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1 capillary electrophoresis (MD-CE) assays for amino acid neurotransmitters.
2 ther calcium binding proteins and inhibitory amino acid neurotransmitters.
3 y of proteins that mediates the packaging of amino acid neurotransmitters.
5 rations, decreased the release of excitatory amino acid neurotransmitters, and decreased the total fr
6 from combinatorial assembly of moieties from amino acid, neurotransmitter, and lipid metabolism in th
7 ective assembly of moieties from nucleoside, amino acid, neurotransmitter, and lipid metabolism, sugg
8 affold to which diverse moieties from lipid, amino acid, neurotransmitter, and nucleoside metabolism
9 between the apoE4 allele and the recovery of amino acid neurotransmitters (aspartate, glutamate, and
10 dic chip that allows for in vivo sampling of amino acid neurotransmitters by low-flow push-pull perfu
11 the peptides into the brain while monitoring amino acid neurotransmitters by microdialysis sampling c
12 he balance between inhibitory and excitatory amino acid neurotransmitters contributes to the control
13 mals to determine the effects of ketamine on amino acid neurotransmitter cycling and glial metabolism
14 banesthetic doses of ketamine increased mPFC amino acid neurotransmitter cycling, as well as neuronal
16 n of the immunohistochemical localization of amino acid neurotransmitters demonstrated that the riMLF
17 ed to include increased de novo synthesis of amino acid neurotransmitters, especially glutamate and G
22 he lateral hypothalamus (LH), the inhibitory amino acid neurotransmitter, GABA, has had a long-standi
23 ons have suggested a role for the inhibitory amino acid neurotransmitter gamma-aminobutyric acid (GAB
26 ty of small organic molecules, including the amino acid neurotransmitters glutamate, aspartate and ta
27 ied by morphology and immunolabeling for the amino acid neurotransmitters glutamate, aspartate, gamma
28 trastructural localization of the excitatory amino acid neurotransmitters, glutamate and aspartate, u
29 ), which mimics the action of the excitatory amino acid neurotransmitter glutamic acid, releases LH-R
30 ontrast, the distributions of the inhibitory amino acid neurotransmitters glycine and GABA were not a
32 e) and inhibitory (gamma-amino butyric acid) amino acid neurotransmitters in brain, and is a source o
33 the growing weight of evidence for a role of amino acid neurotransmitters in pancreatic islets and in
34 ts of histaminergic agents on the release of amino acid neurotransmitters in slices of the medial ves
35 rs sensory processing, we measured levels of amino acid neurotransmitters in spinal dialysates from a
40 h-level exposure to glutamate, an excitatory amino acid neurotransmitter, leads to neuronal death.
41 ule cells normally contain two "fast-acting" amino acid neurotransmitters, one excitatory and one inh
42 C6 family of Na(+) -dependent symporters for amino acids, neurotransmitters, osmolytes, or creatine.
43 e addressed the topographic distribution and amino acid neurotransmitter phenotype of endocannabinoid
45 e, and allopregnanolone, modulate ionotropic amino acid neurotransmitter receptors, and may function
49 involve alterations in the major inhibitory amino acid neurotransmitter system of gamma-aminobutyric
50 lance of the major inhibitory and excitatory amino acid neurotransmitter systems of gamma-aminobutyri
51 ough the differential release of peptide and amino acid neurotransmitters to coordinate reproductive
52 Robust efflux of excitatory and inhibitory amino acid neurotransmitters was detected during ischemi
53 Both markers for inhibitory and excitatory amino acid neurotransmitters were found in varicose axon
55 mate and aspartate are endogenous excitatory amino acid neurotransmitters widely distributed in the m
56 for simultaneous monitoring of all the major amino acid neurotransmitters with 10-s temporal resoluti