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1 ges in consciousness from the effects of the anesthetic drugs.
2 nonneuronal mechanism for sedative action of anesthetic drugs.
3 nel's inner pore overlap with those of local anesthetic drugs.
4 ty's effects on the clinical pharmacology of anesthetic drugs.
5 an systems decreases the margin of safety of anesthetic drugs.
6 eration in rat pups exposed to commonly used anesthetic drugs.
7 ne cardiac-specific external paths for local anesthetic drugs.
8  Each altered arousal state results from the anesthetic drugs acting at multiple targets in the centr
9  results of preclinical studies suggest that anesthetic drugs administered to neonatal animals cause
10 f the immunological effects of commonly used anesthetic drugs and highlight their potential impact on
11 oss and return of consciousness regulated by anesthetic drugs and physiological sleep are used as mod
12 ousness remains unclear, and whether diverse anesthetic drugs and sleep share a common neural pathway
13 o alterations of physiology and in choice of anesthetic drugs and techniques.
14                               The effects of anesthetic drugs and temperature variation on zebrafish
15 oundational implications for biology because anesthetic drugs are effective in organisms ranging from
16                                              Anesthetic drugs are known to interact with GABAA recept
17                                              Anesthetic drugs are thought to mainly target neurons in
18 (A) receptors as the primary targets of most anesthetic drugs, but how these compounds produce parado
19 e than a decade of mounting animal data that anesthetic drugs can cause apoptosis during a critical p
20                                 Knowing that anesthetic drugs can pose immunomodulatory effects, it w
21                  Propofol, like most general anesthetic drugs, can induce both behavioral and electro
22   The neurophysiological mechanisms by which anesthetic drugs cause loss of consciousness are poorly
23              Multiple continuous intravenous anesthetic drugs (CIVADs) are available for the treatmen
24                                          How anesthetic drugs create the state of general anesthesia
25  BIS monitor results in less use of hypnotic anesthetic drugs, decreased time to extubation, decrease
26                                 This retards anesthetic drug development, confounds interpretation of
27         Devices which monitor some aspect of anesthetic drug effects have evolved in the past few yea
28 ofol (2,6-di-isopropylphenol), a widely used anesthetic drug, exerts its effect primarily by modulati
29 ures (p = 0.0077), and in those who required anesthetic drugs for seizure control (p = 0.0035).
30 fractoriness, use of mechanical ventilation, anesthetic drugs for seizure control, and medical compli
31 tal and 90-day mortality, whereas the use of anesthetic drugs for seizure control, mechanical ventila
32 f induction, brought about by elimination of anesthetic drugs from their CNS site(s) of action.
33 tudies is clear and continuing to mount that anesthetic drugs given at the right time and in sufficie
34 e relaxation, and to minimize the amounts of anesthetic drugs given to infants and sick children.
35 oduction of newer less-toxic, shorter-acting anesthetic drugs has reduced the requirement for muscle
36                                         Many anesthetic drugs have been shown to disrupt conscious re
37 anges in practice and the development of new anesthetic drugs have influenced the use of muscle relax
38 This is a consequence of ideal mixing of the anesthetic drugs in the membrane fluid phase and exclusi
39  have shown that prolonged administration of anesthetic drugs, including ketamine, isoflurane, nitrou
40 lly diverse group of sedative, hypnotic, and anesthetic drugs, including the volatile anesthetic isof
41 ulk of reported complications are related to anesthetic drug-induced respiratory depression or airway
42            Malignant hyperthermia (MH) is an anesthetic-drug-induced, life-threatening hypermetabolic
43 e 3 [interquartile range, 2-4]), 24 received anesthetic drug infusions for seizure control.
44  novel ways of reversing the effects of some anesthetic drugs (inhalational anesthetics and muscle re
45 cular mechanisms of antiarrhythmic and local anesthetic drug interactions with hNa(V)1.5 and will be
46 (GABA(A)Rs), and it is assumed that once the anesthetic drug is eliminated, the activity of GABA(A)Rs
47  support for the antidepressant effect of an anesthetic drug, ketamine, by Inverse-Frequency Analysis
48 the deactivation of NsVBa, whereas the local anesthetic drug lidocaine was shown to antagonize NsVBa
49 is were used to determine whether the use of anesthetic drugs, mechanical ventilation, Status Epilept
50                                       Use of anesthetic drugs, medical complications, and mechanical
51 eceptor (NMDAR) antagonists are dissociative anesthetics, drugs of abuse, and are of therapeutic inte
52 us be understood as a differential effect of anesthetic drugs on thalamic nuclei with disparate spati
53 many to investigate the neurotoxic effect of anesthetic drugs on the developing brain.
54 ainly question and undermine the safe use of anesthetic drugs, particularly in pediatric anesthesia,
55                    Use of enantiomeric local anesthetic drugs permits a safer and wider range of post
56 ions (10-15 Hz) induced by the commonly used anesthetic drug propofol are synchronized between the th
57 uccessfully employed in the synthesis of the anesthetic drug Quinisocaine.
58                      Common clinical general anesthetic drugs, such as propofol and isoflurane, have
59 ugh the rho1 receptor is insensitive to many anesthetic drugs that modulate the heteromeric GABA(A) r
60 ors, such as the influence of vasoactive and anesthetic drugs, total muscular relaxation, or the pres
61 ry of epilepsy, previous SE, type of SE, and anesthetic drug used were not associated with functional
62 tations these monitors can be used to reduce anesthetic drug utilization and turnover time.
63                     Propofol, a sedative and anesthetic drug was chosen as a model lipophilic drug in
64 troencephalogram background suppression with anesthetic drugs) was tested.
65                   Ketamine is a dissociative anesthetic drug, which has more recently emerged as a ra
66  anesthetics and consequences of exposure to anesthetic drugs will likely require the evaluation and
67    Fentanyl is a widely abused analgesic and anesthetic drug with a narrow therapeutic window that cr
68 iate seizure activity and the interaction of anesthetic drugs with AEDs.
69 the interactions of antiarrhythmic and local anesthetic drugs with hNa(V)1.5.