ライフサイエンスコーパス: alpha-smooth muscle actin

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1 onectin as well as the myofibroblast marker, alpha-smooth muscle actin.

2 receptor tyrosine kinase 2, fibronectin, and alpha-smooth muscle actin.

3 ated positively with that of fibronectin and alpha-smooth muscle actin.

4 1 in NL-fibroblasts increased collagen-I and alpha-smooth muscle actin.

5 oduction of extracellular matrix proteins or alpha-smooth muscle actin.

6 lphav, beta3, beta1, alpha2(I) collagen, and alpha-smooth muscle actin.

7 in and acquisition of the mesenchymal marker alpha-smooth muscle actin.

8 nail, Slug/Snai2, vimentin, fibronectin, and alpha-smooth muscle actin.

9 markers, including collagen IV, laminin, and alpha-smooth muscle actin.

10 lso formed vascular structures and expressed alpha-smooth muscle actin.

11 nduce expression of the myofibroblast marker alpha-smooth muscle actin.

12 induced a fibroblast phenotype negative for alpha-smooth muscle actin.

13 bers of activated fibroblasts that expressed alpha-smooth muscle actin.

14 infusion also reduced hepatic expression of alpha-smooth muscle actin (0.19 +/- 0.007-fold compared

15 y indicator, calponin loss intermediate, and alpha-smooth muscle actin a later indicator of compromis

16 y cancer cells expressed increased levels of alpha-smooth muscle actin, a marker of CAF, compared wit

17 distinct ASC plaques that were positive for alpha-smooth muscle actin, a marker of EMT.

18 nsitometry analyses showed less staining for alpha-smooth muscle actin, a myofibroblast marker, in th

19 Interestingly, LPS down-modulated alpha-smooth muscle actin (activated HSC marker) and col

20 1, only submesothelial fibroblasts expressed alpha-smooth muscle actin after induction of peritoneal

21 ne in TACE activity, procollagen alpha1 (I), alpha smooth muscle actin (alpha-SMA) and transforming g

22 e TGF-beta1 is critical for the induction of alpha smooth muscle actin (alpha-SMA) and type 1 collage

23 vitro and in vivo, by reducing the levels of alpha smooth muscle actin (alpha-SMA) expression.

24 n of both CLIC4 and the myofibroblast marker alpha smooth muscle actin (alpha-SMA) in stromal fibrobl

25 utroban reduced liver fibrosis and decreased alpha smooth muscle actin (alpha-SMA), collagen-I, and t

26 Molecular assays revealed higher level of alpha smooth muscle actin (alpha-SMA), desmin, calponin,

27 ls expressed several EMT biomarkers, but not alpha smooth muscle actin (alpha-SMA).

28 proliferation in vitro, it robustly induced alpha smooth muscle actin (alpha-SMA).

29 he increased expression of markers including alpha smooth muscle actin (alpha-SMA).

30 Immunostaining with alpha-smooth muscle actin (alpha SMA) revealed a signifi

31 The expression of alpha-smooth muscle actin (alpha-SMA) and Collagen I wer

32 ifferentiation and fibrotic markers, such as alpha-smooth muscle actin (alpha-SMA) and Collagen III (

33 n is associated with increased expression of alpha-smooth muscle actin (alpha-SMA) and collagen in fi

34 r, DDR2 acted via integrin-beta1 to regulate alpha-smooth muscle actin (alpha-SMA) and collagen type

35 n and an increase in the profibrotic markers alpha-smooth muscle actin (alpha-SMA) and fibronectin, a

36 tolic blood pressure and renal expression of alpha-smooth muscle actin (alpha-SMA) and proliferating

37 Corneal sections were stained for alpha-smooth muscle actin (alpha-SMA) and tenascin-C (TN

38 breast tumor models, Cellax therapy reduced alpha-smooth muscle actin (alpha-SMA) content by 82% and

39 ibroblasts undergo activation and convert to alpha-smooth muscle actin (alpha-SMA) expressing myofibr

