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1 in for adenylyl cyclase, in complex with the alpha2 adrenergic receptor.
2 h Galphaz coupled strongly to the endogenous alpha2 adrenergic receptor.
3 K1/2 is inhibited by epinephrine through the alpha2-adrenergic receptor.
4 restin is required for ERK activation by the alpha2-adrenergic receptor.
5  inhibition of presynaptic TRPV1 channels by alpha2 adrenergic receptors.
6 y yohimbine, consistent with contribution of alpha2 adrenergic receptors.
7 irming that it was mediated by activation of alpha2 adrenergic receptors.
8 n is specific for post-junctional alpha1- or alpha2-adrenergic receptors.
9 ined whether this was specific to alpha1- or alpha2-adrenergic receptors.
10 ecline (PHFD) requires central activation of alpha2-adrenergic receptors.
11                          GABAB receptors and alpha2-adrenergic receptors (activated by bath applicati
12               G(i/o)-coupled somatostatin or alpha2-adrenergic receptor activation stimulated beta-ce
13 te whether transdermal administration of the alpha2 adrenergic-receptor agonist, clonidine, can be us
14                               In humans, the alpha2-adrenergic receptor agonist clonidine increases d
15  the NRe and HPC through the infusion of the alpha2-adrenergic receptor agonist UK 14,304 impaired re
16 ermal administration of the centrally acting alpha2-adrenergic receptor agonist, clonidine), and hypo
17        Significance statement: Clonidine, an alpha2-adrenergic receptor agonist, which decreases the
18                                      Whether alpha2-adrenergic receptor agonist-based sedation, compa
19 mpatholytic effects we observed suggest that alpha2 adrenergic-receptor agonists may offer a pharmaco
20 ), partial agonists (eg, buprenorphine), and alpha2-adrenergic receptor agonists (eg, clonidine and l
21                                              alpha2-Adrenergic receptor agonists increase in potency
22  syndrome indicates that chronic exposure to alpha2-adrenergic receptor agonists produces strong depe
23 ess in the anti-allodynic effect elicited by alpha2-adrenergic receptor agonists.
24                                          The alpha2 adrenergic receptor (alpha(2)-AR) antagonist yohi
25 ers in cognition and emotion, by alpha1- and alpha2-adrenergic receptors (alpha1-ARs, alpha2-ARs) in
26                                              alpha2 adrenergic receptor (alpha2-AR) agonists have bee
27                     Activation of inhibitory alpha2-adrenergic receptors (alpha2-A(R)) with UK-14,304
28                Here we show that agonists of alpha2-adrenergic receptors (alpha2-AR) have very strong
29 ole of arrestins in the trafficking of human alpha2-adrenergic receptors (alpha2-ARs) and the effect
30 the evolutionary precursor to the vertebrate alpha2-adrenergic receptors (alpha2-ARs) based upon sequ
31  the regulation of cell surface transport of alpha2-adrenergic receptors (alpha2-ARs) by GGA3 (Golgi-
32 strates subserving the inhibitory actions of alpha2-adrenergic receptors (alpha2-ARs) in the locus co
33                                              alpha2-Adrenergic receptors (alpha2-ARs) mediate a numbe
34               We report that two subtypes of alpha2-adrenergic receptors (alpha2A/D- and alpha2C-AR)
35 ve, and sympatholytic responses in vivo, and alpha2 adrenergic receptor (alpha2AR) agonists are used
36 is study, we investigated how NE release and alpha2-adrenergic receptor (alpha2AR) modulation influen
37                                          The alpha2-adrenergic receptor (alpha2AR) subtype alpha2C10
38                                              alpha2-Adrenergic receptors (alpha2ARs) are essential co
39                                          The alpha2-adrenergic receptors (alpha2ARs) are localized to
40                                              alpha2-Adrenergic receptors (alpha2ARs) present in the b
41 ined the selectivity of interactions between alpha2-adrenergic receptors (alpha2R) and various combin
42 generate unconsciousness selectively through alpha2-adrenergic receptors and related circuits.
43 ng properties on I1 imidazoline receptors vs alpha2-adrenergic receptors and their blood pressure eff
44 sors are tested for the beta2-, alpha1-, and alpha2- adrenergic receptors, and adenosine type 1 recep
45 rolled by negative feedback from presynaptic alpha2-adrenergic receptors, and the targets of the rele
46 bolished the proepileptogenic actions of the alpha2 adrenergic receptor antagonist idazoxan.
