ライフサイエンスコーパス: amylase

1 concentration of uric acid, cholesterol, and amylase.

2 CSP6) showed high affinity in binding alpha-amylase.

3 nto a point-of-care detection tool for alpha-amylase.

4 or because of starch-hydrolysis catalyzed by amylase.

5 ted to the oral environment rich in salivary amylase.

6 digestive enzymes, namely trypsin and alpha-amylase.

7 baked with and without the addition of alpha-amylase.

8 was sensitized also to either flour or alpha-amylase.

9 fic inhibition of the human pancreatic alpha-amylase.

10 of lipase, but slightly catalyzed the alpha-amylase.

11 The other 56% were generated by amylases.

12 radicting results regarding the link between amylase 1 (AMY1) copy numbers (CNs), obesity, and type 2

13 This involves the synergistic action of beta-amylase 1 (BAM1) and alpha-amylase 3 (AMY3)-enzymes that

14 ic action of beta-amylase 1 (BAM1) and alpha-amylase 3 (AMY3)-enzymes that are normally not required

15 ncreased lipase (57 [10%] of 547), increased amylase (31 [6%]), and increased alanine aminotransferas

16 s inhibited alpha-glucosidase (37.8%), alpha-amylase (35.6%), dipeptidyl peptidase-IV (34.4%), reacti

17 sent study investigates the effects of alpha-amylase (6 and 10ppm), xylanase (70 and 120ppm) and cell

18 s) on four enzymes: alpha-glucosidase, alpha-amylase, acetylcholinesterase, and butyrylcholinesterase

19 These results showed that colistin and amylase-activated dextrin-colistin conjugate to a lesser

20 hich possible binding modes within the alpha-amylase active site could be investigated in silico usin

21 uercetagetin additionally binds to the alpha-amylase active site.

22 able to inhibit alpha-glucosidase and alpha-amylase activities.

23 suited method for the determination of alpha-amylase activity and as an easy method to do kinetic stu

24 and negatively with monomeric protein, alpha-amylase activity and sodium carbonate solvent retention

25 ng number (FN) in wheat indicates high alpha-amylase activity associated with poor end-use quality.

26 m was to determine inhibition of human alpha-amylase activity by (poly)phenols using maltoheptaoside

27 Significant inhibition of alpha-amylase activity by plant extracts was also observed.

28 d pancreas by inflammatory cells, and plasma amylase activity compared with control mice given cerule

29 r-sensor for quantitative detection of alpha-amylase activity in human blood serum.

30 , which helps the detection of unknown alpha-amylase activity in the blood serum.

31 The assessment of alpha amylase activity is carried out by the quenching of the

32 2 rs11185098 genotype associated with higher amylase activity may have greater loss of adiposity duri

33 tisfactorily for the assessment of the alpha-amylase activity over activity range (3-321U/L) in diffe

34 In germinating brown rice, alpha-amylase activity was significantly higher in treated gro

35 ds appeared to be potent inhibitors of alpha-amylase activity with an IC50 of (0.075+/-0.010-0.103+/-

36 Less lipid peroxidation and higher alpha-amylase activity, higher ascorbate (RAsA) and TPC were o

37 carrying AG1 + AG2 QTLs showed higher alpha-amylase activity, leading to rapid starch degradation an

38 Plasma amylase activity, pancreatic edema, and myeloperoxidase

39 erity of pancreatitis was evaluated by serum amylase activity, pathological scores, myeloperoxidase a

40 ication of the GOD method in assessing alpha-amylase activity.

41 ead use of the GOD method in assessing alpha-amylase activity.

42 lic acid (DNSA) method for determining alpha-amylase activity.

43 n optical sensor for the assessment of alpha-amylase activity.

44 vity, while C. noveboracense had the highest amylase activity.

45 Enzymes (amylolytic and alpha-amylase) activity increased at both temperatures for LR

46 tracts radical scavenging activity and alpha-amylase, alpha-glucosidase and aldose reductase inhibito

47 owed the higher capacity in inhibiting alpha-amylase, alpha-glucosidase and HT29 cell growth.