40 ncluding an approximately 2-fold increase in alpha-smooth muscle actin (alpha-SMA) expression and str

41 t stress fibers, exhibited greater levels of alpha-smooth muscle actin (alpha-SMA) expression, and ex

42 eased expression of the myofibroblast marker alpha-smooth muscle actin (alpha-SMA) in healthy lung fi

43 inhibited the induction of activation marker alpha-smooth muscle actin (alpha-SMA) in rat and mouse H

44 Alpha-smooth muscle actin (alpha-SMA) is a marker of act

45 Transcription of the contractile protein alpha-smooth muscle actin (alpha-SMA) is mediated by the

46 ollagen deposition and reduced expression of alpha-smooth muscle actin (alpha-SMA) protein indicated

47 HIF-1alpha) activation of the TGF-beta/Smad3/alpha-smooth muscle actin (alpha-SMA) signaling pathway,

48 phosphorylation and the myofibroblast marker alpha-smooth muscle actin (alpha-SMA) were notably overe

49 Quantitative proteomic comparison of mature alpha-smooth muscle actin (alpha-SMA)+ myofibroblasts (v

50 TCP and were analyzed for the expression of alpha-smooth muscle actin (alpha-SMA), a key marker of m

51 ation, cell proliferation, and expression of alpha-smooth muscle actin (alpha-SMA), a marker of PSC a

52 Neoexpression of alpha-smooth muscle actin (alpha-SMA), an established ma

53 including collagen Ialpha1 (colIalpha1) and alpha-smooth muscle actin (alpha-SMA), and cell migratio

54 ch assay cell migration, immunocytochemistry alpha-smooth muscle actin (alpha-SMA), and collagen gel

55 hTM cells, associated with overexpression of alpha-smooth muscle actin (alpha-SMA), and differential

56 ically confirmed via expression of vimentin, alpha-smooth muscle actin (alpha-SMA), and glial fibrill

57 ssions of NADPH oxidase gp91 (phox) subunit, alpha-smooth muscle actin (alpha-SMA), and NFkappaB p65

58 he three major myofibroblast markers, SDF-1, alpha-smooth muscle actin (alpha-SMA), and TGF-beta1, an

59 neal fibroblasts to up-regulate synthesis of alpha-smooth muscle actin (alpha-SMA), collagen 1 (COL1)

60 Expression of alpha-smooth muscle actin (alpha-SMA), collagen I and II

61 ately after UUO injury reduced expression of alpha-smooth muscle actin (alpha-SMA), fibronectin, and

62 cal examination as well as the expression of alpha-smooth muscle actin (alpha-SMA), transforming grow

63 Protein levels of alpha-smooth muscle actin (alpha-SMA), type I collagen,

64 10 nM) did not modify the gene expression of alpha-smooth muscle actin (alpha-SMA), Vimentin (VIM), F

65 -triggered differentiation into contractile, alpha-smooth muscle actin (alpha-SMA)-positive myofibrob

66 CCA has been associated with the presence of alpha-smooth muscle actin (alpha-SMA)-positive myofibrob

67 duction of EMT resulted in the generation of alpha-smooth muscle actin (alpha-SMA)-positive myofibrob

68 en deposition, immune cell infiltration, and alpha-smooth muscle actin (alpha-SMA)-positive myofibrob

69 rotic proteins, such as fibronectin (FN) and alpha-smooth muscle actin (alpha-SMA).

70 FAK expression and causes TGFbeta to induce alpha-smooth muscle actin (alpha-SMA).

71 transforming growth factor (I and III), and alpha-smooth muscle actin (alpha-SMA).

72 mutations in the gene, ACTA2, which encodes alpha-smooth muscle actin (alpha-SMA).

