ライフサイエンスコーパス: angina pectoris

1 al infarction; 90 patients [42.1%], unstable angina pectoris).

2 acute coronary syndrome (82 MI, 44 unstable angina pectoris).

3 pital with new-onset chest pain or worsening angina pectoris.

4 angina pectoris in most patients with stable angina pectoris.

5 ients with ischemic heart disease and stable angina pectoris.

6 ect adverse outcomes in patients with stable angina pectoris.

7 symptoms and myocardial perfusion in stable angina pectoris.

8 f the endovascular approach in the relief of angina pectoris.

9 001) for combined CHD death/nonfatal MI plus angina pectoris.

10 MI); 86 cases of fatal CHD; and 124 cases of angina pectoris.

11 rent myocardial infarction (MI) and unstable angina pectoris.

12 tractile reserve in patients with refractory angina pectoris.

13 48 (5.9%) men and 41 (5.2%) women developed angina pectoris.

14 of an age less than the duration of unstable angina pectoris.

15 lesions develop in association with unstable angina pectoris.

16 had confirmed acute myocardial infarction or angina pectoris.

17 combined incident coronary events including angina pectoris.

18 mic necroses in other patients with unstable angina pectoris.

19 uced the clinical manifestations of unstable angina pectoris.

20 ostic significance in patients with unstable angina pectoris.

21 (MI), 20 cases of fatal CHD, and 60 cases of angina pectoris.

22 mes were procedure-related complications and angina pectoris.

23 anginal symptoms in patients with refractory angina pectoris.

24 iated with sympathetic phenotypes, including angina pectoris.

25 an association exists between edentulism and angina pectoris.

26 less, and 673,810 (2.3%) were diagnosed with angina pectoris.

27 1.90) were more likely to be associated with angina pectoris.

28 rm clinical outcomes in patients with stable angina pectoris.

29 (MI), 44 cases of fatal CHD, and 68 cases of angina pectoris.

30 ients with myocardial infarction or unstable angina pectoris.

31 new treatment for ischaemia in patients with angina pectoris.

32 enal function, congestive heart failure, and angina pectoris.

33 s prognostic benefit in patients with stable angina pectoris.

34 approved for the treatment of chronic stable angina pectoris.

35 ion, in chronic heart failure, and in stable angina pectoris.

36 linical approach to the treatment of chronic angina pectoris.

37 th either acute coronary syndromes or stable angina pectoris.

38 enosis in patients undergoing PCI for stable angina pectoris.

39 progression in patients with chronic stable angina pectoris.

40 on long-term outcome in patients with stable angina pectoris.

41 fects for patients with chronic, symptomatic angina pectoris.

42 new therapeutic approach to the treatment of angina pectoris.

43 c options exist for patients with refractory angina pectoris.

44 nsidered to be normal and those with typical angina pectoris.

45 ed between patients with unstable and stable angina pectoris.

46 pital with new-onset chest pain or worsening angina pectoris.

47 heart failure (0.635 [95% CI, 0.615-0.655]), angina pectoris (0.649 [95% CI, 0.630-0.667]), and ische

48 art disease, 0.45% (95% CI: 0.13%, 0.77%) in angina pectoris, 0.75% (95% CI: 0.38%, 1.13%) in acute m

49 failure ($20 764 [95% CI, $17 500-$24 027]), angina pectoris ($18 428 [95% CI, $16 102-$20 754]), and

50 betes; 5%, a prior myocardial infarction; 5% angina pectoris; 2.3%, intermittent claudication; and 7%

51 Angina pectoris affects at least 6.6 million people in t

52 monstrated the efficacy of TMR for relieving angina pectoris, although no study to date has specifica

53 A total of 95 patients with angina pectoris and angiographically documented coronary

54 primarily for treatment of hypertension and angina pectoris and are thought to act as allosteric mod

55 is prospective trial patients with suspected angina pectoris and at least one cardiovascular risk fac

56 ly high in subjects with personal history of angina pectoris and familial aneurysm.

