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1 eraction of the same alphai subunit with the alpha2A-adrenergic receptor.
2  a well-defined allosteric site on the human alpha2A-adrenergic receptor.
3 0, a PC12 cell line overexpressing the human alpha2A-adrenergic receptor.
4  receptor could be replaced by activation of alpha2A-adrenergic receptors.
5 eurons, while POMC neurons are inhibited via alpha2A - adrenergic receptors.
6 onded to both amphetamine and guanfacine, an alpha2A adrenergic receptor agonist.
7  is specifically stimulated by the selective alpha2A-adrenergic receptor agonist UK14304 and by lysop
8 nstrates that agonist-mediated activation of alpha2A adrenergic receptors (alpha(2A)AR) is voltage-de
9                            Here, we used the alpha2A-adrenergic receptor (alpha(2A)AR) as a model to
10  intracellular (3i) loop of the heptahelical alpha2A-adrenergic receptor (alpha2A AR) is critical for
11             Previous studies have shown that alpha2A-adrenergic receptor (alpha2A-AR) retention at th
12                                          The alpha2A adrenergic receptor (alpha2AAR) is directly deli
13 ne expressing a point mutation (D79N) in the alpha2a adrenergic receptor (alpha2aAR) to investigate t
14 was tethered to the carboxyl terminus of the alpha2A adrenergic receptor (alpha2AAR-Galphai2YFP).
15 inent feature of long-term regulation of the alpha2A-adrenergic receptor (alpha2AAR) is a loss of cel
16 novel direct interaction between APP and the alpha2A-adrenergic receptor (alpha2AAR) that occurs at t
17              Truncations and chimeras of the alpha2A-adrenergic receptor (alpha2AAR) were evaluated t
18 C (PKC) to phosphorylate and desensitize the alpha2A-adrenergic receptor (alpha2AAR).
19 etween delta opioid receptors (deltaORs) and alpha2A adrenergic receptors (alpha2AARs) require protei
20  competing at the one allosteric site on the alpha2A-adrenergic receptor and rules out the possibilit
21  alter subsequent radioligand binding to the alpha2a-adrenergic receptor and the dopaminergic D1 rece
22 293 cells, stimulation of either alpha1B- or alpha2A-adrenergic receptors (ARs) leads to rapid 5-10-f
23 drenergic receptor-encoding mRNA, alpha1 and alpha2A adrenergic receptor-encoding mRNA, and alpha1, a
24      The amyloid precursor protein modulates alpha2A-adrenergic receptor endocytosis and signaling th
25 ith fluorescent proteins and four GPCRs: the alpha2A-adrenergic receptor, GABAB, cannabinoid receptor
26  fluorescently labeled Galphai1 subunits and alpha2A-adrenergic receptor, GABAB, or dopamine receptor
27  receptor complexes between micro-opioid and alpha2A-adrenergic receptors has been demonstrated in tr
28 e RVL, 59% of c-Fos-positive cells contained alpha2A-adrenergic receptor-like immunoreactivity in clo
29 uggest that the effects of the overexpressed alpha2A-adrenergic receptor on PLD activity are mediated
30 tion of Gi-coupled lysophosphatidic acid and alpha2A adrenergic receptors or overexpression of Gbeta1
31                   We show that mu opioid and alpha2A adrenergic receptors reside in close proximity i
32  and 0.97) with the affinities for the human alpha2A adrenergic receptor subtype and poorly correlate
33 this inhibition is relieved by activation of alpha2A-adrenergic receptor, suggesting that the interac
34 f9 cell membranes expressing the recombinant alpha2A-adrenergic receptor were used to assess the cont
35 abeled agonist and antagonist binding to the alpha2a-adrenergic receptor with an IC50 value of 0.5 mi