73 c NK cells were enriched in proximity to the alpha-smooth muscle actin (alpha-SMA+) area within mild

74 h signaling), sox-9 (progenitor marker), and alpha-smooth muscle actin (alpha-SMA; myofibroblast mark

75 rylated AKT [p-AKT](+)), and differentiated (alpha-smooth muscle actin [alpha-SMA](+)) fibrocytes wer

76 Similarly, alpha-smooth muscle actin(alpha-SMA) expression, aortic

77 r with PDZ-binding motif, YAP/TAZ) and late (alpha-smooth muscle actin, alpha-SMA) markers of myofibr

78 cid synthase 2, HAS2, and PDPN increased and alpha-smooth muscle actin alphaSMA mRNA decreased.

79 mice showed fibrotic changes with increased alpha smooth muscle actin (alphaSMA) and desmin-positive

80 on, apoptosis, and number of (proliferating) alpha smooth muscle actin (alphaSMA) neointimal cells.

81 d the expression of the myofibroblast marker alpha smooth muscle actin (alphaSMA).

82 rmal fibroblasts mostly by the expression of alpha smooth muscle actin (alphaSMA).

83 hymal transition (EMT) and the expression of alpha-smooth muscle actin (alphaSMA) and matrix metallop

84 roliferating cell nuclear antigen (PCNA) and alpha-smooth muscle actin (alphaSMA) double-immunostaini

85 on ensuing heart failure, a 38% reduction in alpha-smooth muscle actin (alphaSMA) expression, and a 7

86 4 on HLMF proliferation, collagen secretion, alpha-smooth muscle actin (alphaSMA) expression, and Sma

87 Evaluating spatiotemporal dynamics of alpha-smooth muscle actin (alphaSMA) in reporter mice re

88 AF subpopulation with elevated expression of alpha-smooth muscle actin (alphaSMA) located immediately

89 We used an alpha-smooth muscle actin (alphaSMA) promoter-driven and

90 Tensin1 and alpha-smooth muscle actin (alphaSMA) were strongly co-lo

91 Specific deletion of Tgfbr2 in alpha-smooth muscle actin (alphaSMA)(+) cells revealed t

92 Cells that co-express Thy-1 and alpha-smooth muscle actin (alphaSMA), a CAF marker, were

93 TIN-19, and mesenchymal markers VIMENTIN and alpha-SMOOTH MUSCLE ACTIN (alphaSMA).

94 inhibitory effects on the expression of both alpha smooth muscle actin and extracellular matrix in cu

95 xpressed in mesenchymal cells that expressed alpha-smooth muscle actin and activated BMP signaling pa

96 ens of macular pseudoholes were positive for alpha-smooth muscle actin and anti-glial fibrillary acid

97 Myofibroblasts express alpha-smooth muscle actin and are present in granulation

98 ype characterized by increased expression of alpha-smooth muscle actin and collagen 1.

99 and reduced expression of TGF-beta1-induced alpha-smooth muscle actin and collagen alpha-2(I) but no

100 weeks after radiation by immunostaining for alpha-smooth muscle actin and collagen deposition.

101 pha increased the survival of HSCs and their alpha-smooth muscle actin and collagen I expression, thu

102 Treatment with 77427 abrogated interstitial alpha-smooth muscle actin and collagen.

103 cription factor-1 and the mesenchymal marker alpha-smooth muscle actin and CXCR3 expression was exami

104 otes dedifferentiation of MyoFb with loss of alpha-smooth muscle actin and depolymerization of stress

105 iation is characterized by the expression of alpha-smooth muscle actin and extracellular matrix prote

106 rin, and the increase in mesenchymal markers alpha-smooth muscle actin and fibroblast-specific protei

107 ctivated freshly isolated HSCs (induction of alpha-smooth muscle actin and fibrosis-associated genes)

108 n as assessed by the increased production of alpha-smooth muscle actin and its incorporation into str

109 ce treated with RF ablation were stained for alpha-smooth muscle actin and Ki-67 to establish the rol

110 termined by immunohistological assessment of alpha-smooth muscle actin and Masson's trichrome stainin

111 Rivaroxaban deactivated HSC, with decreased alpha-smooth muscle actin and mRNA expression of other H

112 e EMyT manifested by increased expression of alpha-smooth muscle actin and other contractile proteins

