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1 plays an essential role in enamel formation (amelogenesis).
2  and for the robust nature of the process of amelogenesis.
3 ibute to its physiological regulation during amelogenesis.
4 ssion throughout the developmental stages of amelogenesis.
5 eater than optimal levels of fluoride during amelogenesis.
6 modification of gene expression important to amelogenesis.
7 le for nascent amelogenin polypeptide during amelogenesis.
8 tructure during the early secretory stage of amelogenesis.
9 TP) effectors during events related to early amelogenesis.
10 bility and organization of amelogenin during amelogenesis.
11 oblasts during the early maturation stage of amelogenesis.
12 vention of abnormal crystal formation during amelogenesis.
13  expressed throughout the secretory stage of amelogenesis.
14 namel proteins during the secretory phase of amelogenesis.
15  in reduced ameloblasts at the conclusion of amelogenesis.
16 trix protein (EMP), plays a critical role in amelogenesis.
17 ed nor whether their activity changes across amelogenesis.
18 us in secretory stage, thus participating in amelogenesis.
19 f ACP4 function during normal and pathologic amelogenesis.
20 in endosome/lysosome-related pathways during amelogenesis.
21 e clarified due to a lack of knowledge about amelogenesis.
22 ys regulate dental epithelial stem cells and amelogenesis.
23 ccurs in the developing enamel matrix during amelogenesis.
24 f GPR68 in the enamel organ at all stages of amelogenesis.
25 ence for the crucial function of ACPT during amelogenesis.
26 onal role in subsequent tooth patterning and amelogenesis.
27  material formed by the intricate process of amelogenesis.
28 eral formation during the secretory stage of amelogenesis.
29 ndant enamel matrix protein expressed during amelogenesis.
30  enamel defects reflecting failure of normal amelogenesis.
31 uring the secretory and maturation stages of amelogenesis.
32 s led us to investigate the effect of BPA on amelogenesis.
33 n ameloblasts during the maturation stage of amelogenesis.
34 efect in tooth enamel that reflects impaired amelogenesis.
35 ing ameloblast integrity and function during amelogenesis.
36 f these genes during the maturation stage of amelogenesis.
37 ble function in enamel mineralization during amelogenesis.
38  enamel extracellular matrix proteins during amelogenesis.
39 ing a possible signal for the termination of amelogenesis.
40  enamel matrix during the secretory stage of amelogenesis.
41 t that AE2 and NBCe1 play important roles in amelogenesis.
42 hat accumulate during the secretory stage of amelogenesis?
43                                     Although amelogenesis and dentinogenesis initiate in the molar to
44 ure investigations into mechanisms governing amelogenesis and introduce another feature to consider w
45  of enamel with the developmental process of amelogenesis and structural development, since they are
46  periodontal ligament is required for normal amelogenesis and that periostin is critically required f
47 h decay (dental caries), enamel development (amelogenesis) and associated pathologies (e.g., amelogen
48 ss in our understanding of enamel formation (amelogenesis) and the functional properties of mature en
49 tor (CSF-1) is a key regulatory cytokine for amelogenesis, and ameloblasts synthesize CSF-1.
50 iological control over crystal growth during amelogenesis, and hint at implications for the preservat
51 hoprotein contributes to the early events of amelogenesis, and in particular to those events that res
52 in is a cell adhesion molecule essential for amelogenesis, and it plays a role in maintaining the dif
53 s ameloblastin during the secretory stage of amelogenesis, and we present a hypothesis about the sequ
54 ngth originate during the secretory stage of amelogenesis as 2-nm-thin and 15-nm-wide ribbons that de
55 d a rapid rate of tooth enamel elongation or amelogenesis at 0.24 mm/day with dental tissues common t
56                   Starting with the onset of amelogenesis beneath the future cusp tips, the shape of
57  Data suggest that BPA exerts its effects on amelogenesis by disrupting normal protein removal from t
58 nclusion, miR-153 regulates maturation-stage amelogenesis by targeting key genes involved in the endo
59 tified in this study is sufficient to affect amelogenesis causing AI, but not so severe as to be inco
60 Our findings demonstrate that early and late amelogenesis depend upon cell-matrix interactions.
61 he OMIM database was searched with the terms amelogenesis, enamel, dental, and tooth, and all results
62                     These data suggest rapid amelogenesis evolved among non-hadrosauriform iguanodont
63 and growth of hydroxyapatite crystals during amelogenesis generate a large number of protons that mus
64                  The pivotal role of AMBN in amelogenesis has been confirmed experimentally using mou
65             Among ornithopods, high rates of amelogenesis have been previously observed in hadrosauri
66                     Recent studies on rodent amelogenesis have identified a multitude of gene product
67 om aberrations during the secretory stage of amelogenesis, hypomaturation AI indicates a deficiency o
68  in SLC24A4 in a family with hypomineralized amelogenesis imperfect a (AI), a condition in which toot
69 ciated with autosomal-dominant hypocalcified amelogenesis imperfecta (ADHCAI), which is typically cha
70 q24.3 cause autosomal-dominant hypocalcified amelogenesis imperfecta (ADHCAI).