48 cetin showed the highest inhibition of alpha-amylase, alpha-glucosidase and lipase (IC50: 0.38mg/mL,

49 metabolic syndrome-associated enzymes (alpha-amylase, alpha-glucosidase and lipase) was evaluated.

50 otential inhibitory activities towards alpha-amylase, alpha-glucosidase and tyrosinase.

51 all PSP samples inhibited the enzymes alpha-amylase, alpha-glucosidase and xanthine oxidase.

52 gher antioxidant activity, and greater alpha-amylase, alpha-glucosidase, and ACE inhibitory activity

53 ved the strong inhibitory potential of alpha-amylase, alpha-glucosidase, and dipeptidyl peptidase III

54 ted with metabolic syndrome, including alpha-amylase, alpha-glucosidase, lipase and hydroxyl methyl g

55 ylcholinesterase), anti-diabetic (anti-alpha-amylase, -alpha-glucosidase, -pancreatic lipase) and ant

56 als carrying the A allele (indicating higher amylase amount and activity) showed a greater reduction

57 number variations that affect differences in amylase amount and activity, and AMY1 copies have been a

58 contained clusters of acinar cells devoid of amylase (AMY(-)).

59 he cell wall anchored starch-degrading alpha-amylase, Amy13K of E. rectale harbors five CBMs that all

60 Here we report that the chloroplastic alpha-amylase AMY3, a starch-degrading enzyme, interferes with

61 ulation in their native form or treated with amylases/amyloglucosidase to facilitate the entry of act

62 Drain amylase analysis identifies which moderate/high risk pat

63 Their effect in inhibiting i) alpha-amylase and alpha-glucosidase activities and ii) colorec

64 t properties and inhibitory effects on alpha-amylase and alpha-glucosidase activities.

65 e determined by evaluating the lipase, alpha-amylase and alpha-glucosidase activities.

66 Some monoterpenes inhibited alpha-amylase and alpha-glucosidase activity and stimulated gl

67 The inhibition of the alpha-amylase and alpha-glucosidase activity facilitates the m

68 Differential inhibition of alpha-amylase and alpha-glucosidase activity was observed in r

69 recent evidence regarding the in vitro alpha-amylase and alpha-glucosidase inhibition activities of e

70 nt type are parameters that affect the alpha-amylase and alpha-glucosidase inhibition activities of t

71 ids, alkaloids have been shown to have alpha-amylase and alpha-glucosidase inhibition activities.

72 rization was observed on antidiabetic (alpha-amylase and alpha-glucosidase inhibition) activity, howe

73 omoting benefits (anticancer activity, alpha-amylase and alpha-glucosidase inhibition, angiotensin-co

74 ectively) was investigated, as well as alpha-amylase and alpha-glucosidase inhibition, antihypertensi

75 ultivars showed superior inhibition of alpha-amylase and alpha-glucosidase than foxtail millet cultiv

76 Inhibition of alpha-amylase and alpha-glucosidase was the highest at 65 and

77 ifferent inhibition properties against alpha-amylase and alpha-glucosidases, showing different inhibi

78 Five Pinto bean peptides with alpha-amylase and angiotensin converting enzyme (ACE) inhibito

79 However, alpha-amylase and cellulase incubation caused significant incr

80 esterase, butyrylcholinesterase, tyrosinase, amylase and glucosidase inhibition) were used for screen

81 ities of cyclooxygenase 1 and -2, as well as amylase and glucosidase was recorded for the breads enri

82 his study, kinetics of binding between alpha-amylase and green tea flavonoids were investigated by fl

83 ain starch-degrading enzymes alpha- and beta-amylase and limit dextrinase.

84 Activity of the hydrolytic enzymes alpha-amylase and lipase along with stored food reserves (lipi

85 bolic syndrome, including alpha-glucosidase, amylase and lipase and exhibited antioxidant activity by

86 reover, cerulein- and arginine-induced serum amylase and lipase were significantly higher in panc-PTP

87 suggestive of pancreatic disease and normal amylase and lipase who underwent the examination with th

88 ration, hemorrhage, necrosis, the release of amylase and lipase.

