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2 ic automation and parallelization of Limulus amebocyte lysate (LAL)-based bacterial endotoxin testing
5 ither L-37pA nor D-37pA affected the Limulus amebocyte lysate activity of LPS, indicating that LPS up
6 sessed by LPS-induced coagulation of limulus amebocyte lysate and production of tumor necrosis factor
7 -33 inhibited the coagulation of the Limulus amebocyte lysate and the secretion of TNF-alpha by RAW 2
8 ological activity examined using the Limulus amebocyte lysate assay and nitric oxide production by al
9 aluated levels of plasma LPS (by the Limulus amebocyte lysate assay) and immune activation markers in
10 esence of endotoxin (detected by the Limulus amebocyte lysate assay) was compared to the presence of
11 ain were analyzed for endotoxin/LPS (Limulus amebocyte lysate assay), lipid profile, troponin, and Ig
12 -rich plasma were detected using the limulus amebocyte lysate assay, according to differential antibi
13 eat stable, reacts positively in the Limulus amebocyte lysate assay, and can be inhibited by polymyxi
14 ch is difficult to estimate with the Limulus amebocyte lysate assay, and cannot be completely neutral
15 measured by a kinetic turbidimetric limulus amebocyte lysate assay, and serum NO metabolite (NOx) co
16 and lipopolysaccharide (LPS) by the limulus amebocyte lysate assay, at presentation and after antivi
17 ant showed decreased toxicity by the Limulus amebocyte lysate assay, reduced adherence to human epith
18 nificantly decreased toxicity by the Limulus amebocyte lysate assay, reduced resistance to normal hum
19 s of LPS bioactivity, using modified limulus amebocyte lysate assay, suggest that recombinant factor