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1 other agents which are more specific for the alpha1 adrenergic receptor.
2 was improved by sub-chronically blocking the alpha1-adrenergic receptor.
3 mIPSCs frequency results from activation of alpha1 adrenergic receptors.
4 these modulatory effects are attributable to alpha1 adrenergic receptors.
5 , thereby ruling out [3H]CUMI-101 binding to alpha1-adrenergic receptors.
6 yl cyclase, and protein kinase A, as well as alpha1-adrenergic receptors.
7 E release and that this might be specific to alpha1-adrenergic receptors.
8 nformation describing this drug class at the alpha1-adrenergic receptors.
9 inity for dopamine D2, serotonin 5-HT1A, and alpha1-adrenergic receptors.
10 h suggested an association between Panx1 and alpha1D-adrenergic receptor.
12 onstrated impaired testicular vasomotion via alpha1-adrenergic receptor activation and increased hypo
16 Consistent with this idea, the selective alpha1-adrenergic receptor agonist methoxamine was still
17 cked the induction of these responses by the alpha1-adrenergic receptor agonist phenylephrine, wherea
18 ith either insulin or phenylephrine (PE), an alpha1-adrenergic receptor agonist, induced Ser-9 phosph
19 itoneally) or saline, the response to the a) alpha1-adrenergic receptor agonist, phenylephrine; b) en
20 was perturbed by intravenous injection of an alpha1-adrenergic receptor agonist, unilateral pressure
21 was perturbed by intravenous injection of an alpha1-adrenergic receptor agonist, unilateral pressure
23 sylated Rho and inhibited the characteristic alpha1-adrenergic receptor agonist-induced myofibrillar
26 are induced in cultured myocardial cells by alpha1- adrenergic receptor agonists, such as phenylephr
27 inately induced by growth promoters, such as alpha1-adrenergic receptor agonists (e.g. phenylephrine
28 which were prevented by incubation with the alpha1 adrenergic receptor (alpha1-AR) antagonist prazos
29 through vasopressin receptor (V1A-R) but not alpha1 adrenergic receptor (alpha1-AR), suggesting that
30 rat ventricular myocytes, stimulation of the alpha1-adrenergic receptor (alpha1-AdrR) activates a pro
31 oth muscle, or to phenylephrine, a selective alpha1-adrenergic receptor (alpha1-AR) agonist, was simi
33 alpha1ABKO) mice that lacked cardiac myocyte alpha1-adrenergic receptor (alpha1-AR) binding, aortic c
34 ing mediated by the angiotensin (Ang) II and alpha1-adrenergic receptor (alpha1-AR) pathways is impor
36 elease in vascular smooth muscle coordinates alpha1-adrenergic receptor (alpha1-AR) vasoconstriction
38 serotonin dorsal raphe neurons is driven via alpha1-adrenergic receptors (alpha1-A(R)) activation.
39 n, noradrenaline activates G-protein-coupled alpha1-adrenergic receptors (alpha1-A(R)) causing action
40 n, noradrenaline activates Galpha(q)-coupled alpha1-adrenergic receptors (alpha1-A(R)), causing actio
43 4(SMC) channels to the constrictor effect of alpha1 adrenergic receptor (alpha1AR) stimulation and el
47 loned cDNAs encoding three subtypes of human alpha1-adrenergic receptors (alpha1ARs), alpha1a, alpha1
49 botropic neurotransmitter receptors (mGluRs, alpha1 adrenergic receptors, and muscarinic acetylcholin
50 ease of dopamine, activation of postsynaptic alpha1 adrenergic receptors, and suppression of InsP3-in
51 us losartan (10 mg/kg b.wt) nor prazosin, an alpha1 adrenergic receptor antagonist, at doses of 5 mic
53 e Cl- current to 5.9 nM, and addition of the alpha1-adrenergic receptor antagonist prazosin did not c
54 drenergic receptor antagonist) and prazosin (alpha1-adrenergic receptor antagonist) reduced female bu
56 heteromerize with one or more members of the alpha1-adrenergic receptor (AR) family in recombinant sy
57 61W retinal cells in high glucose and of the alpha1-adrenergic receptor (AR) on vascular lesions of t
60 hat the type 1 muscarinic receptor (m1R) and alpha1-adrenergic receptor (AR)-mediated SRF activation
61 nd novel cytoplasmic binding partners of the alpha1D-adrenergic receptor (AR), a yeast two-hybrid scr
62 ion of vascular smooth muscle, activation of alpha1D-adrenergic receptors (AR) induces smooth muscle
66 of blood pressure through interactions with alpha1-adrenergic receptors (ARs) in vascular smooth mus
67 direct evidence suggests that stimulation of alpha1-adrenergic receptors (ARs) increases smooth muscl
68 ith POTS (98.3%) and all controls (100%) had alpha1 adrenergic receptor autoantibody concentrations a
72 ral-midbrain dopaminergic neurons through an alpha1 adrenergic receptor-dependent mechanism within th
73 tes interfered with the basal expression and alpha1-adrenergic receptor-dependent activation of a TEF
76 he mechanism underlying the critical role of alpha1 adrenergic receptors in the regulation of dopamin
77 ed if insulin or IGF-I induced expression of alpha1 adrenergic receptors in vascular smooth muscle ce
78 hat significantly altered mRNA levels of the alpha1- adrenergic receptor in the basal forebrain as we
79 our recently demonstrated mechanism whereby alpha1-adrenergic receptors in the same cell type regula
82 uctive tract, it has been suggested that the alpha1-adrenergic receptor influences fertility and sper
84 70 gene expression induced by stimulation of alpha1 adrenergic receptors is markedly attenuated, whic
85 concurrent episodic activation of 5-HT2 and alpha1-adrenergic receptors is necessary to produce long
88 ases neural stimulation of BAT and increases alpha1-adrenergic receptor number; however, the alpha1 r
89 t of hypertension and inhibition of systemic alpha1-adrenergic receptor or angiotensin II AT1 recepto
90 that in MDCK-D1 cells, epinephrine acting on alpha1-adrenergic receptors, promotes a rapid increase i
91 ikewise, ANP and 8-bromo-cGMP attenuated the alpha1-adrenergic receptor- stimulated increase in prepr
92 imulated increase in prepro-ANP mRNA and the alpha1-adrenergic receptor-stimulated decrease in sarcop
93 re, in myocytes, 8-bromo-cGMP attenuated the alpha1-adrenergic receptor-stimulated increases in c-fos
95 d signal transduction mechanisms involved in alpha1, adrenergic receptor stimulation of hsp7O gene ex
98 or (ANF) reporter gene expression induced by alpha1-adrenergic receptor stimulation with phenylephrin
99 ing assays were used to examine the basis of alpha1-adrenergic receptor subtype selectivity for a ser
101 with that previously described for the other alpha1 adrenergic receptor subtypes, may shed light on t
102 nsfected with each of the three cloned human alpha1 adrenergic receptor subtypes, norepinephrine stro
103 y episodic but not continuous stimulation of alpha1-adrenergic receptors that requires protein kinase
104 slated and active in costimulating, with the alpha1-adrenergic receptor, the firing of DR 5-HT neuron
107 was to determine the effect of endotoxin on alpha1-adrenergic receptor vasoconstriction and both end
108 tors, is also shown to be a modulator of the alpha1-adrenergic receptors, which suggests a general la
109 ncreases intracellular Ca(2+) similar to the alpha1 adrenergic receptor, while OAMB-K3 additionally s