ライフサイエンスコーパス: anamnestic

1 A antibodies in both mice and humans may be anamnestic.

2 .64), which suggests that responses were not anamnestic.

3 s were functional and able to mount a robust anamnestic Ab response upon revaccination.

4 latter stages of the GC response and in the anamnestic AFC response.

5 we assessed the magnitude of the primary and anamnestic Ag-specific IgG responses of mice to four cli

6 Mitigating the problem of AMBR requires the anamnestic and de novo DSA responses to be prevented and

7 at much lower viral loads and coincided with anamnestic anti-hepatitis B surface (HBs) responses and

8 Anamnestic anti-SEB serum immunoglobulin G (IgG) respons

9 The anamnestic anti-spike IgG response to the first boost wa

10 Anamnestic antibodies (590-fold increased titer) but not

11 1, MSP-2, and MSP-3 that are needed to evoke anamnestic antibody and effector T-cell responses elicit

12 ully vaccinated individuals developed robust anamnestic antibody and T cell responses after infection

13 infection with virulent chlamydiae promotes anamnestic antibody and T cell responses that protect th

14 2 were impaired in their ability to mount an anamnestic antibody response and were more susceptible t

15 Our data suggest that the cross-reactive anamnestic antibody response has a protective capacity d

16 An anamnestic antibody response was also detected.

17 ication, plasma cell differentiation, and an anamnestic antibody response.

18 he absence of viraemia and the absence of an anamnestic antibody response.

19 undetectable viral loads and the lack of an anamnestic antibody response.

20 ses in naive hosts, ICC-1132 elicited potent anamnestic antibody responses in mice primed with P. fal

21 on against SARS-CoV-2 in NHPs and that rapid anamnestic antibody responses in the lung may be a key m

22 s in the peripheral blood and the absence of anamnestic antibody responses postchallenge.

23 sure to pathogens or their antigens triggers anamnestic antibody responses that are higher in magnitu

24 owever, neither route of vaccination induced anamnestic antibody responses to the surface antigens of

25 ty, induced durable immunity, and primed for anamnestic antibody responses, making it an attractive c

26 ble but delayed and potentially dependent on anamnestic antibody responses.

27 Secondary patients had robust anamnestic antibody responses.

28 Anamnestic binding and neutralizing antibody responses w

29 ortant immunologic questions include whether anamnestic CD4 T cell responses drive disease rather tha

30 llowing the CL-13 boost, a greater number of anamnestic CD8 T cells localized to the lymph nodes, exh

31 Anamnestic CD8(+) T cell responses were further enhanced

32 s known regarding how AhR activation affects anamnestic CD8+ T cell responses.

33 6a) did not develop viremia but did show an anamnestic cell-mediated immune response after rechallen

34 Massive early anamnestic cellular immune responses controlled acute-ph

35 However, anamnestic cellular immune responses developed during th

36 bjective molecular clusters, irrespective of anamnestic clinico-morphological features, despite the c

37 d macaques exhibited a rapid and substantial anamnestic CTL response specific for the p11C, C-M Gag e

38 ociated with the evolution of high-frequency anamnestic CTL responses specific for a dominant Mamu-A*

39 from PPD Ag-challenged lungs conferred a Th1 anamnestic cytokine response in recipients.

40 Clinical and anamnestic data were collected using a standardized form

41 Clinical and anamnestic data were systematically collected, and patie

42 , suggests that infection induces suboptimal anamnestic defenses.

43 all four of the immunized animals developed anamnestic E-SPOT responses after challenge.

44 nths, and re-exposure to antigen resulted in anamnestic effector and central memory CD8(+) T-lymphocy

45 e boost vaccination minimizes contraction of anamnestic effectors and maximizes memory CD8+ T cell qu

46 er, prior immunization capable of priming an anamnestic Env antibody response did not accelerate V1/V

47 t in protective immunity was associated with anamnestic Env-specific cellular immunity that developed

48 wever, 13 blips of virus in six macaques and anamnestic Env-specific rectal IgA responses in three of

49 lls are required for protection, there is no anamnestic expansion of SIV-specific CD8(+) T cells in a

50 tibodies against all vaccine genotypes in an anamnestic fashion.

51 able ASC responses to primary infection, and anamnestic fecal IgA responses likely contributed to sel

52 eased both the magnitude and the kinetics of anamnestic Gag-specific T cell responses following HIV-1

53 V-gag/pol/env prime was able to induce rapid anamnestic gp140 antibody response upon SIV encounter.

54 ng of the immune system to induce protective anamnestic heterotypic neutralizing antibody responses u

55 nonvaccinated acutely infected individuals, anamnestic HIV-1-specific B- and T-cell responses appear

56 Strong anamnestic humoral and CMI responses were elicited by 1

57 SV pre-exposed NHPs elicits a dose-dependent anamnestic humoral immune response comparable to a subun

58 memory B cells upon antigen re-exposure for anamnestic humoral responses, but little is known about