57 Secondary endpoints were angina pectoris and hospitalization for heart failure.

58 ally and significantly activated in unstable angina pectoris and is not affected by severity of CAD o

59 Male patients (n = 328) with stable angina pectoris and ischemia on treadmill testing were r

60 Moderate drinking decreases the risk for angina pectoris and myocardial infarction in apparently

61 infarction, 10 angina pectoris, and 12 both angina pectoris and myocardial infarction) after the dia

62 heart by atherosclerotic lesions, leading to angina pectoris and myocardial infarction, damages the h

63 is coronary heart disease, including stable angina pectoris and the acute coronary syndromes.

64 e the population of patients with refractory angina pectoris and to present the therapeutic options c

65 We identified 80 conventional (eg, stable angina pectoris and type 2 diabetes) and unconventional

66 3 first events (11 myocardial infarction, 10 angina pectoris, and 12 both angina pectoris and myocard

67 282) patients with recurrent or deteriorated angina pectoris, and 99 (95% confidence interval 69 to 1

68 n trials of hypertension, diabetes mellitus, angina pectoris, and atrial fibrillation provides even m

69 ovascular disorders, including hypertension, angina pectoris, and cardiac arrhythmia.

70 , myocardial infarction, functional class of angina pectoris, and hospitalizations for unstable angin

71 uelae: nonfatal acute myocardial infarction, angina pectoris, and ischemic heart failure.

72 stroke, heart failure, cardiac dysrhythmias, angina pectoris, and peripheral artery disease), sociode

73 d MI, coronary revascularization procedures, angina pectoris, and sudden CHD deaths.

74 jority of patients (94%) had class III or IV angina pectoris, and two patients (6%) had unstable symp

75 enting with stable angina pectoris, unstable angina pectoris,and ST-segment elevation myocardial infa

76 t disease (CHD); myocardial infarction (MI); angina pectoris; and performance of coronary bypass or a

77 with preserved ejection fraction (HFpEF) and angina pectoris (AP).

78 Although angina pectoris appears to be related statistically to s

79 After we excluded isolated angina pectoris as an initial event, the lifetime risk o

80 Cardiac amyloidosis can present as angina pectoris associated with coronary flow reserve ab

81 time-dependent proportional hazards methods; angina pectoris at 5 years was modeled using univariate

82 dictor of nonfatal myocardial infarction and angina pectoris at 5 years, even after consideration of

83 as latanoprost was linked to conditions like angina pectoris, atrial tachycardia and Meniere's diseas

84 The use of nitroglycerin in the treatment of angina pectoris began not long after its original synthe

85 l results in 60-90% of diseases that include angina pectoris, bronchial asthma, herpes simplex, and d

86 Non-cardiac chest pain mimics angina pectoris but generally originates from the oesoph

87 alcium antagonists are widely prescribed for angina pectoris but their effect on clinical outcome is

88 ambulatory ischemia in patients with stable angina pectoris, but it remains to be established whethe

89 Patients with moderate/severe angina pectoris (Canadian Cardiovascular Society class 2

90 cardiovascular event (myocardial infarction, angina pectoris, cerebrovascular accidents, or major cor

91 h Canadian Cardiovascular Society grading of angina pectoris class 1 (n=1107, 18 events).

92 h Canadian Cardiovascular Society grading of angina pectoris class 2 or higher (n=839, 34 events), in

93 AxCanadian Cardiovascular Society grading of angina pectoris class interaction was observed in SCD ri

94 , Canadian Cardiovascular Society grading of angina pectoris class, and exercise capacity were used a

95 ed as new episodes of myocardial infarction, angina pectoris, congestive heart failure, or stroke.

96 l infarction, CHD death, angiogram-confirmed angina pectoris, coronary artery bypass graft surgery, s

97 he lifetime risks of coronary heart disease (angina pectoris, coronary insufficiency, myocardial infa

98 nce than medical therapy for men with stable angina pectoris due to single-vessel disease.

99 Twenty developed angina pectoris during pacing, while 21 did not.

100 Angina pectoris during RCA occlusion tended to occur in

101 intracoronary ECG ST-segment elevation, and angina pectoris during the same 1-minute coronary occlus

102 tory and duration of clinical improvement in angina pectoris following TMR.

