ライフサイエンスコーパス: amino acid metabolism

1 uxes (Krebs cycle, fatty acid, carbohydrate, amino acid metabolism).

2 iciency, is the most common defect of sulfur amino acid metabolism.

3 e mostly related to carbohydrate, lipid, and amino acid metabolism.

4 genes involved in bile acid, fat, sugar, and amino acid metabolism.

5 face among glycolysis, lipid metabolism, and amino acid metabolism.

6 inuria, the most frequent disorder of sulfur amino acid metabolism.

7 tional regulators for the control of sulphur amino acid metabolism.

8 laborate on the genomics and enzymology of d-amino acid metabolism.

9 s in fatty acid oxidation and branched-chain amino acid metabolism.

10 hat were heavily focused on carbohydrate and amino acid metabolism.

11 escine biosynthesis and pathways involved in amino acid metabolism.

12 ysiological significance of BHMT-2 in sulfur amino acid metabolism.

13 or cell wall function, hormone response, and amino acid metabolism.

14 are functionally linked to carbohydrate and amino acid metabolism.

15 on of sequences assigned to carbohydrate and amino acid metabolism.

16 fer to isoleucine and reduced branched-chain amino acid metabolism.

17 xpression of genes predominantly involved in amino acid metabolism.

18 cluding carbohydrate, nucleotide, lipid, and amino acid metabolism.

19 termediates in carbohydrate, fatty acid, and amino acid metabolism.

20 , suggesting a novel role for this operon in amino acid metabolism.

21 oader role for GcvB as a global regulator of amino acid metabolism.

22 ponse, whereas in CF nasal epithelium it was amino acid metabolism.

23 e in galN-treated rats, reflecting disturbed amino acid metabolism.

24 transcription, especially in the context of amino acid metabolism.

25 l-tRNA formation is intimately tied to amide amino acid metabolism.

26 athways, which can be explained by secondary amino acid metabolism.

27 to all life, such as purine, pyrimidine and amino acid metabolism.

28 s, cytosolic stress response, and purine and amino acid metabolism.

29 only recently shown to play a role in plant amino acid metabolism.

30 e with respect to glucose metabolism but not amino acid metabolism.

31 n-bound amino acids and a dominant locus for amino acid metabolism.

32 arboxylase, are involved in carbohydrate and amino acid metabolism.

33 iosynthetic pathways or in the regulation of amino acid metabolism.

34 uses resistance to the effect of glucagon on amino acid metabolism.

35 es and key enzyme activities associated with amino acid metabolism.

36 ivo role in the regulation of branched chain amino acid metabolism.

37 tosomal recessive disorder of branched-chain amino acid metabolism.

38 that were homologous to proteins involved in amino acid metabolism.

39 ts indicate a novel link between glucose and amino acid metabolism.

40 mechanism for a fungal enzyme essential for amino acid metabolism.

41 ed for studies of cell wall biosynthesis and amino acid metabolism.

42 acids have also been commonly used to trace amino acid metabolism.

43 many regulate nitrogen source utilization or amino acid metabolism.

44 caused the most significant modifications in amino acid metabolism.

45 nuria patients, a rare inherited disorder of amino acid metabolism.

46 supporting the substantial impact of HIIT on amino acid metabolism.

47 c reprogramming, including the disruption of amino acid metabolism.

48 purine metabolism and disrupted glucose and amino acid metabolism.

49 ate an expanded role for SIRT5 in regulating amino acid metabolism.

50 whole-body protein turnover, and splanchnic amino acid metabolism.

51 n autosomal-recessive Mendelian phenotype of amino acid metabolism.

52 ylketonuria, the most common inborn error of amino acid metabolism.

53 any metabolites associated with nitrogen and amino acid metabolism.

54 metabolism, the citric acid (TCA) cycle, and amino acid metabolism.

55 erences in KEGG pathways including lipid and amino acid metabolism.

56 degradation, mitochondrial trafficking, and amino acid metabolism.

57 ctions and investigate whether FXR regulates amino acid metabolism.

58 biosynthesis, fatty acids, nucleotides, and amino acid metabolism.

59 ceted intercompartmental effects on aromatic amino acid metabolism.

60 de variety of chemical reactions involved in amino acid metabolism.

61 y composition, including pathways related to amino acid metabolism.

62 es involving redox imbalance and deficits in amino acid metabolism.

