ライフサイエンスコーパス: alveolar

1 e also observed septal thickening, decreased alveolar air space total volume and, septa surface densi

2 Here we compare Mtb growth in mouse alveolar (AMs), peritoneal (PMs), and liver (Kupffer cel

3 Col6a1(-/-) mouse displayed histopathologic alveolar and airway abnormalities.

4 among transcription factors specifying most alveolar and bronchial epithelial lineages.

5 bedded lung tissue samples and quantified in alveolar and bronchial epithelium.

6 The lung is inhabited by resident alveolar and interstitial macrophages as well as monocyt

7 pression levels of alpha7nAChR in monocytes, alveolar and interstitial macrophages.

8 esults: Antenatal PHi therapy preserves lung alveolar and vascular growth and lung function and preve

9 HIF-2alpha may maintain alveolar architecture by promoting vascular survival and

10 artery is a branch of the posterior superior alveolar artery that supplies the lateral wall of the ma

11 ity assays performed in adenocarcinoma human alveolar basal epithelial (A549) and highly metastatic m

12 includes 2 mineralized tissues, cementum and alveolar bone (AB), both essential for tooth attachment.

13 Linear measurements of alveolar bone (radiographic bone level [rBL]), assessed

14 Teeth are attached to alveolar bone by the periodontal ligament (PDL), which c

15 hanges in bone trabeculation, changes in the alveolar bone can be detected quantitatively.

16 ors related to the resorption of the palatal alveolar bone caused by tooth movement after the maxilla

17 ion and junctional epithelium, cementum with alveolar bone crest destruction, but animals pretreated

18 (MSs) was compared at experimentally-created alveolar bone defects in rats.

19 confirmed decreased bone volume fraction and alveolar bone density.

20 mparable outcomes in terms of maintenance of alveolar bone dimensions, feasibility of implant placeme

21 ontia, microdontia, tooth root deficiencies, alveolar bone hypoplasia, and a range of skeletal malfor

22 Destruction of the alveolar bone in the jaws can occur due to periodontitis

23 C) functions and increased bacterial load in alveolar bone in vivo and whether DC inhibition alone pr

24 During periodontitis, tooth-supporting alveolar bone is resorbed when there is an increased exp

25 Desipramine administration reduced alveolar bone loss as histologically observed, and modul

26 t baseline, 5xFAD mice presented significant alveolar bone loss compared to WT mice.

27 ts surrounding dental implants, and reverses alveolar bone loss following extraction socket remodelin

28 fects the course of chronic inflammation and alveolar bone loss in females.

29 Alveolar bone loss measurements were made on histologica

30 flammation, gingival tissue destruction, and alveolar bone loss through sustained exacerbation of the

31 Alveolar bone loss was analyzed by micro-computed tomogr

32 Alveolar bone loss was significantly reduced in the liga

33 significantly reduced P. gingivalis-induced alveolar bone loss.

34 on of PD, both WT and 5xFAD mice experienced alveolar bone loss.

35 e could eventually arrest the RANKL-mediated alveolar bone loss.

36 4-DPCA/hydrogel), to promote regeneration of alveolar bone lost owing to experimental periodontitis.

37 We hypothesized that alveolar bone osteocytes develop senescence characterist

38 While long bone and alveolar bone osteocytes in Hyp mice overexpressed fibro

39 Alveolar bone osteocytes negatively regulate Gli1+ PDLSC

40 16(Ink4a) mRNA expression were identified in alveolar bone osteocytes with aging.

41 d appeared to be a protective factor against alveolar bone osteonecrosis.

42 Alveolar bone resorption and gingival collagen fibers we

43 Tooth extraction results in alveolar bone resorption and is accompanied by postopera

44 RP-SG) is indicated to attenuate physiologic alveolar bone resorption as a consequence of tooth extra

45 tor and TES on the inflammatory response and alveolar bone resorption associated with ligature-induce

46 main effector Caspase-1 in inflammation and alveolar bone resorption associated with periodontitis.

47 However, the effect of boldine on alveolar bone resorption during periodontitis has not be

48 The alveolar bone resorption is a distinctive feature of per

49 The amount of palatal alveolar bone resorption was measured and various parame

50 n periodontal ligament (PDL) disintegration, alveolar bone resorption, and ultimately tooth loss.