89 roteins, globulin 3A and 3C, chitinase, beta-amylase and LMW glutenins, were identified from the elec

90 larity indexes (SSIM) of 0.925 and 0.920 for amylase and pan-keratin respectively.

91 C (applying a dienzyme treatment with alpha-amylase and protease).

92 Enhanced protease and alpha-amylase and reduced lipase activities were observed in s

93 ve enzymatic chemical reaction between alpha-amylase and starch.

94 ds, their inhibitory activity against fungal amylase and the occurrence of aflatoxins were determined

95 e enzymatic activity was determined by alpha-amylase and tyrosinase enzyme inhibition.

96 er inhibitory activity with respect to alpha-amylase and tyrosinase.

97 However, expression differences of beta-amylases and GLUCAN-WATER DIKINASE1 were not statistical

98 n of storage time, usage of maltogenic alpha-amylases and spatial position in the loaf by texture mea

99 lyphenolic composition) can inhibitdigestive amylases and thereby slow down starch digestion.

100 ation product, probably due to inhibition of amylases and yeast activity.

101 The inhibition of alpha-glucosidase, alpha-amylase, and angiotensin-converting I enzymes, antioxida

102 tifications of immunostaining markers (DAPI, amylase, and cytokeratins; Spearman correlation score =

103 hancement of Ca(2+) oscillations, pancreatic amylase, and pulmonary myeloperoxidase.

104 henolic extracts were able to inhibit fungal amylase, and the PCA analysis confirmed that the relatio

105 , time-dependent starch digestion with alpha-amylase, and the subsequent variation in electrical resp

106 hibition of hydroxyl, nitric oxide and alpha-amylase, as well as a decrease in the inhibition of alph

107 ification (0.025-1000IU/L) compared to alpha-amylase assays in current clinical use.

108 or this purpose, alpha-glucosidase and alpha-amylase assays were assessed; among all bean ecotypes, t

109 alpha-Amylase at 37 degrees C during 15min followed by an alka

110 am1 and bam3 We propose that LSF1 binds beta-amylases at the starch granule surface, thereby promotin

111 Glu233, His299, Asp300 and His305 for alpha-amylase; (b) His353, Ala354, His383, Glu384, His387, Glu

112 bidopsis thaliana) genome contains nine beta-amylase (BAM) genes, some of which play important roles

113 LSF1 interacts with the beta-amylases BAM1 and BAM3, and the BAM1-LSF1 complex shows

114 we report successful immobilization of beta-amylase (bamyl) from peanut (Arachis hypogaea) onto Grap

115 identified key residues that are crucial for amylase binding.

116 We found a unique gene locus encoding an amylase-binding adhesin AbpA and a sortase B in oral str

117 The assessment of the alpha amylase biomarker by the proposed method increases its s

118 by inhibition of alpha-glucosidase and alpha-amylase, both involved in the carbohydrate metabolism.

119 kers are exposed not only to flour and alpha-amylase but also to other 'improver' enzymes, the nature

120 heat was not significantly degraded by alpha-amylase but had developmental changes with an increased

121 ere found to be moderate inhibitors of alpha-amylase, but potent inhibitors of alpha-glucosidase, sho

122 hypothesize starch - the substrate of alpha-amylase, can directly influence hot flour pasting proper

123 hocyanins inhibited the human salivary alpha-amylase catalyzed hydrolysis competitively.

124 However, the presence of xylanase, alpha-amylase, cellulase and lipase resulted in bread with gre

125 he percentage of patients with a drain fluid amylase checked on postoperative day 1 increased (P < 0.

126 (beta-galactosidase, beta-glucosidase, beta-amylase, chitinase, pectate lyase (PL), pectinesterase (

127 h active site residues of T. castaneum alpha-amylase compared to C. chinensis alpha-amylase, which co

128 decreased monotonically with an increase in amylase concentration because the intensity of the refle

129 f the photoresistor were correlated with the amylase concentration in analyte.