47                                Yohimbine, an alpha2 adrenergic receptor antagonist, is a well-validat
48 factor 1 (CRF1) antagonist (antalarmin), and alpha2-adrenergic receptor antagonist (yohimbine; used a
49 d PLD activity was completely blocked by the alpha2-adrenergic receptor antagonist rauwolscine and by
50 can be reversed by a potent centrally acting alpha2 adrenergic receptor (AR) agonist (dexmedetomidine
51                 Activation of the beta2- and alpha2-adrenergic receptors (AR) involves hydrogen bondi
52                                              alpha2-adrenergic receptors (AR) within the bed nucleus
53 ediated via both post-junctional alpha1- and alpha2-adrenergic receptors are blunted in contracting h
54                                     Although alpha2-adrenergic receptors are known to modulate the im
55                                              alpha2-Adrenergic receptors (ARs) play a key role in reg
56 was a result of activation of muscarinic and alpha2-adrenergic receptors because a specific alpha2-ad
57 ms using drug treatments targeting beta- and alpha2-adrenergic receptors, but little is known about t
58 that PHFD does not require the activation of alpha2-adrenergic receptors, but that alpha2-adrenergic
59 ion of alpha2-adrenergic receptors, but that alpha2-adrenergic receptors can modulate resting and pos
60 ctivity, which was increased by 5-HT(1A) and alpha2-adrenergic receptor co-activation in an activator
61 teract with a photoaffinity derivative of an alpha2-adrenergic receptor-derived peptide from the thir
62  voltage-dependent Ca2+ channels mediated by alpha2-adrenergic receptors desensitizes slowly with pro
63 d with pharmacological antagonists targeting alpha2-adrenergic receptors (idazoxan), beta-adrenergic
64 vious studies in several animal species, the alpha2 adrenergic receptors in the human ciliary body, r
65  Stimulated GTPgammaS binding indicated that alpha2-adrenergic receptors in HET hippocampus were more
66 from locus coeruleus project to the DCN, and alpha2-adrenergic receptors inhibit spontaneous spike ac
67                                Activation of alpha2-adrenergic receptors inhibited both NALCN and spi
68 hile rolipram and PDE4D deficiency shortened alpha2 adrenergic receptor-mediated anesthesia, a surrog
69 eleased glutamate in the mNTS and results in alpha2 adrenergic receptor-mediated inhibition of DMV ne
70                We have previously shown that alpha2-adrenergic receptor-mediated coupling to phosphol
71 ctions by approximately 50% and enhanced the alpha2-adrenergic receptor-mediated inhibition of forsko
72 btype influence signaling efficiency for the alpha2-adrenergic receptor-mediated signaling pathway.
73 e from studies in animals demonstrating that alpha2-adrenergic receptor-mediated vasoconstrictor resp
74 he level of arousal by selectively targeting alpha2 adrenergic receptors on LC neurons, resulting in
75 ic actions of norepinephrine are exerted via alpha2 adrenergic receptors residing on targets of norad
76 poptotic effects via activation of beta2 and alpha2 adrenergic receptors, respectively.
77                                Activation of alpha2-adrenergic receptors resulted in a fast, voltage-
78            Here we show that mutation in the alpha2 -adrenergic receptor subtype B (alpha2B -AR) is a
79 cular properties of each Gialpha, Gbeta, and alpha2-adrenergic receptor subtype influence signaling e
80 ecluded understanding the role of individual alpha2 adrenergic receptor subtypes in the antiepileptog
81                       The existence of three alpha2 adrenergic receptor subtypes together with the la
82                                    The three alpha2-adrenergic receptor subtypes (alpha2a, alpha2b, a
83 at low dose SNRIs, by acting on 5-HT(1A) and alpha2-adrenergic receptors, synergistically reduced AMP
84   Here, we show that noradrenaline activates alpha2-adrenergic receptors to control short-term and lo
85 lymorphisms (GPIIb, FcgammaRIIa, P-selectin, alpha2 adrenergic receptor, transforming growth factor [
86 a high binding affinity of M1 muscarinic and alpha2 adrenergic receptors was associated with a greate
87                               The density of alpha2 adrenergic receptors was highest in the iris (440
88                                          The alpha2-adrenergic receptor was found to be markedly more
89 ution and concentration of beta1, beta2, and alpha2 adrenergic receptors were examined in the frontal
90  mice to sedation elicited by stimulation of alpha2 adrenergic receptors, whereas arrestin 3 knockout
91 ne binding sites in plasma membrane and lack alpha2-adrenergic receptors, which recognize many I1-imi