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1 rt striking reciprocal phenotypic effects on androgenetic (AG: two paternal genomes) and parthenogene
2 the mother (gynogenetic [GG]) or the father (androgenetic [AG]) are unique models for studying genomi
3 [95% confidence intervals]) in patients with androgenetic alopecia (37.3 128.4, 46.1]) and alopecia a
4                                              Androgenetic alopecia (AGA) has now become a common diso
5                                              Androgenetic alopecia (AGA) is a common heritable and an
6                                              Androgenetic alopecia (AGA) is a condition of scalp hair
7                                              Androgenetic alopecia (AGA) is associated with trichodyn
8                                        Early androgenetic alopecia (AGA) is patterned hair loss occur
9                                              Androgenetic alopecia (AGA) is the most common type of h
10                                  Presence of androgenetic alopecia (AGA) might be such a predictor.
11  Dermal papilla cells (DPCs) taken from male androgenetic alopecia (AGA) patients undergo premature s
12 ased interest in low-dose oral minoxidil for androgenetic alopecia (AGA) treatment.
13                                              Androgenetic alopecia (AGA), a hereditary disorder that
14                                              Androgenetic alopecia (AGA), also known as common baldne
15 s is used in established scoring systems for androgenetic alopecia (AGA), central centrifugal cicatri
16 l variants implicated in the pathobiology of androgenetic alopecia (AGA), eczema and other complex tr
17 lative efficacy of 3 commonly used drugs for androgenetic alopecia (AGA), namely, minoxidil and the t
18                                 Male pattern androgenetic alopecia (mpAGA), the most common form of h
19 ed patients with telogen effluvium (n = 30), androgenetic alopecia (n = 52), alopecia areata (n = 17)
20 nt follicular cycling in the scalp including androgenetic alopecia (pattern hair loss).
21 ison, skin biopsies from alopecia areata and androgenetic alopecia affected humans were also collecte
22  in the miniaturization of hair follicles of androgenetic alopecia by interfering with the dermal pap
23               Male pattern baldness (MPB) or androgenetic alopecia is one of the most common conditio
24                                              Androgenetic alopecia may begin in adolescence, and topi
25 rpolarization rescues hair loss in aging and androgenetic alopecia models.
26 c skin disorders such as acne, hirsutism, or androgenetic alopecia remains to be established.
27 d occipital region in males with and without androgenetic alopecia revealed that next to the androgen
28 zed by premature entry into catagen, such as androgenetic alopecia, alopecia areata, and telogen effl
29 opecia in adolescence are telogen effluvium, androgenetic alopecia, and alopecia areata.
30 bel in the management of acne, hidradenitis, androgenetic alopecia, and hirsutism.
31 therapy-induced alopecia, telogen effluvium, androgenetic alopecia, cicatricial alopecia, graying).
32 reatment of benign prostatic hyperplasia and androgenetic alopecia, reduced self-administration of mu
33 eptor degraders, both in clinical trials for androgenetic alopecia, with GT20029 also evaluated for a
34      Of 18 patients who had biopsies, 14 had androgenetic alopecia-like features, 2 had cicatricial a
35 aking finasteride over the past 3 months for androgenetic alopecia.
36 ntagonist for sebum control and treatment of androgenetic alopecia.
37 us-to-terminal and terminal-to-vellus during androgenetic alopecia.
38 scopic signs indistinguishable from those of androgenetic alopecia.
39 nuated in the scalp tissues of patients with androgenetic alopecia.
40  masculinizing hormone therapy who developed androgenetic alopecia.
41 al (already demethylated) paternal genome in androgenetic and triploid diandric embryos.
42 etic ova), and in individual gynogenetic and androgenetic blastomeres, both maternal and paternal Igf
43 ion were also present in parthenogenetic and androgenetic cells and in tissues from animals maternall
44  is phenotypically indistinguishable from an androgenetic complete hydatidiform mole, in which abnorm
45    Both maternal (gynogenetic) and paternal (androgenetic) derived cells conveyed long-term, multilin
46 -modified paternal pronucleus should support androgenetic development (i.e., from the paternal pronuc
47 ans by generating exclusively paternal human androgenetic embryonic stem cells (aESCs) and comparing
48 pressed in differentiated cells derived from androgenetic embryonic stem cells and normal embryos but
49 f the coding sequence in parthenogenetic and androgenetic embryonic stem cells.
50 s a difference in developmental potential of androgenetic embryos produced with eggs from females of
51             Specifically, using 8-cell stage androgenetic embryos, a single spermatozoon can yield up
52 licated in the periimplantation lethality of androgenetic embryos.
53                                        Mouse androgenetic haploid embryonic stem cells (AG-haESCs) ca
54   We developed a meiotic mapping panel of 94 androgenetic haploid embryos that were scored for geneti
55 n favor of its plausibility at the origin of androgenetic HM formation in humans.
56 ge is the strongest risk factor for sporadic androgenetic HM, which affects 1 in every 600 pregnancie
57 H5, SYCP2, MEIOB, and HFM1, in patients with androgenetic HMs, including a familial case of 3 affecte
58 enome in sporadic hydatidiform moles (purely androgenetic in complete hydatidiform moles and diandric
59                Although most complete HM are androgenetic in origin, a rare, frequently familial, bip
60 ise to a 46,XX genotype and is thought to be androgenetic in origin.
61 enetic material from other species may allow androgenetic lineages of Corbicula to mitigate the effec
62 NA from other species occur within otherwise androgenetic lineages of Corbicula.
63 of which are indistinguishable from those in androgenetic or gynogenetic embryos.
64 ntire genome is either exclusively paternal (androgenetic) or maternal (parthenogenetic), exhibit dra
65 HM, identical to that seen in complete HM of androgenetic origin (AnCHM).
66 ion mechanisms: in individual blastomeres of androgenetic ova, both paternal Snrpn alleles were activ
67 normal ova were unchanged in gynogenetic and androgenetic ova; the latter contain two maternal and tw
68 lopment that are particularly evident in the androgenetic phenotype, uniparental cells of both parent
69 ions in Drosophila melanogaster that produce androgenetic progeny, we demonstrate that the Wolbachia-
70 al and nuclear loci from multiple sexual and androgenetic species across the global distribution of C
71 the hypothesis of long-term clonality of the androgenetic species.
72 nes have been produced using gynogenetic and androgenetic techniques.
73 arthenogenetic (two maternal chromosomes) or androgenetic (two paternal chromosomes) cells displaying
74 ferences in X-linked gene expression between androgenetic (two paternal genomes), gynogenetic (two ma