103 exercise tolerance test, and stable chronic angina pectoris (for at least 2 months) were recruited i

104 In patients with suspected stable angina pectoris, global longitudinal peak systolic strai

105 Patients with MI and unstable angina pectoris had higher VEGF levels compared with sta

106 New York Heart Association class or comorbid angina pectoris, had lower activity levels, lived in Eas

107 Postprandial angina pectoris has been recognized for more than two ce

108 nifedipine GITS to conventional treatment of angina pectoris has no effect on major cardiovascular ev

109 d aortocoronary vein grafts and uncontrolled angina pectoris have limited options for therapy.

110 -8)) risk for ACS in individuals with stable angina pectoris (hazard ratio, 1.163 [95% CI, 1.082-1.25

111 1]) compared with individuals without stable angina pectoris (hazard ratio, 1.531 [95% CI, 1.497-1.56

112 coronary heart disease/heart failure, angina/angina pectoris, heart attack, and stroke, who provided

113 vascular and cerebrovascular diseases (e.g., angina pectoris, heart failure, cerebral infarction).

114 onary revascularization, hospitalization for angina pectoris, hospitalization for congestive heart fa

115 ar mortality, myocardial infarction, stroke, angina pectoris, hospitalization for heart failure, ESRD

116 drugs, used extensively in the treatment of angina pectoris, hypertension, and arrhythmia.

117 The indication for PTCA was stable angina pectoris in 69 patients, unstable angina in 22 an

118 The standard liquid meal precipitated angina pectoris in all patients.

119 rength of association between edentulism and angina pectoris in Mexican adults aged 35 years and olde

120 s is responsible for myocardial ischemia and angina pectoris in most patients with stable angina pect

121 The standard liquid meal induced angina pectoris in patients with coronary artery disease

122 maging, TMR improved the functional class of angina pectoris in patients with end stage coronary arte

123 CI (with or without FFR guidance) for stable angina pectoris in Sweden between January 2005 and March

124 Stable angina pectoris in women has often been considered a "so

125 iovascular events (myocardial infarction and angina pectoris) in 498 women with systemic lupus erythe

126 apid CAD progression in patients with stable angina pectoris is associated with increased C-reactive

127 Angina pectoris is associated with morbidity and mortali

128 Refractory angina pectoris is defined, and traditional medical ther

129 inking to the risk for other events, such as angina pectoris, is not known.

130 Of 50 diseases monitored, a single one, angina pectoris, is significantly elevated (3.3x) in ici

131 the multiple pathobiological precipitants of angina pectoris, ischaemia and infarction.

132 the multiple pathobiological precipitants of angina pectoris, ischemia, and infarction.

133 ified in network meta-analyses of stroke and angina pectoris, limiting the conclusiveness of findings

134 Angina pectoris may arise from obstructive coronary arte

135 o 1.093] for every 0.26 mmol/L increase) and angina pectoris (multivariate odds ratio, 1.049 [95% con

136 e used to assess risk for a first CHD event (angina pectoris, myocardial infarction, or cardiac death

137 ears, 55 subjects developed coronary events (angina pectoris, myocardial infarction, or coronary deat

138 -up of 7.9 years, 76 subjects developed CEs (angina pectoris, myocardial infarction, or coronary deat

139 infarction (n=5371, 901 deaths), and stable angina pectoris (n=6536, 965 deaths) in 4 age categories

140 ve patients with clinically suspected stable angina pectoris, no previous cardiac history, and normal

141 for the treatment of myocardial ischemia and angina pectoris not amenable to conventional percutaneou

142 for the treatment of myocardial ischemia and angina pectoris not amenable to conventional percutaneou

143 ther self-reported QoL parameters related to angina pectoris, notably in terms of angina frequency an

144 myocardial infarction, but the prevalence of angina pectoris, of smoking, and of chest pain in the at

145 al, 23,860 patients underwent PCI for stable angina pectoris; of these, FFR guidance was used in 3,36

146 Angina pectoris often results from ischemic episodes tha

147 participants (HR = 1.17, 1.05-1.31) and with angina pectoris only in women (HR = 1.55, 1.03-2.33).