51 Hence, its actions on alveolar bone resorption, gingival collagen content and

52 Boldine inhibited the alveolar bone resorption.

53 dy is to evaluate the changes in the palatal alveolar bone thickness and find the factors related to

54 Palatal alveolar bone thickness changes and resorption factors w

55 es of maxillary central incisors and palatal alveolar bone thickness were measured, and the correspon

56 Hyp mandibles demonstrated expanded alveolar bone with accumulation of osteoid, and micro-CT

57 s were disturbed in Hyp versus WT long bone, alveolar bone, and cementum, including osteocyte/cemento

58 tial stem cells (PDLSCs) giving rise to PDL, alveolar bone, and cementum.

59 tructure with surrounding native dentine and alveolar bone, Raman microspectroscopy analysis is used.

60 tion in loads transfer and remodeling of the alveolar bone.

61 tors play in the volumetric reduction of the alveolar bone.

62 that were localized to regions of mucus and alveolar-bronchiolar hyperplasia, proliferations of type

63 We show that alveolar capillaries are mosaics; similar to the epithel

64 s, development, renewal and evolution of the alveolar capillary endothelium.

65 Alveolar capillary microthrombi were 9 times as prevalen

66 minative role in bacterial dissemination and alveolar-capillary barrier dysfunction, and edema toxin

67 we examined the structural integrity of the alveolar-capillary barrier in archival formalin-fixed lu

68 rane of gram-negative bacteria, disrupts the alveolar-capillary barrier, triggering pulmonary vascula

69 Ethambutol concentrations were highest in alveolar cells (alveolar cells:plasma ratio 15.0, 95% CI

70 gulated host response, followed by damage to alveolar cells and lung fibrosis.

71 cCRE linked to SLC6A20, a gene expressed in alveolar cells and with known functional association wit

72 oncentrations in epithelial lining fluid and alveolar cells than plasma.

73 a putative mechanism used for viral entry in alveolar cells.

74 ma, lung/airway epithelial lining fluid, and alveolar cells.

75 ncentrations were highest in alveolar cells (alveolar cells:plasma ratio 15.0, 95% CI 11.4-18.6).

76 sulted in type 2 AEC-like cells (iAEC2) with alveolar characteristics.

77 despite their significant restoration in the alveolar compartment of the lung as well as in the perip

78 e of airway epithelial-like cells within the alveolar compartments of the lung, herein termed bronchi

79 ble), nausea and vomiting, sedation, minimal alveolar concentration of volatile anesthetic, fatigue,

80 Study endpoints included radial alveolar counts (RACs), vessel density, and right ventri

81 ested to determine alveolarization by radial alveolar counts, pulmonary vessel density, and right ven

82 surgical flap was placed within 3 mm of the alveolar crest (286/306 sites) as opposed to 50% when th

83 en the surgical flap was >3 mm away from the alveolar crest (48/96 sites).

84 ioning the surgical flap more closely to the alveolar crest when performing osseous surgery resulted

85 t level, gingival bleeding, and radiographic alveolar crestal height (ACH).

86 ic observations were consistent with diffuse alveolar damage (70/70) and capillary dilatation and con

87 Both lungs showed various stages of diffuse alveolar damage (DAD), including edema, hyaline membrane

88 red in the absence of histologically evident alveolar damage and abundance of neutrophils in the pare

89 The major pulmonary finding was diffuse alveolar damage in the acute or organising phases, with

90 Coronavirus infection causes diffuse alveolar damage leading to acute respiratory distress sy

91 c pattern in the peripheral lung was diffuse alveolar damage with perivascular T-cell infiltration.

92 cient, but drawbacks include muscle atrophy, alveolar damage, and reduced mobility.

93 ted with the pathologic processes of diffuse alveolar damage, capillary dilatation and congestion, an

94 proinflammatory substances, causing diffuse alveolar damage, which is the histopathological basis of

95 damage with organizing pneumonia and diffuse alveolar damage.

96 tocyte growth factor), a protein involved in alveolar development and homeostasis.

97 asia, characterized by interrupted postnatal alveolar development and increased morbidity to respirat

98 lated angiogenesis and impaired vascular and alveolar development.

99 (+) differentiation while promoting adaptive alveolar differentiation into SFTPC(+) epithelium.

100 found to have lymphocytic bronchiolitis and alveolar ductitis with B-cell follicles and emphysema, i

101 ed marked epithelial hyperplasia in proximal alveolar ducts and adjacent alveoli with associated cent

102 Human cystic and alveolar echinococcosis are among the priority neglected

103 Significant alveolar echinococcosis hotspots were detected in the Os

104 gical incidence of cystic echinococcosis and alveolar echinococcosis in Kyrgyzstan.

