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1 eus, and the magnocellular part of the basal amygdaloid.
2 opulus deltoides, and indiscernible in Salix amygdaloides.
3 by combining anterograde labeling of thalamo-amygdaloid afferents with postembedding immunoelectron m
4 usality methods to evaluate task-based intra-amygdaloid and extra-amygdaloid connectivity with the ne
5 mmunoreactive cells were detected in several amygdaloid and hypothalamic nuclei.
6 pothalamic VN target-sites and also in a few amygdaloid and hypothalamic sites outside the traditiona
7 d and sustained expression was noted in most amygdaloid and hypothalamic VN target-sites and also in
8 express neither CRF-R, including the central amygdaloid and paraventricular hypothalamic nuclei, and
9  To determine whether discrete components of amygdaloid and striatal memory systems could interact to
10 tical, olfactory, hippocampal, hypothalamic, amygdaloid and visual structures.
11 nuclei; and in a number of septal, thalamic, amygdaloid, and brainstem nuclei.
12 ntral medial prefrontal cortex and thalamic, amygdaloid, and hippocampal sources.
13 PA and NMDA receptor properties at cortical, amygdaloid, and hippocampal synapses.
14  affected twin had markedly smaller caudate, amygdaloid, and hippocampal volumes, and smaller cerebel
15 the caudal sublenticular region and anterior amygdaloid area (cSLR/AAA) differ from other basal foreb
16 by 70% in the PVN and by 50% in the anterior amygdaloid area, a control region of the brain.
17 ral cortex, hypothalamus, thalamus, cortical amygdaloid area, and white matter tracts did not show an
18 uclei (including anterior cortical, anterior amygdaloid area, periamygdaloid cortex, medial, posterio
19 of the lateral olfactory tract, the anterior amygdaloid area, the posterolateral cortical amygdaloid
20 apsule tracts carried GDNF into the anterior amygdaloid area.
21 ngulate cortex, project to medially situated amygdaloid areas such as the cortical and medial nuclei
22 lar cortex, orbital cortex, and all cortical amygdaloid areas.
23                                          The amygdaloid basal nuclei (BA) receive convergent input fr
24 is study investigated the involvement of the amygdaloid BDNF system in molecular mechanisms underlyin
25 loarchitecture, and chemoarchitecture of the amygdaloid body of the banded mongoose (Mungos mungo) an
26  overall lower myelin density throughout the amygdaloid body than the banded mongoose, a feature that
27 he organization and chemoarchitecture of the amygdaloid body.
28 , and neuropeptide Y (NPY) expression in the amygdaloid brain regions of rats.
29 e ELS demonstrated atypical intra- and extra-amygdaloid CeA-dominant paths with compensatory modulati
30      Furthermore, evidence suggests that the amygdaloid central nucleus (ACe) may influence cardiovas
31 rch sought to determine if activation of the amygdaloid central nucleus (ACe) modulates the activity
32 rostral ventromedial medulla) and forebrain (amygdaloid central nucleus and nucleus parafascicularis
33        These results reveal a novel role for amygdaloid chromatin remodeling in the process of alcoho
34 the amygdalo-hypothalamic projections, intra-amygdaloid circuitry and afferents from the lateral cort
35 essential for controlling the development of amygdaloid circuitry.
36 ey limbic-related cortical/thalamic/striatal/amygdaloid circuits, and the promise of subtle modulatio
37 ange rests on a rather small arcopallial and amygdaloid cluster of neurons.
38 amygdala complex as well as subsequent extra-amygdaloid communication.
39 adult human subjects was used to investigate amygdaloid complex (AC) activity associated with the sto
40                                          The amygdaloid complex (AC) is involved in very relevant cog
41                                          The amygdaloid complex (AC), a key component of the limbic s
42 hypothalamus, the lateral hypothalamus (LH), amygdaloid complex (AD) and thalamus (TH), and to a less
43 ily deactivated sites within the basolateral amygdaloid complex (BLA) or central amygdaloid region (C
44 esent study examined whether the basolateral amygdaloid complex (BLA) participates in the expression
45                       Within the basolateral amygdaloid complex (BLA), neuropeptide Y (NPY) buffers a
46 ateral lesions restricted to the basolateral amygdaloid complex (i.e., not including the Ce) did not
47 080411 genotype by sex was found in the left amygdaloid complex (male risk allele carriers showed les
48 e) plus adjacent portions of the basolateral amygdaloid complex (with either the excitotoxin NMDA or
49 cleus 0.36, remaining nuclei 3.33, and total amygdaloid complex 12.21.
