allozymes were then expressed in COS-1 cells, and five d
170 rotein level was also observed for the Arg39 allozyme, whereas Met201 displayed no significant change
171  similar to the wild-type cytosolic (WT Cyt) allozyme, whereas the R424C and S457F cytosolic variants
172 xpression vector, encoding these 13 SULT2B1a allozymes, which were bacterially expressed in BL21 E. c
173 zyme alleles, the lack of association of PGM allozymes with the cosmopolitan In(3L)P inversion, and t
174  protein was observed for the Arg146 variant allozyme, with 20% of wild-type protein, primarily becau
175 ) 6.2% of the activity of the wild type (WT) allozyme, with 20, 190, 4.4, and 7.9% of the level of WT
176 1, and 11 mum for WT, Trp(173), and Thr(287) allozymes, with K(m) values for sodium arsenite with the

 
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