113 , vimentin, the matrix metalloprotease MMP2, alpha-smooth muscle actin and phospho-Smad2, as well as

114 otoxicity showed significantly greater Nox2, alpha-smooth muscle actin and picrosirius levels compare

115 c-5 expression was associated with increased alpha-smooth muscle actin and plasminogen activator inhi

116 We also observed increased alpha-smooth muscle actin and procollagen type I mRNAs,

117 llagen type I mRNAs, large fibrotic areas in alpha-smooth muscle actin and Sirius red staining, and i

118 Ccn2), associated with reduced expression of alpha-smooth muscle actin and Sox2.

119 The amount of alpha-smooth muscle actin and tgf-beta were significantl

120 duced the expression of myofibroblast marker alpha-smooth muscle actin and the proliferation of renal

121 accompanied subsequently with stimulation of alpha-smooth muscle actin and type I collagen expression

122 ter numbers of myofibroblasts that expressed alpha-smooth muscle actin and vimentin than controls.

123 ere stained for markers of activation (e.g., alpha smooth muscle actin) and were also tested for pres

124 ducing levels of a marker of HSC activation (alpha-smooth muscle actin) and expression of genes that

125 phorylation), myofibroblast differentiation (alpha-smooth muscle actin), and collagen deposition (Mas

126 ctile markers CNN-1 (calponin 1), alpha-SMA (alpha-smooth muscle actin), and SM22-alpha (smooth muscl

127 ctor alpha, transforming growth factor beta, alpha smooth muscle actin, and collagen (P < 0.01).

128 The fibrotic markers f-actin, fibronectin, alpha smooth muscle actin, and collagen type 1 were equa

129 r the fibrotic markers f-actin, fibronectin, alpha smooth muscle actin, and collagen type 1 were perf

130 arkers (collagenIotaalpha1, EDA-fibronectin, alpha smooth muscle actin, and connective tissue growth

131 mentin, fibroblast activation protein alpha, alpha smooth muscle actin, and PDGF receptor beta.

132 lular valve scaffolds expressed CD163, CD31, alpha smooth muscle actin, and vimentin at each time poi

133 hymal markers, including collagen type I and alpha-smooth muscle actin, and a reduction in endothelia

134 In activated mouse HSCs, CCN2, alpha-smooth muscle actin, and collagen 1(alpha1) were s

135 r alpha, monocyte chemoattractant protein-1, alpha-smooth muscle actin, and collagen I and TIMP1 expr

136 albumin, alpha(1) -antitrypsin, glypican-3, alpha-smooth muscle actin, and collagen type 1A2 markers

137 r this purpose, quantification of TGF-beta1, alpha-smooth muscle actin, and collagen type I mRNA and

138 reases in hepatic collagen content, level of alpha-smooth muscle actin, and Enhanced Liver Fibrosis s

139 ] 1, PC2, and KIF3A), fibroblasts (vimentin, alpha-smooth muscle actin, and fibroblast-specific prote

140 of less organized intimal collagen, laminin, alpha-smooth muscle actin, and matrix-rich fibrosis was

141 tren's patients, reduced their expression of alpha-smooth muscle actin, and mediated disassembly of t

142 osition, hepatic expressions of collagen-1a, alpha-smooth muscle actin, and mononuclear cell infiltra

143 ignificantly less expression of collagen IV, alpha-smooth muscle actin, and other markers of disease

144 , collagen (Col) alpha1(I), Col alpha1(III), alpha-smooth muscle actin, and p-Smad2/3 was characteriz

145 pression of the fibroblast markers vimentin, alpha-smooth muscle actin, and S100A4.