59 After challenge, anamnestic IgA and IgG ASC and memory B-cell responses w

60 -VLPs (with or without mLT) developed higher anamnestic IgA and IgG ASC responses in ileum after chal

61 sting with an unadjuvanted protein amplified anamnestic IgA responses in both the upper and the lower

62 We found an anamnestic IgG response in one pretransfusion seropositi

63 at a rapid IgG response to SARS-CoV-2 and an anamnestic IgG response to the nucleoprotein of the CCCs

64 ugate-vaccinated and control G2 mice induced anamnestic IgG- and VH3-positive responses to GXM and wa

65 valent vaccine (HR-Tet) showed a tetravalent anamnestic immune response in 100% (16/16) of AGMs after

66 ers of Clostridium difficile have a systemic anamnestic immune response to toxin A.

67 The observed anamnestic immune response was characterized by (i) more

68 Anamnestic immune responses after rechallenge suggested

69 Strong anamnestic immune responses can be induced by additional

70 erosol challenge model, and that primary and anamnestic immune responses to toxin proteins provide pr

71 Anamnestic immune responses were demonstrated for HI, ne

72 acute viremia that inversely correlated with anamnestic immune responses.

73 ine adjuvants for eliciting both primary and anamnestic immune responses.

74 ivotal role for IL-33 in driving primary and anamnestic immunity against the rodent hookworm Nippostr

75 ere investigated to understand mechanisms of anamnestic immunity at the skin barrier.

76 cleared from the lower respiratory tract by anamnestic immunity.

77 One of these two monkeys also showed no anamnestic increases in antibody levels following challe

78 ung residence of T(H)2 cells and ameliorates anamnestic inflammation in adults.

79 nd matched controls were surveyed on general anamnestic information, eating and drinking habits, and

80 By corroborating clinical, anamnestic, laboratory and imaging data, the case was di

81 Thus, anamnestic memory T cell differentiation is flexible, an

82 Moreover, anamnestic NAb response is undetectable in challenge ani

83 is a promising vector to use to prime for an anamnestic neutralizing antibody response following infe

84 This anamnestic neutralizing antibody response was also detec

85 human immunodeficiency virus (SHIV89.6P), an anamnestic neutralizing antibody response was observed,

86 Although anamnestic neutralizing antibody responses against labor

87 V/6120/DeltaNS2/1030s primed for substantial anamnestic neutralizing antibody responses following nat

88 vaccination elicited comparable primary and anamnestic neutralizing antibody responses.

89 xhibited functional exhaustion and decreased anamnestic potential following secondary antigen challen

90 IMV to induce affinity maturation results in anamnestic production of nonprotective, complement-fixin

91 ation because it takes advantage of both the anamnestic properties of an adaptive immune response and

92 or T-cell-deficient mice lacked the complete anamnestic protection observed in immunocompetent mice.

93 0.1 mL ID of HDCV was given to evaluate the anamnestic rabies antibody response.

94 se to current antigens but instead suggested anamnestic recall of antibody to earlier influenza virus

95 A fourth dog exhibited a strong early anamnestic response (216 BU), with slow decline to 0.8 B

96 xisting FIX inhibitors exhibited a transient anamnestic response (5 Bethesda units) at 2 weeks after

97 presence of a detectable antibody or a rapid anamnestic response (ie, priming).

98 vage and a stronger rectal anti-envelope IgA anamnestic response 2 weeks postchallenge.

99 However, anamnestic response after reinfection was variant-depend

100 Development of an anamnestic response after vaccination would suggest prev

101 with recombinant virus, animals exhibited an anamnestic response against SNV.

102 n, and levels of both increased in a typical anamnestic response following a challenge infection.

103 acaques exhibited a CD4(+) and CD8(+) T cell anamnestic response following booster immunization.

104 Anamnestic response following homologous or heterologous

105 on Beta/B.1.351 (rS-Beta) produces a robust anamnestic response in baboons against SARS-CoV-2 varian

106 The anamnestic response in immunized cattle was specific for

107 rs from a third dose suggests a brisk B-cell anamnestic response in patients with cancer.

108 y responses following challenge indicated an anamnestic response in the vaccinated monkeys.

109 V, both CD4 Th1 and Th2 memory cells made an anamnestic response in vivo.

110 The anamnestic response is the property of the immune system

111 In experiments designed to compare the anamnestic response of susceptible and resistant mouse s

112 peak anti-Nef titers, a more rapid anti-Nef anamnestic response postchallenge, and expanded CD8(CM)

113 After hepatitis B vaccination, the anamnestic response rate in HIV-1-positive subjects who

114 The overall anamnestic response rate was 16% and was not significant

115 The anamnestic response rate was higher in subjects who test

116 However, such patients are at risk for an anamnestic response resulting from a proinflammatory res

117 They also developed a potent and fast anamnestic response to a subsequent parenteral boost wit

118 fter the initial immunization resulted in an anamnestic response to C-GTF resulting in 10- and 100-fo

119 This may have represented an anamnestic response to HCV antigens translated directly

120 r SIV challenge, apparently the result of an anamnestic response to SIV antigens.

