ライフサイエンスコーパス: alveolar fluid clearance

1 by reducing lung inflammation and enhancing alveolar fluid clearance.

2 nary edema by up-regulating sodium-dependent alveolar fluid clearance.

3 eolar epithelial fluid transport measured as alveolar fluid clearance.

4 ndothelial barrier permeability and restored alveolar fluid clearance.

5 mechanisms may contribute to the decrease in alveolar fluid clearance.

6 bited both basal and beta agonist-stimulated alveolar fluid clearance.

7 is, neutrophil activation and clearance, and alveolar fluid clearance.

8 elial permeability to protein, and decreased alveolar fluid clearance.

9 ed elevated lung inflammation and attenuated alveolar fluid clearance.

10 a level comparable to maximal cAMP-dependent alveolar fluid clearance.

11 samples were associated with slower rates of alveolar fluid clearance.

12 genous catecholamines did not correlate with alveolar fluid clearance.

13 in and an approximately 50% reduction in net alveolar fluid clearance.

14 id instillation led to a 50% decrease in net alveolar fluid clearance.

15 ANTU-induced edema formation by potentiating alveolar fluid clearance.

16 e may be of more benefit than improvement of alveolar fluid clearance.

17 mild pulmonary oedema (24/29 [83%]), intact alveolar fluid clearance (17/23 [74%]), and normal or mi

18 ith serial samples, there was a high rate of alveolar fluid clearance (19 +/- 9%/h, mean +/- SD).

19 lar epithelial dysfunction, as determined by alveolar fluid clearance (AFC) and intra-alveolar levels

20 Furthermore, measurement of alveolar fluid clearance (AFC) demonstrated that A2BAR s

21 h acute lung injury (ALI) who retain maximal alveolar fluid clearance (AFC) have better clinical outc

22 Moreover, induced Na,K-ATPase improved alveolar fluid clearance (AFC) in IAV-infected mice.

23 of the NO donor, DETANONOate, would decrease alveolar fluid clearance (AFC) in the rabbit in vivo.

24 nfected with M. pulmonis for measurements of alveolar fluid clearance (AFC) in vivo and isolation of

25 reactive byproducts inhibit Na(+)-dependent alveolar fluid clearance (AFC) in vivo and the activity

26 Alveolar fluid clearance (AFC) is necessary for the reso

27 Whether these receptors are essential for alveolar fluid clearance (AFC) or if other mechanisms ar

28 by alveolar edema accumulation with reduced alveolar fluid clearance (AFC), alveolar-capillary barri

29 Alveolar fluid clearance (AFC), an index of active Na(+)

30 Alveolar fluid clearance (AFC), an index of alveolar act

31 A(2a)R- or A(3)R-specific agonists increased alveolar fluid clearance (AFC), whereas physiologic conc

32 g the inhibition of the c-AMP stimulation of alveolar fluid clearance (ALC) in rats.

33 aspiration-induced lung injury by increasing alveolar fluid clearance and decreasing endothelial perm

34 hese data suggest that claudin-4 may promote alveolar fluid clearance and demonstrate that the amount

35 subunit plasmid showed a twofold increase in alveolar fluid clearance and Na(+),K(+)-ATPase activity

36 ce to lung injury, db/db mice had diminished alveolar fluid clearance and reduced Na,K-ATPase functio

37 In isolated perfused lungs, alveolar fluid clearance and secretion were determined b

38 nce, fibrinogenesis, inflammatory cytokines, alveolar fluid clearance, and endothelial injury and act

39 panied by a 2.4-fold increase in the rate of alveolar fluid clearance at 4 hrs in the salmeterol-trea

40 Recently, we showed that impairment of alveolar fluid clearance because of inhibition of epithe

41 with control, LTD4 (1 x 10(-11) M) increased alveolar fluid clearance by 41% (p < 0.001) in isolated,

42 in BALB/c mice increased amiloride-sensitive alveolar fluid clearance by approximately 30%, consisten