50 f the anterior and posterior portions of the amygdaloid complex and its surrounding cortical regions
51 cingulate bundle connecting the centromedial amygdaloid complex and middle cingulate cortex to the do
52 ex, and the ventral pallium-derived extended amygdaloid complex and olfactory centers.
53  with bilateral ibotenic acid lesions of the amygdaloid complex and the hippocampus or were sham oper
54 s mediating anxiety responses, including the amygdaloid complex and the paraventricular hypothalamic
55 describe some quantitative parameters of the amygdaloid complex and their main nuclei that could help
56 ta-opioid receptors is also decreased in the amygdaloid complex and ventral putamen of Alzheimer's di
57   Projection neurons of the basolateral (BL) amygdaloid complex are regulated by an intrinsic inhibit
58 n animals implicates stress hormones and the amygdaloid complex as key, interacting modulators of mem
59 f neurons was estimated in the control human amygdaloid complex by using stereological sampling.
60                                   The intact amygdaloid complex from one hemisphere of 10 brains was
61 hological changes in neuronal density in the amygdaloid complex have been associated with various neu
62   Single-unit activity was recorded from the amygdaloid complex in freely moving rats during an infus
63  as well as the critical contribution of the amygdaloid complex in modulation of memory by emotional
64                                          The amygdaloid complex is a prominent temporal lobe region t
65       To investigate whether activity in the amygdaloid complex is altered with anxiolytic effects of
66                                   The rodent amygdaloid complex is composed of numerous subnuclei imp
67 evelopmental profile of ER expression in the amygdaloid complex is not available.
68                                          The amygdaloid complex is the primary neuronal site that ini
69                                          The amygdaloid complex is thought to be a major site of acti
70  of the nuclear and cortical portions of the amygdaloid complex is very similar in both species.
71                                          The amygdaloid complex mediates learning, memory, and emotio
72 azabemide to MAO-B was measured in the right amygdaloid complex of 15 major depressive subjects and 1
73 ns by injecting anterograde tracers into the amygdaloid complex of Macaca fascicularis monkeys and ex
74 V1 by injecting anterograde tracers into the amygdaloid complex of Macaca fascicularis monkeys.
75 ine (DA) transporter and D2 receptors in the amygdaloid complex of subjects with major depression ind
76 nd D2/D3 receptors have been observed in the amygdaloid complex of subjects with major depression.
77                                  The primate amygdaloid complex projects to a number of visual cortic
78 We conclude from these observations that the amygdaloid complex provides an excitatory input to areas
79                                          The amygdaloid complex represents a group of telencephalic n
80 ing magnetic resonance imaging guidance, the amygdaloid complex was lesioned bilaterally in six rhesu
81                                   The entire amygdaloid complex was outlined and then further partiti
82                                          The amygdaloid complex was outlined on coronal sections then
83 x (Brodmann area 11/32) bilaterally, and the amygdaloid complex were affected, but no significant atr
84 tion could be relayed from the cortex to the amygdaloid complex were investigated by using the antero
85  lesions of the hippocampal formation or the amygdaloid complex were tested on concurrent discriminat
86 l, basal, and accessory basal) nuclei of the amygdaloid complex were the source of most connections b
