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1 y secretions that could be attributed to the anesthetic agent.
2 s a powerful anxiolytic, anticonvulsant, and anesthetic agent.
3 t that was developed over 50 years ago as an anesthetic agent.
4 endent on a known interaction site for local anesthetic agents.
5 sm, which alter the pharmacokinetics of many anesthetic agents.
6 losporine A, radiocontrast dyes and volatile anesthetic agents.
7 imental evidence supports neuroprotection by anesthetic agents.
8 ro- and anti-convulsant effects of the newer anesthetic agents.
9 as performed to compare ketamine and 7 other anesthetic agents.
11 and enflurane, three commonly used volatile anesthetic agents, affect glutamate receptor-modulated n
12 ve considerable potential to be developed as anesthetic agents and as drugs to treat brain disorders
13 r any of the commonly available inhalational anesthetic agents and each can be used for general anest
14 associated with the use of lower amounts of anesthetic agents and faster recovery from anesthesia in
15 e cells, and are molecular targets for local anesthetic agents and intracellular free Ca(2+) ([Ca(2+)
16 1 measurements obtained with several inhaled anesthetic agents and propofol dissolved in saline were
19 ystem development and are being developed as anesthetic agents and treatments for psychiatric disease
20 he level of consciousness in volunteers with anesthetic agents and visualizing the resultant changes
21 Epidemiological studies suggest exposures to anesthetic agents and/or sedative drugs (AASDs) in child
24 idence for neuroprotective effects of common anesthetic agents, and presents potential mechanisms inv
25 mparison to other small hydrophobic poisons, anesthetic agents, and protein toxins that inhibit ion c
26 ECT intervention with ketamine and/or other anesthetic agents; and (3) measures included: depressive
30 channels, which are putative targets for the anesthetic agents, as well as advancements in high-perfo
31 visiae was used to investigate the action of anesthetic agents because of its powerful molecular gene
32 rcotics, antipsychotics, inotropes, digoxin, anesthetic agents, bronchodilators, and drugs that cause
33 corporating standard monitoring of end-tidal anesthetic-agent concentration (ETAC) for the prevention
34 esthetic management directed by an end-tidal anesthetic-agent concentration protocol is equally effec
35 uch higher (toxic) concentrations of certain anesthetic agents did alter lipid bilayer properties.
37 ty of surgical wound infiltration with local anesthetic agents during cesarean delivery for the reduc
39 lness, escalating and constant levels of two anesthetic agents (experiment 1, n = 39), and during sle
41 ed mutation at an interaction site for local anesthetic agents (F1760A) partially attenuated the effe
44 It is less widely known that several other anesthetic agents have also shown antidepressant effects
48 Ca(2+), changes in internal and external pH, anesthetic agents, heat, stretch and membrane deformers.
49 the pharmacokinetics and pharmacodynamics of anesthetic agents in the obese population, we specify ce
53 that anesthetic management and even specific anesthetic agents may worsen outcomes in high-risk patie
55 a method for studying the interaction of two anesthetic agents, Morphine and Midazolam, acting simult
57 ting or motion sickness, young age, volatile anesthetic agents, nitrous oxide, and the administration
58 sics, benzodiazepines, opioids, inhalational anesthetic agents, nitrous oxide, ketamine, barbiturates
62 evaluation of patients, discovery of better anesthetic agents, pain control, and the evolution of pe
69 s of initiating brexanolone infusion without anesthetic agent reinstatement in the following 24 hours
70 cological postconditioning with the volatile anesthetic agent sevoflurane (n = 48), intermittent clam
73 pport a neuroprotective potential of several anesthetic agents, specifically isoflurane and xenon, co
74 erefore, we propose that amphiphilic general anesthetic agents such as 1-alkanols may modulate gating
78 uring surgery and also to optimize dosing of anesthetic agents to maximize the desired/undesired effe
79 nificant correlation between the duration of anesthetic agent use and DeltaVBR was found (Spearman r
81 s, perhaps because of confounding effects of anesthetic agents used in most animal experiments on fun
83 esized, and the utility of these steroids as anesthetic agents was evaluated through determination of
86 Xenon and dichloromethane are inhalational anesthetic agents whose binding to myoglobin has been de
88 EG parameters to increasing concentration of anesthetic agents with a dosing plateau response over a
89 models determined associations of cumulative anesthetic agents with the primary neurocognitive outcom
91 lum can be targeted with US for injection of anesthetic agents, with adequate spread of injected meth
92 001) and receipt of intravenous propofol, an anesthetic agent without preservative, which was adminis