146 Snai1, and Snai2; by immunofluorescence for alpha-smooth muscle actin; and by Western blot analysis

147 rocollagen type I, procollagen type III, and alpha-smooth muscle actin, areas of pronounced collagen

148 terized using whole transcriptome profiling, alpha smooth muscle actin (ASMA) expression and cell imp

149 geny of individual VSMCs contributes to both alpha smooth muscle actin (aSma)-positive fibrous cap an

150 in the expression of HSC-activation markers (alpha smooth muscle actin, beta platelet-derived growth

151 fibrosis stage, hepatic collagen content and alpha-smooth muscle actin (by morphometry), NAFLD Activi

152 d with mesenchymal smooth muscle epitopes of alpha-smooth muscle actin, caldesmon and calponin, which

153 ssion of the cardinal myofibroblast products alpha smooth muscle actin, calponin, and collagen in pri

154 n mothers against decapentaplegic homolog 4, alpha-smooth muscle actin, CD31, phospho-vascular endoth

155 howed normal outgrowth of mouse ERG-positive/alpha-smooth muscle actin coimmunolocalized cells; howev

156 ssical fibrocytes, they display cell surface alpha smooth muscle actin, collagen I/V, and mediate ang

157 reased expression of three fibrotic markers: alpha-smooth muscle actin, collagen 1, and fibronectin.

158 lpha, led to a significant downregulation of alpha-smooth muscle actin, collagen I and III and decrea

159 TGF-beta as confirmed by strong induction of alpha-smooth muscle actin, collagen type I (COL1A1), and

160 he expression of the fibrosis-related genes, alpha-smooth muscle actin, collagen-1, and collagen-3; r

161 F-actin, phosphorylated myosin light chain, alpha-smooth muscle actin, collagen-1A, and total collag

162 n types I and VI, and the profibrotic factor alpha-smooth muscle actin compared with placebo in subcu

163 cores, and hepatic stellate cell activation (alpha-smooth muscle actin) compared to Long-Evans Tokush

164 fibrotic components, including fibronectin, alpha-smooth muscle actin, connective tissue growth fact

165 , Yap activation, increases in the levels of alpha-smooth muscle actin, connective tissue growth fact

166 sed expression of fibronectin-1, collagen I, alpha-smooth muscle actin, connective tissue growth fact

167 ression levels of fibronectin-1, collagen I, alpha-smooth muscle actin, CTGF, and PAI-1, but decrease

168 transition (EndoMT) may be a source of those alpha-smooth muscle actin-expressing cells.

169 teoblasts, endothelial cells, Schwann cells, alpha-smooth muscle actin-expressing macrophages and mes

170 ) involves predominantly the accumulation of alpha-smooth muscle actin-expressing mesenchymal-like ce

171 fibrosis (IPF) involves the accumulation of alpha-smooth muscle actin-expressing myofibroblasts aris

172 serve as a receptor for the HU177 epitope in alpha-smooth muscle actin-expressing stromal cells and s

173 nesis and, surprisingly, the accumulation of alpha-smooth muscle actin-expressing stromal cells.

174 on to a mesenchymal phenotype with increased alpha smooth muscle actin expression and suppression of

175 s, coupled to a reduction in fibronectin and alpha smooth muscle actin expression in stromal fibrobla

176 tion together with increased fibronectin and alpha-smooth muscle actin expression and decreased in E-

177 cation of troglitazone significantly reduced alpha-smooth muscle actin expression and haze in the str

178 SSc fibroblasts showed increased alpha-smooth muscle actin expression and improved capabi

179 ice led to more abundant vascular and airway alpha-smooth muscle actin expression and inflammatory pu

180 d both MMP-9-induced TGF-beta activation and alpha-smooth muscle actin expression by displacing MMP-9

181 In contrast, alpha-smooth muscle actin expression by infarct myofibro

182 M(-/-), mice promoted increased collagen and alpha-smooth muscle actin expression from lung fibroblas

183 gest that both pro- and active MMP-9 trigger alpha-smooth muscle actin expression in cultured fibrobl

184 knockdown of GalR1 in cholangiocytes reduced alpha-smooth muscle actin expression in LX-2 cells treat

185 Exposure to mtDNA augments alpha-smooth muscle actin expression in NHLFs.

186 FPR stimulation promoted alpha-smooth muscle actin expression in normal fibroblas

187 nectin, transforming growth factor-beta, and alpha-smooth muscle actin expression in obstructed kidne