87  SREB2 expression (hippocampal formation and amygdaloid complex).
88 s found in limbic system areas: (1) extended amygdaloid complex, (2) lateral septum, and (3) infralim
89 PH cortices are also interconnected with the amygdaloid complex, although comparatively little is kno
90 dorsolateral prefrontal cortex, centromedial amygdaloid complex, and middle cingulate cortex volumes
91                             The neostriatum, amygdaloid complex, and most hypothalamic areas express
92  Brain areas analyzed were caudate, putamen, amygdaloid complex, hippocampal formation and various ce
93 on of NK(1) receptor immunoreactivity in the amygdaloid complex, induction of NK(1) receptor endocyto
94 he medial temporal polar cortex, most of the amygdaloid complex, most or all of the entorhinal cortex
95 euniens and anterior medial thalamic nuclei, amygdaloid complex, piriform cortex and subfornical orga
96 undant nitrergic cells in the basal ganglia, amygdaloid complex, preoptic area, basal hypothalamus, m
97 which include the lateral hypothalamic area, amygdaloid complex, septal-ventral striatal areas, and i
98  the components of the basal ganglia and the amygdaloid complex, the alar and basal hypothalamic regi
99                      In the basolateral (BL) amygdaloid complex, the excitability of projection cells
100 shed afferent to the ventral striatum is the amygdaloid complex, which projects throughout the shell
101 ods used to define subdivisions of the human amygdaloid complex.
102 f sensory information that occurs within the amygdaloid complex.
103 to the lateral nucleus of the macaque monkey amygdaloid complex.
104 ly involved in feedback inhibition in the BL amygdaloid complex.
105 incides with altered volume and shape of the amygdaloid complex.
106 nerable brain regions of the hippocampus and amygdaloid complex.
107  the bed nucleus of the stria terminalis and amygdaloid complex.
108 nd innervate interneurons in the basolateral amygdaloid complex.
109 gdala (CEm), the major output nucleus of the amygdaloid complex.
110 cleus (Me) is a superficial component of the amygdaloid complex.
111 btained for the basal ganglia and septal and amygdaloid complexes in amphibians (anamniotic tetrapods
112 een suggested to play a role in cochlear and amygdaloid complexes.
113                  The cortical, thalamic, and amygdaloid connections of the rodent temporal cortices w
114 aluate task-based intra-amygdaloid and extra-amygdaloid connectivity with the network underlying impl
115 onal relationships for both intra- and extra-amygdaloid connectivity, while ELS demonstrated atypical
116  orbitofrontal cortex (OFC)-CeA-driven extra-amygdaloid connectivity.
117                                  The role of amygdaloid corticotropin-releasing factor (CRF) in alcoh
118                                     Neonatal amygdaloid damage mildly impaired acquisition at the 24-
119 ults provide evidence for the involvement of amygdaloid dopamine in regulating surprise in fear and t
120    In conclusion, these results suggest that amygdaloid dopamine transmission at D2 receptors contrib
121               Modularity analysis identified amygdaloid elements consistent with the main anatomical
122 olved in high-order functions in prefrontal, amygdaloid, entorhinal, and perirhinal cortex, to which
123 he surface of the telencephalic vesicles, an amygdaloid group of cells originated in the caudal pole
124             Acute ethanol exposure decreased amygdaloid HDAC activity and HDAC2 protein levels, incre
125                                  We measured amygdaloid HDAC activity, levels of HDAC isoforms, and h
126 tudy examined the effects of bilateral intra-amygdaloid infusions of the D2 receptor antagonist, etic
127  investigated the effects of bilateral intra-amygdaloid infusions of the selective D1 receptor antago
128                       Animals received intra-amygdaloid injection of kainic acid (KA) to induce seizu
129 , caudal ventral striatal areas that receive amygdaloid inputs also receive significant innervation b
130 he AMPA/NMDA ratio increased at cortical and amygdaloid inputs but not at hippocampal inputs.
131                               There are also amygdaloid inputs to CaBP-positive areas outside the she
132                                              Amygdaloid inputs to the CaBP-poor zone in the lateral a
133    Evidence based on cortical, thalamic, and amygdaloid inputs to the striatum suggests a medio-later
134 nterior insula, which integrates sensory and amygdaloid inputs, projects to the classic ventral stria
135 rea and charted the distribution of specific amygdaloid inputs.
136 ons (< 1%), we focused on cortical and intra-amygdaloid inputs.