71             Autosomal dominant hypocalcified amelogenesis imperfecta (ADHCAI; OMIM #130900) is a gene
72  is significant because RELT mutations cause amelogenesis imperfecta (AI) and this directly links ADA
73                                          The Amelogenesis Imperfecta (AI) are a group of clinically a
74                                              Amelogenesis Imperfecta (AI) can be caused by the defici
75   Hereditary enamel disorders referred to as amelogenesis imperfecta (AI) can severely affect the dev
76                                              Amelogenesis imperfecta (AI) comprises a group of rare,
77                                              Amelogenesis imperfecta (AI) describes a broad group of
78                                              Amelogenesis imperfecta (AI) describes a clinically and
79                                              Amelogenesis imperfecta (AI) describes a heterogeneous g
80                                Patients with amelogenesis imperfecta (AI) have defective enamel; ther
81 ge would lead to an enamel defect similar to amelogenesis imperfecta (AI) in humans, we generated tra
82  generalized hypoplastic autosomal-recessive amelogenesis imperfecta (AI) in individuals from six app
83                                              Amelogenesis imperfecta (AI) is a broad group of heredit
84                                              Amelogenesis imperfecta (AI) is a collection of genetic
85                                Non-syndromic amelogenesis imperfecta (AI) is a collection of isolated
86                                              Amelogenesis imperfecta (AI) is a collective term for fa
87                                              Amelogenesis imperfecta (AI) is a diverse group of inher
88                                              Amelogenesis imperfecta (AI) is a group of inherited con
89                                              Amelogenesis imperfecta (AI) is a group of inherited def
90                                              Amelogenesis imperfecta (AI) is a heterogeneous group of
91                                              Amelogenesis imperfecta (AI) is a heterogeneous group of
92                                              Amelogenesis imperfecta (AI) is a heterogeneous group of
93                                              Amelogenesis imperfecta (AI) is a heterogeneous group of
94                                              Amelogenesis imperfecta (AI) is a heterogeneous group of
95                                              Amelogenesis imperfecta (AI) is an innate disorder that
96                                              Amelogenesis imperfecta (AI) is group of inherited disor
97                                              Amelogenesis Imperfecta (AI) represents a group of hered
98 s the first gene involved in the etiology of amelogenesis imperfecta (AI) that does not encode a secr
99 ively inherited cone-rod dystrophy (CRD) and amelogenesis imperfecta (AI) was first reported by Jalil
100 ort stature with brachyolmia and hypoplastic amelogenesis imperfecta (AI) with almost absent enamel.
101  genes, FAM20A mutations are associated with Amelogenesis Imperfecta (AI) with gingival hyperplasia a
102 ied a family in which pitted hypomineralized amelogenesis imperfecta (AI) with premature enamel failu
103 f26, as a cause of recessive hypomineralized amelogenesis imperfecta (AI), a disease in which the for
104 in gene a prime candidate in the etiology of amelogenesis imperfecta (AI), a genetic disease in which
105              Dental enamel malformations, or amelogenesis imperfecta (AI), can be isolated or syndrom
106 d displayed severe enamel defects that mimic amelogenesis imperfecta (AI), with a rough, irregular en
107 AM gene have been found to cause hypoplastic amelogenesis imperfecta (AI), with phenotypes ranging fr
108 sulting in an inherited tooth enamel defect, amelogenesis imperfecta (AI).
109 d improve the clinical diagnosis of X-linked amelogenesis imperfecta (AI).
110 usly linked to an autosomal-dominant form of amelogenesis imperfecta (AI).
111 el phenotypes that are collectively known as amelogenesis imperfecta (AI).
112 s on X-chromosomal amelogenin cause X-linked amelogenesis imperfecta (AI).
113 spanic families with generalized hypoplastic amelogenesis imperfecta (AI).
114 alformed tooth enamel that phenocopies human amelogenesis imperfecta (AI).
115  altered conditions is collectively known as amelogenesis imperfecta (AI).
116    Defects in the enamelin gene (ENAM) cause amelogenesis imperfecta (AI).
117 ions in ACP4 cause non-syndromic hypoplastic amelogenesis imperfecta (AI1J, OMIM#617297).
118 ysin (MMP20) genes cause autosomal-recessive amelogenesis imperfecta (ARAI).
119                                     X-linked amelogenesis imperfecta (MIM 301200), a phenotypically d
120 features and inheritance pattern of X-linked amelogenesis imperfecta (XAI).
121 tin1, and Pax9 as candidate genes related to amelogenesis imperfecta and the NRF2-mediated pathway.
122    We furthermore develop a disease model of amelogenesis imperfecta in a three-dimensional (3D) orga
123  mutated amelogenin (TgP70T), which leads to amelogenesis imperfecta in humans, have heterogeneous en
124 ese findings suggest that hypoplastic pitted amelogenesis imperfecta is a feature of Kindler epidermo
125 logenesis) and associated pathologies (e.g., amelogenesis imperfecta or molar hypomineralization), an
126 hreonine, as in some cases of human X-linked amelogenesis imperfecta or when tyrosyl residues were su
127 hreonine, as in some cases of human X-linked amelogenesis imperfecta or when tyrosyl residues were su
128 c regions similar to those in enamel of male amelogenesis imperfecta patients with an identical mutat
129 in X-linked forms of the human enamel defect amelogenesis imperfecta resulting from amelogenin gene m
130 z syndrome, an epileptic encephalopathy with amelogenesis imperfecta that has parallels to V-ATPase-r
131 ferentiation and that its failure results in amelogenesis imperfecta through ectopic NRF2 activation.