43 ung and restored the normal up-regulation of alveolar fluid clearance by catecholamines after prolong

44 e lung prevented the normal up-regulation of alveolar fluid clearance by catecholamines following hem

45 Alveolar fluid clearance contributes to graft function a

46 Intact alveolar fluid clearance correlated with less histologic

47 Because a decrease in net alveolar fluid clearance could be due to lung endothelia

48 Alveolar fluid clearance driven by active epithelial Na(

49 The two patients with no net alveolar fluid clearance had persistent hypoxemia and mo

50 Patients with maximal alveolar fluid clearance had significantly lower mortali

51 edema and acute lung injury, we measured net alveolar fluid clearance in 79 patients with acute lung

52 -mediated inhibition of beta-agonist-induced alveolar fluid clearance in a murine model of trauma-sho

53 ring the beta- adrenergic agonist-stimulated alveolar fluid clearance in acute lung injury, an effect

54 The unimpaired alveolar fluid clearance in AQP5-null mice indicates tha

55 nfection has been shown to reduce Na+-driven alveolar fluid clearance in BALB/c mice in vivo.

56 g injury and the loss of claudin-4 decreases alveolar fluid clearance in mice.

57 in contrast to hydrostatic pulmonary edema, alveolar fluid clearance in patients with acute lung inj

58 K-ATPase in alveolar epithelial cells and on alveolar fluid clearance in rat lungs.

59 The high rates of alveolar fluid clearance indicate that the fluid transpo

60 red in the majority of patients, and maximal alveolar fluid clearance is associated with better clini

61 Airway and alveolar fluid clearance is mainly governed by vectorial

62 However, impaired alveolar fluid clearance is present in most of the patie

63 Of the patients, 56% had impaired alveolar fluid clearance (< 3%/h), 32% had submaximal cl

64 Measurement of alveolar fluid clearance may be useful to assess the sev

65 ins (including ZO-1) was not associated with alveolar fluid clearance or claudin-4 levels.

66 nction protein claudin-4 are associated with alveolar fluid clearance or clinical measures of lung fu

67 3) and in patients with an absence of intact alveolar fluid clearance (p =.03).

68 Maximal cAMP-dependent alveolar fluid clearance rate was 32.9 +/- 10.9 %/hr (p

69 Alveolar fluid clearance rates were measured in ex vivo

70 human lungs, mesenchymal stem cells restored alveolar fluid clearance, reduced inflammation, and exer

71 ing demonstrated a positive correlation with alveolar fluid clearance (Spearman rank correlation [r(s

72 Concomitantly, we observed a reversal of alveolar fluid clearance, suggesting that reversed trans

73 ts with acute lung injury (ALI) have reduced alveolar fluid clearance that has been associated with h

74 , and isoproterenol 10(-6) M each stimulated alveolar fluid clearance to a level comparable to maxima

75 -grade human mesenchymal stem cells restored alveolar fluid clearance to a normal level, decreased in

76 across the alveolar epithelium and restored alveolar fluid clearance to normal.

77 th hydrostatic pulmonary edema, in whom mean alveolar fluid clearance was 13%/h; only 25% had impaire

78 Mean alveolar fluid clearance was 6%/h.

79 Basal alveolar fluid clearance was 7.6 +/- 2.2 %/hr.

80 Net alveolar fluid clearance was calculated from sequential

81 Alveolar fluid clearance was calculated from serial samp

82 Compared with basal rates, alveolar fluid clearance was increased by both racemic a

83 Alveolar fluid clearance was measured by change in conce

84 Alveolar fluid clearance was measured in anesthetized, v

85 Alveolar fluid clearance was measured in living ventilat

86 Alveolar fluid clearance was measured in vivo.

87 r without electroporation, and 3 days later, alveolar fluid clearance was measured.

88 Alveolar fluid clearance was preserved in SP-C-GM mice i

89 Acute lung injury with maximal alveolar fluid clearance were more likely to be female (

90 eduction of Na,K-ATPase expression decreases alveolar fluid clearance, which in turn leads to pulmona