105 surgical cases of cystic echinococcosis and alveolar echinococcosis reported through the national ec

106 in Uch-Dobo (Alay district, Osh region) for alveolar echinococcosis.

107 nococcus multilocularis causes in humans the alveolar echinococcosis.

108 ovel positive regulator of ST2 expression in alveolar ECs to generate retinoic acid (RA) and supports

109 nd supports the synthesis of RA from type II alveolar ECs to suppress excessive activation of innate

110 the epithelium that lines the alveolus, the alveolar endothelium is made up of two intermingled cell

111 l ICs offer a unique resource to study human alveolar epithelial biology.

112 Rationale: Alveolar epithelial cell (AEC) injury and dysregulated r

113 how amelioration of lung function, decreased alveolar epithelial cell apoptosis, and fibroblast proli

114 ly benefit from reproducible availability of alveolar epithelial cells (AEC).

115 murine lower airway tissues, primary type II alveolar epithelial cells (AECIIs), and the mouse lung c

116 When alveolar epithelial cells (AECs) lacked Sting or gap jun

117 We treated HO-exposed mice and primary alveolar epithelial cells (AECs) with the novel TREK-1 a

118 dent AMs can blunt inflammatory signaling in alveolar epithelial cells (ECs) by transcellular deliver

119 that miR-17 and miR-548b were upregulated in alveolar epithelial cells after CI/EVR, which merit furt

120 r cell polarity (PCP) pathway is required in alveolar epithelial cells and myofibroblasts for alveolo

121 Primary alveolar epithelial cells can be derived from human lung

122 To overcome the scarcity of primary human alveolar epithelial cells for lung research, and the lim

123 Atypical alveolar epithelial cells in IPF express molecular marke

124 epithelial cells, with reduced expression in alveolar epithelial cells in IPF lungs.

125 unknown etiology; however, apoptosis of lung alveolar epithelial cells plays a role in disease progre

126 ung is determined by their interactions with alveolar epithelial cells, in particular alveolar type 1

127 Alveolar epithelial cells, myofibroblasts and endothelia

128 Using lung microvascular endothelial and alveolar epithelial cells, we demonstrated that N-WASP d

129 is expressed predominantly in bronchial and alveolar epithelial cells, with reduced expression in al

130 ired for SARS-CoV-2 viral infection of human alveolar epithelial cells.

131 led bacterial growth in both macrophages and alveolar epithelial cells.

132 positive signals of the 2 miR expression in alveolar epithelial cells.

133 ell as TGF-beta-mediated permeability across alveolar epithelial cells.

134 en (KL)-6 is pathophysiological biomarker of alveolar epithelial damage.

135 ed a new method to immortalise primary human alveolar epithelial lung cells using a non-viral vector

136 In all four settings, alveolar epithelial progenitor (AT2) cells expressing on

137 ifferentiation that allows us to investigate alveolar epithelial progenitor cell differentiation in v

138 of the STAT3-BDNF-TrkB axis in orchestrating alveolar epithelial regeneration.

139 s reveals novel mesenchymal and transitional alveolar epithelial states unique to LAM lung.

140 kinetics in Vero E6 cells and primary human alveolar epithelial tissues were not affected.

141 ions in adult mice by reducing the number of alveolar epithelial type 2 (AT2) cells.

142 Alveolar epithelial type 2 cells (AEC2s) are the faculta

143 The virus infects alveolar epithelial type 2 cells (AT2s), leading to lung

144 as organoids derived from single adult human alveolar epithelial type II (AT2) or KRT5(+) basal cells

145 -derived air-liquid interface (ALI) model of alveolar epithelial type II (ATII)-like cell differentia

146 Sftpc-cRaf-IKK2DN) in cRaf-induced tumors in alveolar epithelial type II cells restricted tumor forma

147 structure and composition of the airway and alveolar epithelium in regions of fibrosis.

148 Here, we show that collaboration with the alveolar epithelium is critical for controlling infectio

149 The absence of ex vivo models of human alveolar epithelium is hindering an understanding of cor

150 how Legionella-infected macrophages use the alveolar epithelium to metabolically process myeloid cel

151 IL-1 induces the alveolar epithelium to produce granulocyte-macrophage co

152 at simulates the initial apical infection of alveolar epithelium with SARS-CoV-2 by using induced plu

153 This highlights the alveolar epithelium's importance as a key signaling brid

154 lecular characteristics of the healthy human alveolar epithelium, we have developed a new method to i

155 imic the cellular heterogeneity in the human alveolar epithelium.

