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1 oped as a tool for risk stratification after aneurysmal subarachnoid hemorrhage.
2 ic stiffness occur during the early phase of aneurysmal subarachnoid hemorrhage.
3 d with extent of hemorrhage in patients with aneurysmal subarachnoid hemorrhage.
4 een used to prevent cerebral vasospasm after aneurysmal subarachnoid hemorrhage.
5 mprove the outcome among patients with acute aneurysmal subarachnoid hemorrhage.
6 er craniotomy among good-grade patients with aneurysmal subarachnoid hemorrhage.
7 arted at a median (IQR) of day 2 (1-2) after aneurysmal subarachnoid hemorrhage.
8 hol use, and family history of aneurysms and aneurysmal subarachnoid hemorrhage.
9 dings support the use of lumbar drains after aneurysmal subarachnoid hemorrhage.
10 ly assigned critically ill adults with acute aneurysmal subarachnoid hemorrhage and anemia to a liber
12 the epidemiology of types of stroke, such as aneurysmal subarachnoid hemorrhage and cerebral vein thr
13 agnosing cerebral vasospasm in patients with aneurysmal subarachnoid hemorrhage and for guiding trans
14 sent with intracranial hemorrhage, including aneurysmal subarachnoid hemorrhage and spontaneous intra
15 se-wave velocity were improved between acute aneurysmal subarachnoid hemorrhage and stable state (p <
16 atients in coma with traumatic brain injury, aneurysmal subarachnoid hemorrhage, and intracranial hem
17 n ischemic stroke, intracerebral hemorrhage, aneurysmal subarachnoid hemorrhage, and traumatic brain
19 vasospasm (VSP) is a common phenomenon after aneurysmal subarachnoid hemorrhage (aSAH) and contribute
20 irment in cerebral autoregulation (CA) after aneurysmal subarachnoid hemorrhage (aSAH) is associated
22 le quantifiable scoring system that predicts aneurysmal subarachnoid hemorrhage (aSAH) mortality, del
23 dies revealed the prognosis differed between aneurysmal subarachnoid hemorrhage (aSAH) patients with
25 driver of adverse outcomes in patients with aneurysmal subarachnoid hemorrhage (aSAH), defining an u
26 butor to delayed cerebral ischemia following aneurysmal subarachnoid hemorrhage (aSAH), leading to hi
27 Cerebral vasospasm is a dreaded sequelae of aneurysmal subarachnoid hemorrhage (aSAH), requiring tim
28 emia is associated with worse outcomes after aneurysmal subarachnoid hemorrhage (aSAH), there is no c
29 y contributes to a devastating outcome after aneurysmal subarachnoid hemorrhage (aSAH), with limited
34 ay be used to prevent early rebleeding after aneurysmal subarachnoid hemorrhage, but anticoagulation
35 anal-modified protein levels in humans after aneurysmal subarachnoid hemorrhage correlate with the de
36 model to predict 60-day case fatality after aneurysmal subarachnoid hemorrhage developed from the In
37 Ninety-four percent of participants with aneurysmal subarachnoid hemorrhage experienced augmented
39 or delayed cerebral ischemia treatment after aneurysmal subarachnoid hemorrhage has been challenged r
41 ignificant morbidity and mortality following aneurysmal subarachnoid hemorrhage; however, the effect
42 ologies including migraine, ischemic stroke, aneurysmal subarachnoid hemorrhage, intracerebral hemato
43 diagnosis as primary cerebral vasculitis and aneurysmal subarachnoid hemorrhage is common because of
45 is trial, prophylactic lumbar drainage after aneurysmal subarachnoid hemorrhage lessened the burden o
46 tracerebral hemorrhage (n = 104 [32.7%]), or aneurysmal subarachnoid hemorrhage (n = 81 [25.5%]) and
47 A total of 287 adult patients with acute aneurysmal subarachnoid hemorrhage of all clinical grade
50 ligible patients had traumatic brain injury, aneurysmal subarachnoid hemorrhage, or intracerebral hem
52 ated at two time points: on admission (acute aneurysmal subarachnoid hemorrhage phase) and at least 2
53 luences hemorrhage severity in patients with aneurysmal subarachnoid hemorrhage, potentially through
54 common, treatable condition in patients with aneurysmal subarachnoid hemorrhage (SAH) and has been as
56 cause ischemic neurological damage following aneurysmal subarachnoid hemorrhage (SAH) have remained e
60 thophysiology of early ischemic injury after aneurysmal subarachnoid hemorrhage (SAH) is not understo
61 Mortality and morbidity can be reduced if aneurysmal subarachnoid hemorrhage (SAH) is treated urge
62 In this prospective observational study of aneurysmal subarachnoid hemorrhage (SAH) patients, we ex
64 are is recommended to improve outcomes after aneurysmal subarachnoid hemorrhage (SAH), but understand
69 n is associated with poor outcome, but after aneurysmal subarachnoid hemorrhage, this has not been in
72 uscle atrophy and may improve outcomes after aneurysmal subarachnoid hemorrhage We sought to identify
73 rovide early identification of patients with aneurysmal subarachnoid hemorrhage who are at high risk
74 at risk for delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage with hemoglobin 7-13