148 OR heart failure OR myocardial infarction OR angina pectoris OR acute coronary syndrome OR coronary a

149 in 72 h of presentation with either unstable angina pectoris or acute myocardial infarction.

150 re American region, older age, no history of angina pectoris or asthma, no use of hypoglycemic agent,

151 or revascularization, or with a diagnosis of angina pectoris or CHD defined by angiography.

152 the duration of symptoms in patients who had angina pectoris or myocardial infarctions.

153 in 444 of 1473 patients with either unstable angina pectoris or non-Q-wave myocardial infarction (NQW

154 d 18 years or older, with stable or unstable angina pectoris or patients who had a myocardial infarct

155 26.64), and less likely to have a history of angina pectoris (OR 0.58, 95% CI 0.34-0.99) compared wit

156 rial fibrillation, renal dysfunction, stable angina pectoris, or advanced New York Heart Association

157 acute coronary arteriography and 2) syncope, angina pectoris, or drug-related adverse event.

158 ntrol for adults with myocardial infarction, angina pectoris, or following coronary artery bypass gra

159 ry of heart failure, coronary heart disease, angina pectoris, or myocardial infarction.

160 atients had stable angina pectoris, unstable angina pectoris, or non-ST-elevation myocardial infarcti

161 t CVD, defined as new myocardial infarction, angina pectoris, or stroke, which developed between base

162 We studied 124 chronic stable angina pectoris patients (84 men; mean age, 61+/-10 year

163 is at rest in patients with suspected stable angina pectoris predicts the presence of coronary artery

164 Candidates with angina pectoris, previous myocardial infarction, or cong

165 ring patch-off hours in patients with stable angina pectoris receiving a beta-adrenergic blocking age

166 TMLR performed in patients with refractory angina pectoris reduces ischemic wall motion abnormaliti

167 cident CHD, including myocardial infarction, angina pectoris, revascularization, and coronary death,

168 nt cohorts of patients with suspected stable angina pectoris (SAP) (3033 patients; median 10.7 y foll

169 ve coronary angiography for suspected stable angina pectoris (SAP) (n = 4131) and an independent coho

170 ients referred for angiography due to stable angina pectoris (SAP) or acute coronary syndrome (ACS).

171 elevation AMI and unstable angina, or stable angina pectoris (SAP).

172 t-elevation myocardial infarction and stable angina pectoris , similar patterns were found albeit les

173 without self-reported CHD (heart attack and angina pectoris), stroke, peripheral vascular disease, a

174 ecently, all with significant improvement in angina pectoris that appears both rapid and sustained.

175 linical outcomes in patients with refractory angina pectoris treated with transmyocardial laser revas

176 75 in the best available therapy group) and angina pectoris (two [3%] of 74 in the ruxolitinib group

177 rdial infarction (MI) (n = 205) and unstable angina pectoris (UAP) (n = 185).

178 ocardial infarction (MI) (n =57) or unstable angina pectoris (UAP) (n =60) were consecutively recruit

179 Patients (n=141) with stable angina pectoris undergoing PCI had serial venous blood s

180 Eligible patients had stable angina pectoris, unstable angina pectoris, or non-ST-ele

181 t plaques in patients presenting with stable angina pectoris, unstable angina pectoris,and ST-segment

182 Overall improvement in angina pectoris was sustained at 1 year by at least one

183 Women with angina pectoris were less likely to undergo cardiac cath

184 with the duration of the episode of unstable angina pectoris were observed in 6 of 21 patients who di

185 ) undergoing coronary angiography for stable angina pectoris were studied.

186 including acute coronary syndrome and stable angina pectoris, were independent predictors of MACE.

187 oposed catheter-based therapy for refractory angina pectoris when bypass surgery or angioplasty is no

188 MR) is an operative treatment for refractory angina pectoris when bypass surgery or percutaneous tran

189 We observed a patient with unstable angina pectoris who developed foci of ischemic necroses

190 PVI were observed in 10 patients with stable angina pectoris, with well-defined single vessel coronar

191 s validated using CT images of patients with angina pectoris without known valvular disease (n = 95).

192 However, in the group with angina pectoris, women were considerably less likely to

193 However, a diagnosis of stable angina pectoris yielded a differential association betwe