188 KO fibroblasts did not increase alpha-smooth muscle actin expression in response to TGF-

189 knockdown of Has2 in SF, significantly less alpha-smooth muscle actin expression was detected in co-

190 L290 significantly reduced collagen content, alpha-smooth muscle actin expression, and cell prolifera

191 oblasts populating collagen pads, attenuated alpha-smooth muscle actin expression, and stimulated mat

192 ted with fibronectin deposition, increase in alpha-smooth muscle actin expression, and the release of

193 on and genetic silencing of Cat B diminished alpha-smooth muscle actin expression, delayed fibroblast

194 d Akt signaling axis, increased collagen and alpha-smooth muscle actin expression, distinct gene expr

195 ured epithelial cells promote proliferation, alpha-smooth muscle actin expression, F-actin expression

196 s lacking endogenous sFRP-1 showed increased alpha-smooth muscle actin expression, higher cell prolif

197 onists tested in diverse cell models blocked alpha-smooth muscle actin expression, myofibroblast diff

198 Hyalinosis was accompanied by loss of alpha-smooth muscle actin expression, whereas the endoth

199 phenotype defined by induction of fibroblast alpha-smooth muscle actin expression.

200 lase inhibitor ODQ increased osteocalcin and alpha-smooth muscle actin expression.

201 tion of F4/80(+) and CD11b(+) leukocytes and alpha-smooth muscle actin(+) fibroblasts before attenuat

202 ological changes including the expression of alpha-smooth muscle actin, fibronectin and TGFbeta-1 com

203 gation of beta1-integrin and upregulation of alpha-smooth muscle actin, fibronectin, collagen 1A, vim

204 y quantitative polymerase chain reaction for alpha-smooth muscle actin, fibronectin, collagen type 1a

205 HSCs to express tumor-promoting factors, and alpha-smooth muscle actin, fibronection, and CTGF, marke

206 connective tissue growth factor (CTGF), and alpha-smooth muscle actin gene expression at 7 days, com

207 y substantial downregulation of collagen and alpha-smooth muscle actin gene expression.

208 as Thbs1(-/-)) mice and from mice expressing alpha-smooth muscle actin-green fluorescent protein that

209 for hematoxylin and eosin, Masson trichrome, alpha-smooth muscle actin, IL-33, CD8, and IL-13 and ana

210 /7 receptor inhibitors blocked expression of alpha-smooth muscle actin in ASC in the absence of direc

211 ardiomyogenic markers cardiac troponin T and alpha-smooth muscle actin in CPCedeltaB compared with CP

212 ent to normalize the decreased expression of alpha-smooth muscle actin in dermal blood vessels and im

213 ly induces collagen I, fibronectin, elastin, alpha-smooth muscle actin in human adult dermal (HDFs) a

214 was notably upregulated along with Col-1 and alpha-smooth muscle actin in pleural thickening in the c

215 ially decreased with a de novo expression of alpha-smooth muscle actin in PTCs.

216 d expression of fibronectin, collagen I, and alpha-smooth muscle actin in the TM in mouse eyes.

217 of the myofibroblastic markers vimentin and alpha-smooth-muscle actin in developing kidneys.

218 pressions of two EMT markers (E-cadherin and alpha-smooth muscle actin) in the hGE cells after CsA tr

219 imulated with cholangiocyte supernatants and alpha-smooth muscle actin levels were measured.

220 e 4 alpha 1, matrix metalloproteinase 2, and alpha-smooth muscle actin) markers.

221 (transforming growth factor-beta protein and alpha-smooth muscle actin mRNA), whereas EX increased th

222 and contained abnormally persistent alveolar alpha-smooth muscle actin myofibroblasts.