156 tissue factor and C5a), and multifocal intra-alveolar fibrin deposition.

157 Conditional ablation of IGF1 in alveolar fibroblasts or deletion of the IGF-1 receptor f

158 ing acute eosinophilic pneumonia and diffuse alveolar hemorrhage, have also been reported.

159 ild-type strain caused high mortality, intra-alveolar hemorrhages, extensive alveolar septal sequestr

160 on important for surfactant biosynthesis and alveolar homeostasis.

161 ty (high ADC values) suggesting diagnosis of alveolar hydatid.

162 genation and lung aeration but may result in alveolar hyperinflation and hemodynamic alterations.

163 lung and could reduce both the occurrence of alveolar hypoxia and absorption atelectasis, thus optimi

164 Neutrophils may contribute to the diffuse alveolar inflammation seen in acute respiratory distress

165 ng microbiota are altered and correlate with alveolar inflammation.

166 uding high levels of inflammatory cytokines, alveolar inflammatory infiltrates and vascular microthro

167 duction of alveolar type 1 cells, failure of alveolar inflation and early postnatal lethality in mous

168 on the lung, provoking both inflammation and alveolar injury.

169 Rhinoceros bronchial alveolar lavage fluid (BALF) was found to have an inhibi

170 ctors Stat1 and Rorc Additionally, bronchial alveolar lavage fluid from infected IL-8R2-deficient mic

171 ce, histological lung injury scores, broncho-alveolar lavage protein levels and cell counts, and IL-6

172 ginated Obstruction Response (POOR) in which alveolar leak leads to surfactant dysfunction and increa

173 oid models to finely map the trajectories of alveolar-lineage cells during injury repair and lung reg

174 physiologically relevant human bronchial and alveolar lung mucosa models cultured at air-liquid inter

175 he lung cells, especially on the predominant alveolar macrophage (AM) population, is limited.Objectiv

176 intracellular depot of ciprofloxacin to the alveolar macrophage compartment that was sustained over

177 lammatory cytokine, plays a critical role in alveolar macrophage homeostasis, lung inflammation and i

178 controlling CXCL13 gene expression in human alveolar macrophages (AM) and monocyte-derived macrophag

179 Alveolar macrophages (AM) are the most prominent immune

180 Alveolar macrophages (AMs) and epithelial cells (ECs) ar

181 Alveolar macrophages (AMs) are highly abundant lung cell

182 Alveolar macrophages (AMs) derived from embryonic precur

183 Resident alveolar macrophages (AMs) suppress allergic inflammatio

184 he number of classical (SiglecFhighCD11bneg) alveolar macrophages (AMs) was reduced by approximately

185 tion of transcriptomes and open chromatin of alveolar macrophages (AMs), interstitial macrophages (IM

186 Interestingly, alveoli outnumber alveolar macrophages (AMs), which favors alveoli devoid

187 one marrow and an increase in ferroportin on alveolar macrophages (AMs).

188 Tissue-resident (TR) alveolar macrophages (APhi) are long-lived, self-renew a

189 axis and differentiate into monocyte-derived alveolar macrophages (Mo-AMs), which is a cell populatio

190 n mouse models, depletion of tissue-resident alveolar macrophages (TRAMs) attenuated neutrophil recru

191 two macrophage populations, tissue-resident alveolar macrophages and interstitial macrophages, which

192 over lnc-IL7R levels were reduced in lavaged alveolar macrophages and primary human small airway epit

193 Alveolar macrophages are among the first immune cells th

194 After injury, tissue-resident alveolar macrophages are depleted, and monocytes from th

195 el self-promoting mechanism of activation of alveolar macrophages by arachidonate 15-lipoxygenase-der

196 n to humans, the bacteria proliferate within alveolar macrophages causing pneumonia.

197 R2 expression in a subset of mouse and human alveolar macrophages further highlights EGR2 as a conser

198 e airway immune cell repertoire shifted from alveolar macrophages in healthy control subjects to a pr

199 Here, we investigated the impact of alveolar macrophages on A. baumannii pneumonia using a m

200 Apoptotic cell uptake by lung alveolar macrophages suppressed HDM-driven allergic asth

201 h and was only found in lung parenchyma and alveolar macrophages thereafter.