137 APDC, were tested on both the development of amygdaloid kindling and on fully developed stage 5 amygd
138                           The development of amygdaloid kindling was significantly retarded in 2R,4R-
139                                  The lateral amygdaloid (LA) nucleus is the main input station of the
140                                           No amygdaloid labeling was observed.
141  LTP, and genetic modifications that disrupt amygdaloid LTP eliminate fear conditioning.
142  antagonists of the NMDA receptor block both amygdaloid LTP induction and fear conditioning, fear con
143 hypothalamic arcuate and dorsomedial nuclei, amygdaloid medial and centromedial nuclei, thalamic cent
144 uated for 0-90 min in rats receiving central amygdaloid microinjection of RX 77368 (0.1, 0.5 or 1.0 m
145 d the bed n of the stria terminalis), medial amygdaloid n; the thalamus (anteromedial thal. n; reunie
146  reverse the amphetamine-induced increase in amygdaloid neuronal activity and required more than 20 m
147 trates that a small group of arcopallial and amygdaloid neurons constitute a wide range of contralate
148 reduce anxiety and dendritic arborization of amygdaloid neurons of adult male Wistar rats.
149  for glutamate and aspartate to determine if amygdaloid neurons projecting to different targets in th
150 ls of glutamate and aspartate in basolateral amygdaloid neurons projecting to the prefrontal cortex a
151               However, the great majority of amygdaloid neurons with projections to the mediodorsal n
152 ted calcium channels in acutely isolated rat amygdaloid neurons.
153 vation in GC secretion might directly affect amygdaloid neurons.
154  for fear conditioning in rats trained under amygdaloid NMDA receptor blockade.
155  animals suggests that structures within the amygdaloid nuclear complex (ANC) are critical for acquir
156 nal cortex of the inferior colliculis (2.1), amygdaloid nuclei (1.5-2.1), cingulate cortex (1.8), CA1
157 sory olfactory tract, and innervates several amygdaloid nuclei (nucleus of the accessory olfactory tr
158 unts for the amygdala and each of four major amygdaloid nuclei (the lateral, basal, accessory basal,
159 rolateral cortical, and posterior basomedial amygdaloid nuclei and amygdalostriatal transition zone.
160 estigated, notably in the striatum/pallidum, amygdaloid nuclei and in the hippocampus it was more div
161                     Anatomically, individual amygdaloid nuclei are connected with many neural systems
162                                   Individual amygdaloid nuclei have been shown to project to various
163 levation in binding to MAO-B observed in all amygdaloid nuclei in major depressive subjects as compar
164 dala and to assess the separate roles of the amygdaloid nuclei in social behavior in primates.
165 t distinct neural systems involving separate amygdaloid nuclei mediate different types of conditioned
166 but no double labeled cells were seen in the amygdaloid nuclei or the hypothalamic region.
167 f the AC; also, volume of the neurons in the amygdaloid nuclei was estimated using the point-sampled
168 sed in the amygdala (lateral and basolateral amygdaloid nuclei) and hippocampal formation (pyramidal
169 reflect the known neurobiology of individual amygdaloid nuclei, allowing for human imaging studies to
170 nterior hypothalamus, the medial and central amygdaloid nuclei, and the lateral septum.
171 nd in cortical regions, central and anterior amygdaloid nuclei, and the olfactory bulb.
172 lexus of Meynert, basolateral and basomedial amygdaloid nuclei, anterior pretectal and interpeduncula
173 presubiculum, parasubiculum, CA1-CA4 fields, amygdaloid nuclei, basal forebrain, thalamus, hypothalam
174 r nucleus, zona incerta, medial and cortical amygdaloid nuclei, cerebellum, nucleus of the solitary t
175 omotor cortices, the limbic system including amygdaloid nuclei, hippocampus and septum, the basal gan
176 of the stria terminalis, medial and cortical amygdaloid nuclei, preoptic area, lateral habenula, peri
177 l formation, presubiculum and parasubiculum, amygdaloid nuclei, thalamic nuclei, locus coeruleus, and
178 agonal band of Broca, the basal and cortical amygdaloid nuclei, the entopeduncular nucleus, the subth
179                            These include two amygdaloid nuclei, the horizontal limb of the diagonal b
180  brain regions connected to the BSTL are the amygdaloid nuclei, the paraventricular and ventromedial
181                            The activation of amygdaloid nuclei, the ventral perirhinal cortex (vPRh),
182 ells were present in the cortical and medial amygdaloid nuclei, which are known to have strong recipr
183 nces were found in the medial or basolateral amygdaloid nuclei.
