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1 Expected survival rates are good, as are the anginal and functional classifications, but there is a h
3 e patients with coronary disease and > or =3 anginal attacks per week despite maximum recommended dos
4 among several compounds suggested to trigger anginal chest pain; however, the pH reached when a coron
5 The statistically significant reduction in anginal class and strong positive trends for remaining e
7 th complete 12-month follow-up (n =27), mean anginal class improved from 3.5+/-0.5 pre-TMR to 2.8+/-0
9 d with improvements in myocardial perfusion, anginal complaints, and quality of life score </=12 mont
11 onship of ranolazine, a potentially new anti-anginal compound, on symptom-limited exercise duration.
12 hat is unknown, however, is whether any anti-anginal drugs (beta-blockers, long-acting nitrates, calc
14 e anti-ischemic therapy for their qualifying anginal episode and less likely to undergo invasive proc
16 disease, were more likely than men to report anginal episodes and had more recorded ischemic periods
18 Many patients with chronic angina experience anginal episodes despite revascularization and antiangin
20 ary outcome was the average weekly number of anginal episodes over the last 6 weeks of the study.
21 The outcomes pooled were indices of angina (anginal episodes, Canadian Cardiovascular Society angina
22 d by improvements in surrogate end points of anginal episodes, use of antianginal medications, Canadi
23 s with troponin-positive acute chest pain or anginal equivalent and inconclusive diagnosis after clin
24 number of patients with refractory angina or anginal equivalent symptoms despite maximal medical ther
25 or = 50%), severe anginal symptoms (> or = 2 anginal events in preceding 24 h), use of aspirin in the
26 lectrocardiogram at presentation, at least 2 anginal events in prior 24 hours, use of aspirin in prio
27 everal dimensions of HRQOL including reduced anginal frequency and bodily pain as well as improved di
28 diovascular outcome, notably higher rates of anginal hospitalization, repeat catheterization, and gre
31 th angina-limited exercise discontinued anti-anginal medications and were randomized into a double-bl
33 cular clinical relevance because chest pain, anginal or otherwise, has been shown to be a frequent bu
34 case history of an individual with possible anginal pain and asked how respondents would react to ex
42 tients with Stable Coronary Artery Disease), anginal status was mapped each year in patients without
43 ciation between repeat revascularization and anginal status, according to the type of initial revascu
44 The patient-oriented composite endpoint, anginal status, and exercise testing, were not statistic
46 younger women presenting with stable angina-anginal symptom characterization may align with older wo
47 AD), known CAD (stenosis > or = 50%), severe anginal symptoms (> or = 2 anginal events in preceding 2
48 ss surgery persisted among those with severe anginal symptoms (31 percent of blacks underwent surgery
49 clinical presentation with exercise-related anginal symptoms (chest pain or dyspnea) with or without
50 sts because of their efficacy in controlling anginal symptoms (Dihydropyridine calcium channel blocke
51 Conversely, there was no association between anginal symptoms and CAD (63.2% [67 of 106 patients] vs
54 s may explain how aortic stenosis can induce anginal symptoms and their prompt relief after PAVR.
56 4, P<10-7) identified inducible ischemia and anginal symptoms as the most powerful predictors (83%, 6
58 en CPET parameters, myocardial ischaemia and anginal symptoms in patients with chronic coronary syndr
59 ith improvements in myocardial perfusion and anginal symptoms in patients with refractory angina pect
60 ter likelihood of seeking immediate care for anginal symptoms than Europeans; this finding indicates
62 tatus and self-reported CVD among those with anginal symptoms was determined in multivariable-adjuste
65 ar follow-up, 43 (72.9%) reported persistent anginal symptoms, 5 (8.5%) were taking an antiplatelet,
66 ly to be on dialysis (83% vs 44%), had fewer anginal symptoms, and were more likely to have coronary
67 ention (PCI) is usually performed to relieve anginal symptoms, but data are emerging to suggest that
68 icrovascular coronary spasm in patients with anginal symptoms, despite angiographically normal corona
69 eral trials have demonstrated a reduction in anginal symptoms, increases in exercise tolerance, and o
76 tective effects of preinfarction angina, the anginal "warm-up phenomenon," and studies performed on h