ライフサイエンスコーパス: angiographically

1 iodontal disease and chronic CAD as assessed angiographically.

2 networks have been detected in human tumors angiographically.

3 ention in-stent tissue, and (3) was not seen angiographically.

4 eous coronary intervention that are not seen angiographically.

5 tratify cardiac risk have not been validated angiographically.

6 rabbits; arterial occlusions were documented angiographically.

7 E did not demonstrate any neovascularization angiographically.

8 cute myocardial infarction (MI) were studied angiographically; 1,848 patients had coronary artery dis

9 Angiographically, 549 pt had severe (>60% coronary steno

10 31) underwent coronary angiography; 781 were angiographically acceptable; 454 (58% of eligible) patie

11 oing angiography as well as do the number of angiographically affected arteries.

12 Angiographically, all arteries remained open.

13 e is particularly helpful in intermediate or angiographically ambiguous lesions in the absence of non

14 In 55 patients with an angiographically ambiguous LMCS, a pressure guidewire wa

15 dogrel and aspirin for 6 months and followed angiographically and clinically.

16 tments on these structures was also assessed angiographically and histologically.

17 Closure of the CNV was assessed both angiographically and histologically.

18 but also in 12 (15%) of 80 segments without angiographically apparent coronary disease (p = 0.004, a

19 e suppression and correlated the presence of angiographically apparent plaque with (18)F-FDG uptake i

20 Likewise, the presence of angiographically apparent thrombus was associated with a

21 ents suffering severe late rejection develop angiographically apparent TxCAD rapidly and must be moni

22 eromas that have large plaque burden despite angiographically appearing mild.

23 disease out of proportion to their effect on angiographically assessed lumen stenosis.

24 sease (prior myocardial infarction or proven angiographically) between September 2011 and November 20

25 raphic score, as measured histologically and angiographically, compared with vehicle or empty viral v

26 ous coronary interventions for patients with angiographically complex lesions confers additive long-t

27 The IVUS-detected ruptured plaques had angiographically complex morphology (95%) with ulceratio

28 were independent predictors of the number of angiographically complex stenoses.

29 , 1.52; 95% confidence interval, 1.17-1.98), angiographically confirmed angina (1.91; 1.59-2.29), cor

30 ression analyses utilizing the data from 256 angiographically confirmed CAD patients and 250 non-CAD

31 atients from Kuopio University Hospital with angiographically confirmed CHD and 250 age-, gender-, an

32 e patients with ACS and 12 CSA patients with angiographically confirmed coronary artery disease and 9

33 Among older acute MI patients with angiographically confirmed coronary artery disease disch

34 ult volunteers and seven patients with X-ray angiographically confirmed coronary artery disease under

35 ry intima-media thickness, 442 patients with angiographically confirmed coronary artery disease, and

36 In patients with angiographically confirmed coronary atherosclerosis, lep

37 plasma leptin and prognosis in patients with angiographically confirmed coronary atherosclerosis.

38 rtery disease, and 351 patients without such angiographically confirmed disease.

39 We included adults aged 18-85 years with angiographically confirmed large vessel occlusion stroke

40 45 years +/- 10.4 [standard deviation]) with angiographically confirmed Moyamoya (n = 8) or internal

41 This case series included 173 patients with angiographically confirmed SCAD enrolled between January

42 ter cohort study identified 87 patients with angiographically confirmed SCAD.

43 plicated in only one death in a patient with angiographically-confirmed PE at initial presentation.

44 lood sera was reported previously to predict angiographically defined advanced coronary artery diseas

45 domly assigned 423 postmenopausal women with angiographically defined atherosclerosis (321 women had

46 ere collected from 1412 patients with severe angiographically defined CAD (stenosis >/=70%).

47 predictive power of the same methodology for angiographically defined CAD using plasma samples from g

48 n the HSP70-2 gene +1267A>G polymorphism and angiographically defined CAD within an Iranian populatio

49 In patients with angiographically defined CAD, tHCY is a significant pred

50 iated death among patients with significant, angiographically defined CAD.

51 associated with the presence and severity of angiographically defined CAD.