63 al changes in peripheral glucose, lipid, and amino acid metabolism.

64 ylketonuria, the most common inborn error of amino acid metabolism.

65 L2, particularly those involved in lipid and amino-acid metabolism.

66 The largest group of such reactions involves amino-acid metabolism.

67 , a key metabolic compound closely linked to amino-acid metabolism.

68 blocks derived from lipid beta-oxidation and amino-acid metabolism.

69 in renal acidic amino-acid re-absorption and amino-acid metabolism.

70 ted with glycerophospholipid, fatty acid and amino acid metabolisms.

71 largely involved in nitrogen, nucleoside and amino acid metabolisms.

72 f the 6 named metabolites, 4 are involved in amino acid metabolism, 1 (prolylhydroxyproline) is a dip

73 oteins and related processes (18% of genes), amino acid metabolism (12% of genes), and cofactor and p

74 g pathway; 2) affect broiler liver lipid and amino acid metabolism; 3) induce liver cell immune respo

75 ls lacking sla1(+) have increased mRNAs from amino acid metabolism (AAM) genes and, furthermore, exhi

76 ncluded those associated with hyphal growth, amino acid metabolism, adherence, drug resistance, ergos

77 is, serine biosynthesis, and one-carbon- and amino acid metabolism, all of which have been identified

78 , Krebs cycle, fatty acid beta oxidation and amino acid metabolism, alluding to reduced energy biogen

79 of pathway involved in both EPS assembly and amino acid metabolism along with the availability of sug

80 y with infant age, such as valerobetaine and amino acid metabolism, among others.

81 lso expressed are genes encoding enzymes for amino acid metabolism, anaerobic aldehyde oxidation, hyd

82 Other clones contained genes required for amino acid metabolism, anaerobic respiration, DNA repair

83 Amino acid metabolism analysis revealed the inability to

84 toxic serum nephritis model was inhibited by amino acid metabolism and a protective autophagic respon

85 ic activities, including enzymes involved in amino acid metabolism and biosynthesis of neurotransmitt

86 ed with two distinct gene sets, representing amino acid metabolism and cell proliferation, respective

87 dation, reduced protein synthesis, decreased amino acid metabolism and depressed cell growth were rel

88 protein provides a useful method to examine amino acid metabolism and determine conditional amino ac

89 used to study how KG fuels bioenergetic and amino acid metabolism and DNA, RNA, and protein hydroxyl

90 r dysfunction characterized by downregulated amino acid metabolism and elevated Casp12.

91 showed that these plants had both perturbed amino acid metabolism and enhanced levels of GSLs.

92 Alterations in amino acid metabolism and epigenetic changes in the cere

93 olism in relapsed LSCs, which activates both amino acid metabolism and fatty acid oxidation to drive

94 riod as an animal nutritionist interested in amino acid metabolism and genetic variation in nutrient

95 also play an important role and that hepatic amino acid metabolism and glucagon are linked in a mutua

96 a role of IGF2BP2 and m(6)A modification in amino acid metabolism and highlight the potential of tar

97 nd may help to elucidate the roles of MPC in amino acid metabolism and hypoglycemia.

98 safely and positively influence protein and amino acid metabolism and improve overall daily activity

99 forming and breaking reactions in sugar and amino acid metabolism and in biosynthetic pathways via a

100 in skeletal muscle may contribute to reduced amino acid metabolism and insulin resistance in haemodia

101 in skeletal muscle may contribute to reduced amino acid metabolism and insulin resistance in MHD pati

102 the current understanding of branched-chain amino acid metabolism and its role in cancer.

103 metabolic perturbations, particularly at the amino acid metabolism and Krebs cycle level.

104 is might impair glucagon's action on hepatic amino acid metabolism and lead to hyperaminoacidemia and

105 athway and network analyses revealed altered amino acid metabolism and lipid metabolism, especially f

106 significant variations in energy production, amino acid metabolism and oxidative phosphorylation-rela

107 t) lies at an intersection of nucleotide and amino acid metabolism and performs a multitude of metabo

108 ulating biochemical pathways associated with amino acid metabolism and redox status.

109 By reducing BCAT1 levels, DDX6 KO reprograms amino acid metabolism and sensitizes AML cells to cytara

110 tudies have examined the interaction between amino acid metabolism and TAG biosynthesis.