132           The severity of hypoplastic pitted amelogenesis imperfecta varied from generalized to local
133 ions in amelogenin observed in patients with amelogenesis imperfecta who demonstrate defects in ename
134                                             'Amelogenesis imperfecta' (AI) describes a group of inher
135 ous variants in GPR68 in three families with amelogenesis imperfecta, a genetically and phenotypicall
136 e non-syndromic enamel malformations, termed amelogenesis imperfecta, and ablation of Mmp20 in mice r
137 ic changes in enamel phenotype, resulting in amelogenesis imperfecta, enamel that is defective and ea
138 APH, OMIM *614829) cause autosomal recessive amelogenesis imperfecta, however, the function of ODAPH
139 omes were the presence of hypoplastic pitted amelogenesis imperfecta, intraoral wounds, gingivitis an
140 erized by sensorineural hearing loss (SNHL), amelogenesis imperfecta, nail abnormalities, and occasio
141  Fam20 family lead to human diseases such as amelogenesis imperfecta, nephrocalcinosis, lethal and no
142 human enamelin gene cause autosomal dominant amelogenesis imperfecta.
143 tosomal-recessive hypoplastic-hypomaturation amelogenesis imperfecta.
144 efects have been identified in kindreds with amelogenesis imperfecta.
145 of undiagnosed autosomally dominant cases of amelogenesis imperfecta.
146  enamel are grouped under the classification amelogenesis imperfecta.
147 es the need for family-specific diagnosis of amelogenesis imperfecta.
148 the enamelin gene in kindreds suffering from amelogenesis imperfecta.
149 hreonine, as in some cases of human X-linked amelogenesis imperfecta.
150 ation identified in a case of human X-linked amelogenesis imperfecta.
151  defects in the enamel, a condition known as amelogenesis imperfecta.
152 opment result in enamel anomalies, including amelogenesis imperfecta.
153 Atg3(F/F) conditional knockout mice) exhibit amelogenesis imperfecta.
154 ability, early-onset epileptic seizures, and amelogenesis imperfecta.
155  or mutated, resulting in a condition called amelogenesis imperfecta.
156 consistent with the diagnosis of hypoplastic amelogenesis imperfecta.
157              Amelx and Mmp20 mutations cause amelogenesis imperfecta.
158 because defects in KLK4 cause hypomaturation amelogenesis imperfecta.
159                       Microcephaly (38%) and amelogenesis imperfecta/enamel dysplasia (42%) were addi
160 tivities that define the maturation stage of amelogenesis include ion (e.g., calcium and phosphate) t
161 rgeting of the molecular pathways regulating amelogenesis is an ongoing challenge in dental research,
162 y which the enamel organ regulates pH during amelogenesis is largely unknown.
163 s, we determined that the secretory stage of amelogenesis is not affected in Odaph(C41*/C41*) mice.
164 upting incisor enamel was normal, suggesting amelogenesis is only sensitive to MIH-causing agents dur
165                                              Amelogenesis is the process of dental enamel formation,
166                                              Amelogenesis is the process of enamel formation.
167 fecta, however, the function of ODAPH during amelogenesis is unknown.
168                            Enamel formation (amelogenesis) is a two-step process whereby crystals par
169                            Enamel formation (amelogenesis) is the result of highly orchestrated extra
170 ages of tooth development, especially during amelogenesis, it is unclear how mutant forms cause ARS d
171 hat, while FAM20C is a molecule essential to amelogenesis, its inactivation in the dental epithelium
172 al and biochemical processes observed during amelogenesis provided limited information on how extrace
173 hat PMCAs are functional Ca(2+) pumps during amelogenesis regulating (c) Ca(2+) upon low and/or moder
174 rrogate the relationship of cytoskeletal and amelogenesis-related defects.
175                               Any failure in amelogenesis results in defects in the enamel, a conditi
176  tooth number, crown and root morphology and amelogenesis that is likely due to a functional role of
177  from the secretory and maturation stages of amelogenesis, the serine protease chymotrypsin C (caldec
178 ogical role in organizing ameloblasts during amelogenesis, this study strengthens the hypothesis that
179                                          For amelogenesis to proceed, the cells of the inner enamel e
180 eral structure during the secretory stage of amelogenesis, utilizing Mmp20-null mice that lack this e
181                  The enamel matrix pH during amelogenesis was studied in 10 normal and 10 CF mice.
182 s that AMELX phosphorylation is critical for amelogenesis, we generated and characterized a hemizygou
183       To investigate the role of ACP4 during amelogenesis, we generated and characterized Acp4(R110C)
184 l-time PCR showed DPPI expression throughout amelogenesis, with highest expression at maturation, and

 
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