52 med an association between 9p21 and CAD with angiographically defined cases and control subjects (poo

53 rstanding of the anatomic characteristics of angiographically defined CNV lesion subtypes.

54 onducted in subjects 37 to 67 years old with angiographically defined coronary artery disease.

55 Study who were 37 to 67 years of age and had angiographically defined coronary artery disease.

56 sk factors with the presence and severity of angiographically defined coronary atherosclerosis was an

57 S-derived patterns with 3.2-y progression of angiographically defined coronary atherosclerosis.

58 area under the curve, 0.87 [0.64-0.97]) and angiographically defined moderate-to-severe CAV, and CZT

59 A cohort of 7,220 individuals with angiographically defined significant CAD (> or =70%) was

60 ficant burden of ischemia remains even after angiographically defined successful revascularization.

61 iabetic nonsmoking patients (n = 4,811) with angiographically defined, clinically significant CAD (>

62 y was to investigate the association between angiographically-defined CAD and periodontal disease.

63 There is no angiographically demonstrable obstructive coronary arter

64 in women with myocardial infarction without angiographically demonstrable obstructive coronary arter

65 ferent myocardial diseases in the absence of angiographically demonstrable stenosis of the epicardial

66 t mortality in 985 consecutive patients with angiographically demonstrated CAD (stenosis >/=70%).

67 uals with stable presentation and those with angiographically demonstrated clean coronaries are not a

68 Effects were larger for those with angiographically demonstrated coronary artery disease.

69 Moreover, in the largest GWAS for angiographically derived coronary atherosclerosis perfor

70 n evaluating the physiologic significance of angiographically detectable coronary artery stenoses via

71 ation in heart transplant recipients without angiographically detectable disease.

72 Angiographically detectable systemic venous collateral c

73 dial perfusion defects on SPECT studies with angiographically detected CAD and with human expert visu

74 This study sought to correlate angiographically detected complex lesions and intracoron

75 ory of complex lesions and specifically with angiographically detected ICT and decreased TIMI flow.

76 to test the ability to predict the extent of angiographically determined coronary artery disease (CAD

77 Spiral CT flow index agreed with angiographically determined flow in 85% (95% CI: 0.77, 0

78 nd CABG arms of the all-comers SYNTAX trial, angiographically determined ICR has a detrimental impact

79 EN IV trial, OCT-guided PCI in patients with angiographically determined moderately or severely calci

80 more predictive of adverse outcome than was angiographically determined multivessel disease.

81 Angiographically determined prePTCA minimal lumen diamet

82 dy of 734 type 2 diabetes patients (322 with angiographically diagnosed CAD and 412 with no evidence

83 Angiographically diagnosed ischemic HF is associated wit

84 l determinants of mortality in patients with angiographically diagnosed ischemic or nonischemic cardi

85 a are from examinations of 375 patients with angiographically diagnosed PE who participated in the Pr

86 t late lumen loss was 0.27 mm (SD 0.37), and angiographically discernable vasomotion was documented i

87 Coronary vasoconstriction in angiographically diseased arteries varies with hemodynam

88 sion showed that log score but not number of angiographically diseased vessels significantly predicte

89 e CAD, we monitored 167 stable patients with angiographically documented 3-vessel CAD (average follow

90 Fifteen adults with angiographically documented CAD ingested 7.7+/-1.2 mL.kg

91 (type 2 and type 1) patients with or without angiographically documented CAD.

92 d with improved CV survival in patients with angiographically documented CAD.

93 (age 63+/-10 years, 230 men) of whom 289 had angiographically documented coronary artery disease (> o

94 otal of 95 patients with angina pectoris and angiographically documented coronary artery disease were

95 e 2 diabetes mellitus and clinically stable, angiographically documented coronary artery disease were

96 In normolipidemic patients with angiographically documented coronary artery disease who

97 therapy; these men had angina (determined by angiographically documented coronary artery disease).