111 that the PDV are key organs with respect to amino acid metabolism and that the intestines use a disp

112 ia (AML) patients are selectively reliant on amino acid metabolism and that treatment with the combin

113 pression of genes involved in branched-chain amino acid metabolism and the Ami permease; and upregula

114 Here we exploit amino acid metabolism and the promiscuity of SPT to modu

115 rial network composed of enzymes involved in amino acid metabolism and the TCA cycle affects the diet

116 nes to those coding for proteins involved in amino acid metabolism and transport suggest that FIST do

117 ates genes involved in redox homeostasis and amino acid metabolism and transport.

118 strating its broad role in the regulation of amino acid metabolism and transport.

119 as a second intracellular pathway for N-acyl amino acid metabolism and underscore enzymatic division

120 eceptor (GCGR), PEPCK, and genes involved in amino acid metabolism and urea production.

121 s identified; these belonged to pathways for amino acid metabolism and vitamin K-2 biosynthesis.

122 s direct evidence for a relationship between amino-acid metabolism and protein biosynthesis.

123 tem, fatty acid biosynthesis, branched chain amino acids metabolism), and molecular chaperones (GroEL

124 tabolic pathways including sugar metabolism, amino acid metabolism, and an increase in production of

125 esis, fructose and glucose metabolism, basic amino acid metabolism, and bile acid transport.

126 id and bile acid synthesis, gluconeogenesis, amino acid metabolism, and blood coagulation.

127 ress contributes to changes in TCA cycle and amino acid metabolism, and cell death, which is suppress

128 oid and phytol metabolism, a limited role in amino acid metabolism, and dual sets of the SUF pathway

129 subsets, the molecular intersection between amino acid metabolism, and effective T cell immunity and

130 ycolysis/gluconeogenesis, citric acid cycle, amino acid metabolism, and fatty acid metabolism were fo

131 epair, RNA metabolism, chromatin remodeling, amino acid metabolism, and heat shock response.

132 mitochondrial function, neurotransmitter and amino acid metabolism, and lipid biosynthesis.

133 The carbohydrate metabolism pathway, amino acid metabolism, and membrane transport were the f

134 egulation in translation initiation factors, amino acid metabolism, and methanogenesis from methanol,

135 f nitrogen metabolism via ureide catabolism, amino acid metabolism, and ornithine cycle intermediates

136 racellular degradative enzyme production and amino acid metabolism, and processes not previously know

137 ncluding metal homeostasis, nucleic acid and amino acid metabolism, and protein fate.

138 Changes in energy metabolism, amino acid metabolism, and RNase gene expression were co

139 cesses, including central carbon metabolism, amino acid metabolism, and secondary metabolism.

140 ain-length FAs originate from branched chain amino acid metabolism, and serve as primers for elongati

141 cell functions, including energy production, amino acid metabolism, and signal transduction, where th

142 t, the downregulation of gluconeogenesis and amino acid metabolism, and the upregulation of protein d

143 es in key pathways related to fatty acid and amino acid metabolism are associated with HF-related end

144 Glucose and amino acid metabolism are critical for glioblastoma (GBM

145 genes involved in nitrogen assimilation and amino acid metabolism are induced by iron deprivation, w

146 ribosome biogenesis, nuclear transport, and amino acid metabolism are more likely to be targeted for

147 ntral carbon pathways such as glycolysis and amino acid metabolism are shown to synchronize their res

148 pport Wolbachia genome-based predictions for amino acid metabolism as a primary energy source.

149 the liver is a major handler of protein and amino acid metabolism as it is responsible for the major

150 Larvae had disruptions in amino acid metabolism as well as GABA and catecholamine

151 crobiota being related to central carbon and amino acid metabolism as well as the biosynthesis of sec

152 cells are unable to engage in glycolysis or amino acid metabolism but have an increased rate of fatt

153 es not only cell-autonomous proteostasis and amino acid metabolism, but also affects non-cell-autonom

154 a and NH(4)Cl differs in terms of endogenous amino acid metabolism, but is similar in relation to mic

155 n, lyso-PC stimulated intracellular cationic amino acid metabolism by inducing ornithine decarboxylas

156 olic energy generation, DNA replication, and amino-acid metabolism by linking biochemical pathways in

157 al potential primarily focused on metabolism-amino acid metabolism, carbohydrate metabolism, and ener

