ライフサイエンスコーパス: aluminum fluoride

1 ) ACAP1 onto the endosome in the presence of aluminum fluoride.

2 nce of the ATP transition state analogue ADP-aluminum fluoride.

3 nted the increase in PKD activity induced by aluminum fluoride.

4 as the transitional state of Rop mimicked by aluminum fluoride.

5 e GAP domain with the GDP-bound Rop, as does aluminum fluoride.

6 vated by the beta-adrenergic receptor and by aluminum fluoride.

7 nzymatically activated in cells treated with aluminum fluoride.

8 o alpha-agarose and eluted specifically with aluminum fluoride.

9 ilament generated in the presence of ATP and aluminum fluoride.

10 ng was achieved by the formation of an (18)F-aluminum fluoride ((18)F-AlF) complex, and (64)Cu labeli

11 Aluminum fluoride, a powerful inhibitor of some switch p

12 cate that the transition state analog, MgADP-aluminum fluoride-acetate, forms an abortive complex in

13 ing cell lysates revealed that RGS3 bound to aluminum fluoride-activated Galpha11 and to a lesser ext

14 50 values of 30 and 96 nM, respectively, for aluminum fluoride-activated wild type alphao and alphai1

15 hown to indirectly activate the ARF1 GTPase, aluminum fluoride (AIF) treatment of ARF6-transfected ce

16 describe 4 new probes using either (64)Cu or aluminum fluoride (Al(18)F) chelated to 2 NOTA derivativ

17 y a ring-opening reaction in the presence of aluminum fluoride (AlF(3) ) to create SPEs inside LiNi(0

18 Beryllium fluoride (BeF(3-)) and aluminum fluoride (AlF(4-)), inorganic phosphate analogu

19 Aluminum fluoride (AlF) treatment of ARF6-transfected ce

20 the alphaT*(R238E) mutant did not respond to aluminum fluoride (AlF4(-)), as read out by changes in T

21 sults in decreased activation by receptor or aluminum fluoride (AlF4-) and increased basal GDP releas

22 t with TGF alpha and the G protein activator aluminum fluoride (AlF4-), we determined if TGF alpha or

23 ch as vanadate, beryllium fluoride (BeFx) or aluminum fluoride (AlF4-), yields stable complexes which

24 also blocked by pre-treatment of cells with aluminum fluoride (AlF4-).

25 Beryllium fluoride and aluminum fluoride also reduce this rate, and they increa

26 ivators PspF and NifA in the presence of ADP-aluminum fluoride, an analog of ATP in the transition st

27 nding of phosphate, or the phosphate analogs aluminum fluoride and beryllium fluoride, to actin filam

28 ass II-containing complexes was inhibited by aluminum fluoride and brefeldin A, indicating the involv

29 nd (15)N-ChiT in the presence and absence of aluminum fluoride and guanosine 5'-3-O-(thio)triphosphat

30 of vanadate trapped MgADP (MgADPVi-S1) with aluminum fluoride and scandium fluoride trapped MgADP (M

31 We speculate that beryllium fluoride and aluminum fluoride bind to the HPO4(2-) binding site.

32 (18)F-AlF-PSMA-11 using direct labeling with aluminum fluoride can be produced in NH(4)OAc, pH 6.9; s

33 constituted with G(betagamma) as assessed by aluminum fluoride-dependent changes in intrinsic tryptop

34 ADP-aluminum fluoride-dependent interactions and considerati

35 Another ATP analog, ADP-aluminum fluoride, does not promote unwinding.

36 Ca(2+)-ATPases to form stable complexes with aluminum fluoride (E2.AlF) and beryllium fluoride (E2.Be

37 Aluminum fluoride has become an important tool for inves

38 Inclusion of aluminum fluoride in the chase media reversibly inhibite

39 either adenosine 5'-O-(thiotriphosphate) or aluminum fluoride in the presence of Mg(2+).

40 tide binding or hydrolysis, or activation by aluminum fluoride, indicating that the effects on recept

41 Recent work demonstrating aluminum fluoride-induced complex formation between Ras

42 on of cofilin phosphorylation in response to aluminum fluoride induction in B16-F1 cells, which event

43 e phosphate analogues beryllium fluoride and aluminum fluoride led to complete inhibition of ATPase a

44 lipid vesicles was observed upon addition of aluminum fluoride or excess unlabeled alpha subunit, ind

45 n, but activation of F-alpha with either GDP/aluminum fluoride or guanosine 5'-O-(3-thiotriphosphate)

46 and gamma-phosphate analogues (beryllium and aluminum fluoride) or when dialyzed into guanosine 5'-3-

47 Activation of G proteins by treatment with aluminum fluoride produced an accumulation within the er

48 o first-order inhibition is dependent on the aluminum fluoride species, AlF4, and is linear with [Al]

49 rresponding reduction in charge in the bound aluminum fluoride species, which changes to a trifluoroa

50 The kinetics of aluminum fluoride-stimulated dissociation were slower th

51 locked activation of PKD by RhoQ63L, Lbc, or aluminum fluoride-stimulated Galpha(13).

52 in PKD activity induced by RhoQ63L, Lbc, or aluminum fluoride-stimulated Galpha(13).

53 ls injected with GTPgammaS or incubated with aluminum fluoride, suggesting a general role for GTPases

54 e) or 5'-adenylylimido-diphosphate), ADP, or aluminum fluoride that is postulated to trap ATPases at

55 Addition of aluminum fluoride to cells co-transfected with PKD and w

56 The addition of aluminum fluoride to cells co-transfected with PKD and w

57 rescence and subcellular fractionation after aluminum fluoride treatment, suggesting that it occurs w

58 hGBP-1 from efficiently redistributing after aluminum fluoride treatment.

59 P-1 remained cytosolic in cells treated with aluminum fluoride unless the cells were preincubated wit

60 Region I to activator in the presence of ADP-aluminum fluoride was shown and inferred from in vivo su

61 lpha(T)(*) linker mutants were responsive to aluminum fluoride, which binds to alpha-GDP complexes an