130 or the point-of-care-testing (POCT) of alpha-amylase concentration in serum.

131 An increase of systemic amylase concentration was associated with POPFs.

132 d ischemia time, intensive care unit length, amylase concentration), pancreas procurement, isolation

133 ieved a highly linear response against alpha-amylase concentration.

134 Here we assess amylase copy numbers in a global sample of 480 high cove

135 lack of bedside monitoring devices for alpha-amylase detection has hitherto restricted the clinical p

136 negligible/low risk patients and drain fluid amylase (DFA) was measured on postoperative day 1 (POD 1

137 ded by postoperative day (POD) 1 drain fluid amylase (DFA-1), is associated with reduced rates of cli

138 to the changes in the conformation of alpha-amylase due to ethanol-induced denaturation.

139 demonstrate the importance of salivary alpha-amylase during oro-gastric processing of starchy foods.

140 dingly, the contribution of pancreatic alpha-amylase during the intestinal phase was lower for bread

141 Salivary and pancreatic amylases (encoded by AMY1 and AMY2 genes, respectively)

142 id metabolism as well as AMY1B, the salivary amylase-encoding gene in the polar bear.

143 lower density kernels displayed higher alpha-amylase, endoxylanase, and peptidase activities as well

144 itate mixing and promote the specific starch-amylase enzymatic reaction.

145 Activity of alpha-amylase enzyme in human serum indicates the onset of pan

146 for the assessment of the activity of alpha amylase enzyme in urine and serum samples for early diag

147 Diastase alpha-amylase extracted from malt, catalyses break down of sta

148 on of the AMY1 locus, which encodes salivary amylase, facilitating starch digestion.

149 rves obtained using porcine pancreatic alpha-amylase for a range of particle size fractions.

150 st effective (IC50: 0.25mg/mL) against alpha-amylase; Fraction V from black turtle bean was the most

151 The immobilization of maltogenic alpha-amylase from Bacillus stearothermophilus (BsMa) onto nov

152 n with observations regarding salivary alpha-amylase from other fields of knowledge.

153 hydrolysis of native starch by a novel beta-amylase from peanut (Arachis hypogaea).

154 gelatinization for maltose production, beta-amylase from peanut could be a useful alternative in the

155 Particularly, alpha-amylases from Helicoverpa armigera (Lepidoptera) were no

156 showed differential inhibition against alpha-amylases from human, insects, fungi and bacteria.

157 The amylase gene (AMY), which codes for a starch-digesting e

158 onflicting associations between the salivary amylase gene (AMY1) copy number and obesity.

159 tigated whether genetic variants determining amylase gene copies are associated with 2-year changes i

160 inconsistent link observed between salivary amylase gene copy number (AMY1 CN) and weight management

161 volved in sugar- and hormone-regulated alpha-amylase gene expression in rice.

162 urthermore, increased expression of the beta-amylase gene in leaves and storage roots also accelerate

163 volved in sugar- and hormone-regulated alpha-amylase genes expression in rice.

164 Humans have more copies of amylase genes than other primates.

165 a result, some GH families, including alpha-amylases (GH13), have their chemical steps concealed kin

166 Enzymes including papain, alpha-amylase, glucose oxidase and phytase stabilized dough st

167 % in both alpha-glucosidase (>99%) and alpha-amylase (>=70%).

168 -reactivity to mealworm tropomyosin or alpha-amylase, hexamerin 1B precursor and muscle myosin, respe

169 The inhibition of human pancreatic alpha-amylase (HPA) enzyme activity can offer facile routes to

170 order of seconds) with Human Salivary alpha-amylase (HSA) and Porcine Pancreatic alpha-amylase (PPA)

171 on Aspergillus oryzae fermentation and alpha-amylase hydrolysis are also proposed.