98 65 patients (aged 18-85 years) with angiographically documented coronary artery disease, a p

99 2.3 years) with chronic stable angina due to angiographically documented coronary artery disease, all

100 of oxidized LDL are strongly associated with angiographically documented coronary artery disease, par

101 oronary atherosclerosis in 825 patients with angiographically documented coronary artery disease.

102 4.5 g of OTC or placebo in 48 subjects with angiographically documented coronary artery disease.

103 trial was performed recruiting patients with angiographically documented coronary disease (n=96) and

104 ascular ultrasonography in 502 patients with angiographically documented coronary disease.

105 ascular ultrasonography in 408 patients with angiographically documented coronary disease.

106 differences at presentation and severity of angiographically documented disease.

107 suspected renovascular hypertension and (b) angiographically documented hemodynamically significant

108 In-stent restenosis was angiographically documented in 282 patients with 409 les

109 re measured in 405 consecutive patients with angiographically documented multivessel coronary disease

110 valuable prognostic marker in patients with angiographically documented single- and double-vessel di

111 In patients with angiographically documented stable CAD without heart fai

112 de polymorphism rs2383206 and CAD defined as angiographically documented stenosis greater than 50% in

113 Twenty-seven (0.4%) had angiographically documented subacute closure <1 week aft

114 d patients with type 2 diabetes mellitus and angiographically documented, stable coronary disease to

115 Technical success was evaluated angiographically during and after placement.

116 our weeks later, vasoreactivity was assessed angiographically during infusion of acetylcholine (Ach)

117 further medical therapy (307), and 781 were angiographically eligible for random allocation; 454 of

118 tion of the brachial artery in patients with angiographically established CAD.

119 The observed close correlation between an angiographically established parameter of flow-dependent

120 The rate of angiographically established restenosis was 40.8 percent

121 Among angiographically evaluable patients (n = 754), the prima

122 owing heart transplantation do not result in angiographically evident acceleration of transplant CAD.

123 e coronary artery calcium are likely to have angiographically evident atherosclerosis.

124 a sensitive but not a specific indicator of angiographically evident atherosclerosis; sensitivity is

125 cations and to determine the relationship of angiographically evident complications to elevations of

126 the association of cardiac risk factors and angiographically evident coronary artery disease with co

127 at raising HDL-cholesterol in patients with angiographically evident coronary artery disease.

128 otic changes, even before the development of angiographically evident endothelial dysfunction; theref

129 ation with coil and particle embolization of angiographically evident SPC vessels.

130 should target CRP-associated risk as well as angiographically evident stenosis.

131 ath, revascularization of the target lesion, angiographically evident thrombosis, or myocardial infar

132 esized that combination therapy would reduce angiographically evident thrombus (AET) and would increa

133 myocardial infarction in 41.4% of the cases, angiographically evident thrombus in 20.9%, and abrupt o

134 inine, compared to placebo-treated controls; angiographically evident vascularity in the ischemic lim

135 single image, now affords us the ability to angiographically examine the parts of the retina previou

136 ed in situ for 5 days, at which time animals angiographically exhibiting thrombus were randomly assig

137 patients without acute MI who were assessed angiographically for coronary artery disease (CAD) and w

138 revious treatment were included and observed angiographically for up to 18 months and clinically for

139 timate associations between BPA exposure and angiographically graded coronary atherosclerosis.

140 This study compared angiographically graded coronary blood flow with intraco

141 ce has demonstrated a modest benefit with an angiographically guided approach; but patients having ne

142 After angiographically guided PCI, patients were randomized 1:

143 After angiographically guided PTCA of 104 lesions in 102 patie

144 ter 12 months of follow-up) between complete angiographically guided revascularization (n=154) or str

145 group and 22 (14%) patients of the complete angiographically guided revascularization group (hazard

146 e assessed the clinical outcomes of complete angiographically guided revascularization versus stress

147 y not be significantly different to complete angiographically guided revascularization, thereby reduc

148 s were administered intramuscularly along an angiographically guided target artery path on days 0, 28

149 guided approach was superior to the standard angiographically-guided approach for percutaneous revasc

150 The presence of angiographically identified intracoronary thrombus has b

151 Angiographically identified restenosis (stenosis of 50 p

152 or QOL measures by adding ranolazine in this angiographically-identified population.