184  refinement of intrinsic connections between amygdaloid nuclei.
185 leus, with little or no innervation of other amygdaloid nuclei.
186 basal forebrain regions, thalamic nuclei and amygdaloid nuclei.
187 serotonergic input, such as hypothalamic and amygdaloid nuclei.
188 e seen in the parabrachial nucleus or in the amygdaloid nuclei.
189 arse labeling in the lateral and basolateral amygdaloid nuclei.
190                        The anterior cortical amygdaloid nucleus (ACo) is a chemosensory area of the c
191 tly higher in female rats, and in the medial amygdaloid nucleus (Am), GAD(67) mRNA was higher in male
192 of patients with anxiety and the basolateral amygdaloid nucleus (BLA) in chronic stress mice.
193                     Induction in the central amygdaloid nucleus (CeA) and in the bed nucleus of the s
194                                  The central amygdaloid nucleus (CeA) is a key limbic structure invol
195 x (PR), and moderate labeling in the lateral amygdaloid nucleus (L).
196                                   The medial amygdaloid nucleus (Me) is a key nodal point in the neur
197 iform cortex (Pir), posteriolateral cortical amygdaloid nucleus (PLCo), and the amygdalopiriform tran
198  piriform cortex and posterolateral cortical amygdaloid nucleus (PLCo).
199 area of the hypothalamus and also the medial amygdaloid nucleus and CA1 subfield of the hippocampus.
200 l in naked mole-rats include the basolateral amygdaloid nucleus and dentate gyrus, but the septohippo
201 nuclei, hippocampal CA3 region, centromedial amygdaloid nucleus and thalamic paraventricular and reun
202 mporal lobe was associated with the cortical amygdaloid nucleus and the pyramidal cells of the hippoc
203 n neocortex and magnocellular cells in basal amygdaloid nucleus are also intensely CRF(1)-ir.
204  monkeys (orbitofrontal region) or in the BL amygdaloid nucleus in cats.
205 intaining a central role for the basolateral amygdaloid nucleus in the acquisition and storage of thi
206 ing increased c-fos expression in the medial amygdaloid nucleus of both DOM and SUB males as well as
207 striatal area is more related to the central amygdaloid nucleus than to the striatum.
208 ampus and magnocellular cells in basolateral amygdaloid nucleus were also intensely FLNa immunoreacti
209  levels of mRNA of DA receptors in the basal amygdaloid nucleus were measured postmortem in subjects
210 olus vulgaris-leucoagglutinin in the central amygdaloid nucleus were shown to contact cSLR/AAA cholin
211 s-expression by 3.7-times in the basolateral amygdaloid nucleus, 1.2-times in the supraoptic nucleus
212 c area, several hypothalamic nuclei, central amygdaloid nucleus, amygdalohippocampal area, dorsal per
213  followed by moderate labeling of the medial amygdaloid nucleus, amygdalostriatal zone and caudoputam
214 amygdaloid area, the posterolateral cortical amygdaloid nucleus, and the dorsolateral part of the lat
215 al parvocellular part of the basal accessory amygdaloid nucleus, and the magnocellular part of the ba
216 alon: IRP-LI was concentrated in the central amygdaloid nucleus, bed nucleus of stria terminalis and
217 al ventricular ridge, PDVR, and dorsolateral amygdaloid nucleus, DLA) subdivisions.
218 at in Cape mole-rats include the basolateral amygdaloid nucleus, hippocampal CA3 subfield, and dentat
219  specifically, the lateral septum and medial amygdaloid nucleus, indicate greater c-fos mRNA inductio
220 ory tubercle, nucleus accumbens, basolateral amygdaloid nucleus, rostroventrolateral medulla and nucl
221 ustrum, bed nucleus of the stria terminalis, amygdaloid nucleus, ventral posterior division of the th
222 in the lateral septal nucleus and the medial amygdaloid nucleus, which have numerous Fos-stained nucl
223  is remarkably similar to that of the medial amygdaloid nucleus.