172 relevant effect on enzyme inhibition (alpha-amylase: IC(50)-42.34 ug/mL; alpha-glucosidase: IC(50):6

173 885 +/- 85.4 mug/ml, respectively) and alpha-amylase (IC50 343 +/- 26.2 and 167 +/- 6.12 mug/ml, resp

174 ns displayed strong inhibition towards alpha-amylase [IC50, 108.68 mug/ml (bran) and 148.23 mug/ml (h

175 ida showed inhibitory activity against alpha-amylase, IC50 0.74+/-0.02mg/ml and 0.81+/-0.03mg/ml, res

176 beta-Amylase immobilization onto GO-CNT (bamyl@GO-CNT) and Fe

177 We have developed a highly sensitive alpha-amylase immunosensor platform, produced via in situ elec

178 correlation with the concentration of alpha-amylase in buffer, which helps the detection of unknown

179 efficient in detecting unknown quantities of amylase in human blood serum.

180 Concentration of alpha-amylase in human serum is a key indicator of various pan

181 option of an enzymatic treatment, with alpha-amylase in order to reduce the paste viscosity of the re

182 ace to enable a sensitive detection of alpha-amylase in serum (25 - 100 U/l) at a quick response time

183 eous starch-FeSO(4) solution to detect alpha-amylase in serum.

184 h-lab tests while detecting unknown level of amylase in serum.

185 correlated to detect the level of the alpha-amylase in the analyte.

186 t variation exists in the use of drain fluid amylase in the management and timing of surgical drain r

187 We aim to quantitate the levels of amylase in the terminal ileum.

188 of sensitization to enzymes other than alpha-amylase in UK supermarket bakers; in only a small propor

189 as a very effective inhibitor of human alpha-amylase in vitro, comparable to the drug acarbose.

190 nti-diabetic properties because strong alpha-amylase inhibition generally causes undesired side effec

191 alpha-Amylase inhibition of WSEs were >34% in both milk types

192 Their effect on alpha-amylase inhibition was evaluated.

193 tissues (85% alpha-glucosidase and 8% alpha-amylase inhibition).

194 t of lectins (48%), trypsin inhibitor (57%), amylase inhibitor (49%) and phytic acid (56%).

195 The smallest 32 amino acid alpha-amylase inhibitor from Amaranthus hypochondriacus (AAI)

196 combined as deemed fit to enhance the alpha-amylase inhibitor peptide discovery.

197 Antioxidant and alpha-amylase inhibitor peptides were successfully extracted f

198 t peptides, whereas seven peptides for alpha-amylase inhibitor.

199 Cystine knot alpha-amylase inhibitors are cysteine-rich, proline-rich pepti

200 Multiple amylase inhibitors were also identified.

201 disulfide bond conserved within known alpha-amylase inhibitors were present in AhAI.

202 lic compounds and alginates are potent alpha-amylase inhibitors, thereby potentially retarding glucos

203 ied as potential alpha-glucosidase and alpha-amylase inhibitors.

204 The antioxidant, antitumor and alpha-amylase inhibitory activities of exopolysaccharide-C47 p

205 tro antioxidant, alpha-glucosidase and alpha-amylase inhibitory activities of various crudes and frac

206 The alpha-amylase inhibitory activities ranged from 52.5 to 67.2mu

207 atio and temperature, gave the highest alpha-amylase inhibitory activity (57.5%).

208 , ABTS (42.2%) and FRAP (0.81 mM)) and alpha-amylase inhibitory activity (62.1%), was then subjected

209 e-based method was developed to assess alpha-amylase inhibitory activity (GOD method).

210 otein co-precipitates showed increased alpha-amylase inhibitory activity compared to non-sonicated sa

211 or the detection and quantification of alpha-amylase inhibitory activity using the glucose assay kit

212 The range of the values for alpha-amylase inhibitory activity was 10.03-23.33mM, whereas t

213 ited the highest alpha-glucosidase and alpha-amylase inhibitory activity with IC50=1.1+/-0.1mug/ml an

214 l phenolic compounds and alginates for alpha-amylase inhibitory effects.