153 the photosensitizer verteporfin was assessed angiographically in CNV lesions, to determine the optima

154 ye, highlighting looping patterns visualized angiographically in human tumors.

155 r location in follow-up studies was verified angiographically in relation to branch vessels.

156 to distal microembolization or spasm, and/or angiographically inapparent dissection or residual steno

157 Early after MI, IRAs frequently have angiographically indeterminant lesions.

158 und (IVUS)-guided strategy for patients with angiographically indeterminate left main coronary artery

159 r ultrasound is an accurate method to assess angiographically indeterminate lesions of the LMCA.

160 conducted IVUS studies on 214 patients with angiographically indeterminate LMCA lesions, and deferra

161 Of the patients with angiographically indeterminate LMCAs, 83 (38.8%) had an

162 tandard to determine hemodynamic severity of angiographically intermediate coronary lesions.

163 associated with a higher probability that an angiographically intermediate coronary stenosis is funct

164 9, and September 30, 2017, and had SIHD with angiographically intermediate disease (40% to 69% diamet

165 continents with coronary artery disease and angiographically intermediate severity stenoses who unde

166 Evidence: A recommendation to perform FFR in angiographically intermediate stenoses in the absence of

167 In patients with SIHD and angiographically intermediate stenoses, use of FFR has s

168 omes among unselected patients with SIHD and angiographically intermediate stenoses.

169 rtery bypass graft surgery having at least 1 angiographically intermediate stenosis.

170 ysfunction in arteries not yet clinically or angiographically involved in thromboangiitis obliterans.

171 The equivalent odds ratios for angiographically irregular versus smooth plaque were 6.3

172 ery prevalence, 48% versus 24%; P<0.001) and angiographically (mean Japan-chronic total occlusion sco

173 tin plus niacin provides marked clinical and angiographically measurable benefits in patients with co

174 esions responsible for follow-up events were angiographically mild at baseline (mean [+/-SD] diameter

175 e coronary syndromes often occur at sites of angiographically mild coronary-artery stenosis.

176 PCI of angiographically mild lesions with large plaque burden w

177 ble for unanticipated events were frequently angiographically mild, most were thin-cap fibroatheromas

178 that plaque rupture (PR) is associated with angiographically minimally occlusive lesions.

179 Forty-eight patients with one angiographically moderate-to-severe stenosis were includ

180 ed NIRS and intravascular ultrasound detects angiographically non-obstructive lesions with a high lip

181 Patients with an angiographically nonobstructive stenosis not intended fo

182 age, 53.0+/-10.1 years) with chest pain and angiographically nonsignificant coronary artery disease

183 patients during cardiac catheterization with angiographically normal anterior descending arteries.

184 s: 85 patients with chest pain syndromes and angiographically normal arteries (group 1); 21 patients

185 Coronary vasodilatory reserve was higher in angiographically normal arteries in patients with chest

186 vasodilatory reserve values in patients with angiographically normal arteries who had atypical chest

187 able coronary artery disease (CAD) (n = 17), angiographically normal coronary arteries (n = 8), and f

188 rs (healthy control subjects), patients with angiographically normal coronary arteries (patient contr

189 rding the range of normal values obtained in angiographically normal coronary arteries in patients wi

190 /- 10 years) with angina-like chest pain and angiographically normal coronary arteries underwent exer

191 terquartile range, 51-64]; 15 men, 43%) with angiographically normal coronary arteries were randomly

192 onary artery disease and in 12 subjects with angiographically normal coronary arteries who were free

193 We studied 26 patients with angiographically normal coronary arteries, 10 without ri

194 Despite angiographically normal coronary arteries, heterogeneous

195 METHODS AND In patients with angiographically normal coronary arteries, intracoronary

196 ssels to be distinguished from subjects with angiographically normal coronary arteries, with a specif

197 patients with coronary risk factors but with angiographically normal coronary arteries.

198 of endothelial function in the subgroup with angiographically normal coronary arteries.

199 m in patients with anginal symptoms, despite angiographically normal coronary arteries.