121 ing immunization with NYVAC/SIV(gpe) and the anamnestic response to SIV(mac251) at 48 h after challen

122 n there is an association between a systemic anamnestic response to toxin A, as evidenced by increase

123 d an immune reaction compatible with a local anamnestic response upon contact with the replicating FM

124 after primary CHIKV immunization and then an anamnestic response upon subsequent VEEV vaccination.

125 An anamnestic response was defined as an anti-HBs titer of

126 timing of humoral response was similar, and anamnestic response was driven by the variant involved d

127 An anamnestic response was observed in all monkeys after th

128 A classic anamnestic response was observed in patients dosed with

129 A strong anamnestic response was observed when convalescent baboo

130 Following challenge infection, the dominant anamnestic response was solely in the B-cell compartment

131 imals and showed little or no evidence of an anamnestic response when measured with SIVsmE660.

132 or T cells, memory is often equated with the anamnestic response, the ability of secondary lymphoid t

133 y levels in serum, suggesting the lack of an anamnestic response.

134 booster vaccination after 1 year induced an anamnestic response.

135 al immunization, DCFHP-alum elicits a robust anamnestic response.

136 A-1273-vaccinated hamsters coincided with an anamnestic response.

137 ted bacterial clearance, and orchestrated an anamnestic response.

138 but lacking detectable antibody abrogates an anamnestic response.

139 patients and provide treatment to prevent an anamnestic response.

140 tive persons marked the capacity to mount an anamnestic response.

141 of the serotype-cross-reactive "recall" or "anamnestic" response.

142 zyme-linked immunosorbent assay demonstrated anamnestic responses after each boost.

143 ely), indicating that the vaccine primed for anamnestic responses after natural RSV exposure.

144 survival after secondary challenge and rapid anamnestic responses directed against toxin proteins.

145 Booster vaccination elicited anamnestic responses in all participants.

146 Anamnestic responses in front-line tissues are thus an i

147 e, concentrations were extremely low with no anamnestic responses in mice with secondary infection.

148 odies (DSA) that are most often generated as anamnestic responses in sensitized recipients or de novo

149 trachoma vaccine and their peripheral T cell anamnestic responses studied.

150 ry B cell subsets as potential predictors of anamnestic responses to additional immunizations.

151 rise to long-lived memory T cells capable of anamnestic responses to antigenic rechallenge.

152 Anamnestic responses to boosting without sustained high

153 nd IgG antibodies were induced in serum with anamnestic responses to secondary infection.

154 y illness (RSV-MAALRI) and primed for potent anamnestic responses upon natural exposure to wild-type

155 monstrated that both vaccines induced potent anamnestic responses upon virus challenge, potentiated i

156 Anamnestic responses were observed in preimmune individu

157 stsurveillance without RSV-MAARI, indicating anamnestic responses without significant illness after i

158 assay to study antibody-maturation patterns, anamnestic responses, and cross-immunity to the common-c

159 nogenicity and primed vaccine recipients for anamnestic responses, encouraging further evaluation of

160 both proximal and distal sites contribute to anamnestic responses, T cells from proximal IC-LNs prefe

161 unoglobulin G responses, and near absence of anamnestic responses.

162 cines were immunogenic and primed for strong anamnestic responses.

163 cipally by CD4(+) and CD8(+) T cell-mediated anamnestic responses.

164 at rabies booster immunization elicits rapid anamnestic responses.

165 nerated long-lived memory T cells capable of anamnestic responses.

166 ion with nonconjugated Pfs25H induced strong anamnestic responses.

167 oire of cancers depends, on one hand, on the anamnestic retention of their ontogenesis and, on the ot

168 subsequent winter was associated with strong anamnestic RSV-antibody responses.

169 By use of a murine model, the anamnestic serologic response was characterized followin

170 el, which is likely to be a reflection of an anamnestic serum IgG response.

171 In contrast, the anamnestic SIV Gag-specific CD4(+) T cell response in BA

172 However, the anamnestic, SIV Gag-specific CD8(+) T cell response to S

173 peptides as immunogens to prime for strong, anamnestic, strain-cross-reactive type 1 immune response

174 of these antigen delivery systems to induce anamnestic systemic and secretory responses to the clone

175 a chlamydial component drives a deleterious anamnestic T cell response upon oviduct reinfection.

176 Anamnestic T cell responses to Ag85A/b were not detected

177 s in the vaccine phase, and occurred without anamnestic T-cell responses.

178 emia during persistence could result from an anamnestic Th lymphocyte response to conserved regions o

179 Anamnestic uncertainty, found in 88 families (8.5%), if

180 mutation rate; and evaluation of cases with anamnestic uncertainty.

181 eropositive individuals may have resulted in anamnestic, vector-specific CD4(+) T lymphocytes that co

182 ion rate), and all groups showed significant anamnestic virus-specific IgG and IgA ASC responses.