215 elop an efficient workflow to discover alpha-amylase inhibitory peptides from cumin seed.

216 Salivary amylase initiates starch digestion in the oral cavity; s

217 Therefore, we histologically assessed amylase, insulin, glucagon, lipase, and/or trypsinogen i

218 L. pneumophila injects the effector LamA, an amylase, into the cytosol of human macrophage (hMDMs) an

219 The effect is larger when alpha-amylase is added.

220 alpha-amylase is an established marker for diagnosis of pancre

221 Amylase is elevated in the foregut and has been used to

222 Clinical outcomes are best when drain fluid amylase is low and operatively placed drains are removed

223 play help an immediate presentation of alpha-amylase level in the serum, comparable to the clinically

224 nge, 30-120 U/L [0.50-2.0 mukat/L]), a serum amylase level of 210 U/L (3.50 mukat/L) (normal range, 3

225 Serum amylase level was within normal limits at 84 U/L (1.40 u

226 control diet with restored serum lipase and amylase levels (p < 0.05).

227 rates increased linearly with salivary alpha-amylase levels (r(2) = 0.6, p = 0.02).

228 Low serum salivary amylase levels have been associated with a range of meta

229 Median daily amylase levels ranged from 4470 U/L to 23,000 U/L, with

230 Amylase levels remain highly elevated as the enzyme tran

231 Serum lipase and amylase levels were analyzed at the end of the study.

232 Starch is degraded primarily by beta-amylases, liberating maltose, but this activity is prece

233 vity for three different classes of enzymes (amylase, lipase, and sulfatase), relying on two distinct

234 The inhibition of alpha-glucosidase, alpha-amylase, lipase, cyclooxygenases-1 and -2 (COX-1/COX-2),

235 otein 3beta/pancreatitis-associated protein, amylase, lipase, glucose, and creatinine levels were qua

236 atinization (-0.14 to -0.46J/g), enthalpy of amylase-lipid complex (4329-2293J/g), total flavonoid co

237 e genomes and find that regions flanking the amylase locus show notable depression of genetic diversi

238 72 h at 40 degrees C, only trypsin and alpha-amylase maintained high activity.

239 lmonella used its two GHs sialidase nanH and amylase malS for internalization by targeting different

240 itated with the simple and readily available amylase measurements employed with serum tests.

241 during the first hour of fermentation, while amylase-mediated sugar release was predominant in the la

242 The effect of xylanase and alpha-amylase on DON content depended on the fermentation temp

243 Addition of different dosage of alpha-amylase on the biosensor selectively depletes starch sta

244 nd autoimmune disorder (one [5%]), increased amylase (one [5%]), myositis (one [5%]), and dysphonia (

245 nzymes without sensitization to either alpha-amylase or flour.

246 st fluid analysis (carcinoembryonic antigen, amylase, or mucin presence).

247 , whereas the output of proteins, especially amylase (P < 0.05), was decreased.

248 for lipase -p = 0.009-, and quinoa for alpha-amylase -p < 0.001-).

249 neously co-immobilizing three enzymes; alpha-amylase, pectinase and cellulase onto amino-functionaliz

250 While amylase-positive (AMY(+)) acinar cells were detectable i

251 a-amylase (HSA) and Porcine Pancreatic alpha-amylase (PPA).

252 of potato starch by porcine pancreatic alpha amylase (PPAA) was investigated using isolated starch an

253 gene-based community analysis that providing amylase-pretreated wheat bran as the sole added energy s

254 ation of trienzyme treatment combining alpha-amylase, protease and gamma-carboxy peptidase allowing c

255 rs) or even different hydrolases (e.g. alpha-amylase/protease inhibitors preventing both early germin

256 detection of immunosuppressant drugs, alpha-amylase protein, or protease activity of thrombin and Fa

257 linking) and two enzymes modification (alpha-amylase/pullulanase) falls under the former classificati

258 ns and the AuNPs present catalyse the starch-amylase reaction on the PANI surface to enable a sensiti

259 herapeutic dose of drugs used to treat alpha-amylase related diseases.