158 e sucrose metabolism, fatty acid metabolism, amino acid metabolism, carbon fixation, and the biosynth

159 ystinuria (HCU) is an inborn error of sulfur amino acid metabolism caused by deficient activity of cy

160 onectin corrected the altered branched-chain amino acid metabolism caused by HFD and corrected increa

161 synthesis and partitioning, N-allocation and amino acid metabolism, cell wall biosynthesis, and hormo

162 Other processes that were up-regulated were amino acid metabolism, cell wall metabolism, and genetic

163 ates the expression of genes responsible for amino acid metabolism, cellular redox state, and anti-st

164 -limiting enzymes of choline, gloxylate, and amino acid metabolism coincided with the altered urinary

165 a decrease in levels of proteins regulating amino acid metabolism, cytoskeletal proteins, and cellul

166 Altered amino acid metabolism, decreased blood vessel density, a

167 t but is not involved in photorespiration or amino acid metabolism, demonstrated that the greater ABA

168 ty of m-Tyr may result from interfering with amino acid metabolism, destabalizing a large number of p

169 cative of a variety of pathologies including amino acid metabolism disorders and kidney malfunction.

170 ective analysis was to investigate the brain amino acid metabolism during epileptic seizures by (18)F

171 indings underscore the potential key role of amino acid metabolism early in the pathogenesis of diabe

172 metry flux, or metabolites of fatty acid and amino acid metabolism.Ex vivomuscle function in extensor

173 Nucleotide and amino acid metabolism exemplified the earliest responses

174 Glucose and amino acid metabolism generates hexosamine precursors th

175 NGR, while twenty two metabolites related to amino acid metabolism, glycerophospholipid metabolism an

176 the brain included those involved in acidic amino acid metabolism, Golgi apparatus, and ion and phos

177 issociation of insulin effect on glucose and amino acid metabolism has been reported in type 2 diabet

178 Enzymes involved in Staphylococcus aureus amino acid metabolism have recently gained traction as p

179 c energy mobilization, fatty acid oxidation, amino acid metabolism, heme biosynthesis and apoptosis.

180 ridoxal 5'-phosphate play essential roles in amino acid metabolism in all organisms.

181 identified mTORC2 as a critical regulator of amino acid metabolism in cancer via phosphorylation of t

182 entify an unanticipated mechanism regulating amino acid metabolism in cancer, enabling tumor cells to

183 Herein, we have investigated amino acid metabolism in cerebella of NPC1-deficient mic

184 omprehensive assessment of acylcarnitine and amino acid metabolism in clinical research studies using

185 The objective of this study was to assess amino acid metabolism in cob and spikelet tissues during

186 ided novel insights into the significance of amino acid metabolism in controlling immunity.

187 Amino acid metabolism in different cells and their cross

188 PAH deficiency is the most common defect of amino acid metabolism in humans, brain dysfunction in in

189 CRC cell lines also showed altered amino acid metabolism in hypoxia and hypoxia-responsive

190 ight the crucial role of enzymes involved in amino acid metabolism in immune cell biology.

191 the brain demonstrates the conservation of D-amino acid metabolism in mammals with implications for t

192 microbiota, enhancing bacterial motility and amino acid metabolism in mice with high parasite burden

193 Disruption of amino acid metabolism in Ni-D foliage resulted in accumu

194 The subcellular sites of branched-chain amino acid metabolism in plants have been controversial,

195 that GLNT1 is a general mechanism regulating amino acid metabolism in sap-feeding insects.

196 reases nutrient stress by altering lipid and amino acid metabolism in skeletal muscle.

197 cation and glucocorticoids on branched-chain amino acid metabolism in specific organs are unknown, th

198 many other metabolic processes, the role of amino acid metabolism in T cell activation has not been

199 was to construct a dynamic model of hepatic amino acid metabolism in the lactating dairy cow that co

200 d revealed unexpected concomitant changes in amino acid metabolism in the liver and at extrahepatic s

201 as part of a more complex system describing amino acid metabolism in the whole ruminant.

202 sulin is associated with profound changes in amino acid metabolism in type 1 diabetes.

203 sful forward engineering of complex aromatic amino acid metabolism in yeast, with the best machine le

204 ach by investigating the dynamic response of amino acids metabolism in mammalian cells upon activatio

205 igated the effect of dopamine depletion on D-amino acids metabolism in the brain of MPTP-lesioned Mac

206 amples of metabolomics evaluations of sulfur amino-acid metabolism in psychiatry, neurology, and neur

207 a role for various alcohols, by-products of amino acid metabolism, in altering cellular morphology.