223 nd tissue inhibitor metalloproteinase-1, and alpha-smooth muscle actin (myofibroblasts) were evaluate

224 xpression of selective VSMCs markers such as alpha smooth muscle actin, myosin heavy chain 11, and sm

225 enotype from neural/glial antigen 2 positive/alpha-smooth muscle actin negative to neural/glial antig

226 d effectors, including collagens I and V and alpha-smooth muscle actin, on the transcript and protein

227 ession of CCN2 downstream targets, including alpha smooth muscle actin or collagen.

228 Green fluorescent protein costaining with alpha-smooth muscle actin or collagen 1alpha in left ven

229 of the adenoma stroma; cells that expressed alpha-smooth muscle actin or desmin were lost, along wit

230 ith bean-shaped condensed nuclei, absence of alpha-smooth muscle actin or stress fibres, and a corres

231 ed miR-199a-5p-dependent inhibition of CCN2, alpha-smooth muscle actin, or collagen 1(alpha1) in acti

232 ficantly decreases the profibrogenic protein alpha-smooth muscle actin (P < 0.05).

233 nd the amniotic membrane at 6 hours, whereas alpha-smooth muscle actin, phosphorylated Smad3, and col

234 peroxide, hydroxynonenal), and fibrogenesis (alpha-smooth muscle actin, picrosirius red, trichrome, v

235 d ATP but also increased basal expression of alpha-smooth muscle actin, plasminogen activator inhibit

236 timulation resulted in stably differentiated alpha-smooth muscle actin(+)/platelet-derived growth fac

237 ced fibrogenic activity (e.g., expression of alpha smooth muscle actin, platelet-derived growth facto

238 The perfused vessel area, number of alpha smooth muscle actin positive vessels and hypoxic a

239 eveloped severe TV with intimal hyperplasia (alpha-smooth muscle actin positive cells in the neointim

240 N629D/N629D aortas showed fewer alpha-smooth muscle actin positive cells that were not c

241 negative to neural/glial antigen 2 positive/alpha-smooth muscle actin positive in some pericytes (PC

242 t inhibition reduced fibrosis as measured by alpha-smooth muscle actin positive myofibroblasts and co

243 In addition, alpha-SMA (alpha-smooth muscle actin)-positive cells were reduced i

244 ne of the coagulation zone was surrounded by alpha-smooth muscle actin-positive activated myofibrobla

245 ng growth factors and cytokines, decrease of alpha-smooth muscle actin-positive ASFs, and finally in

246 th factor receptor-alpha-positive cells, and alpha-smooth muscle actin-positive blood vessels were as

247 wth factor receptor-alpha-positive cells, 4) alpha-smooth muscle actin-positive blood vessels, and 5)

248 erized by increased macrophage and decreased alpha-smooth muscle actin-positive cell population, fibr

249 a isoform co-localized with CD31-positive or alpha-smooth muscle actin-positive cells at different de

250 Cs and a 1.35-fold increase in proliferative alpha-smooth muscle actin-positive cells in the lungs of

251 ll thickening, and abnormal proliferation of alpha-smooth muscle actin-positive cells, as well as inc

252 dentified the presence von Willebrand factor/alpha-smooth muscle actin-positive endothelial cells in

253 elial-to-mesenchymal transition, detected as alpha-smooth muscle actin-positive endothelial cells, si

254 rthermore, treatment with cysteamine reduced alpha-smooth muscle actin-positive interstitial myofibro

255 lial fibroblasts are specific progenitors of alpha-smooth muscle actin-positive myofibroblasts that a

256 n of matrix molecules, renal accumulation of alpha-smooth muscle actin-positive myofibroblasts, and b

257 and Gli2-positive cells differentiated into alpha-smooth muscle actin-positive myofibroblasts.

258 e phenotype, as indicated by the presence of alpha-smooth muscle actin-positive pericytes.

259 eptor-alpha), but generally lacked alphaSMA (alpha-smooth muscle actin) positivity.

260 vated fibroblasts, shown by up-regulation of alpha-smooth muscle actin, pro-collagen 1, and F-actin e

261 relaxin-2 down-regulates type I collagen and alpha smooth muscle actin production and increases intra

262 ss cytotoxic, and a more potent inhibitor of alpha-smooth muscle actin protein expression than CCG-20

263 tenuated fibrotic markers such as diminished alpha-smooth muscle actin, reduced collagen deposition.