260 vation by the low gastric pH, salivary alpha-amylase released about 80% of the starch in bread and 30

261 lytic enzymes (salivary and pancreatic alpha-amylases) remains a matter of debate.

262 h) and cholecystokinin (CCK-8), including 1) amylase secretion, 2) exocytosis, 3) intracellular Ca(2+

263 se regardless of LogP, the presence of alpha-amylase selectively reduces the headspace concentration

264 Dispensing alpha-amylase solution on the starch-iodine coated paper reduc

265 Covalently binding an alpha-amylase specific antibody to a polyaniline (PANI) layer

266 C. platycarpus viz., chitinase (CHI4), Alpha-amylase/subtilisin inhibitor (IAAS) and Flavonoid 3_5 hy

267 Since the action of already known plant beta-amylases (sweet potato and soybean) on native starch gra

268 s have multiple copies of the gene for alpha-amylase, the enzyme that breaks down starchy foods, and

269 ction was employed to detect the activity of amylase, the sensor was found to be equally efficient in

270 he binding of hydrophobic compounds to alpha-amylase, thereby increasing their headspace concentratio

271 n were elevated lipase (four [5%]), elevated amylase (three [4%]), and fatigue, maculopapular rash, d

272 lications may be an essential first step for amylase to be expressed in saliva.

273 and can potentially be used with industrial amylases to convert starch into a fermentable carbohydra

274 The beta-amylase treatment significantly increased the complexed

275 rch complexes with linoleic acid when a beta-amylase treatment was applied to acetylated and debranch

276 at a common dietary protein component, wheat amylase trypsin inhibitors (ATI), stimulate intestinal m

277 Amylase trypsin inhibitors (ATIs), a component of wheat,

278 acked wheat or gluten, with or without wheat amylase trypsin inhibitors (ATIs), for 1 week.

279 BACKGROUND & AIMS: Wheat amylase-trypsin inhibitors (ATIs) are nutritional activa

280 Wheat amylase-trypsin inhibitors (ATIs) are nutritional activa

281 Amylase-trypsin inhibitors (ATIs) have recently been ide

282 Amylase-trypsin inhibitors (ATIs) in wheat and related c

283 Alpha-amylase/trypsin bi-functional inhibitors (ATIs) are non-

284 ased concentrations of lipase (six [8%]) and amylase (two [3%]).

285 erases (one [2%] of 46), increased lipase or amylase (two [4%]), and pancreatitis (one [2%]).

286 stigate the relationship between drain fluid amylase value on the first postoperative day (DFA1) and

287 interrupting gastric amylolysis by salivary amylase via a preliminary acidification of gastric conte

288 tory activity on pancreatic lipase and alpha-amylase was assessed by traditional in vitro methods (wi

289 nsity of the color with the concentration of amylase was estimated in three stages: (i) initially, th

290 e susceptibility of granular starch to alpha-amylase was not affected.

291 Drain fluid amylase was recorded on POD1 for 1285 (27.3%) patients w

292 Serum lipase and amylase were lower in the DR group than in the control a

293 Callosobruchus chinensis (Coleoptera) alpha-amylases were completely inhibited.

294 alpha-amylase compared to C. chinensis alpha-amylase, which could be the rationale behind the dispari

295 g properties and its susceptibility to alpha-amylase, which further affects viscosity.

296 osity and high susceptibility to wheat alpha-amylase, which further facilitates the decrease of visco

297 ared particularly important to inhibit alpha-amylase, while the hydroxyl group (OH) at C3 of the C-ri

298 was found to be a potent inhibitor of alpha-amylase with an IC50 value of 0.046+/-0.004mg/ml.

299 bition of bacterial and human salivary alpha-amylases with IC50 values of 0.11 and 0.04mumol, respect

300 Additionally, combination of alpha-amylase, xylanase and cellulase had a synergetic effect