208 e modulated, including purine metabolism and amino acid metabolism, in the group with KJT.

209 at multiple pathways are impacted, including amino acid metabolism, increasing the confidence in each

210 rform a wide variety of functions, including amino acid metabolism, ion transport, and gene regulatio

211 functional groups including carbohydrate and amino acid metabolism, ion transporters, stress and defe

212 the results of this investigation show that amino acid metabolism is a component of the acid-adaptiv

213 Consequently, d-amino acid metabolism is an attractive target for antibi

214 Abnormal amino acid metabolism is associated with vascular diseas

215 Amino acid metabolism is crucial for inflammatory proces

216 Amino acid metabolism is deregulated in many cancers, wi

217 1-1 toward amino acid feeding indicates that amino acid metabolism is generally deregulated in pig1-1

218 The phenotype suggested that amino acid metabolism is important for acid adaptation,

219 recognized, its role in systemic protein and amino acid metabolism is less well-appreciated.

220 ms or if they are part of overall changes in amino acid metabolism is unclear.

221 tanding and targeting such pathways, such as amino acid metabolism, is potentially useful for develop

222 ation of two intermediates in branched-chain amino acid metabolism, isovaleryl-Coenzyme A (CoA) and i

223 15) is an important regulator of glucose and amino acid metabolism, its endogenous role in lipid home

224 enetoclax and azacitidine (ven/aza) inhibits amino acid metabolism, leading to cell death.

225 Amino acid metabolism, lipid metabolism, alkaloid biosyn

226 s such as demethylation of DNA and proteins, amino acid metabolism, lipid peroxidation and one carbon

227 sponse identified oxidative phosphorylation, amino acid metabolism, matrisome (extracellular matrix [

228 Emerging insights about amino acid metabolism may lead to new strategies in clin

229 Abnormal amino acid metabolism may thus be an important factor in

230 ansferase system), EPS assembly (epsG1D) and amino acid metabolism (methionine, cysteine/arginine met

231 corresponding genes cause inborn diseases of amino acid metabolism, most of which are treated by diet

232 nstrated that it comprises genes involved in amino acid metabolism, nitrogen assimilation as well as

233 methods has allowed the study of protein and amino acid metabolism not only in the healthy newborn bu

234 nctional SNPs were found in pathways such as amino acid metabolism, nucleotide biosynthesis, and cell

235 ey source of nitrogen fueling the integrated amino acid metabolism of the aphid-Buchnera partnership.

236 The impact of altered amino acid metabolism on cancer progression is not fully

237 ion or result from abnormalities in branched amino acid metabolism (organic acidemias), have been sho

238 uding AOX, TCA cycle, fatty acid metabolism, amino acid metabolism, organic acid metabolism, and ethy

239 Major metabolic pathways were branched-chain amino acid metabolism (partially independent of insulin

240 The gut plays a major role in whole body amino acid metabolism, particularly arginine homeostasis

241 demonstrated distinct metabolic shifts, with amino acid metabolism pathways being consistently enrich

242 e targeting of miR-21 and miR-30d and/or the amino acid metabolism pathways may offer translational o

243 ch and sucrose, nitrogen, and branched-chain amino acids metabolism pathways.

244 cise mechanisms involved, it is evident that amino acid metabolism plays a role in ASCVD.

245 ) is an inherited disorder of branched-chain amino acid metabolism presenting with life-threatening c

246 ) is an inherited disorder of branched chain amino acid metabolism presenting with neonatal encephalo

247 d-associated CpGs were enriched in lipid and amino acid metabolism processes.