200 The remaining 120 (70%) patients had an angiographically normal CS.

201 The CS is angiographically normal in most patients.

202 ltrasound was performed on 121 patients with angiographically normal LMCAs to determine the lower ran

203 ore stenting in the culprit vessel and in an angiographically normal nonculprit vessel in patients wi

204 iography for assessment of stable angina had angiographically normal or near normal coronary arteriog

205 died a second group of 10 cocaine users with angiographically normal or near-normal arteries.

206 For comparison, evaluation of an angiographically normal reference vessel from the same s

207 after angioplasty, after stenting, and in an angiographically normal reference vessel.

208 n vascular pathology, diabetic patients with angiographically normal retinas have been found to exhib

209 a deleterious effect of gamma-irradiation on angiographically normal uninjured reference segments in

210 city and MPR ratios between poststenotic and angiographically normal vascular beds were comparably re

211 All segments studied were angiographically normal.

212 Plaque occupied a mean of 51+/-15% of the angiographically "normal" reference segments.

213 IVUS commonly detects occult disease in angiographically "normal" sites.

214 algorithm for noninvasive identification of angiographically obstructive three-vessel and/or left ma

215 ts with Coats' disease diagnosed clinically, angiographically, or both from 1995 through 2015.

216 he right or left prostatic artery through an angiographically placed microcatheter.

217 In both groups, 50% were angiographically positive for emboli.

218 fect of equivalent size to the effusion were angiographically positive for PE.

219 agnetic resonance images in 21 patients with angiographically proved dural AV fistula of the cavernou

220 nd pulmonary angiograms of 104 patients with angiographically proved PE were reviewed by two nuclear

221 Fifteen (32%) of 47 patients had angiographically proved pulmonary embolism.

222 We present a case of angiographically proven asymptomatic left internal carot

223 teen patients with chronic stable angina and angiographically proven CAD (>70% stenosis in at least 1

224 Twenty-six patients with angiographically proven CAD and 29 normal volunteers und

225 Thirty-one men with angiographically proven CAD were recruited; 16 were trea

226 s of 19 eyes of 19 consecutive patients with angiographically proven CCF and 19 eyes of 19 age- and s

227 Cases were men aged over 40 years with angiographically proven CHD.

228 Twenty-seven patients with angiographically proven coronary artery disease and 5 su

229 cification than age-matched individuals with angiographically proven coronary artery disease.

230 nd placebo-controlled study of patients with angiographically proven coronary artery disease.

231 tors, duration of follow-up, the presence of angiographically proven obstructive CAD (>/=50% stenosis

232 Further study of angiographically proven patients with coronary artery di

233 d men and women aged 60 years and older with angiographically proven stable ischaemic heart disease o

234 Of the 163 patients, 57 (35%) had angiographically-proven PE, 77 (47%) had at least one la

235 nsfection, VEGF-transfected animals had more angiographically recognizable collateral vessels (angiog

236 .43 mL x min(-1) x 100 g(-1), P<0.001), more angiographically recognizable collateral vessels (angios

237 Thirty days later, angiographically recognizable collateral vessels and his

238 se large atheromatous vulnerable plaques may angiographically seem mild.

239 Angiographically severe calcium was confirmed by the cor

240 risk factor analysis can rule in or rule out angiographically severe disease, i.e., three-vessel and/

241 ons had as much target lesion calcium as did angiographically severe lesions.

242 nts with angina (or equivalent symptoms), no angiographically severe stenosis and fractional flow res

243 udication or ischemic pain while at rest and angiographically significant atherosclerotic lesions to

244 4-year cardiovascular risk in women without angiographically significant CAD (hazard ratio 1.41, 95%

245 We monitored 2315 patients with angiographically significant CAD (stenosis > or =70%) fo

246 uited in a case-control study: 250 cases had angiographically significant CAD (stenosis > or =70%), a

247 ere stratified by the presence or absence of angiographically significant CAD at study entry, in wome

248 aphy, determination of pretest likelihood of angiographically significant CAD by the invasive angiogr

