1 carcinomas, 11 small cell carcinomas, and 27 anaplastic carcinomas.

2 omising therapy for ATC and other aggressive anaplastic carcinomas.

3 e only detected in poorly differentiated and anaplastic carcinomas.

4 lowing loss of p53 function, may progress to anaplastic carcinomas.

5 PTCs), 12 were follicular carcinomas, 9 were anaplastic carcinomas, 5 were Hurthle cell carcinomas, 2

6 was associated with aggressive medullary and anaplastic carcinomas, and its expression pattern in med

7  thyroid cancer to poorly differentiated and anaplastic carcinomas, both in terms of the presence of

8  of 2 papillary carcinoma cell lines, 4 of 5 anaplastic carcinoma cell lines, 1 of 2 follicular carci

9 nt thyroid tumor cell lines, such as thyroid anaplastic carcinoma cells (ARO cells), while either age

10 choalveolar cell carcinoma) or Calu 6 (human anaplastic carcinoma) NSCLC cells.

11  in pulmonary adenocarcinomas and large-cell anaplastic carcinomas (P=0.006).

12  (one PTC, one follicular carcinoma, and one anaplastic carcinoma) showed evidence of hemizygous dele

13 nd 11q13, and loss of 5q11-31 were unique to anaplastic carcinomas, suggesting they may be markers fo

14 cell cancer of the right lung (microcellular anaplastic carcinoma), was admitted with focal neurologi