202 e production, reduced phagocytic function of alveolar macrophages, and consequently, increased pneumo

203 en genes, IgG4-rich immune complexes coating alveolar macrophages, and increased immunostaining for p

204 e alpha7nAChR in granulocytes, monocytes and alveolar macrophages, and low expression levels of alpha

205 s and Main Results: Adding IFN-beta to MDMs, alveolar macrophages, and PBECs prior to, but not after,

206 461630

207 the airways and expressed on the surface of alveolar macrophages, dendritic cells, innate lymphoid t

208 hyperoxia caused a shift in the phenotype of alveolar macrophages, increasing proportion of cells wit

209 an primates, Siglec-1 is highly expressed by alveolar macrophages, whose abundance correlates with pa

210 cterium Francisella tularensis, that infects alveolar macrophages.

211 gic airway inflammation through an action on alveolar macrophages.

212 and protected intracellular localization in alveolar macrophages.

213 s of myeloid regulatory cells, monocytes and alveolar macrophages.

214 rs postnatally and is thought to require the alveolar myofibroblast (AMF).

215 al mandible fractures involving the inferior alveolar nerve (15 affected IAN and 15 unaffected IAN fr

216 col for direct visualization of the inferior alveolar nerve in the setting of mandibular fractures.

217 derlying changes in respiratory quotient and alveolar oxygen tension during venovenous extracorporeal

218 Bilateral alveolar pattern (odds ratio = 1.67 [1.03-2.69]; p = 0.0

219 , we found pulmonary emphysematous-appearing alveolar patterns in the lungs of mgR mice.

220 oliferate and transdifferentiate into type I alveolar pneumocytes (AT1 cells).

221 This process occurs when type II alveolar pneumocytes (AT2 cells) proliferate and transdi

222 n significant declines in community-acquired alveolar pneumonia (CAAP) and overall chest radiography

223 Community-acquired alveolar pneumonia (CAAP) is considered a bacterial dise

224 bsorption atelectasis, and maintain the same alveolar PO2, by increasing the extracorporeal oxygen de

225 nt of the native lung and thus influence the alveolar PO2.

226 ing growth failure, renal disease, pulmonary alveolar proteinosis, autoimmune disorders and osteoporo

227 ons shows how an inflammatory niche controls alveolar regeneration by controlling stem cell fate and

228 ient progenitors (DATPs), that arises during alveolar regeneration.

229 e Krt8 + transitional stem cell state during alveolar regeneration.

230 atter with notable deposition in the fragile alveolar region of the lungs.

231 ed and type 2 pneumocyte cells in airway and alveolar regions, respectively.

232 be used in the future to study modulation of alveolar repair by (pharmaceutical) compounds.

233 ALI-iAEC2 were used to study alveolar repair over a period of 2 weeks following mecha

234 lts demonstrated the feasibility of studying alveolar repair using hiPSC-AEC2 cultured at the ALI and

235 marked by high H2-K1 expression critical for alveolar repair.

236 multiple miEC populations and contribute to alveolar revascularization after injury.

237 The clinically aggressive alveolar rhabdomyosarcoma (RMS) subtype is characterized

238 nfavorable prognosis of PAX3-FOXO1 fusion in alveolar rhabdomyosarcoma.

239 ssment of grafted sites following horizontal alveolar ridge augmentation.

240 in less buccal plate resorption and a wider alveolar ridge by day 21.

241 resh extraction socket may partly reduce the alveolar ridge contraction and that several factors like

242 let-rich fibrin (PRF) membranes can preserve alveolar ridge dimension after tooth extraction.

243 After minimally traumatic tooth extraction, alveolar ridge dimensions were measured using a custom-f

244 at the favorable effects of PRF membranes in alveolar ridge preservation may be attributed, at least

245 Alveolar ridge preservation via socket grafting (ARP-SG)

246 ical and radiographic dimensional changes in alveolar ridge width with an average horizontal bone gai

247 e identify GORAB as a regulator of embryonic alveolar sac formation as genetically disrupting the Gor

248 ung mesenchyme fibroblasts, and suggest that alveolar sac formation resembles a patterning event that

249 Embryonic development of the alveolar sac of the lung is dependent upon multiple sign

250 asts could, respectively, mimic or attenuate alveolar sac-like phenotypes in a co-culture model.