248 egulates the expression of genes involved in amino acid metabolism, redox homeostasis and ER stress r

249 34) defined a new disorder of branched-chain amino acid metabolism resembling human maple syrup urine

250 e (MtaR) that regulate methionine transport, amino acid metabolism, resistance to neutrophil-mediated

251 arm of the UPR, including genes involved in amino acid metabolism, resistance to oxidative stress, a

252 (GA-I) is an autosomal recessive disorder of amino acid metabolism resulting from a deficiency of glu

253 mTORC1/c-Myc pathway of stromal glucose and amino acid metabolism, resulting in increased stromal IL

254 These wholesale changes in amino acid metabolism segregate as a suite of traits res

255 re, plasma metabolites related to energy and amino acid metabolism significantly varied between CE an

256 many cancers, with changes in branched-chain amino acid metabolism specifically affecting cancer cell

257 ified genes are associated with respiration, amino acids metabolism, stress response and small RNAs,

258 These include defects in amino acid metabolism such as low alanine levels and ele

259 Our analysis revealed amino acid metabolism, such as arginine, proline, aspart

260 aminoadipic acid levels, indicating enhanced amino acid metabolism supporting nucleotide synthesis an

261 vacuoles contributed to the perturbation of amino acid metabolism, TCA cycle and oxidative stress.

262 ts at least 14 metabolic pathways, including amino acid metabolism, TCA cycle, gluconeogenesis, gluta

263 iched in enzymes for lipid, carbohydrate and amino acid metabolism, tetrapyrrole biosynthesis and pro

264 family and advances our knowledge of plant d-amino acid metabolism that is currently largely unexplor

265 nt for KLF15, a transcriptional regulator of amino acid metabolism that is induced by glucocorticoids

266 tides can therefore provide an assessment of amino acid metabolism that is specific to subcellular, c

267 e expression of genes involved in energy and amino acid metabolism, the data suggest that indole is a

268 ck loop that degrades glucagon and regulates amino acid metabolism to coordinately control glucose ho

269 at LSCs in patients with de novo AML rely on amino acid metabolism to drive oxidative phosphorylation

270 Mutations in cancer reprogram amino acid metabolism to drive tumor growth, but the mol

271 nt and mTOR-independent mechanisms that link amino acid metabolism to ferroptosis sensitivity.

272 c waste product but is recycled into central amino acid metabolism to maximize nitrogen utilization.

273 ons of major pathways for central carbon and amino acid metabolisms to biofilm pH homeostasis.

274 F4), the transcriptional master regulator of amino acid metabolism, to supply glucose-derived glycine

275 fatty acid oxidation and enhanced ketogenic amino acid metabolism, together with alterations in mito

276 l expression of genes functionally linked to amino acid metabolism, transcriptional regulation and si

277 These differences in amino acid metabolism translate into distinct pathway de

278 CD40 signaling mostly regulated amino acid metabolism, tricarboxylic acid cycle (TCA), a

279 ate uptake and metabolism, cation transport, amino acid metabolism, ubiquinone and menaquinone biosyn

280 XRalpha-mediated gene expression involved in amino acid metabolism was examined using the NIA Mouse 1

281 Amino acid metabolism was particularly evident among pat

282 synthesis, isoprenoid, sulfur, nitrogen, and amino acid metabolism, was obtained.

283 , which links cell proliferation with sulfur amino acid metabolism, was significantly affected by K11

284 etabolites broadly reflective of glucose and amino acid metabolism were associated with at least 1 me

285 Imbalances in amino acid metabolism were evident from increased branch

286 Protein rebalancing and amino acid metabolism were focal points of the metabolic

287 rnithine-glutamine shunt, and branched-chain amino acid metabolism were hypothesized as the primary i

288 2-D gel analysis, NlpD and genes involved in amino acid metabolism were identified by subtractive hyb

289 osolic and mitochondrial) involved in energy/amino acid metabolism were not significantly affected by

290 In addition, glycolysis and amino acid metabolism were significantly up-regulated by

291 that encode homologs of proteins involved in amino acid metabolism were similarly upregulated in S(pr

292 e transport, gluconeogenesis/glycolysis, and amino acid metabolism were up-regulated after wounding.

293 er published viromes, while carbohydrate and amino acids metabolisms were 3.7- and 4.2-fold less abun

294 etabolism, pigment biosynthesis and aromatic amino acid metabolism, were significantly modified by th

295 is limiting but N is abundant and upregulate amino acid metabolism when both light and N are limiting

296 matostatin and evaluated hepatic glucose and amino acid metabolism when glucagon was at basal levels

297 ome samples containing more genes related to amino acid metabolism, whereas N. aquilinus (a wood-feed

298 Disturbances in amino acid metabolism, which have been observed in Hunti

299 impacted the microbiome and host-associated amino acid metabolism, with a strong downregulation of m

300 B was a key regulator of glycogen, lipid and amino acid metabolism, with SEC22B-S137 phosphorylation