ライフサイエンスコーパス: alveolar wall

1 pectrum of injury and abnormal repair of the alveolar wall.

2 membrane to directly disseminate through the alveolar wall.

3 e lung to the most peripheral vessels in the alveolar wall.

4 hyperoxia-induced apoptosis of cells in the alveolar wall.

5 the epithelium of bronchi, bronchioles, and alveolar walls.

6 ed pulmonary inflammation and destruction of alveolar walls.

7 nd with a decrease in apoptotic cells in the alveolar walls.

8 ury and caspase-3 activation in cells of the alveolar walls.

9 l airways, airway epithelium, submucosa, and alveolar walls.

10 modeling process in the connective matrix in alveolar walls.

11 alveolar surface area due to destruction of alveolar walls.

12 ified the strain experienced in the adjacent alveolar walls.

13 t localized in intrapulmonary airways and in alveolar walls.

14 ha5 is prominent in the basement membrane of alveolar walls, airways, and pleura in developing and ad

15 nd overall inflammation in small airways and alveolar walls, along with alveolar wall thickening in 6

16 blast migration and proliferation within the alveolar wall and airspace after lung injury can lead to

17 iopsy showed lymphocytic infiltration of the alveolar wall and interlobular septa.

18 and pneumomediastinum due to the rupture of alveolar walls and barotrauma in mechanically ventilated

19 n the lungs of CD18-/- mice by 68 and 35% in alveolar walls and by 28 and 26% in venous walls, respec

20 the amplitude of the strain in the adjacent alveolar walls and extended the strain amplification dee

21 y, absence of TLOs attenuated destruction of alveolar walls and inflammation in bronchoalveolar lavag

22 ormal lungs only showed hyaluronan in intact alveolar walls and perivascular tissue.

23 zed initially by lymphocytic infiltration of alveolar walls and spaces, followed by an exuberant mono

24 incorporation of fibroblastic material into alveolar walls, and (c) cigarette smoke-related inflamma

25 ith a type II cell phenotype, grew along the alveolar walls, and projected into the alveolar septa.

26 alveolar septa; congested capillaries within alveolar walls; and congested capillaries within the wal

27 peroxia demonstrated only modest increase in alveolar wall apoptosis compared to room air.

28 prilysin had no effect on baseline airway or alveolar wall architecture, vessel density, cardiac func

29 tronger structural breakdown at the level of alveolar walls, as well as accelerated decay of stiffnes

30 expressed by epithelial cells that lined the alveolar walls, as well as by inflammatory cells and slo

31 rs pulmonary host defense, beyond effects on alveolar wall barrier function.

32 al bronchioles accompanied by destruction of alveolar walls, but without obvious fibrosis.

33 pase-3, Bax, and p53) were more prevalent in alveolar wall cells from the patients who died with ALI

34 aspase 3 demonstrated increased apoptosis in alveolar wall cells in wild-type mice in hyperoxia compa

35 eads to fixed narrowing of small airways and alveolar wall destruction (emphysema).

36 t adeno-associated virus markedly attenuated alveolar wall destruction and oxidative stress caused by

37 Emphysema is characterized by alveolar wall destruction induced mainly by cigarette sm

38 sema is a pulmonary disease characterized by alveolar wall destruction, resulting in enlargement of g

39 d inflammation in bronchoalveolar lavage and alveolar wall destruction.

40 the edge of a lung tumor and in surrounding alveolar walls experience increased strain during respir

41 The degree of alveolar space cellularity, alveolar wall fibrosis, and cellularity did not affect s

42 we show that a subset of AMs attached to the alveolar wall form connexin 43 (Cx43)-containing gap jun

43 BAL fluid probably reflect a high burden of alveolar wall granulomas and/or the removal of granuloma

44 ngs at day 4 revealed less disruption of the alveolar wall in SP-C-GM mice compared to wild-type mice

45 cinoma, the tumour cells slowly spread along alveolar walls in a non-destructive manner and have low

46 fter zoster revealed SVV antigen in the lung alveolar wall, in ganglionic neurons and nonneuronal cel

47 ter tail vein injection of tumor cells, with alveolar wall infiltration seen with AQP1-expressing tum

48 ons were characterized by hyperplasia of the alveolar walls, infiltration of polymorphonuclear leukoc

49 Emphysema causes a permanent destruction of alveolar walls leading to airspace enlargement, loss of

50 2 activity in turn causes the destruction of alveolar walls leading to emphysema, making it potential

51 gs show that IAV infection blocked defensive alveolar wall liquid (AWL) secretion and induced airspac

52 compartments, studies on the composition of alveolar wall liquid, and neutrophil and platelet traffi

53 Mechanical stress to alveolar walls may cause progressive damage after an ear

54 interstitial cells were demonstrated in the alveolar wall of normal Lewis rat lung.

55 valuate the binding kinetics of f-PPE in the alveolar walls of normal mouse lungs.

56 To explore the mechanism of alveolar wall remodeling after elastolytic injury, we ex

57 ion associated with collagen degradation and alveolar wall rupture.

58 Multiple gross disruptions of the alveolar walls, suggestive of stress fractures.

59 ppearance of an inflammatory reaction in the alveolar wall that can be triggered by different antigen

60 e COPD: in spite of the destructive process, alveolar wall thickening and focal fibrosis may be detec

61 ulmonary neutrophil infiltration and reduced alveolar wall thickening by 45% (p < 0.05).

62 Modeling alveolar wall thickening by encircling the tumor with th

63 small airways and alveolar walls, along with alveolar wall thickening in 67 subjects undergoing lung

64 Histopathological correlates of acid injury (alveolar wall thickening, edema, and leukocyte infiltrat

65 prenatal hormone exposure was a decrease in alveolar wall thickness and an increase in aerated paren

66 ar myofibroblast proliferation and increased alveolar wall thickness compared with saline-treated con

67 Measurements of the alveolar wall thickness in clinical and mouse model samp

68 Alveolar wall thickness was increased in all smokers, bu

69 ormone exposure, but there was a decrease in alveolar wall thickness with advancing gestation and at

70 eal pressure, lung aeration measured via CT, alveolar wall thickness, cell infiltration, and surfacta

71 cient mice exhibited significantly decreased alveolar wall thickness, inflammatory cell infiltration,

72 After septation, the alveolar walls thin to allow efficient gas exchange.

73 en and elastin fibers to represent pulmonary alveolar wall tissue.

74 which Mycobacterium tuberculosis crosses the alveolar wall to establish infection in the lung is not

75 fibroblastic foci, an abnormal adjacency of alveolar walls was seen, suggesting alveolar collapse.

76 ent stages of bacterial crossing through the alveolar wall, we established a two-layer transwell syst

77 Alveolar walls were thinner (5.5 +/- 0.1 vs 7.8 +/- 0.2

78 ficantly larger in stretched than in relaxed alveolar walls with a linear relation between k(off) and

79 sease in which fibroblasts accumulate in the alveolar wall within a type I collagen-rich matrix.