264 ASC migration toward EC and upregulation of alpha-smooth muscle actin, SM22alpha, and calponin expre

265 Although some TECs strikingly upregulate alpha smooth muscle actin (SMA), a principal marker of E

266 es, adiponectin stimulated the expression of alpha-smooth muscle actin (SMA) and extracellular matrix

267 the nucleus, correlating with an increase in alpha-smooth muscle actin (SMA) expression.

268 sion as well as alcohol-induced TGFbeta1 and alpha-smooth muscle actin (SMA) protein expression in PL

269 s were tested for cell viability, apoptosis, alpha-smooth muscle actin (SMA), fibronectin (FN) produc

270 on and remodeling of extracellular matrix by alpha-smooth muscle actin (SMA)-expressing myofibroblast

271 enal tubular epithelial cells transform into alpha-smooth muscle actin (SMA)-expressing myofibroblast

272 performed to detect the myofibroblast marker alpha-smooth muscle actin (SMA).

273 sing SERS-labelled antibodies direct against alpha-smooth muscle actin (SMA).

274 scle actin; and by Western blot analysis for alpha-smooth muscle actin, SNAIL1, SNAIL2, and the alpha

275 growth factor receptor-alpha, vimentin, and alpha-smooth muscle actin, specifying myofibroblasts as

276 10 nM concentration reduced the intensity of alpha-smooth muscle actin staining by 56% and periostin

277 of fibrosis-related genes, smaller areas of alpha-smooth muscle actin staining, and low collagen pro

278 sed by gene expression, picrosirius red, and alpha-smooth muscle actin staining, hydroxyproline assay

279 cell type characterized by the appearance of alpha-smooth muscle actin stress fibers.

280 oliferative or nonproliferative, with mature alpha-smooth muscle actin stress fibers.

281 ion, as >60% of fibroblasts formed alphaSMA (alpha-smooth muscle actin) stress fibers and expressed m

282 portant in lung myofibroblast collagen-1 and alpha-smooth-muscle actin synthesis.

283 dherin and PECAM1 and increases in collagen, alpha-smooth muscle actin, TGFbeta receptor 1, and the t

284 ncreased both mRNA and protein expression of alpha-smooth muscle actin, transgelin, and calponin vers

285 roblast differentiation and up-regulation of alpha-smooth muscle actin, type I collagen, and fibronec

286 or submesothelial fibroblasts each expressed alpha-smooth muscle actin under the influence of TGF-bet

287 oblast differentiation (ED-A fibronectin and alpha-Smooth Muscle Actin) upon treatment with TGF-beta1

288 by their expression of CD45, collagen I, and alpha-smooth muscle actin using flow cytometry.

289 erin, and Integrins), cytoskeletal proteins (alpha-Smooth Muscle Actin, Vimentin, and beta-catenin),

290 corporation of EdU and protein expression of alpha-smooth muscle actin was analyzed by immunocytochem

291 Expression of the contractile protein alpha-smooth muscle actin was high in arterioles and low

292 l differentiation markers p63, calponin, and alpha-smooth muscle actin was observed in the mouse myoe

293 MesoMT marker alpha-smooth muscle actin was reduced in 9ING41-treated

294 ated genes type I collagen, fibronectin, and alpha-smooth muscle actin were highly expressed.

295 and zona occludens-1, whereas collagen-I and alpha-smooth muscle actin were increased in ATII cells i

296 roblasts was reduced, whereas collagen-I and alpha-smooth muscle actin were markedly elevated.

297 Fibrotic markers, such as collagen and alpha smooth muscle actin, were also significantly decre

298 , corneal fibrosis markers, Collagen III and alpha-smooth muscle actin, were significantly downregula

299 as indicated by the decreased expression of alpha smooth muscle actin, when compared with convention

300 addition, we observed a significant loss of alpha-smooth muscle actin, which indicates a difference