224 ed to be a possible homologue of the central amygdaloid nucleus.
225  is theorized to be homologous to the medial amygdaloid nucleus.
226 ield CA1 and mainly targeted the basolateral amygdaloid nucleus.
227 us, and the dorsolateral part of the lateral amygdaloid nucleus.
228 GABAergic, and project to the medial CEA (an amygdaloid output region), this may be a novel mechanism
229 chanism whereby conditioned fear potentiates amygdaloid output.
230 brachial area (part of the spino-parabrachio-amygdaloid pain pathway) in brain slices from control ra
231  could be useful to understand the impact of amygdaloid pathology in these processes, both globally a
232 monstrated narrowly defined BLA-driven intra-amygdaloid paths and concise orbitofrontal cortex (OFC)-
233  and robust CeA-facilitated intra- and extra-amygdaloid paths.
234                              The spino-ponto-amygdaloid pathway is a major ascending circuit relaying
235 e pathway, the spino-(trigemino)-parabrachio-amygdaloid pathway, was performed using an alpha-herpes
236           The fear component is regulated by amygdaloid pathways, while the contextual component is,
237                                              Amygdaloid projections from the posterior insular cortex
238                      Cortical, thalamic, and amygdaloid projections of the rat anterior and posterior
239                                              Amygdaloid projections targeted the ventromedial subdivi
240              We examined the organization of amygdaloid projections to visual cortical areas TE and V
241          Interestingly, only arcopallial and amygdaloid projections were reciprocally organized, and
242 olateral amygdaloid complex (BLA) or central amygdaloid region (CeA) via GABA(A) agonist (muscimol) i
243 higher FLI than the tested groups in all the amygdaloid regions and in the hypothalamic paraventricul
244 ntal motor cortex, hippocampus, thalamus and amygdaloid) regions based on their relevance to ASD.
245 artially overlapping brain mechanisms (i.e., amygdaloid-reticular projections) mediate these effects.
246 as higher in 6ByJ relative to 6J mice in the amygdaloid ridge, hypothalamus, and midbrain.
247  nigra and three closely related species: S. amygdaloides, S. gooddingii, and S. caroliniana.
248 , warming shifted both snowmelt peaks and S. amygdaloides seed release 21 d earlier, maintaining thei
249 CI, Pandey and colleagues identify a central amygdaloid signaling pathway involved in anxiety-like an
250 t is expressed in the majority of in thalamo-amygdaloid spines and that within these spines, the vari
251 he prevalence of the NR2B subunit in thalamo-amygdaloid spines provides morphological evidence suppor
252                 A high percentage of thalamo-amygdaloid spines was immunoreactive for GluR2 (80%), Gl
253 ratios of GluRs were measured within thalamo-amygdaloid spines.
254 lamic 'attack' site in the absence of medial amygdaloid stimulation suppressed predatory attack, thus
255 ttack, thus simulating the effects of medial amygdaloid stimulation.
256 in anxiety levels or on CREB function in the amygdaloid structures of NP rats.
257                    Among the nonchemosensory amygdaloid structures, the PDVR receives afferents from
258 define a unique set of molecular markers for amygdaloid subnuclei and provide tools to genetically di
259 ields, dysfunctional pulvinar, claustrum and amygdaloid subnuclei of the amygdala, the latter progres
260 ellular and neurochemical composition of the amygdaloid subnuclei suggests their clustering into subu
261 pes of DA receptor mRNA were detected in all amygdaloid subnuclei, although D1, D2, and D4 receptor m
262 ivate) or bicuculline (to activate) to these amygdaloid subregions.
263 ovide support for the hypothesis that medial amygdaloid suppression of lateral hypothalamically elici
264 second limb of the pathway subserving medial amygdaloid suppression of predatory attack behavior proj
265  Drug infusion produced a blockade of medial amygdaloid suppression of predatory attack in a time- an
266 sent study tested the hypothesis that medial amygdaloid suppression of predatory attack is mediated,
267 ning, fear conditioning induces increases in amygdaloid synaptic transmission that resemble LTP, and
268 d anxiolysis was measured in adult rats, and amygdaloid tissues were used for miRNA profiling by micr

 
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