249 na typicality-based pretest probabilities of angiographically significant CAD derived from invasive c

250 wer in predicting the severity and extent of angiographically significant CAD in symptomatic patients

251 higher accuracy for detecting patients with angiographically significant CAD than the analysis of wa

252 ignificant CAD at study entry, in women with angiographically significant CAD, the metabolic syndrome

253 Compared with patients without angiographically significant CAD, the number of EPCs was

254 calcium scores in a model for prediction of angiographically significant coronary artery disease (CA

255 have higher sensitivity for the detection of angiographically significant coronary artery disease, wh

256 ise, which may be observed in the absence of angiographically significant coronary artery stenosis.

257 omatic cardiac transplant recipients without angiographically significant coronary disease, FFR and t

258 the occurrence of myocardial ischemia but no angiographically significant coronary stenoses.

259 <0.0001) or the clinical model combined with angiographically significant coronary stenosis (P=0.0007

260 e hospitalized for chest pain but who had no angiographically significant coronary-artery obstruction

261 Of the 283 patients without angiographically significant disease, 124 had negative u

262 s increased and specificity is decreased for angiographically significant disease.

263 and the calcium score for the probability of angiographically significant disease.

264 ntions were infrequent even in patients with angiographically significant lesions.

265 ure of atherosclerosis, occurring in >90% of angiographically significant lesions.

266 ither complete revascularization with PCI of angiographically significant nonculprit lesions or no fu

267 ete revascularization with additional PCI of angiographically significant nonculprit lesions or to no

268 cent) of the 35 women with chest pain and no angiographically significant stenosis had decreases in t

269 at sites that did not previously exhibit an angiographically significant stenosis.

270 e aim of this study was to determine whether angiographically silent early coronary intimal thickenin

271 ant coronary abnormalities with OCT that are angiographically silent in children with a history of co

272 or significant thickening, 88% of which were angiographically silent.

273 Most of these findings were angiographically silent.

274 Early SVG failures were angiographically smaller than late failures (reference:

275 ary artery disease (CAD) and 8 patients with angiographically smooth coronary arteries (normal), we i

276 The PCI was considered angiographically successful in 93%, stents were placed i

277 ovascular resistance (HMR) immediately after angiographically successful PCI predicts MVI at cardiova

278 n develop microvascular injury (MVI) despite angiographically successful primary percutaneous coronar

279 For example, angiographically ulcerated plaques were much more likely

280 ification at a remote site is a predictor of angiographically undetected target lesion calcium.

281 according to whether the treated lesion was angiographically unstable or stable, and we investigated

282 We hypothesized that angiographically unsuspected atheromatous remodeling wit

283 ransient ischemic attack or stroke caused by angiographically verified 50 to 99 percent stenosis of a

284 r transient ischaemic attack attributable to angiographically verified 50-99% stenosis of a major int

285 ciate significantly with the age of onset of angiographically verified coronary artery disease (hazar

286 Patients with angiographically verified coronary artery disease (n=194

287 In clinical samples from patients with angiographically verified coronary artery disease, APOB(

288 randomly assigned a total of 309 women with angiographically verified coronary disease to receive 0.

289 In patients with angiographically verified diffuse DME, the mean improvem

290 All patients were aged >50 years and had angiographically verified exudative AMD.

291 m by ultrasound was greater in patients with angiographically visible calcification (175 degrees +/-

292 Of 120 patients without angiographically visible calcium at the target lesion si

293 Angiographically visible coronary collaterals were prese

294 nce the behavior of both the small number of angiographically visible lesions and the large number of

295 We sought to evaluate the impact of angiographically visible thrombus on short- and long-ter

296 sus 0.77+/-0.06; P<0.005), reduced number of angiographically visible vessels (angiographic score=0.4

297 When it is angiographically visible, the arc of calcium is likely t

298 he area of diffuse vascular leakage measured angiographically were significantly larger with DEX impl

299 prospective cohort of 985 patients diagnosed angiographically with severe CAD (stenosis > or =70%) an

300 transcutaneous ultrasound were widely patent angiographically, with TIMI grade 3 flow.