251 The local density of IMs was greater in the alveolar septa than in the connective tissue surrounding

252 Of the IMs, 78% were located within the alveolar septa, 14% around small vessels, and 7% around

253 IM density was greatest in the alveolar septa.

254 ality, intra-alveolar hemorrhages, extensive alveolar septal sequestration of bacteria and neutrophil

255 lays a critical role in ensuring appropriate alveolar septation during alveologenesis.

256 egulates pulmonary angiogenesis resulting in alveolar simplification mimicking BPD in neonatal mice,

257 proved elastin fiber organization, decreased alveolar simplification, and preserved lung function in

258 rogressed to severe airspace enlargement and alveolar simplification, with concurrent thinning in the

259 bundance of neutrophils in the parenchyma or alveolar space did not change at these time points.

260 helial Vegfa deletion; without Car4 ECs, the alveolar space is aberrantly enlarged despite the normal

261 M) are the most prominent immune cell in the alveolar space.

262 confirm prominent hyaluronan exudates in the alveolar spaces of Covid-19 lungs, supporting the notion

263 The relative abundance of AMs in the alveolar spaces, alongside their potential for nonspecif

264 changes and recruitment of neutrophils into alveolar spaces, which might be linked to a decrease in

265 d with bone morphogenetic protein signaling, alveolar specification, and tumor suppression.

266 ith lineage tracing revealed that airway and alveolar stem cells converge on a unique Krt8 + transiti

267 ca-induced fibrosis by reestablishing normal alveolar structure and decreasing both collagen accumula

268 al disease involving destruction of the lung alveolar structure.

269 that a local inflammatory niche develops in alveolar structures and drives the disease process.

270 d immune activation and cytokine response in alveolar structures.

271 ains low surface tensions and stabilizes the alveolar surface.

272 using synthetic peptide surrogates) act like alveolar surfactant proteins to rapidly bind and stabili

273 of ACE2 were significantly increased in both alveolar tissue and bronchial epithelium of patients wit

274 Compared to normoxia, the controls' alveolar-to-arterial oxygen gradient significantly incre

275 Vegfa is predominantly expressed by alveolar type 1 (AT1) cells and locally required to spec

276 d that loss of Cyp26b1 leads to reduction of alveolar type 1 cells, failure of alveolar inflation and

277 ith alveolar epithelial cells, in particular alveolar type 1 cells.

278 Alveolar type 2 (AT2) cells represent a metabolically ac

279 se of a present pandemic, infects human lung alveolar type 2 (hAT2) cells.

280 reatment increased lung endothelial cell and alveolar type 2 cell proliferation.Conclusions: Postnata

281 ked to SARS-CoV-2 host entry gene TMPRSS2 in alveolar type 2 cells, which had immune regulatory signa

282 the propagation and differentiation of human alveolar type 2 cells/pneumocytes derived from primary l

283 This includes PDGF secretion from alveolar type I and type II cells, cell shape changes of

284 rotein concentrates in the basal membrane of alveolar Type I epithelial cells.

285 Grp78, a key regulator of ER homeostasis, in alveolar type II (AT2) cells, progenitors of distal lung

286 ulture model, comprising immortalised bovine alveolar type II (BATII) epithelial cells and bovine pul

287 ing and a reduced proliferative potential of alveolar type II cells and club cells, increased cellula

288 mor development is restricted to a subset of alveolar type II cells expressing Hnf1b.

289 Alveolar Type II progenitor cell density and self-renewa

290 cells and their terminal differentiation to alveolar type-1 cells.

291 MHC-II on alveolar type-II (AT-II) cells is associated with immune

292 The hPSC-LOs (particularly alveolar type-II-like cells) are permissive to SARS-CoV-

293 s that orchestrate the ejection of milk from alveolar units and its passage along the mammary ductal

294 hypoperfusion that is likely due to abnormal alveolar ventilation and perfusion.

295 eal pressure, lung aeration measured via CT, alveolar wall thickness, cell infiltration, and surfacta

296 and pneumomediastinum due to the rupture of alveolar walls and barotrauma in mechanically ventilated

297 ormal lungs only showed hyaluronan in intact alveolar walls and perivascular tissue.

298 2 activity in turn causes the destruction of alveolar walls leading to emphysema, making it potential

299 After septation, the alveolar walls thin to allow efficient gas exchange.

300 Alveolar walls were thinner (5.5 +/- 0.1 vs 7.8 +/- 0.2